Arhive etichetă | vitamin E cancer

ULEIUERI si GRASIMI PERMISE IN CANCER,

Acest articol este un extras din cartea Dr italian Giuseppe Nacci , M.D. , „1000 plante impotriva cancerului FARA chimio-terapie”,una din cele mai solid argumentate carti de profil , 650 pagini in care se citeaza  2,050 publicatii stintifice officiale si 2,100 referinte bibliografice. Extrasul se refera la ULEIUERI si GRASIMI PERMISE IN CANCER, care ar trebui sa fie SURSA PRINCIPALA DE ENERGIE ( de preferat in locul carbohidratilor)

CLICK AICI pentru  limba ROmana(traducerea “aproximativa” utilizand google translate).

Chapter 1.c: FATS and OILS (“Fatty acids”)
Looking at Table 1 (Chapter 1.a) you will notice that oils are extremely rich as a source of energy
(900 Kilocalories per 100 grams olive oil, sunflower oil, flax oil, grape seed oil, corn oil, etc).

Fats are very rich in energy as well.
Chemically speaking, both can be considered to be “fatty acids”.

There are “essential” oils and fats that contain vitamins (vitamin E, vitamin F, etc…). They are called “essential” because our body is not able to synthesize them.
However, in the modern world we talk a lot about severe health problems linked to a diet rich in fats and oils. Actually, the problem needs to be further explained.
Chemically speaking, fats and oils contain three types of “fatty acids”:
Saturated fatty acids (dangerous for health);
monounsaturated fatty acids (not dangerous for health, can contain vitamins);
polyunsaturated fatty acids (not dangerous for health, can contain vitamins).
Saturated fats, or “bad” fats, are present in most animal fats, in margarine and in fats used in pastry shops.

GM food has been recently suspected to contain them as well (1207).

Saturated fatty acids, or “bad” fats, cause very severe alterations to cell membranes. They replace vitamin F (“good” fatty acids), and cause severe forms of cell wall impermeability for many
substances like glucose (that might cause Type 2 Diabetes), apoptotic vitamins (that might cause cancer and tumours in general), and other substances that are vital for cells, such as vitamin C (that
might cause heart attack, strokes etc).
On the contrary, unsaturated fats (“good fats”) are made of cis-cis fatty acids, that are typically contained in cold-worked vegetable oils.
Unsaturated fats (vegetable oils) are present in many plant seeds (SEE Table 2), and in some animals, such as some fat fish that live in cold waters (salmons, herrings – caution to heavy metals contamination).
Fatty acids are vital for muscle cells to produce energy for them during physical activity and to relax them (1208).
Furthermore, “good” fatty acids control blood coagulation (1209).
They also influence the release of CCK, a hormone that tells our brain that we ate enough and that we should stop eating (1210).
They contribute to maintaining conduction speed in motor and sensory nerves as well (1211).
They can keep our skin healthy (1212).
They reduce high blood pressure (1213).
They reduce lung cancer (Pardini R.S.: Nutritional Intervention with Omega-3 Fatty Acids in a case of Malignant
Fibrous Histiocytoma of the Lungs, Nutrition and Cancer 2005, 52 (2) , pp.: 121-129
It is useful to give a bit more detail about the vitamins contained in these oils.

Alpha-Lipoic Acid
Alpha-lipoic acid is an essential fatty acid that contains organic sulphur (as it is organic, that is, linked to biological molecules, it is not toxic). It directly helps making brain, muscle and skeleton
energy available during physical activity (1208), controlling diabetes as well (1214).

Alpha-Linolenic Acid
Alpha-linolenic acid is a cis polyunsaturated fatty acid present in cold-pressed flax seed oil. It is transformed into EPA and DHA (Omega 3 fats), that are difficult to find in food.

Table 2: percentage of vitamins contained in oils

oils

Conclusions are:

Flax Seeds Excellent omega 3
Pumpkin Seeds Excellent for vitamin E

SoyBean (NO GM) Good but Avoid soy

Sunflower(NO GM) Excellent but AVOID Polyunsaturated oils (e.g. corn oils ) as they promote the growth of small blood vessels (by promoting the production of “bad” prostaglandins). This is bad for cancer because it allows tumors to grow new blood vessels. However, these small blood vessels are good for preventing strokes. Thus, ONLY WHILE  on a cancer diet avoid polyunsaturated oils.
Walnuts(NO GM) Good 
Rice (NO GM) Good if organic, coldworked
Safflower Good
Grape Seed Good
Sweetcorn (NO GM) Inadvisable for Multiple Sclerosis, also AVOID in cancer
Extravirgin Olive Excellent for vitamin E! moderation
Sesame Good
Rapeseed (NO GM) Good if does not contain  erucic acid
Almonds Advisable! (whole RAW almonds are also great source of protein an vitamin B17)

Peanuts (NO GM) Inadvisable
Palm  Negative 

Palmist Negative

Coconut  Negative(whole raw young coconut allowed in cancer)

Other oils that are great in cancer :

Seabuckthorn oil.  cannabis oil hemp oil, avocado oil(whole avocado) and hazelnuts (whole raw hazelnuts – alune de PADURE).

Other sources of good, healthy fats are various fruit seeds( melons, grapes, berries, prunes, nectarines, apricots(vitamin b17), apples, pears, and others).

Note. If you are not a very weak person that should immediately go on a diet rich in omega3, water-soluble fats(see my book for details) , it is advisable you eat whole RAW foods rather than oils extracted from them.

Best of health,

Cristian

P.S.: Dozele, ca in orice lucru destinat sa combata  cancerul(o conditie severa) sunt RIDICATE: MINIM 6-8 linguri ulei omega 3 hidrosolubil zilnic(NON GMO, atentie inclusiv la in), minim un Pumn de nuci sau migdale zilnic, 40-50 samburi caise zilnic(care NU sunt toxici in aceste doze a se vedea si aici click si aici click si in cartea versiunea in engleza),etc.

Nu mai spun ca unii dintre dvs inca se infometeaza cu Breuss. MARE RESEALA!

 

Scientific Studies Database PAGE 2

CLICK AICI pt. limba ROmana

Vitamins in alphabetical order

Vitamin A

There is an extensive bibliography on the anti-tumoral action of Vitamin A.

In particular the combined use of Retinoids in the proper proportions (beta-Carotene: retinol = 4 : 1), establishes a synergism which is higher than the sum of the single components.

Studies on Vitamin A and cancer (MANY STUDIES -see NOTES below)

science.naturalnews.com/V/Vitamin_A_and_cancer.html

NOTE 1:

The link above contains multiple studies and the same do many links below( I will try to indicate by ”MANY STUDIES”)

Vitamin A, retinoids and carotenoids as chemopreventive agents for prostate cancer.
2004 The Journal of urology

An international evaluation of the cancer preventive potential of vitamin A.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of dietary vitamin A, carotenoids, and other antioxidants with the risk of ovarian cancer.

2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of vitamin A, vitamin C and zinc with laryngeal cancer.
2003 Indian journal of cancer

Vitamin A, retinol, and carotenoids and the risk of gastric cancer: a prospective cohort study.
2007 The American journal of clinical nutrition

The effect of Vitamin A status in children treated for cancer.
2008 Pediatric hematology and oncology

Oxidant, Vitamin A and angiogenic markers in laryngeal cancer patients.
2003 The Journal of laryngology and otology

The role of antioxidants and Vitamin A in ovarian cancer: results from the Women’s Health Initiative.
2008 Nutrition and cancer

Can Vitamin A modify the activity of docetaxel in MCF-7 breast cancer cells?
2007 Folia histochemica et cytobiologica / Polish Academy of Sciences, Polish Histochemical and Cytochemical Society

Does Vitamin A prevent high-dose-methotrexate-induced D-xylose malabsorption in children with cancer?
2004 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Intake of vitamin A-rich foods and lung cancer risk in Taiwan: with special reference to garland chrysanthemum and sweet potato leaf consumption.
2007 Asia Pacific journal of clinical nutrition

Vitamin A and apoptosis in prostate cancer.
2002 Endocrine-related cancer

Skin, sun, and vitamin A: from aging to cancer.
2001 The Journal of dermatology

Vitamin A metabolism is impaired in human ovarian cancer.
2009 Gynecologic oncology

Effectiveness of Vitamin A acetate for enhancing the production of lung cancerspecific monoclonal antibodies.
1999 Cytotechnology

A population-based case-control study of carotenoid and Vitamin A intake and ovarian cancer (United States).
2001 Cancer causes & control : CCC

Recent advances in the use of Vitamin A (retinoids) in the prevention and treatment of cancer.
2000 Nutrition (Burbank, Los Angeles County, Calif.)

Fruit, vegetable, Vitamin A intakes, and prostate cancer risk.
2008 Prostate cancer and prostatic diseases

The Vitamin A family can significantly decrease the expression of ERbeta of ERs positive breast cancer cells in the presence or absence of ER ligands and paclitaxel.
2009 Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology

Induction of apoptosis in maxillary sinus cancer cells by 5-fluorouracil, Vitamin A and radiation (FAR) therapy.
1999 European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology – Head and Neck Surgery

EUROSCAN, a randomized trial of Vitamin A and N-acetylcysteine in patients with head and neck cancer or lung cancer. For the EUropean Organization for Research and Treatment of cancer Head and Neck and Lung cancer Cooperative Groups.
2000 Journal of the National cancer Institute

Rapid HPLC method for the determination of Vitamin A and E and cotinine concentration in human serum in women with CIN and cervical cancer.
2009 Ginekologia polska

Estradiol and tamoxifen differently affects the inhibitory effects of Vitamin A and their metabolites on the proliferation and expression of alpha2beta1 integrins in MCF-7 breast cancer cells.
2009 Advances in medical sciences

Renaissance of the biologically active Vitamin A derivatives: established and novel directed therapies for cancer and chemoprevention.
2003 Current pharmaceutical design

Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
2009 Asian Pacific journal of cancer prevention : APJCP

Whole-food sources of Vitamin A more effectively inhibit female rat sexual maturation, mammary gland development, and mammary carcinogenesis than retinyl palmitate.
2007 The Journal of nutrition

[Chemoprevention and FAR therapy regimen comprised of 5-fluorouracil, Vitamin Aand radiation].
2001 Gan to kagaku ryoho. cancer & chemotherapy

Prostate cancer:

Breast cancer:

Ovarian cancer:

Lung cancer:

Lung, head & neck cancers:

2000 Journal of the National cancer Institute

Cervical cancer:

Gastric cancer:

Laryngeal cancer:

Children cancer:

Maxillary sinus cancer :

Studies on Vitamin A and Antiproliferative

http://science.naturalnews.com/V/Vitamin_A_and_Antiproliferative.html

Studies on Vitamin A and radiation

http://science.naturalnews.com/V/Vitamin_A_and_radiation.html

There is an extensive bibliography on the anti-tumoral action of Vitamin  A cited from LAST PAGE OF THIS DATABASE: 1,3,10,13,14,17,18,23,24,28,34,35,36,45,56,69,73,76,92,93,94,100,106,111,120,121,129,131,137,138,165,187,200,202,203,208,209,21,213,214,216,218,222,235,255,256,257,263,264,265,266,280,282,286,287,288,298,303,304,305,307,313,315,322,324,325,326,334,338,340,341,347,38,352,354,362,363,365,382,383,385,390,398,402,404,405,409,410,420,425,426,427,445,446,447,448,454,457,461,463,468,469,470,471,473,477,48,493,508,512):

This studies conclude  the following about vitamin A:

Anti-tumoral action in general

Vitamin A and retinoids have an anti neoplastic action, shown both in vivo and in vitro, in various tumors: basilomas, scaly carcinomas, melanomes, skin cancers, fungoid mycosis, acute promyelocyte leucemia, ovarian cancer, breast cancer, lung cancer, cancer of the bladder and follicular carcinomas of the thyroid.

Preventative action

Anti-tumoral action has been amply shown indirectly, that is with a preventative purpose. Various  studies have, in fact, shown that low plasmatic levels of beta-Carotene, vitamin C and vitamin E are  connected to an increase in the incidence of lung cancer.

Direct action at a receptor level

Only beta RAR (retinoic acid receptor) seems to be involved. More specifically, vitamin A (and its derivatives) acts by binding with specific receptors. In this way it can both inhibit the proteic synthesis of DNA and RNA, and also perform an anti-promotion  action to determine the return of a cellular differentiation. Furthermore its ability to inhibit oncogenes should be noted.

Apoptosis induction 

Apoptosis means the activation of specific endonucleases which break up the DNA, acting at a level of nucleosomic sites that make up the primary structural unit of the nuclear cromatine of the cell.  Vitamin A and retinoids in general can induce apoptosis in neoplastic cells, by activating intracellular proteolytic enzymes, called caspase 2 and caspase 3, which provoke deterioration  through proteolysis by a transcription factor, called Spl.  If this basal cellular transcription is altered, death is caused by Apoptosis.

In prostate cancer the retinoids intervene by reducing the level of the bcl 2 gene, whose function is to protect the cells from death by Apoptosis.  Carotenoids induced apoptosis in prostate cancer

Dă clic pentru a accesa carotenoidi%20sono%20fattori%20attivi%20contro%20il%20cancro%20della%20prostata.pdf

Inhibition of the cellular cycle

The retinoids block the passage of the cell from phase G1 to phase S (reducing the activity of a protein, called cycline D1): this passage, if not blocked, would lead the cell to mytosis.

Reduction of phosphorilation

Retinoids intervene in cancer causing a reduction in phosphorilation of the pRb, thus increasing survival; this protein is active in suppressing cellular growth.

Synergetic interaction with interferons

A second mechanism, apart from the proteic-enzymatic one, can be traced back to the intervention of interferons which, like retinoids, act as anti-proliferative factors. In practice, acting synergetically, they induce the expression of proteins capable of inhibiting neoplastic cellular proliferation.

The intake of natural carotenoids from food 

There is very little evidence to show that carotenoids taken in from food can increase the levels ofvitamin A: an extra portion a day of green leaf vegetables is not able to increase the hematic level of vitamin A; on the contrary, a mixed diet of foods particularly rich in beta-Carotene will give a  significant increase in vitamin A present in the blood. (627).

If you oblige healthy individuals to eat carrots (270g), broccoli (600g) or tomato juice (180g) it does not establish any significant changes in the hematic levels of the carotenoids: you only find an extremely wide variation (even up to 3-4 times) in the efficiency of gastro-intestinal absorption of  the carotenoids and therefore in their subsequent bio-availability at a hematic level (628) .

Optimal values of anti-oxidants in normal individuals

Vitamin C: >50 microMols/Liter

Vitamin E: >30 microMols/Liter

Vitamin A: >2.2 microMols/Liter

Beta-Carotene: >0.4 microMols/Liter

Association of vitamin A with vitamin E

In an experimental model of a cellular membrane the possibility of a positive interaction between anti-oxidant liposollubles such as beta-Carotene and alpha-Tocopherol has been investigated; the  result showed that there exists a synergetic action between beta-Carotene and alpha-Tocopherol  together which inhibits the processes of lipidic peroxidization compared with when they are used alone (629). THUS:

NOTE 2:

Vitamins , antioxidants and nutrients should be taken from WHOLE, ORGANIC(note below) FOODS (as much as possible) NOT from supplements as they need other vitamins and nutrients for proper usage.

The difference between synthetic vitamins and natural vitamins

The difference between synthetic vitamins and natural ones can be easily exemplified by the experimental case of synthetic beta-Carotene (made up entirely of isomeric trans-beta-Carotene), and of natural beta-Carotene (made up of both isomeric trans-beta-Carotene and isomeric cis-beta- Carotene): the study showed a strong discrimination between the two isomers, with a serious decrease (impoverishment) induced at the level of Lycopene present in the LDLs (…). THUS:

3.It would be better to use the NATURAL (juice) ORGANIC WHOLE foods rich in vitamin A such as  Daucus carota (carrots ), avocados, cantalopues, etc. rather than the synthetic pharmaceutical vitamin products based on Vitamin A.

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B Vitamins 

Studies on Vitamins B and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B_and_cancer.html
A prospective study of dietary folate and Vitamin B and colon cancer according to microsatellite instability and KRAS mutational status.
2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer.
2003 Journal of the National cancer InstituteCancer incidence and mortality after treatment with folic acid and vitamin B12.
2009 JAMA : the journal of the American Medical AssociationOne-carbon metabolism-related nutrients and prostate cancer survival.
2009 The American journal of clinical nutritionCirculating concentrations of folate and vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study.
2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Reduced thiamine (vitamin B1) levels following gastrectomy for gastric cancer.
2002 Gastric cancer : official journal of the International Gastric cancer Association and the Japanese Gastric cancer Association

Dietary folate intake, MTHFR genetic polymorphisms, and the risk of endometrialcancer among Chinese women.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Localized folate and vitamin B-12 deficiency in squamous cell lung cancer is associated with global DNA hypomethylation.
2000 Nutrition and cancer

Dietary factors of one-carbon metabolism and prostate cancer risk.
2006 The American journal of clinical nutrition

Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.
2009 BMC cancer

Folate, vitamin B(6), and vitamin B(12) intake and the risk of breast cancer among Mexican women.
2006 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Dietary folate intake and breast cancer risk: results from the Shanghai Breast cancerStudy.
2001 Cancer research

Vitamin B6 and risk of colorectal cancer: a meta-analysis of prospective studies.
2010 JAMA : the journal of the American Medical Association

One-carbon metabolism and prostate cancer risk: prospective investigation of seven circulating B vitamins and metabolites.
2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Folate, vitamin B6, multivitamin supplements, and colorectal cancer risk in women.
2006 American journal of epidemiology

Relationship of folate, vitamin B-6, vitamin B-12, and methionine intake to incidence of colorectal cancers.
2002 Nutrition and cancer

Association of the B-vitamins pyridoxal 5′-phosphate (B(6)), B(12), and folate with lungcancer risk in older men.
2001 American journal of epidemiology

Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.
2009 Nutrition and cancer

The role of vitamin B6 in the prevention of haematological toxic effects of linezolid in patients with cancer.
2008 The Journal of antimicrobial chemotherapy

Methylenetetrahydrofolate reductase, diet, and risk of colon cancer.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer

Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer

Vitamin B(12) and Folate Status in Head and Neck Cancer.
2002 Asian Pacific journal of cancer prevention : APJCP

Vitamins B2 and B6 and genetic polymorphisms related to one-carbon metabolism as risk factors for gastric adenocarcinoma in the European prospective investigation intocancer and nutrition.
2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Associations of dietary methyl donor intake with MLH1 promoter hypermethylation and related molecular phenotypes in sporadic colorectal cancer.
2008 Carcinogenesis

Vitamin B6 points PC6 injection during acupuncture can relieve nausea and vomiting in patients with ovarian cancer.
2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society

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Vitamin B2 – Riboflavin

Studies on Riboflavin and cancer
Biochemical characterization of Riboflavin carrier protein (RCP) in prostate cancer.
2009 Frontiers in bioscience : a journal and virtual library
Multidrug transporter ABCG2/breast cancer resistance protein secretes Riboflavin(vitamin B2) into milk.
2007 Molecular and cellular biologyIntracellular processing of Riboflavin in human breast cancer cells.
2008 Molecular pharmaceuticsAmeliorating effect of coenzyme Q10, Riboflavin and niacin in tamoxifen-treated postmenopausal breast cancer patients with special reference to lipids and lipoproteins.
2007 Clinical biochemistry

Effect of Coenzyme Q(10), Riboflavin and Niacin on Tamoxifen treated postmenopausal breast cancer women with special reference to blood chemistry profiles.
2009 Breast cancer research and treatment

Design of riboflavin-presenting PAMAM dendrimers as a new nanoplatform for cancer-targeted delivery.
2010 Bioorganic & medicinal chemistry letters

Co-enzyme Q10, Riboflavin and niacin supplementation on alteration of DNA repair enzyme and DNA methylation in breast cancer patients undergoing tamoxifen therapy.
2008 The British journal of nutrition

Riboflavin deficiency and esophageal cancer: a case control-household study in the Caspian Littoral of Iran.
2005 Cancer detection and prevention

[Apoptosis of human gastric cancer cell induced by photochemical riboflavin].
2003 Ai zheng = Aizheng = Chinese journal of cancer

Riboflavin carrier protein: a serum and tissue marker for breast carcinoma.
2001 International journal of cancer. Journal international du cancer

Studies on Riboflavin and Anti-Angiogenic

http://science.naturalnews.com/R/riboflavin_and_Anti-Angiogenic.html

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Vitamin B3(Niacin)

Studies on Niacin and cancer

2007 Biological & pharmaceutical bulletin
2005 Bioscience, biotechnology, and biochemistry
2005 The American journal of gastroenterology
2007 Clinical biochemistry
2009 Breast cancer research and treatment
2008 The British journal of nutrition
2008 Nutrition and cancer
2003 Nutrition and cancer

Other studies:

ANTI-invasive activities of Niacin against cancer  cells; http://science.naturalnews.com/…/3260742_Anti_invasive_activity_of_niacin_and_ trigonelline_against_cancer_cells.html

Studies on Niacin and breast cancer

http://science.naturalnews.com/N/Niacin_and_breast_cancer.html (MANY STUDIES in this link)  

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Vitamin B5 ( pantotenic acid )

Pantothenic Acid for Stage IV Breast Cancer Treatment Information

https://www.medify.com/…/pantothenicacid-treatment-stage-iv-breast-ca

Vitamins B2, B3,B5 are respiratory vitamins(increasing cellular oxygenation –  see above Otto Wartburg proposals).

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Vitamin B6

science.naturalnews.com/V/Vitamin_B6_and_cancer.html
2003 Journal of the National cancer Institute
2008 JAMA : the journal of the American Medical Association
2008 The Journal of antimicrobial chemotherapy
2009 BMC cancer
2009 Nutrition and cancer
2007 Gastroenterology
2005 Journal of the National cancer Institute
2007 Cancer research
2006 American journal of epidemiology
2005 Nutrition and cancer
2008 Gastroenterology
2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society
2010 JAMA : the journal of the American Medical Association
2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
2005 Gastroenterology

Vitamin B12 

science.naturalnews.com/V/Vitamin_B12_and_cancer.html
Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer
Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer
2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
Effect of combined folic acid, vitamin B6, and Vitamin B12 on cancer risk in women: a randomized trial.
2008 JAMA : the journal of the American Medical AssociationPlasma Vitamin B12 concentrations and the risk of colorectal cancer: a nested case-referent study.
2008 International journal of cancer. Journal international du cancerDietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.
2009 BMC cancer

Circulating concentrations of folate and Vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study.

2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.
2009 Nutrition and cancer

Cancer incidence and mortality after treatment with folic acid and vitamin B12.
2009 JAMA : the journal of the American Medical Association

Plasma folate, vitamin B6, vitamin B12, and homocysteine and pancreatic cancer risk in four large cohorts.
2007 Cancer research

Plasma homocysteine, folate, and Vitamin B12 levels in patients with laryngeal cancer.
2008 Archives of otolaryngology–head & neck surgery

A polymorphism of the methionine synthase gene: association with plasma folate, vitamin B12, homocyst(e)ine, and colorectal cancer risk.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Folate, vitamin B6, vitamin B12, and vitamin B2 intake, genetic polymorphisms of related enzymes, and risk of colorectal cancer in a hospital-based case-control study in Japan.
2005 Nutrition and cancer

Elevated serum Vitamin B12 levels associated with CRP as a predictive factor of mortality in palliative care cancer patients: a prospective study over five years.
2000 Journal of pain and symptom management

Folate, Vitamin B12 and postmenopausal breast cancer in a prospective study of French women.
2006 Cancer causes & control : CCC

Vitamin B12 deficiency: a new risk factor for breast cancer?
1999 Nutrition reviews

Toxic epidermal necrolysis related to pemetrexed and carboplatin with Vitamin B12and folic acid supplementation for advanced non-small cell lung cancer.
2009 Onkologie

Methylenetetrahydrofolate reductase C677T genotype affects promoter methylation of tumor-specific genes in sporadic colorectal cancer through an interaction with folate/vitamin B12 status.
2008 World journal of gastroenterology : WJG

Efficacy and safety of two doses of pemetrexed supplemented with folic acid andVitamin B12 in previously treated patients with non-small cell lung cancer.
2008 Clinical cancer research : an official journal of the American Association forcancer Research

Serum Vitamin B12 and folate status among patients with chemotherapy treatment for advanced colorectal cancer.
2009 Upsala journal of medical sciences

Circulating folate, vitamin B12, homocysteine, Vitamin B12 transport proteins, and risk of prostate cancer: a case-control study, systematic review, and meta-analysis.
2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Dietary folate and Vitamin B12 intake before diagnosis decreases gastric cancermortality risk among susceptible MTHFR 677TT carriers.
2010 Nutrition (Burbank, Los Angeles County, Calif.)

Increased serum CA-15.3 levels in patients with megaloblastic anemia due to Vitamin B12 deficiency.
2004 Oncology

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Vitamin B17(Laetrile/ Amygdalin) (very important vitamin)

Note: “Laetrile” acronym for “LAEvomandeloniTRILE-glucoside”) as Amygdalin:

Laetrile has two molecules of glucose, Amygdalin has more.

Indeed, the chemical structure of Laetrile is D-1 mandelonitrile–beta-glucuronide, while

for Amygdalin it is D-mandelonitrile-bi-glucoside.

B17/ Amygdalin studies science.naturalnews.com/amygdalin.html

 How laetrile or B17 from apricot seeds kills only cancer cells ·

apricot studies science.naturalnews.com/apricot.html

Apricot Seeds Kill Cancer Cells without Side Effects

See also:  http://worldwithoutcancer.org.uk/success.html,   http://www.apricotsfromgod.info/journal.htm

Much more details(including treatments & CLINICAL CASES with B17) in my BOOK + DR. Binzel’s FREE EBOOK on this topic + Dr. Nacci’s FREE EBOOK on this topic.

NOTE:

Vitamins(including B group) should be taken from WHOLE FOODS(organic) (as much as possible),NOT from supplements as vitamins and nutrients need other vitamins and nutrients for proper usage within the body.

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Vitamin C (very important vitamin)

Vitamin C   is a respiratory vitamin- it stimulates the body to produce its own hydrogen peroxide (increasing cellular oxygenation –  see above Otto Wartburg proposals).

Therefore, megadoses of vitamin C are administered orally and intravenously by orthomolecular physicians to treat cancer.

60 references to studies published that show the importance of vitamin C:

www.doctoryourself.com / biblio_cameron.html http://www.doctoryourself.com/cancer_hoffer.html.

Even National Institutes of Health is actively sponsoring studies of high-dose intravenous vitamin C therapy.

Other MULTIPLE studies on  vitamin’s C cancer fighting effects: science.naturalnews.com/…/4854330_Vitamin_C_in_alternative_cancer_ treatment_historical_background.html

High doses of vitamin C and cancer :

science.naturalnews.com/…/2387600_High_dose_vitamin_C_supplement_ use_is_associated_with_self.html

Vitamin C and breast cancer studies: science.naturalnews.com / V / Vitamin_C_and_breast_cancer.html

Vitamin C , vitamin E & selenium in breast cancer : science.naturalnews.com/…/1198191_Dietary_beta_carotene_vitamin_C_and _E_intake_and_breast.html science.naturalnews.com/…/3959947_Association_between_breast_cancer_ and_vitamin_C_vitamin_E_and.html

Vitamin C ,vitamin A & zinc  in laryngeal cancer  :

science.naturalnews.com/…/4508069_Association_of_vitamin_A_vitamin_C _and_zinc_with_laryngeal.html

Vitamin C &  gastric cancer:

science.naturalnews.com/…/3603872_Plasma_and_dietary_vitamin_C_levels _and_risk_of_gastric.html science.naturalnews.com/…/3628783_Prediagnostic_plasma_vitamin_C_ levels_and_the_subsequent_risk_of.html science.naturalnews.com/…/4790082_Effect_of_physiological_ concentrations_of_vitamin_C_on_gastric_cancer.html

Also see: http://orthomolecular.org/resources/omns/v04n19.shtml

There is also an extensive bibliography on the anti-tumoral action of vitamin C (25,33,47,54,83,91,122,129,181,197,202,218,244,246,270,299,311,335,339,367,404,405,414,415,416,489,496,510,511)

See the bibliography and studies quoted on last page of databse.

The pioneers of this oncological therapy were Pauling, who received the Nobel Prize for Chemistry, and the Italian Pantellini.

Ascorbic acid is mainly known for its ability to reduce metallic ions in various enzymatic processes and above all for its ability to act as an anti-oxidant agent, thus able to remove free radicals, reducing the damage caused at a genome level.

Furthermore it may be able to block the formation of nitrose at a gastro-intestinal level, as well as carrying out a preventative action on the formation of adenomatose polyps.

Even though ascorbic acid is well known for its collagene forming action, and the well known effect of scurvy in cases of reduction or absence of this acid in the diet, this vitamin is also important, alongside vitamins A and E as a first class anti-tumoral agent.

It is a respiratory vitamin(like vitamins E, B3, B5)  that helps increase cellular oxygen levels.

It reinforces the intercellular bond and forestalls the destructive action of the hyalurons produced by many neoplastic cells.

Vitamin C stimulates the Natural killer lymphocytes, it supports the macrophagic activity, the chemio-tactical mobility of the white corpuscles, the production of antibodies and the response of the T cytotoxic lymphocytes to the antigenes.

Cameron found very low plasmatic levels of vitamin C in cancer patients (0.26 mg/100 mL) compared to normal plasmatic values (54).

In other studies, 154 cancer patients, undergoing analysis, were found to have low levels of vitamin C not only in their plasma (0.31 mg/100 mL), but also in their leucocytes (15.9 mg/10 E+8), with a positive correlation (r = 0.42) between these two values; in particular the authors attributed the alteration in the immune response to the tumor, especially of the phagocytosis, to the low  concentration of vitamin C in the white blood corpuscles.

Already in 1974 Goetz had shown that vitamin C, in vitro, was capable of stimulating the motility and the chemiotaxis of neutrophiles.

The doses advised in literature for anti-neoplastic therapy are about 3-10 grams a day, reaching even 40 grams daily, because the vitamin is not toxic, at high doses it only has a laxative effect.

It would be better to take it by eating fresh fruit, but it is difficult to reach such high doses of the vitamin in this way.

Integrating fresh fruit (kiwi, oranges, lemons and grapefruit) with the juice of sea buckthorn  , Rosa canina (dog rose, wild rose) to reach a daily dosage of at least 5- 8 grams of natural vitamin C, avoiding however, the use of pharmaceutically prepared vitamin C tablets.

Natural vitamin C, rich in its metabolites and other components called bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercitin, Pychnogenol, etc) is more powerful and efficient, and furthermore is devoid of unpleasant gastric effects which are the result of high doses of synthetic vitamin C.

Natural vitamin C is moreover, characterized by a significant reduction in the formation of Calcium oxalate in the kidneys, as opposed to synthetic vitamin C. It is also easier for the intestine to absorb and has greater bio-availability, above all through its most important metabolites such as tronic acid, lixonic acid, xilonic acid etc.

This bio-availability has a critical importance in the immune defense system because the white blood cells tend to absorb Natural vitamin C 4 times more than they absorb synthetic vitamin C.

Recently, Myrciaria paraensis (camu-camu) has appeared on the European market. It is a small exotic fruit, similar to a small orange, but it contains 50 times more natural vitamin C than Citrus aurantium (orange), and it could therefore provide the daily dosage, of at least 3 grams, of vitamin  .

Also Malpighia punicifolia (acerola), a cherry from the Antilles, is very rich in vitamin C, containing 50 to 100 times more than citrus fruits.

Natural vitamin C is therefore efficient because it is naturally associated to the bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercetine,Quercitine, Pychnogenol, etc..) and other molecules, in plants often characterized by an immune  stimulating activity (Echinacea purpurea, Plantago major, Capsicum frutescens)

Note: With very high therapeutic doses (>8-10 grams a day), Magnesium (e.g. Dolomite) must also be taken to avoid the risk of kidney stones.

Here enclosed scientific papers extracted from Catherine Kousmine (“Save your body”, page 129,

“Effects of C vitamin on our body according to Linus Pauling, edition Tecniche Nuove):

“…an intake of 1500 milligrams of ascorbic acid by mouth determines a concentration of 1.5 milligrams of C vitamin for each 100 millilitres of blood. By increasing the intake, the concentration suddenly increases up to 2.5 milligrams and then goes back to 1.5 millilitres for each 100 millilitres blood. There are enzymes which help the conversion of most

ascorbates into useful oxidation products. If the intake remains high, the body increases the amount of enzymes useful to the conversion; otherwise, if the ascorbic acid dose is suddenly reduced for some days, an excess of conversion enzymes and then a too law level of vitamin C in the blood occur. This means a number of disorders, as for example a higher sensibility to infections. The adaptation to a lower proportion takes place by reducing the number of conversion enzymes: it is necessary to gradually decrease the dose of vitamin C. By taking 100 milligrams a day and in presence of a plasmatic level of 1 milligram for every 100 millilitres blood, urines do not contain ascorbic acid because it is reabsorbed by renal tubules. If the intake is higher than 100 milligrams, i.e. 1-2 grams a day, 25% go in urines and the rest is kept by the body. Healthy people, who lack in vitamin C for some months, have to take 2-4 grams in order to eliminate them through urines. In case of cancer patients, who are used to take high doses of ascorbic acid, an interruption of some days requires an intake of 50 grams (fifty grams) of vitamin C so that this one can be found in urines”.

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Vitamin D (very important vitamin)

Cancer prevention by a MINIMUM of 77% cases just by sun exposure(best source of vitamin D): http://www.naturalnews.com/035063_vitamin_D_cancer_facts.html

Cancer treatment with sun exposure(vitamin D): http://www.polymvasurvivors.com/what_you_know.html#Vitamin%20D%20

NOTE:The link above contains multiple studies on sun exposure(vitamin D) and it’s crucial importance in fighting cancer:

Circulating Vitamin D levels in the Blood Dramatically Reduces Death Rate

* For each 10ng/ml increase in the blood level of vitamin D, the death rate from cancer would be reduced by 29%. The relationship between vitamin D and colorectal cancer is particularly strong.

The death rate for colorectal cancer would be reduced by 49%. (30 – Read Supporting Scientific Article on PUBMED)

Vitamin D Levels est during Summer = Improves survival by 40%

A Norwegian study showed that cancer patients diagnosed in the summer when vitamin D levels are highest had up to 40% better survival rates than patients diagnosed in the winter when vitamin D levels are at their lowest.

(56 – Read Supporting Scientific Article on PUBMED )

Low Vitamin D Levels Greater chance of Death & Metastasis

* Breast cancer patients with low levels of vitamin D followed over eleven years had a 70% greater chance of dying and twice the rate of developing metastasis than patients with high levels of vitamin D.

(90 – Read Supporting Scientific Article )

Lung Cancer Patients Higher levels = Dramatic increase in Survival

Early stage lung cancer patients who were diagnosed in the summer and had the highest levels of vitamin D had a five-year survival of 73% compared to 30% for those diagnosed in the winter with low vitamin D levels.

(60 – Read Supporting Scientific Article on PUBMED)

Lung – Colon – Prostate – Renal – Endometrial Cancers Higher Vit D levels dramatically decreases Risk

*High vitamin D levels (based on latitude and UV exposure) also correlate with a decreased risk of developing lung cancer, a 45% and 65% reduction in men and women respectively. Similar effects have been demonstrated in colonprostaterenal and endometrial cancer. (33 – Read Supporting Scientific Article on BMJ(British Medical Journal))

Advanced Colorectal Patients Risk of death greatly Improved

The risk of death for colorectal cancer patients with advanced disease but with high levels of vitamin D was reduced by over 60% compared to patients with low vitamin D levels. (64 – Read Supporting Scientific Article on PUBMED)

Prostate Cancer Patients

*Prostate Cancer Patients with Vitamin D levels in the mid and high range had a 60% and 85% respectively reduced risk of death from the condition compared to patients with low levels of Vitamin D. This is almost a seven fold increase in the risk of death in those with low vs high vitamin D levels. (152 – Read Supporting Scientific Article on PUBMED)

There is also an extensive bibliography on the anti-tumoral action of vitamin D- see last page of this database:  (28,157,160,188,208,209,231,240,246, 254,302,323,479,489)

Vitamin D is BEST taken from skin exposure to the sun, MINIMUM 30 mins daily,without any sunscreen or protection. Avoid burning and avoid the interval 11 am – 16 pm.

Natural vitamin D, contained in some plants, is however preferable to the synthetic type, because the latter is about 10 times more capable of binding with Magnesium, taking it away from the organism, thus causing all the damage that the loss of this incurs (osteoporosis, kidney stones).

Vitamin D induces the inhibition of neoplastic cellular growth: this has been shown in vitro in neoplastic cellular lines; especially of the hematopoietic system, of the CNS, of the prostate; the  colon, the ovaries and the breasts.

This action is thought to be expressed at various levels, in particular:

a) by means of apoptosis induction, through the activation of p21, that is, the inhibitor of the kinase proteins;

b) the inhibition of neoplastic cellular growth, which would be blocked in the G1 phase because of the action of the IGF1 inhibitor;

c) by means of cellular differentiation.

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Vitamin E(respiratory vitamin)

This liposoluble substance consists of a group of various components, called Tocopherols.

Seven of these exist in nature; alpha-Tocopherol, beta-Tocopherol, gamma- Tocopherol, delta-Tocopherol, epsilon-Tocopherol, zetaTocopheros and eta-Tocopherol.

Studies on Vitamin E and cancer

Supplemental Vitamin E intake and prostate cancer risk in a large cohort of men in the United States.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

The role of Vitamin E in the prevention of cancer: a meta-analysis of randomized controlled trials.

2007 Annals of Saudi medicine
Vitamin E and selenium supplementation and risk of prostate cancer in the Vitamins and lifestyle (VITAL) study cohort.
2008 Cancer causes & control : CCCVitamin E suppresses telomerase activity in ovarian cancer cells.
2007 Cancer detection and preventionEffects of long-term Vitamin E supplementation on cardiovascular events and cancer: a randomized controlled trial.
2005 JAMA : the journal of the American Medical AssociationSupplemental and dietary Vitamin E intakes and risk of prostate cancer in a large prospective study.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Antioxidants vitamin C and Vitamin E for the prevention and treatment of cancer.
2006 Journal of general internal medicine

Vitamin E: the evidence for multiple roles in cancer.
2003 Nutrition and cancer

Vitamin E supplements and risk of prostate cancer in U.S. men.
2004 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Serum and dietary Vitamin E in relation to prostate cancer risk.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of variants in two Vitamin E transport genes with circulating Vitamin Econcentrations and prostate cancer risk.
2009 Cancer research

Association between breast cancer and vitamin C, Vitamin E and selenium levels: results of a case-control study in India.
2005 Asian Pacific journal of cancer prevention : APJCP

A short-term dietary supplementation with high doses of Vitamin E increases NK cell cytolytic activity in advanced colorectal cancer patients.
2007 Cancer immunology, immunotherapy : CII

SELECT: the next prostate cancer prevention trial. Selenum and Vitamin E cancerPrevention Trial.
2001 The Journal of urology

Vitamin C and Vitamin E supplement use and colorectal cancer mortality in a large American cancer Society cohort.
2001 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Vitamin E and breast cancer.
2004 The Journal of nutrition

SELECT: the selenium and Vitamin E cancer prevention trial.
2003 Urologic oncology

The selenium and Vitamin E cancer prevention trial.
2003 World journal of urology

Selenium and Vitamin E cancer prevention trial.
2004 Annals of the New York Academy of Sciences

Selenium and Vitamin E cancer Prevention Trial: a nutrient approach to prostatecancer prevention.
2009 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

A nutrient approach to prostate cancer prevention: The Selenium and Vitamin Ecancer Prevention Trial (SELECT).
2010 Nutrition and cancer

Null activity of selenium and Vitamin E as cancer chemopreventive agents in the rat prostate.
2010 Cancer prevention research (Philadelphia, Pa.)

Vitamin E and cancer.
2007 Vitamins and hormones

Clinical models for testing chemopreventative agents in prostate cancer and overview of SELECT: the Selenium and Vitamin E cancer Prevention Trial.
2003 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

Cancer Preventive Effects of Vitamin E.
2011 Current pharmaceutical biotechnology

Vitamin E but not St. John’s wort mitigates leukopenia caused by cancerchemotherapy in rats.
2006 Translational research : the journal of laboratory and clinical medicine

Vitamin C and Vitamin E supplement use and bladder cancer mortality in a large cohort of US men and women.
2002 American journal of epidemiology

Review of Vitamin E and selenium in the prevention of prostate cancer: implications of the selenium and Vitamin E chemoprevention trial.
2002 Integrative cancer therapies

Vitamin E and breast cancer prevention: current status and future potential.
2003 Journal of mammary gland biology and neoplasia

Vitamin E and breast cancer prevention: current status and future potential.
2003 Journal of mammary gland biology and neoplasia

Molecular epidemiologic studies within the Selenium and Vitamin E cancerPrevention Trial (SELECT).
2001 Cancer causes & control : CCC

Selenium and vitamin E: cell type- and intervention-specific tissue effects in prostate cancer.

2009 Journal of the National cancer Institute

Tocopherol transfer protein sensitizes prostate cancer cells to vitamin E.
2010 The Journal of biological chemistry

Selenium and Vitamin E for prostate cancer: post-SELECT (Selenium and Vitamin Ecancer Prevention Trial) status.
2011 Molecular medicine (Cambridge, Mass.)

Vitamin E and cancer: An insight into the anticancer activities of Vitamin E isomers and analogs.
2008 International journal of cancer. Journal international du cancer

Vitamin E intake, alpha-tocopherol status, and pancreatic cancer in a cohort of male smokers.
2009 The American journal of clinical nutrition

The roles of alpha-vitamin E and its analogues in prostate cancer.
2007 Vitamins and hormones

Combined lycopene and Vitamin E treatment suppresses the growth of PC-346C human prostate cancer cells in nude mice.
2006 The Journal of nutrition

Vitamin E, alpha- and gamma-tocopherol, and prostate cancer.
1999 Seminars in urologic oncology

Synergistic effect of Vitamin E and selenium in human prostate cancer cell lines.
2004 Prostate cancer and prostatic diseases

Gamma (gamma) tocopherol upregulates peroxisome proliferator activated receptor (PPAR) gamma (gamma) expression in SW 480 human colon cancer cell lines.
2003 BMC cancer

Vitamin E succinate inhibits colon cancer liver metastases.
2002 The Journal of surgical research

Lycopene and Vitamin E interfere with autocrine/paracrine loops in the Dunning prostate cancer model.
2004 The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Higher baseline serum concentrations of Vitamin E are associated with lower total and cause-specific mortality in the Alpha-Tocopherol, Beta-Carotene cancerPrevention Study.
2006 The American journal of clinical nutrition

Alpha-tocopheryl succinate, the most effective form of Vitamin E for adjuvant cancertreatment: a review.
2003 Journal of the American College of Nutrition

gamma-Tocopherol or combinations of Vitamin E forms induce cell death in human prostate cancer cells by interrupting sphingolipid synthesis.
2004 Proceedings of the National Academy of Sciences of the United States of America

Experimental basis for cancer prevention by vitamin E.
2000 Cancer investigation

Vitamin E analogues as a novel group of mitocans: anti-cancer agents that act by targeting mitochondria.
2007 Molecular aspects of medicine

Vitamin E succinate decreases lung cancer tumor growth in mice.
2005 The Journal of surgical research

RRR-alpha-vitamin E succinate potentiates the antitumor effect of calcitriol in prostatecancer without overt side effects.
2009 Clinical cancer research : an official journal of the American Association forcancer Research

Serum levels of folate, lycopene, ß-carotene, retinol and Vitamin E and prostatecancer risk.
2010 European journal of clinical nutrition

Trace elements and Vitamin E status in Nigerian patients with prostate cancer.
2010 African health sciences

Designing the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2005 Journal of the National cancer Institute

Vitamin E analogues and immune response in cancer treatment.
2007 Vitamins and hormones

Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Vitamin E serum levels and gastric cancer: results from a cohort of patients in Tuscany, Italy.
2000 Cancer letters

Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Effects of topical L-selenomethionine with topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
2003 Journal of the American Academy of Dermatology

Vitamin E and the Y4 agonist BA-129 decrease prostate cancer growth and production of vascular endothelial growth factor.
2002 The Journal of surgical research

Pro-apoptotic mechanisms of action of a novel Vitamin E analog (alpha-TEA) and a naturally occurring form of Vitamin E (delta-tocotrienol) in MDA-MB-435 human breast cancer cells.
2004 Nutrition and cancer

Minority recruitment to the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2005 Clinical trials (London, England)

Salubrious effect of vitamin C and Vitamin E on tamoxifen-treated women in breastcancer with reference to plasma lipid and lipoprotein levels.
2000 Cancer letters

Design of Physicians’ Health Study II–a randomized trial of beta-carotene, vitamins E and C, and multivitamins, in prevention of cancer, cardiovascular disease, and eye disease, and review of results of completed trials.
2000 Annals of epidemiology

Vitamin E succinate promotes breast cancer tumor dormancy.
2000 The Journal of surgical research

Mitocans as anti-cancer agents targeting mitochondria: lessons from studies withVitamin E analogues, inhibitors of complex II.
2007 Journal of bioenergetics and biomembranes

Centralized blood processing for the selenium and Vitamin E cancer prevention trial: effects of delayed processing on carotenoids, tocopherols, insulin-like growth factor-I, insulin-like growth factor binding protein 3, steroid hormones, and lymphocyte viability.
2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

The therapeutic and preventive effect of RRR-alpha-vitamin E succinate on prostatecancer via induction of insulin-like growth factor binding protein-3.
2007 Clinical cancer research : an official journal of the American Association forcancer Research

Vitamin E and prostate cancer: is Vitamin E succinate a superior chemopreventive agent?
2005 Nutrition reviews

Aspirin prevents stroke but not MI in women; Vitamin E has no effect on CV disease or cancer.
2006 Cleveland Clinic journal of medicine

Effect of Vitamin E on tamoxifen-treated breast cancer cells.
2006 Surgery

Dietary administration of the proapoptotic Vitamin E analogue alpha-tocopheryloxyacetic acid inhibits metastatic murine breast cancer.
2006 Cancer research

Combination of Vitamin E and selenium causes an induction of apoptosis of human prostate cancer cells by enhancing Bax/Bcl-2 ratio.
2008 The Prostate

Non-hormonal treatment of hot flushes in breast cancer survivors: gabapentin vs. vitamin E.
2009 Climacteric : the journal of the International Menopause Society

Vitamin E analog alpha-TEA, methylseleninic acid, and trans-resveratrol in combination synergistically inhibit human breast cancer cell growth.
2008 Nutrition and cancer

Poly(L-lactide)-vitamin E TPGS nanoparticles enhanced the cytotoxicity of doxorubicin in drug-resistant MCF-7 breast cancer cells.
2010 Biomacromolecules

Vitamin E succinate induced apoptosis and enhanced chemosensitivity to paclitaxel in human bladder cancer cells in vitro and in vivo.
2010 Cancer science

Tocopherol-associated protein suppresses prostate cancer cell growth by inhibition of the phosphoinositide 3-kinase pathway.
2005 Cancer research

Differential retention of alpha-vitamin E is correlated with its transporter gene expression and growth inhibition efficacy in prostate cancer cells.
2007 The Prostate

Vitamin E succinate (VES) induces Fas sensitivity in human breast cancer cells: role for Mr 43,000 Fas in VES-triggered apoptosis.
1999 Cancer research

Effects of topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
2000 Nutrition and cancer

Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
2003 Cancer research

Vitamin E analogs trigger apoptosis in HER2/erbB2-overexpressing breast cancercells by signaling via the mitochondrial pathway.
2005 Biochemical and biophysical research communications

Vitamin E and prostate cancer.
2002 The Urologic clinics of North America

Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
2003 Cancer research

Peptide YY augments gross inhibition by Vitamin E succinate of human pancreaticcancer cell growth.
2000 The Journal of surgical research

Supplemental and dietary vitamin E, beta-carotene, and vitamin C intakes and prostatecancer risk.
2006 Journal of the National cancer Institute

Vitamin E in the primary prevention of cardiovascular disease and cancer: the Women’s Health Study: a randomized controlled trial.
2005 JAMA : the journal of the American Medical Association

Dietary intakes of retinol, beta-carotene, vitamin D and Vitamin E in the European Prospective Investigation into cancer and Nutrition cohort.
2009 European journal of clinical nutrition

Mitochondrial targeting of Vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II.
2011 The Journal of biological chemistry

Impact of supplemental site grants to increase African American accrual for the Selenium and Vitamin E cancer Prevention Trial.
2010 Clinical trials (London, England)

The Outcome of Selenium and Vitamin E cancer Prevention Trial (SELECT) reveals the need for better understanding of selenium biology.
2009 Molecular interventions

Critical roles for JNK, c-Jun, and Fas/FasL-Signaling in Vitamin E analog-induced apoptosis in human prostate cancer cells.
2008 The Prostate

Effect of selenium and Vitamin E on risk of prostate cancer and other cancers: the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2009 JAMA : the journal of the American Medical Association

Tocotrienols, the Vitamin E of the 21st century: its potential against cancer and other chronic diseases.
2010 Biochemical pharmacology

Mixed PEG-PE/vitamin E tumor-targeted immunomicelles as carriers for poorly soluble anti-cancer drugs: improved drug solubilization and enhanced in vitro cytotoxicity.
2008 European journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft für Pharmazeutische Verfahrenstechnik e.V

Nanoparticles of poly(lactide)/vitamin E TPGS copolymer for cancer chemotherapy: synthesis, formulation, characterization and in vitro drug release.
2006 Biomaterials

Involvement of JNK/p73/NOXA in Vitamin E analog-induced apoptosis of human breast cancer cells.
2008 Molecular carcinogenesis

Vitamin E supplementation in cardiovascular disease and cancer prevention: Part 1.
2005 The Annals of pharmacotherapy

A peptide conjugate of Vitamin E succinate targets breast cancer cells with high ErbB2 expression.
2007 Cancer research

Vitamin E succinate inhibits NF-kappaB and prevents the development of a metastatic phenotype in prostate cancer cells: implications for chemoprevention.
2007 The Prostate

The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

Vitamin E succinate and cancer treatment: a Vitamin E prototype for selective antitumour activity.
2003 British journal of cancer

Vitamin E down-modulates iNOS and NADPH oxidase in c-Myc/TGF-alpha transgenic mouse model of liver cancer.
2004 Journal of hepatology

Vitamin E inhibits the high-fat diet promoted growth of established human prostate LNCaP tumors in nude mice.
1999 The Journal of urology

Vitamin E analog alpha-TEA and celecoxib alone and together reduce human MDA-MB-435-FL-GFP breast cancer burden and metastasis in nude mice.
2004 Breast cancer research and treatment

Development of gamma (gamma)-tocopherol as a colorectal cancer chemopreventive agent.
2003 Critical reviews in oncology/hematology

Vitamin E analogues: a new class of inducers of apoptosis with selective anti-cancer effects.

2004 Current cancer drug targets

The role of PC-SPES, selenium, and Vitamin E in prostate cancer.
2002 Oncology (Williston Park, N.Y.)

Differential response of human ovarian cancer cells to induction of apoptosis byVitamin E Succinate and Vitamin E analogue, alpha-TEA.
2004 Cancer research

A short-term dietary supplementation of high doses of Vitamin E increases T helper 1 cytokine production in patients with advanced colorectal cancer.
2002 Clinical cancer research : an official journal of the American Association forcancer Research

Role of caspase-8 activation in mediating vitamin E-induced apoptosis in murine mammary cancer cells.
2003 Nutrition and cancer

High dietary level of synthetic Vitamin E on lipid peroxidation, membrane fatty acid composition and cytotoxicity in breast cancer xenograft and in mouse host tissue.
2003 Cancer cell international

[Roles of ERK1/2 MAPK in Vitamin E succinate-induced apoptosis in human gastriccancer SGC-7901 cells].
2003 Wei sheng yan jiu = Journal of hygiene research

Alpha-vitamin E derivative, RRR-alpha-tocopheryloxybutyric acid inhibits the proliferation of prostate cancer cells.
2007 Asian journal of andrology

Roles of Fas signaling pathway in Vitamin E succinate-induced apoptosis in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

Mechanisms mediating the antiproliferative and apoptotic effects of Vitamin E in mammary cancer cells.
2005 Frontiers in bioscience : a journal and virtual library

Cellular and molecular effects of alpha-tocopheryloxybutyrate: lessons for the design of Vitamin E analog for cancer prevention.
2004 Anticancer research

Vitamin E and cancer.
2009 Nutrition and cancer

Pentoxifylline and Vitamin E treatment for prevention of radiation-induced side-effects in women with breast cancer: a phase two, double-blind, placebo-controlled randomised clinical trial (Ptx-5).
2009 European journal of cancer (Oxford, England : 1990)

Vitamin E prevents lipid raft modifications induced by an anti-cancer lysophospholipid and abolishes a Yap1-mediated stress response in yeast.
2010 The Journal of biological chemistry

Analogs of Vitamin E epitomized by alpha-tocopheryl succinate for pancreatic cancertreatment: in vitro results induce caution for in vivo applications.
2010 Pancreas

Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
2009 Asian Pacific journal of cancer prevention : APJCP

[The study of apoptosis induction effect of Vitamin E succinate on Tca8113 human tongue cancer cells].
2008 Hua xi kou qiang yi xue za zhi = Huaxi kouqiang yixue zazhi = West China journal of stomatology

Antioxidant status and risk of cancer in the SU.VI.MAX study: is the effect of supplementation dependent on baseline levels?
2005 The British journal of nutrition

Double-blind placebo-controlled randomised trial of Vitamin E and pentoxifylline in patients with chronic arm lymphoedema and fibrosis after surgery and radiotherapy for breast cancer.
2004 Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology

Vitamin E succinate induces apoptosis in human prostate cancer cells: role for Fas inVitamin E succinate-triggered apoptosis.
2000 Nutrition and cancer

Vitamin E and other antioxidants inhibit human prostate cancer cells through apoptosis.
2000 The Prostate

Vitamin E succinate suppresses prostate tumor growth by inducing apoptosis.
2006 International journal of cancer. Journal international du cancer

In vitro and in vivo evaluation of anticancer actions of natural and synthetic Vitamin Eforms.
2008 Molecular nutrition & food research

Vitamin E analogues as anticancer agents: lessons from studies with alpha-tocopheryl succinate.
2006 Molecular nutrition & food research

Prospective study of serum Vitamin E levels and esophageal and gastric cancers.
2003 Journal of the National cancer Institute

Systematic review on „vitamin E and prevention of colorectal cancer”.
2010 Pakistan journal of pharmaceutical sciences

Vitamin E analogues as mitochondria-targeting compounds: from the bench to the bedside?
2009 Molecular nutrition & food research

science.naturalnews.com/…/1364373_Vitamin_E_and_cancer_An_insight_ into_the_anticancer_activities.html

There is also an extensive bibliography on the anti-tumoral action of vitamin E -see last page of DATABASE (6,19,20,30,45,91,95,112, 125,129,142,165,167,190,202,228,229,246,261,280,332,404,405,452,494)

This liposoluble substance consists of a group of various components, called Tocopherols. Seven of these exist in nature; alpha, beta, gamma, delta, epsilon, zeta and eta.

Alpha-Tocopherol has an anti-oxidant effect on the lipidic membranes in synergy with Melatonin(see below), carrying out a preventative action on the peroxidization of the cellular membrane induced by  ionizing radiation and by chemical carcinogenes.

Vitamin E also carries out an anti-oxidative action in a wider sense, acting as a ‘scavenger’ of the free radicals, similar to vitamin C.

It performs a stimulating activity on the immune system; it induces cellular differentiation; it inhibits, in a selective way, cellular growth intervening at DNA and RNA synthesis level.

Various studies have shown its ability to induce apoptosis in cellular lines of breast carcinomas and lymphoma B.

It is inactivated by Iron, therefore it is essential that any medicines based on Iron are not taken at the same time as vitamin E, but at a distance of at least 10-12 hours.

Aluminum, which is often present in pharmaceutical products, also deactivates vitamin E.

The uncooked oil of Triticum sativum (wheat germ; note: has vitamin B12) contains about twice as much vitamin E as the uncooked oil from the seeds of Helianthus annuus (sunflower) and the latter  contains about five times as much as uncooked olive oil. Furthermore all these seeds are rich in  essential unsaturated fats, an important part of the diet for cancer patients.

In anti-neoplastic therapy much is being discussed about: the raw seeds of Helianthus annuus

(which the author personally considers useful in therapy), wheat shoots (the author is not in favor),

Saccharomyces cerevisiae (yeast, of which the author is not in favor), and the shoots of soya  lecithin (of dubious use and the author is against their use because of the transgenic risk).

Natural vitamins are by far preferable to industrially produced ones: Dracontium loretense, for example, which is considered one of the best  plants for its specific anti-oxidant potential, is of superior quality in its anti-oxidant ability compared to synthetic vitamin E (566). WHOLE, ORGANIC FOODS are BEST due to COMPLEXES of NUTRIENTS !

As an already extracted natural product, together with or without other vitamins, vitamin E must be  given in addition to high quantities of raw seeds of Helianthus annuus (also containing vitamin A,all the vitamin B compounds, vitamin D, Manganese, Zinc and Magnesium) and of high quantities of raw Triticum sativum (which is rich in the precious alpha-lipoic acid): both are also very rich in vitamin B6 (pyridoxin), the latter is important for the immune system, but it is difficult to find in other compatible foods for a suitable diet for cancer patients. Pyridoxin, in fact, is contained especially in Saccharomyces cerevisiae, the latter is a food which the author does not regard favorably for an anti-neoplastic diet, because it contains high quantities of folic acid.

Natural Octacosanol, extracted from the oil of Triticum sativum, has a synergetic action with vitamin E, but it is, in any case, better to consume it with all the uncooked oil of Triticum sativum  and/or Triticum sativum itself rather than taking it already extracted, as a pharmaceutical product  (because it loses its active principles).

Vitamin E is particularly efficient in combination with Selenium, which is contained in Aloe species, Solanum lycopersicum (tomatoes), Equisetum species, Allium cepa (onions).

There is also an extensive bibliography on Selenium (see last page of DATABASE) :

79,108,112,129,133,136,143,156,228,229,276,338,339,364,367,404,405,407,443,452,458,501,510,511)

Both vitamin E and Selenium are in their turn synergetic with Zinc in inhibiting the production of inflammatoryprostoglandins and leukotrienes.

Vitamin E conversion factors

Vitamin E is expressed in milligrams of tocopheral equivalents (T.E.)

1 milligram of Tocopherol is equal to:

= 1 milligram of D-alpha Tocopherol

= 2 milligrams of D-beta Tocopherol

= 5 milligrams of D-gamma Tocopherol

==========================================================================================================

Vitamin F  (very important vitamin-see omega 3)

Polyunsaturated fatty acids :

arachidonic acid, Linoleic cis-cis natural acid (vitamin F1) as: alpha-lipoic acid,  alpha-linolenic acid,etc.

Alpha-lipoic acid

Food Sources: 

spinach, broccoli, tomato, peas, Brussels sprouts, rice bran

Mechanism of action:

Alpha-lipoic acid acts as a lipophilic free radical scavenger. Dihydrolipoic acid (DHLA), a reduced form of lipoic acid, has more potent antioxidant effects. It can assist in repairing oxidative damage and regenerate endogenous antioxidants such as vitamin C, vitamin E, and glutathione.

Both DHLA and lipoic acid also have metal chelating capacities.

As a lipoamide, alpha-lipoic acid functions as a cofactor in various multienzyme systems involved in the decarboxylation of alpha-keto acids such as pyruvate. (13) (14) (15)

Alpha-lipoic acid caused cell cycle arrest in G0/G1 in FaDu and Jurkat human tumor cell lines (1).

Alpha lipoic acid was also found to scavenge reactive oxygen species in MCF-7 breast cancer cells (16).

Reduction of reactive oxygen species was then followed by cancer cell growth arrest and apoptosis (16).

For studies quoted see http://www.mskcc.org/cancer-care/herb/alpha-lipoic-acid  

Other studies:

science.naturalnews.com/A/AlphaLipoic_Acid_and_cancer.html

Alpha-Linolenic Acid(omega 3)

Food sources:flaxseed, avocado, walnuts, almonds, fishoils

Mechanism of action:

The alpha-linolenic acid (vitamin F), for instance, is a cis-polyunsaturated fatty acid that is contained in linseed cold-pressed oil: it is transformed into EPA and DHA (Omega-3 fatty acids) and is quite effective against malignant tumours, as shown by Pardini ( 1647 );

Moreover, Noguchi has proved that Omega-3 fatty acids, unlike Omega-6 fatty acids, help reduce tumour masses, although

Omega-6 fatty acids are unsaturated fatty acids, too ( 1654 ).

see  last page of DATABASE for studies quoted.

science.naturalnews.com/A/AlphaLinolenic_Acid_and_cancer.html
Alpha-Linolenic Acid and risk of prostate cancer: a case-control study in Uruguay.
2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
HER2 (erbB-2)-targeted effects of the omega-3 polyunsaturated fatty acid, Alpha-Linolenic Acid (ALA; 18:3n-3), in breast cancer cells: the „fat features” of the „Mediterranean diet” as an „anti-HER2 cocktail”.
2006 Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National cancer Institute of MexicoInhibitory effect of conjugated Alpha-Linolenic Acid from bifidobacteria of intestinal origin on SW480 cancer cells.
2009 LipidsProspective studies of dietary Alpha-Linolenic Acid intake and prostate cancer risk: a meta-analysis.
2010 Cancer causes & control : CCC

Dietary Alpha-Linolenic Acid is associated with reduced risk of fatal coronary heart disease, but increased prostate cancer risk: a meta-analysis.
2004 The Journal of nutrition

Growth-inhibitory and proapoptotic effects of Alpha-Linolenic Acid on estrogen-positive breast cancer cells.
2009 Annals of the New York Academy of Sciences

Comparison of stearidonic acid and Alpha-Linolenic Acid on PGE2 production and COX-2 protein levels in MDA-MB-231 breast cancer cell cultures.
2005 The Journal of nutritional biochemistry

Conjugation with Alpha-Linolenic Acid improves cancer cell uptake and cytotoxicity of doxorubicin.
2009 Bioorganic & medicinal chemistry letters

Does Alpha-Linolenic Acid in combination with linoleic acid influence liver metastasis and hepatic lipid peroxidation in BOP-induced pancreatic cancer in Syrian hamsters?
2000 Prostaglandins, leukotrienes, and essential fatty acids

science.naturalnews.com/…/542108_Conjugation_with_alpha_linolenic_acid _improves_cancer_cell_uptake_and.html
Conjugation with alpha linolenic acid improves cancer cell uptake and cytotoxicity of doxorubicin.
science.naturalnews.com/…/2462104_Inhibitory_effect_of_conjugated_alpha _linolenic_acid_from_bifidobacteria_of.html
Inhibitory effect of conjugated alpha linolenic acid from bifidobacteria of intestinal origin on SW480 cancer cells. Publication: Lipids Publication Date: 2009
science.naturalnews.com/…/1637124_Growth_inhibitory_and_proapoptotic_ effects_of_alpha_linolenic_acid_on.html
We tested the anticarcinogenic effect of AlphaLinolenic Acid (ALA) as a single compound. To test the role of ALA in breast cancer cells (MCF-7), we analyzed 
science.naturalnews.com/…/2859351_Effects_of_omega_3_fatty_acids_on_ cancer_risk_a.html
Effects of omega 3 fatty acids on cancer risk: a systematic review.
science.naturalnews.com/…/3442786_Modulation_of_prostate_cancer_ genetic_risk_by_omega_3_and.html
Modulation of prostate cancer genetic risk by omega 3 and omega 6 fatty acids.
science.naturalnews.com/…/3897405_Effect_of_altering_dietary_omega_6_ omega_3_fatty_acid.html
Effect of altering dietary omega 6 omega 3 fatty acid ratios on prostate cancer membrane composition, cyclooxygenase 2, and prostaglandin E2.
science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html
Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids prostate cancer (PCa) risk, whereas a diet rich in omega3 decreases risk.
science.naturalnews.com/…/296576_Beneficial_effects_of_omega_3_long_ chain_fatty_acids_in.html
Beneficial effects of omega 3 long chain fatty acids in breast cancer and cardiovascular diseases: voltage gated sodium channels as a common feature?
science.naturalnews.com/…/1200161_Fatty_fish_and_fish_omega_3_fatty_ acid_intakes_decrease.html
Although it is believed that fish omega3 fatty acids may decrease breast cancer risk, epidemiological evidence has been inconclusive. This study examined the 
science.naturalnews.com/…/759291_Omega_3_fatty_acid_supplements_in_ women_at_high_risk.html
BACKGROUND : Preclinical evidence of the preventive benefits of omega3 (n-3) polyunsaturated fatty acids (PUFAs) in breast cancer continues to fuel interest 
science.naturalnews.com/…/256930_Decreased_severity_of_ovarian_cancer_ and_increased_survival_in_hens.html
science.naturalnews.com/…/2359858_Prevention_and_treatment_of_ pancreatic_cancer_by_curcumin_in_combination.html
Prevention and treatment of pancreatic cancer by curcumin in combination with omega 3 fatty acids. Publication: Nutrition and cancer. Publication Date: 2008
science.naturalnews.com/…/1101398_The_importance_of_the_omega_6_ omega_3_fatty_acid.html
A ratio of 2.5/1 reduced rectal cell proliferation in patients with colorectal cancer, whereas a ratio of 4/1 with the same amount of omega3 PUFA had no effect.
science.naturalnews.com/…/1136096_Impact_of_postoperative_omega_3_ fatty_acid_supplemented_parenteral_nutrition.html
Impact of postoperative omega 3 fatty acid supplemented parenteral nutrition on clinical outcomes and immunomodulations in colorectal cancer patients.
science.naturalnews.com/…/383170_Calcium_Mobilization_in_Ovarian_ Cancer_Cells_in_Response_to_Lysophospholipids.html
Herbal Remedy Essiac® and Cancer (press release) · Omega3 fatty acids slash colorectal cancer risk in men by 66 percent · Fatty Acid Blood Tests Help 
science.naturalnews.com/…/1201162_Growth_of_human_gastric_cancer_ cells_in_nude_mice_is.html
Growth of human gastric cancer cells in nude mice is delayed by a ketogenic diet supplemented with omega 3 fatty acids and medium chain triglycerides.
science.naturalnews.com/Neuroblastoma.html
Cancer science. Omega3 fatty acid supplementation delays the progression of neuroblastoma in vivo. International journal of cancer. Journal 
science.naturalnews.com/…/945592_omega_3_Fatty_acid_containing_diet_ Racol_reduces_toxicity_of.html
omega 3 Fatty acid containing diet (Racol) reduces toxicity of chemoradiation therapy for patients with esophageal cancer].
science.naturalnews.com/…/2597039_Modulation_of_lipid_rafts_by_Omega _3_fatty_acids_in.html
Modulation of lipid rafts by Omega 3 fatty acids in inflammation and cancer: implications for use of lipids during nutrition support.
Omega 3, on their own, are crucial for a cancer patient:

-promote optimal functioning of the immune system.

A team of researchers based in China have published results of their work on omega-3 and breast cancer risk in the British Medical Journal (BMJ), stating that a high intake of fatty acids found in fish is associated with a reduction 14 percent risk of breast cancer later in life. To conduct their study, the scientists reviewed and analyzed a group of 26 studies in the United States, Europe and Asia, which included more than 800,000 participants and 20,000 cases of breast cancer.

promote natural cancer cell death and reduce cancer risk.

After analyzing the data to determine the impact of long chain fats on the risk of breast cancer, the researchers determined the difference between the lowest and highest intake category of marine polyunsaturated fatty acids was associated with a 14 percent reduced risk of disease .

In addition, for an increase of 0.1 grams per day of consumption of omega-3 fatty acids in fish, the risk of breast cancer is reduced by 5 percent. These results clearly demonstrate a significantly reduced risk of affecting the development of breast cancer and future progression of the disease.

The study authors concluded „Our current study provides solid evidence and robust as omega3 are inversely associated with risk of breast cancer. „

Scientists understand from previous studies that omega-3 fats, DHA and EPA, are preferentially accumulated in cell membranes and prevent cancer development through increased transfer of nutrients and oxygen to the cell nucleus.

With DHA metabolism, metabolites are produced that destroy cancer cells. Researchers believe that this mechanism is responsible for reducing the risk of cancers found in this study. (1200-2400 mg EPA / DHA combined daily to reduce the risk associated with breast cancer and other disease lines). Higher doses are required in cancer.

-omega 3 stops spread of cancer

Omega-6, found in most vegetable oils, increased spread of cancer cells in the bone marrow.

Nevertheless, the spread was blocked by omega-3

Comments Dr. Mercola:

„The evidence continues to grow. Not all unsaturated fats are good for you. Even if they come from such healthy foods such as sunflower seeds grown biodynamic. See, most vegetable oils are rich in omega-6 fat and very few people are deficient in these fats.

In addition, omega-3 provides a wonderful benefits for patients with prostate cancer by blocking the functioning of omega-6 fats that cancer cells use them as an energy source. (Therefore, only during cancer your treatment, avoid omega 6 polyunsaturated fats because the thinner blood vessels supply  tumors. thin blood vessels in the body are necessary for optimal functioning, but avoid in cancer high doses of omega-6) This happens at the molecular level when omega-3  (EPA  andDHA), reduces the action of a metabolite of the omega-6 arachidonic acid, prostaglandin E2. The sole point agreed on this study is that scientists have recommended omega 3 as a preventative for prostate cancer as and to stop its development. „

Dr. Clapp compiled a comprehensive program that has achieved national recognition USA. 1990 –was a very successful year of the program – had 16 cures of prostate cancer based on deep cleansing of toxins accumulated in the body and prostate, then flooding the cells with omega 3 fats. „Perhaps the most interesting aspect of this study is clear evidence of the mechanisms that omega-6 fats stimulate the growth of prostate cancer, clearly debunking industry hypethat  vegetable oils are good for our health. „

We saw dramatically reduced overall inflammation (measured by CRP testing) and especially in reducing enlarged prostate, reducing also any tumor mass, monitored / evaluated by Doppler sonograms.

-Omega 3 promote apatosis(programmed cell death) in cancer cells

Two new studies suggest that omega-3 fatty acids may inhibit the growth of liver cancer cells, suggesting that this may be an effective therapy to treat and prevent liver cancer.

Like vitamin B17/laetrile,and other natural substances(turmeric, ginger) omega 3 trigger apatosis.

The first study examined the effects of omega-3 and omega-6 fats in hepatocellular carcinoma cells .

Carcinoma represent 90 percent of all cases of liver cancer and is usually rapidly fatal. Omega-6 had no effect on cancer cells, but omega-3 – as  (DHA) and (EPA) – induced apoptosis (programmed cell death).

In the second study, omega-3 fats have been shown as effective in the treatment of cholangiocarcinoma tumor cells, an aggressive and fatal type of cancer that forms in the liver bile ducts.

Researchers said omega-3 fatty acids may not only be an effective treatment for liver cancer, but could also protect the liver from hepatic steatosis, chronic liver disease thought to be a precursor to cancer.

These above are just a few specific examples of cancer and Omega3 .Omega are great in any cancer.

Omega-3 Builds Muscle Mass in Cancer Patients, thus PREVENT and HELP IN CACHEXIA: http://articles.mercola.com/sites/articles/archive/2011/03/24/omega3-builds-muscle-mass-in-cancer-patients.aspx

Omega 3 fats are highly recommended and among the few fats accepted in cancer (either from seeds, nuts, avocado or fish, although fish oils might be contaminated with heavy metals from the fish )

These and many other roles that help in cancer, including stimulation of the immune system, reducing inflammation , direct relationship to sun tolerance by the body and regulation of vitamin D levels (often those with cancer can not tolerate the sun, therefore can not benefit either its energy or the important natural Vitamin D, the sun being main source of vitamin D, but omega 3 fixes this, also regulating levels of vitamin D)

http://articles.mercola.com/sites/articles/archive/2010/07/27/omega3s-reduce-your-risk-of-breast-cancer.aspx

See also:

For prostate cancer risk and omega3 controversy see this articles:

http://articles.mercola.com/sites/articles/archive/2013/07/31/omega-3-fats.aspx

http://articles.mercola.com/sites/articles/archive/2001/07/21/flaxseed-part-two.aspx

science.naturalnews.com/…/520717_The_relation_of_alpha_linolenic_acid_ to_the_risk_of.html
The relation of alpha linolenic acid to the risk of prostate cancer: a systematic review and meta analysis.
science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html
Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids prostate cancer (PCa) risk, whereas a diet rich in omega3 decreases risk.

Much more about  OMEGA 3 as well a POWERFUL (stage IV) cancer treatment based on AWARDED 2 NOBEL PRIZES  in my book.

============================================================================================

Vitamin K

 (i.e: in algae, greens, grapes, etc) Also see antagonists such as cinnamon, etc,
science.naturalnews.com/V/Vitamin_K_and_cancer.html
Serum undercarboxylated osteocalcin as biomarker of Vitamin K intake and risk of prostate cancer: a nested case-control study in the Heidelberg cohort of the European prospective investigation into cancer and nutrition.
2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
Long-term use of Vitamin K antagonists and incidence of cancer: a population-based study.
2011 BloodCancer prevention and Vitamin K antagonists: an overview.
2010 Thrombosis researchSorafenib combined Vitamin K induces apoptosis in human pancreatic cancer cell lines through RAF/MEK/ERK and c-Jun NH2-terminal kinase pathways.

2010 Journal of cellular physiology

Efficacy of low- molecular- weight- heparin versus Vitamin K antagonists for long term treatment of cancer-associated venous thromboembolism in adults: a systematic review of randomized controlled trials.
2009 Thrombosis research

A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).
2004 Hepato-gastroenterology

Low-molecular-weight heparins are superior to Vitamin K antagonists for the long term treatment of venous thromboembolism in patients with cancer: a cochrane systematic review.
2008 Journal of experimental & clinical cancer research : CR

Oxygen free radical generating mechanisms in the colon: do the semiquinones ofVitamin K play a role in the aetiology of colon cancer?
2001 Biochimica et biophysica acta

A study of the prevalence of Vitamin K deficiency in patients with cancer referred to a hospital palliative care team and its association with abnormal haemostasis.
2008 Journal of clinical pathology

Production of superoxide and dissipation of mitochondrial transmembrane potential by vitamin K2 trigger apoptosis in human ovarian cancer TYK-nu cells.
2006 Apoptosis : an international journal on programmed cell death

Vitamin K2-mediated apoptosis in cancer cells: role of mitochondrial transmembrane potential.
2008 Vitamins and hormones

Vitamins K2, K3 and K5 exert antitumor effects on established colorectal cancer in mice by inducing apoptotic death of tumor cells.
2007 International journal of oncology

Inhibitory effects of vitamin K3 on DNA polymerase and angiogenesis.
2008 International journal of molecular medicine

A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).2004 Hepato-gastroenterology

Vitamin K antagonists

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Scientific Studies Database PAGE 2

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PAGE 2 CONTENTS:

  • Vitamins in alphabetical order:
  1. Vitamin A
  2. B Vitamins
    Vitamin B2 – Riboflavin, Vitamin B3(Niacin), Vitamin B5 ( pantotenic acid )(respiratory vitamins), Vitamin B6,B12, B17
  3. Vitamin C (very important vitamin, also respiratory vitamin)
  4. Vitamin D (very important vitamin)
  5. Vitamin E (respiratory vitamin)
  6. Vitamin F(including Omega 3)
  7. Vitamin K & Vitamin K antagonists

Vitamins in alphabetical order

Vitamin A

There is an extensive bibliography on the anti-tumoral action of Vitamin A.

In particular the combined use of Retinoids in the proper proportions (beta-Carotene: retinol = 4 : 1), establishes a synergism which is higher than the sum of the single components.

Studies on Vitamin A and cancer (MANY STUDIES -see NOTES below)

science.naturalnews.com/V/Vitamin_A_and_cancer.html

NOTE 1:

The link above contains multiple studies and the same do many links below( I will try to indicate by ”MANY STUDIES”)

Prostate cancer:

Breast cancer:

Ovarian cancer:

Lung cancer:

Lung, head & neck cancers:

2000 Journal of the National cancer Institute

Cervical cancer:

Gastric cancer:

Laryngeal cancer:

Children cancer:

Maxillary sinus cancer :

Studies on Vitamin A and Antiproliferative

http://science.naturalnews.com/V/Vitamin_A_and_Antiproliferative.html

Studies on Vitamin A and radiation

http://science.naturalnews.com/V/Vitamin_A_and_radiation.html

There is an extensive bibliography on the anti-tumoral action of Vitamin  A cited from LAST PAGE OF THIS DATABASE: 1,3,10,13,14,17,18,23,24,28,34,35,36,45,56,69,73,76,92,93,94,100,106,111,120,121,129,131,137,138,165,187,200,202,203,208,209,21,213,214,216,218,222,235,255,256,257,263,264,265,266,280,282,286,287,288,298,303,304,305,307,313,315,322,324,325,326,334,338,340,341,347,38,352,354,362,363,365,382,383,385,390,398,402,404,405,409,410,420,425,426,427,445,446,447,448,454,457,461,463,468,469,470,471,473,477,48,493,508,512):

This studies conclude  the following about vitamin A:

Anti-tumoral action in general

Vitamin A and retinoids have an anti neoplastic action, shown both in vivo and in vitro, in various tumors: basilomas, scaly carcinomas, melanomes, skin cancers, fungoid mycosis, acute promyelocyte leucemia, ovarian cancer, breast cancer, lung cancer, cancer of the bladder and follicular carcinomas of the thyroid.

Preventative action

Anti-tumoral action has been amply shown indirectly, that is with a preventative purpose. Various  studies have, in fact, shown that low plasmatic levels of beta-Carotene, vitamin C and vitamin E are  connected to an increase in the incidence of lung cancer.

Direct action at a receptor level

Only beta RAR (retinoic acid receptor) seems to be involved. More specifically, vitamin A (and its derivatives) acts by binding with specific receptors. In this way it can both inhibit the proteic synthesis of DNA and RNA, and also perform an anti-promotion  action to determine the return of a cellular differentiation. Furthermore its ability to inhibit oncogenes should be noted.

Apoptosis induction 

Apoptosis means the activation of specific endonucleases which break up the DNA, acting at a level of nucleosomic sites that make up the primary structural unit of the nuclear cromatine of the cell.  Vitamin A and retinoids in general can induce apoptosis in neoplastic cells, by activating intracellular proteolytic enzymes, called caspase 2 and caspase 3, which provoke deterioration  through proteolysis by a transcription factor, called Spl.  If this basal cellular transcription is altered, death is caused by Apoptosis.

In prostate cancer the retinoids intervene by reducing the level of the bcl 2 gene, whose function is to protect the cells from death by Apoptosis.  Carotenoids induced apoptosis in prostate cancer

Dă clic pentru a accesa carotenoidi%20sono%20fattori%20attivi%20contro%20il%20cancro%20della%20prostata.pdf

Inhibition of the cellular cycle

The retinoids block the passage of the cell from phase G1 to phase S (reducing the activity of a protein, called cycline D1): this passage, if not blocked, would lead the cell to mytosis.

Reduction of phosphorilation

Retinoids intervene in cancer causing a reduction in phosphorilation of the pRb, thus increasing survival; this protein is active in suppressing cellular growth.

Synergetic interaction with interferons

A second mechanism, apart from the proteic-enzymatic one, can be traced back to the intervention of interferons which, like retinoids, act as anti-proliferative factors. In practice, acting synergetically, they induce the expression of proteins capable of inhibiting neoplastic cellular proliferation.

The intake of natural carotenoids from food 

There is very little evidence to show that carotenoids taken in from food can increase the levels ofvitamin A: an extra portion a day of green leaf vegetables is not able to increase the hematic level of vitamin A; on the contrary, a mixed diet of foods particularly rich in beta-Carotene will give a  significant increase in vitamin A present in the blood. (627).

If you oblige healthy individuals to eat carrots (270g), broccoli (600g) or tomato juice (180g) it does not establish any significant changes in the hematic levels of the carotenoids: you only find an extremely wide variation (even up to 3-4 times) in the efficiency of gastro-intestinal absorption of  the carotenoids and therefore in their subsequent bio-availability at a hematic level (628) .

Optimal values of anti-oxidants in normal individuals

Vitamin C: >50 microMols/Liter

Vitamin E: >30 microMols/Liter

Vitamin A: >2.2 microMols/Liter

Beta-Carotene: >0.4 microMols/Liter

Association of vitamin A with vitamin E

In an experimental model of a cellular membrane the possibility of a positive interaction between anti-oxidant liposollubles such as beta-Carotene and alpha-Tocopherol has been investigated; the  result showed that there exists a synergetic action between beta-Carotene and alpha-Tocopherol  together which inhibits the processes of lipidic peroxidization compared with when they are used alone (629). THUS:

NOTE 2:

Vitamins , antioxidants and nutrients should be taken from WHOLE, ORGANIC(note below) FOODS (as much as possible) NOT from supplements as they need other vitamins and nutrients for proper usage.

The difference between synthetic vitamins and natural vitamins

The difference between synthetic vitamins and natural ones can be easily exemplified by the experimental case of synthetic beta-Carotene (made up entirely of isomeric trans-beta-Carotene), and of natural beta-Carotene (made up of both isomeric trans-beta-Carotene and isomeric cis-beta- Carotene): the study showed a strong discrimination between the two isomers, with a serious decrease (impoverishment) induced at the level of Lycopene present in the LDLs (…). THUS:

3.It would be better to use the NATURAL (juice) ORGANIC WHOLE foods rich in vitamin A such as  Daucus carota (carrots ), avocados, cantalopues, etc. rather than the synthetic pharmaceutical vitamin products based on Vitamin A.

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B Vitamins 

Studies on Vitamins B and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B_and_cancer.html

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Vitamin B2 – Riboflavin

Studies on Riboflavin and cancer

Studies on Riboflavin and Anti-Angiogenic

http://science.naturalnews.com/R/riboflavin_and_Anti-Angiogenic.html

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Vitamin B3(Niacin)

Studies on Niacin and cancer

  • Other studies:

ANTI-invasive activities of Niacin against cancer  cells; http://science.naturalnews.com/…/3260742_Anti_invasive_activity_of_niacin_and_ trigonelline_against_cancer_cells.html

Studies on Niacin and breast cancer

http://science.naturalnews.com/N/Niacin_and_breast_cancer.html (MANY STUDIES in this link)  

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Vitamin B5 ( pantotenic acid )

Pantothenic Acid for Stage IV Breast Cancer Treatment Information

https://www.medify.com/…/pantothenicacid-treatment-stage-iv-breast-ca

Vitamins B2, B3,B5 are respiratory vitamins(increasing cellular oxygenation –  see above Otto Wartburg proposals).

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Vitamin B6

science.naturalnews.com/V/Vitamin_B6_and_cancer.html

Vitamin B12 

science.naturalnews.com/V/Vitamin_B12_and_cancer.html

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Vitamin B17(Laetrile/ Amygdalin) (very important vitamin)

Note: “Laetrile” acronym for “LAEvomandeloniTRILE-glucoside”) as Amygdalin:

Laetrile has two molecules of glucose, Amygdalin has more.

Indeed, the chemical structure of Laetrile is D-1 mandelonitrile–beta-glucuronide, while for Amygdalin it is D-mandelonitrile-bi-glucoside.

In 1845, fifteen years after the first French scientific experiences, the French scientific journal “Gazette Medicale de Paris”, (1188) and afterwards the German journal “Journal für die Chirurgie und Augenheil-kunde”(1189), described the first case of metabolic therapy with vitamin B17 to “cure cancer”, created by Russian doctor Inosmetzeff, professor at the Imperial University of Russia in Moscow. Therapy was performed on a twenty-one-year-old boy affected by cancer, and consisted of 46 grams of Amygdalin administered for 3 months. Inosmetzeff had also cured a 48-year-old woman with extended metastases due to womb cancer. In 1845 this woman was still alive, 11 years after metabolic therapy with Amygdalin. In both cases, Inosmetzeff said that he never noticed any side effects with vitamin B17.

For instance, doctor Francisco Contreras, the current managing director of the Oasis of Hope hospital in Tijuana, Mexico, treated more that 60,000 patients with a vegetarian therapy and vitamin B17 in 35 years of activity (1187). http://www.mednat.cancro/Contreras.pdf

Doctor Ernesto Contreras has been using Laetrile since 1963, and thinks that

“…The majority of most frequent cancers, such as lung, breast, colon, ovarian, stomach,esophagus, prostate cancer, and lymphoma, can improve dramatically with Laetrile” (1187).

Clinical Case history

 

Amygdalin taken orally has been known to be a poison since ancient times, though amygdalin-laden black and brown bitter seeds were described as antitumor agents in the pharmacopeia of ancient China (1497)

Egyptian, Greek, Roman and Arabic physicians also used amygdalin to treat tumors (1498).

In a study conducted in 1958, Prof Marco Tasca, head of the radiology department of the Civil Hospital in Sanremo, treated 21 Italian terminally ill patients – 3 suffering from seminomas, 4 from breast cancers, 1 from womb cancer, 2 from laryngitis cancers, 7 from lung cancers, 1 from cancer of the oesophagus, 2 from stomach cancers, 1 from Hodgkin’s disease – with intramuscular injections of Laetrile. He noticed that patients showed good drug tolerance, their clinical conditions improved during the entire treatment period and only one month – on average – after the interruption of the therapy the neoplastic pathology resumed its progression. He pointed out only two complications:

hemorrhage and icterus. The former probably caused by necrotic eschars coming off the tissues, the latter induced by a direct toxic action on hepatic cells, which rarely happens though (5% of his case histories). The article is available in PDF format (1373) at http://www.fiocco59.altervista.org/images/tasca.pdf  or http://www.mednat.org/cancro/tasca.pdf.

In the 1966 report, Proceedings of the Ninth International Cancer Congress, Rossi cites a ten-year trial in Europe involving 150 patients that found „50 percent of all cases in treatment showed objective improvement” and concluded that laetrile was „an extremely useful chemotherapeutic drug.”(1382) http://fiocco59.altervista.org/vitamina_b_17.htm

In 1994, professor Dr .Binzel published the results he obtained treating patients with Laetrile between 1974 and 1991. His case history included 180 patients with primary cancer (with no metastasis and limited to only one organ or tissue). 131 patients were still alive in 1991, when the report was published. At that time, 58 patients had been followed for 2 to 4 years, while 80 of them had had a medical follow-up for 5 to 18 years. Out of the 42 patients that had died by 1991, 23 had died from cancer, 12 from “unrelated causes” and 7 of “unknown causes” (Binzel E.P.: “Alive and Well”). http://www.mednat.org/cancro/ALIVE_AND_WELL.pdf

Among patients with metastasis, 32 out of 108 had died from their disease, 6 from “unrelated causes” and 9 from “unknown causes”. Out of the 61 patients that were still alive in 1991, 30 had had a medical follow-up of 2-4 years, 31 had been followed for 5-18 years.

Doctor John A. Richardson’s case history of 1976 reports over 6,000 cases that show a positive effect of vitamin B17 against cancer. (1187)

There are 4,800 cases reported and carefully studied by doctor Ernesto Contreras. Those were selected among 10,000 case sheets collected in 14 years of experiences with Laetrile.

Doctor Paul Wedel from Oregon reported about 4,000 cases of metabolic treatment. He survived cancer himself with vitamin B17 and a diet similar to the gersonian one (1187).

1,000 cases were reported by doctor Manuel Navarro of Santo Tomas University in Manila, the Philippines. The Mexican government is even monitoring about 100 patients that are being treated with metabolic therapy and vitamin B17, under the guidance of doctor Mario Soto de Leon, medical director of the Cydel Clinic in Tijuana (1187).

In Germany, doctor Hans Nieper reported about 1,000 cases. (http://www.mwt.net/~drbrewer  )

It is interesting to notice that cases such as that of Mr. Glen Rutherford from Kansas, who healed completely in Tijuana, are recorded in tribunal archives as “cures” (1187).

Clinical Trial of Chemotherapeutic treatment of advanced cancers with Leatrile

Guidetti Ettore

Rossi Benedetto

Deckers Christian

Presented at the 9th International Cancer Congress in Tokyo, October 1966

From 1954 to 1966 we gave 150 patients the above-mentioned therapy, chiefy at San Cottolengo Hospital, Turin; DosioHospital, Milan; and Louvain University Cancer Institute. All patients were in the terminal stage of the disease, the majority of them prey to cachezia, and all other therapies had failed.

The following table summarizes the cases treated, classified according to the site of the tumor, and showing the number of patients for each degree of reaction to therapy. We use the sign ++ to denote patients who reacted in an objectively favourable manner, by which we mean diminution of volume of the tumor or at least all interruption of its evolution, improvement in the roentgenographic picture, and improvement in laboratory findings. The mark + and + indicates patients who showed a more or less distinct subjective improvement, and the mark – those who reacted negatively to the treatment.

Cases corresponding to ++ represent about 20% of those treated.

We again underline the fact that the majority of these cases were simultaneously subjected to an immunotype therapy, which might have some bearing on the number of positive results observed, grouped under the signs ++ and + totalling about half the number of cases treated.

Cancer Site        No. cases            ++    +    +-           

Toruli tactiles                    26                           5             6             6             9

Breast                                  25                           3             8             7             7

Uterus                                 24                           7             7             4             6

Rectum                                               20                           2             9            2             7

Ovary (with infusion)    10                           2             2             2             4

Other types                       30                           9             7             2             12

Totals                                   135                        28           39           23           45

We have separately considered neoplasms of the pleura with effusion (15 cases), where the product was used direct by injection in the pleural cavity. In these cases we observed our best results, as generally we obtained reduction and then on occasion complete disappearance of the effusion, associated with a distinct improvement in the patients’ condition.

Conclusion:

On the basis of our clinical trial, we are able to state that L-mandelonitrile-beta-diglucoside may be considered an extremely useful chemotherapeutic drug for palliative medical treatment of malign neoplasms, from the standpoint both of its therapeutic effect and its very low toxicity.

B17/ Amygdalin studies science.naturalnews.com/amygdalin.html

 How laetrile or B17 from apricot seeds kills only cancer cells ·

apricot studies science.naturalnews.com/apricot.html

Apricot Seeds Kill Cancer Cells without Side Effects
  • Monographic Summary of AMYGDALIN
  • Monographic Analysis of Amygdalin (over 70 pages)
  • Nutritional Implications by Ernest T. Krebs, Jr. John Beard Memorial Foundation (Privately published: 1964).
  • The Nitrilosides(Vitamin B-17) Their Nature, Occurence and Metabolic Significance (Antineoplastic Vitamin B-17) Ernst T. Krebs, Jr. Reprinted from the Journal of Applied Nutrition, Volume 22, Numbers 3 and 4, 1970.
  • The Ultimate Cancer Conspiracy Vitamin B17 Laetrile by Joe Vialls.
  • Ernest T. Krebs Jr. Biography (Discoverer of B-17).
  • Success Stories – Laetrile Testimonials, Case Histories, Success Stories.
  • THE UNITARIAN OR TROPHOBLASTIC THESIS OF CANCER by Ernest T. Krebs, Jr.,* Ernst T. Krebs, Sr.,** and Howard H. Beard*** (Reprinted From the Medical Record, 163:149-174, July 1950) MEDICAL RECORD AN INTERNATIONAL JOURNAL OF MEDICINE AND SURGERY Founded 1843 Vol. 163, No.7 JULY, 1950 Whole No.2809
  • The Four Optional forms of Cancer Therapy Dr. Robert D. Sullivan, Department of Cancer Research at the Lahey Clinic Foundation.
  • The Laetrile Quacks The names, professional standings, medical achievements, and clinical findings of some of the more prominent doctors who endorse Laetrile; the beneficial side-effects produced by its use; a suggested anti-cancer diet; and a brief description of vitamin B15.
  • The Hoax Of The “Proven” Cancer Cures The effects of surgery and radiation in the treatment of cancer; a comparison showing that those who receive no treatment at all live just as long, if not longer, than those who are treated.
  • Laetrile and Cyanide Read about the life saving substance called cyanide.
  • Ralph Moss on Cancer Politics – Ralph Moss on Chemotherapy, Laetrile, Coley’s Toxins, Burzynski, & Cancer Politics – Laura Lee radio show, 1994.
  • Metabolic Therapy in Cancer – METABOLIC THERAPY IN CANCER
    AND ITS OBJECTIVE The use of Amygdalin (Vitamin B-17) LAETRILE (Vitamin B-17) TREATMENT by Harold W. Manner, Ph.D. Metabolic Research Foundation.
  • The Truth about Surgeries and Biopsies – According to Clinical Oncology for Medical Students and Physicians (published jointly by the University of Rochester School of Medicine and the American Cancer Society)
  • Laetrile: Cancer Cure Or Quackery? – Excerpts from: Los Angeles Herald-Examiner, Sunday, July 21, 1974.

 

See also:  http://worldwithoutcancer.org.uk/success.html,   http://www.apricotsfromgod.info/journal.htm

Much more details(including treatments & CLINICAL CASES with B17) in my BOOK + DR. Binzel’s FREE EBOOK on this topic + Dr. Nacci’s FREE EBOOK on this topic.

NOTE:

Vitamins(including B group) should be taken from WHOLE FOODS(organic) (as much as possible),NOT from supplements as vitamins and nutrients need other vitamins and nutrients for proper usage within the body.

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Vitamin C (very important vitamin)

Vitamin C   is a respiratory vitamin- it stimulates the body to produce its own hydrogen peroxide (increasing cellular oxygenation –  see above Otto Wartburg proposals).

Therefore, megadoses of vitamin C are administered orally and intravenously by orthomolecular physicians to treat cancer.

60 references to studies published that show the importance of vitamin C:

www.doctoryourself.com / biblio_cameron.html http://www.doctoryourself.com/cancer_hoffer.html.

Even National Institutes of Health is actively sponsoring studies of high-dose intravenous vitamin C therapy.

Other MULTIPLE studies on  vitamin’s C cancer fighting effects: science.naturalnews.com/…/4854330_Vitamin_C_in_alternative_cancer_ treatment_historical_background.html

High doses of vitamin C andi cancer :

science.naturalnews.com/…/2387600_High_dose_vitamin_C_supplement_ use_is_associated_with_self.html

Vitamin C and breast cancer studies: science.naturalnews.com / V / Vitamin_C_and_breast_cancer.html

Vitamin C , vitamin E & selenium in breast cancer : science.naturalnews.com/…/1198191_Dietary_beta_carotene_vitamin_C_and _E_intake_and_breast.html science.naturalnews.com/…/3959947_Association_between_breast_cancer_ and_vitamin_C_vitamin_E_and.html

Vitamin C ,vitamin A & zinc  in laryngeal cancer  :

science.naturalnews.com/…/4508069_Association_of_vitamin_A_vitamin_C _and_zinc_with_laryngeal.html

Vitamin C &  gastric cancer:

science.naturalnews.com/…/3603872_Plasma_and_dietary_vitamin_C_levels _and_risk_of_gastric.html science.naturalnews.com/…/3628783_Prediagnostic_plasma_vitamin_C_ levels_and_the_subsequent_risk_of.html science.naturalnews.com/…/4790082_Effect_of_physiological_ concentrations_of_vitamin_C_on_gastric_cancer.html

Also see: http://orthomolecular.org/resources/omns/v04n19.shtml

There is also an extensive bibliography on the anti-tumoral action of vitamin C (25,33,47,54,83,91,122,129,181,197,202,218,244,246,270,299,311,335,339,367,404,405,414,415,416,489,496,510,511)

See the bibliography and studies quoted on last page of databse.

The pioneers of this oncological therapy were Pauling, who received the Nobel Prize for Chemistry, and the Italian Pantellini.

Ascorbic acid is mainly known for its ability to reduce metallic ions in various enzymatic processes and above all for its ability to act as an anti-oxidant agent, thus able to remove free radicals, reducing the damage caused at a genome level.

Furthermore it may be able to block the formation of nitrose at a gastro-intestinal level, as well as carrying out a preventative action on the formation of adenomatose polyps.

Even though ascorbic acid is well known for its collagene forming action, and the well known effect of scurvy in cases of reduction or absence of this acid in the diet, this vitamin is also important, alongside vitamins A and E as a first class anti-tumoral agent.

It is a respiratory vitamin(like vitamins E, B3, B5)  that helps increase cellular oxygen levels.

It reinforces the intercellular bond and forestalls the destructive action of the hyalurons produced by many neoplastic cells.

Vitamin C stimulates the Natural killer lymphocytes, it supports the macrophagic activity, the chemio-tactical mobility of the white corpuscles, the production of antibodies and the response of the T cytotoxic lymphocytes to the antigenes.

Cameron found very low plasmatic levels of vitamin C in cancer patients (0.26 mg/100 mL) compared to normal plasmatic values (54).

In other studies, 154 cancer patients, undergoing analysis, were found to have low levels of vitamin C not only in their plasma (0.31 mg/100 mL), but also in their leucocytes (15.9 mg/10 E+8), with a positive correlation (r = 0.42) between these two values; in particular the authors attributed the alteration in the immune response to the tumor, especially of the phagocytosis, to the low  concentration of vitamin C in the white blood corpuscles.

Already in 1974 Goetz had shown that vitamin C, in vitro, was capable of stimulating the motility and the chemiotaxis of neutrophiles.

The doses advised in literature for anti-neoplastic therapy are about 3-10 grams a day, reaching even 40 grams daily, because the vitamin is not toxic, at high doses it only has a laxative effect.

It would be better to take it by eating fresh fruit, but it is difficult to reach such high doses of the vitamin in this way.

Integrating fresh fruit (kiwi, oranges, lemons and grapefruit) with the juice of sea buckthorn  , Rosa canina (dog rose, wild rose) to reach a daily dosage of at least 5- 8 grams of natural vitamin C, avoiding however, the use of pharmaceutically prepared vitamin C tablets.

Natural vitamin C, rich in its metabolites and other components called bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercitin, Pychnogenol, etc) is more powerful and efficient, and furthermore is devoid of unpleasant gastric effects which are the result of high doses of synthetic vitamin C.

Natural vitamin C is moreover, characterized by a significant reduction in the formation of Calcium oxalate in the kidneys, as opposed to synthetic vitamin C. It is also easier for the intestine to absorb and has greater bio-availability, above all through its most important metabolites such as tronic acid, lixonic acid, xilonic acid etc.

This bio-availability has a critical importance in the immune defense system because the white blood cells tend to absorb Natural vitamin C 4 times more than they absorb synthetic vitamin C.

Recently, Myrciaria paraensis (camu-camu) has appeared on the European market. It is a small exotic fruit, similar to a small orange, but it contains 50 times more natural vitamin C than Citrus aurantium (orange), and it could therefore provide the daily dosage, of at least 3 grams, of vitamin  .

Also Malpighia punicifolia (acerola), a cherry from the Antilles, is very rich in vitamin C, containing 50 to 100 times more than citrus fruits.

Natural vitamin C is therefore efficient because it is naturally associated to the bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercetine,Quercitine, Pychnogenol, etc..) and other molecules, in plants often characterized by an immune  stimulating activity (Echinacea purpurea, Plantago major, Capsicum frutescens)

Note: With very high therapeutic doses (>8-10 grams a day), Magnesium (e.g. Dolomite) must also be taken to avoid the risk of kidney stones.

Here enclosed scientific papers extracted from Catherine Kousmine (“Save your body”, page 129,

“Effects of C vitamin on our body according to Linus Pauling, edition Tecniche Nuove):

“…an intake of 1500 milligrams of ascorbic acid by mouth determines a concentration of 1.5 milligrams of C vitamin for each 100 millilitres of blood. By increasing the intake, the concentration suddenly increases up to 2.5 milligrams and then goes back to 1.5 millilitres for each 100 millilitres blood. There are enzymes which help the conversion of most

ascorbates into useful oxidation products. If the intake remains high, the body increases the amount of enzymes useful to the conversion; otherwise, if the ascorbic acid dose is suddenly reduced for some days, an excess of conversion enzymes and then a too law level of vitamin C in the blood occur. This means a number of disorders, as for example a higher sensibility to infections. The adaptation to a lower proportion takes place by reducing the number of conversion enzymes: it is necessary to gradually decrease the dose of vitamin C. By taking 100 milligrams a day and in presence of a plasmatic level of 1 milligram for every 100 millilitres blood, urines do not contain ascorbic acid because it is reabsorbed by renal tubules. If the intake is higher than 100 milligrams, i.e. 1-2 grams a day, 25% go in urines and the rest is kept by the body. Healthy people, who lack in vitamin C for some months, have to take 2-4 grams in order to eliminate them through urines. In case of cancer patients, who are used to take high doses of ascorbic acid, an interruption of some days requires an intake of 50 grams (fifty grams) of vitamin C so that this one can be found in urines”.

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Vitamin D (very important vitamin)

NOTE:The link above contains multiple studies on sun exposure(vitamin D) and it’s crucial importance in fighting cancer:

Circulating Vitamin D levels in the Blood Dramatically Reduces Death Rate

* For each 10ng/ml increase in the blood level of vitamin D, the death rate from cancer would be reduced by 29%. The relationship between vitamin D and colorectal cancer is particularly strong.

The death rate for colorectal cancer would be reduced by 49%. (30 – Read Supporting Scientific Article on PUBMED)

Vitamin D Levels est during Summer = Improves survival by 40%

A Norwegian study showed that cancer patients diagnosed in the summer when vitamin D levels are highest had up to 40% better survival rates than patients diagnosed in the winter when vitamin D levels are at their lowest.

(56 – Read Supporting Scientific Article on PUBMED )

Low Vitamin D Levels Greater chance of Death & Metastasis

* Breast cancer patients with low levels of vitamin D followed over eleven years had a 70% greater chance of dying and twice the rate of developing metastasis than patients with high levels of vitamin D.

(90 – Read Supporting Scientific Article )

Lung Cancer Patients Higher levels = Dramatic increase in Survival

Early stage lung cancer patients who were diagnosed in the summer and had the highest levels of vitamin D had a five-year survival of 73% compared to 30% for those diagnosed in the winter with low vitamin D levels.

(60 – Read Supporting Scientific Article on PUBMED)

Lung – Colon – Prostate – Renal – Endometrial Cancers Higher Vit D levels dramatically decreases Risk

*High vitamin D levels (based on latitude and UV exposure) also correlate with a decreased risk of developing lung cancer, a 45% and 65% reduction in men and women respectively. Similar effects have been demonstrated in colonprostaterenal and endometrial cancer. (33 – Read Supporting Scientific Article on BMJ(British Medical Journal))

Advanced Colorectal Patients Risk of death greatly Improved

The risk of death for colorectal cancer patients with advanced disease but with high levels of vitamin D was reduced by over 60% compared to patients with low vitamin D levels. (64 – Read Supporting Scientific Article on PUBMED)

Prostate Cancer Patients

*Prostate Cancer Patients with Vitamin D levels in the mid and high range had a 60% and 85% respectively reduced risk of death from the condition compared to patients with low levels of Vitamin D. This is almost a seven fold increase in the risk of death in those with low vs high vitamin D levels. (152 – Read Supporting Scientific Article on PUBMED)

There is also an extensive bibliography on the anti-tumoral action of vitamin D- see last page of this database:  (28,157,160,188,208,209,231,240,246, 254,302,323,479,489)

Vitamin D is BEST taken from skin exposure to the sun, MINIMUM 30 mins daily,without any sunscreen or protection. Avoid burning and avoid the interval 11 am – 16 pm.

Natural vitamin D, contained in some plants, is however preferable to the synthetic type, because the latter is about 10 times more capable of binding with Magnesium, taking it away from the organism, thus causing all the damage that the loss of this incurs (osteoporosis, kidney stones).

Vitamin D induces the inhibition of neoplastic cellular growth: this has been shown in vitro in neoplastic cellular lines; especially of the hematopoietic system, of the CNS, of the prostate; the  colon, the ovaries and the breasts.

This action is thought to be expressed at various levels, in particular:

a) by means of apoptosis induction, through the activation of p21, that is, the inhibitor of the kinase proteins;

b) the inhibition of neoplastic cellular growth, which would be blocked in the G1 phase because of the action of the IGF1 inhibitor;

c) by means of cellular differentiation.

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Vitamin E (respiratory vitamin)

This liposoluble substance consists of a group of various components, called Tocopherols.

Seven of these exist in nature; alpha-Tocopherol, beta-Tocopherol, gamma- Tocopherol, delta-Tocopherol, epsilon-Tocopherol, zetaTocopheros and eta-Tocopherol.

Studies on Vitamin E and cancer

science.naturalnews.com/…/1364373_Vitamin_E_and_cancer_An_insight_ into_the_anticancer_activities.html

There is also an extensive bibliography on the anti-tumoral action of vitamin E -see last page of DATABASE (6,19,20,30,45,91,95,112, 125,129,142,165,167,190,202,228,229,246,261,280,332,404,405,452,494)

This liposoluble substance consists of a group of various components, called Tocopherols. Seven of these exist in nature; alpha, beta, gamma, delta, epsilon, zeta and eta.

Alpha-Tocopherol has an anti-oxidant effect on the lipidic membranes in synergy with Melatonin(see below), carrying out a preventative action on the peroxidization of the cellular membrane induced by  ionizing radiation and by chemical carcinogenes.

Vitamin E also carries out an anti-oxidative action in a wider sense, acting as a ‘scavenger’ of the free radicals, similar to vitamin C.

It performs a stimulating activity on the immune system; it induces cellular differentiation; it inhibits, in a selective way, cellular growth intervening at DNA and RNA synthesis level.

Various studies have shown its ability to induce apoptosis in cellular lines of breast carcinomas and lymphoma B.

It is inactivated by Iron, therefore it is essential that any medicines based on Iron are not taken at the same time as vitamin E, but at a distance of at least 10-12 hours.

Aluminum, which is often present in pharmaceutical products, also deactivates vitamin E.

The uncooked oil of Triticum sativum (wheat germ; note: has vitamin B12) contains about twice as much vitamin E as the uncooked oil from the seeds of Helianthus annuus (sunflower) and the latter  contains about five times as much as uncooked olive oil. Furthermore all these seeds are rich in  essential unsaturated fats, an important part of the diet for cancer patients.

In anti-neoplastic therapy much is being discussed about: the raw seeds of Helianthus annuus

(which the author personally considers useful in therapy), wheat shoots (the author is not in favor),

Saccharomyces cerevisiae (yeast, of which the author is not in favor), and the shoots of soya  lecithin (of dubious use and the author is against their use because of the transgenic risk).

Natural vitamins are by far preferable to industrially produced ones: Dracontium loretense, for example, which is considered one of the best  plants for its specific anti-oxidant potential, is of superior quality in its anti-oxidant ability compared to synthetic vitamin E (566). WHOLE, ORGANIC FOODS are BEST due to COMPLEXES of NUTRIENTS !

As an already extracted natural product, together with or without other vitamins, vitamin E must be  given in addition to high quantities of raw seeds of Helianthus annuus (also containing vitamin A,all the vitamin B compounds, vitamin D, Manganese, Zinc and Magnesium) and of high quantities of raw Triticum sativum (which is rich in the precious alpha-lipoic acid): both are also very rich in vitamin B6 (pyridoxin), the latter is important for the immune system, but it is difficult to find in other compatible foods for a suitable diet for cancer patients. Pyridoxin, in fact, is contained especially in Saccharomyces cerevisiae, the latter is a food which the author does not regard favorably for an anti-neoplastic diet, because it contains high quantities of folic acid.

Natural Octacosanol, extracted from the oil of Triticum sativum, has a synergetic action with vitamin E, but it is, in any case, better to consume it with all the uncooked oil of Triticum sativum  and/or Triticum sativum itself rather than taking it already extracted, as a pharmaceutical product  (because it loses its active principles).

Vitamin E is particularly efficient in combination with Selenium, which is contained in Aloe species, Solanum lycopersicum (tomatoes), Equisetum species, Allium cepa (onions).

There is also an extensive bibliography on Selenium (see last page of DATABASE) :

79,108,112,129,133,136,143,156,228,229,276,338,339,364,367,404,405,407,443,452,458,501,510,511)

Both vitamin E and Selenium are in their turn synergetic with Zinc in inhibiting the production of inflammatoryprostoglandins and leukotrienes.

Vitamin E conversion factors

Vitamin E is expressed in milligrams of tocopheral equivalents (T.E.)

1 milligram of Tocopherol is equal to:

= 1 milligram of D-alpha Tocopherol

= 2 milligrams of D-beta Tocopherol

= 5 milligrams of D-gamma Tocopherol

==========================================================================================================

Vitamin F  (very important vitamin-see omega 3)

Polyunsaturated fatty acids :

arachidonic acid, Linoleic cis-cis natural acid (vitamin F1) as: alpha-lipoic acid,  alpha-linolenic acid,etc.

Alpha-lipoic acid

Food Sources: 

spinach, broccoli, tomato, peas, Brussels sprouts, rice bran

Mechanism of action:

Alpha-lipoic acid acts as a lipophilic free radical scavenger. Dihydrolipoic acid (DHLA), a reduced form of lipoic acid, has more potent antioxidant effects. It can assist in repairing oxidative damage and regenerate endogenous antioxidants such as vitamin C, vitamin E, and glutathione.

Both DHLA and lipoic acid also have metal chelating capacities.

As a lipoamide, alpha-lipoic acid functions as a cofactor in various multienzyme systems involved in the decarboxylation of alpha-keto acids such as pyruvate. (13) (14) (15)

Alpha-lipoic acid caused cell cycle arrest in G0/G1 in FaDu and Jurkat human tumor cell lines (1).

Alpha lipoic acid was also found to scavenge reactive oxygen species in MCF-7 breast cancer cells (16).

Reduction of reactive oxygen species was then followed by cancer cell growth arrest and apoptosis (16).

For studies quoted see http://www.mskcc.org/cancer-care/herb/alpha-lipoic-acid  

Other studies:

science.naturalnews.com/A/AlphaLipoic_Acid_and_cancer.html

Alpha-Linolenic Acid(omega 3)

Food sources:flaxseed, avocado, walnuts, almonds, fishoils

Mechanism of action:

The alpha-linolenic acid (vitamin F), for instance, is a cis-polyunsaturated fatty acid that is contained in linseed cold-pressed oil: it is transformed into EPA and DHA (Omega-3 fatty acids) and is quite effective against malignant tumours, as shown by Pardini ( 1647 );

Moreover, Noguchi has proved that Omega-3 fatty acids, unlike Omega-6 fatty acids, help reduce tumour masses, although

Omega-6 fatty acids are unsaturated fatty acids, too ( 1654 ).

see  last page of DATABASE for studies quoted.

science.naturalnews.com/A/AlphaLinolenic_Acid_and_cancer.html
Omega 3, on their own, are crucial for a cancer patient:

-promote optimal functioning of the immune system.

A team of researchers based in China have published results of their work on omega-3 and breast cancer risk in the British Medical Journal (BMJ), stating that a high intake of fatty acids found in fish is associated with a reduction 14 percent risk of breast cancer later in life. To conduct their study, the scientists reviewed and analyzed a group of 26 studies in the United States, Europe and Asia, which included more than 800,000 participants and 20,000 cases of breast cancer.

promote natural cancer cell death and reduce cancer risk.

After analyzing the data to determine the impact of long chain fats on the risk of breast cancer, the researchers determined the difference between the lowest and highest intake category of marine polyunsaturated fatty acids was associated with a 14 percent reduced risk of disease .

In addition, for an increase of 0.1 grams per day of consumption of omega-3 fatty acids in fish, the risk of breast cancer is reduced by 5 percent. These results clearly demonstrate a significantly reduced risk of affecting the development of breast cancer and future progression of the disease.

The study authors concluded „Our current study provides solid evidence and robust as omega3 are inversely associated with risk of breast cancer. „

Scientists understand from previous studies that omega-3 fats, DHA and EPA, are preferentially accumulated in cell membranes and prevent cancer development through increased transfer of nutrients and oxygen to the cell nucleus.

With DHA metabolism, metabolites are produced that destroy cancer cells. Researchers believe that this mechanism is responsible for reducing the risk of cancers found in this study. (1200-2400 mg EPA / DHA combined daily to reduce the risk associated with breast cancer and other disease lines). Higher doses are required in cancer.

-omega 3 stops spread of cancer

Omega-6, found in most vegetable oils, increased spread of cancer cells in the bone marrow.

Nevertheless, the spread was blocked by omega-3

Comments Dr. Mercola:

„The evidence continues to grow. Not all unsaturated fats are good for you. Even if they come from such healthy foods such as sunflower seeds grown biodynamic. See, most vegetable oils are rich in omega-6 fat and very few people are deficient in these fats.

In addition, omega-3 provides a wonderful benefits for patients with prostate cancer by blocking the functioning of omega-6 fats that cancer cells use them as an energy source. (Therefore, only during cancer your treatment, avoid omega 6 polyunsaturated fats because the thinner blood vessels supply  tumors. thin blood vessels in the body are necessary for optimal functioning, but avoid in cancer high doses of omega-6) This happens at the molecular level when omega-3  (EPA  andDHA), reduces the action of a metabolite of the omega-6 arachidonic acid, prostaglandin E2. The sole point agreed on this study is that scientists have recommended omega 3 as a preventative for prostate cancer as and to stop its development. „

Dr. Clapp compiled a comprehensive program that has achieved national recognition USA. 1990 –was a very successful year of the program – had 16 cures of prostate cancer based on deep cleansing of toxins accumulated in the body and prostate, then flooding the cells with omega 3 fats. „Perhaps the most interesting aspect of this study is clear evidence of the mechanisms that omega-6 fats stimulate the growth of prostate cancer, clearly debunking industry hypethat  vegetable oils are good for our health. „

We saw dramatically reduced overall inflammation (measured by CRP testing) and especially in reducing enlarged prostate, reducing also any tumor mass, monitored / evaluated by Doppler sonograms.

-Omega 3 promote apatosis(programmed cell death) in cancer cells

Two new studies suggest that omega-3 fatty acids may inhibit the growth of liver cancer cells, suggesting that this may be an effective therapy to treat and prevent liver cancer.

Like vitamin B17/laetrile,and other natural substances(turmeric, ginger) omega 3 trigger apatosis.

The first study examined the effects of omega-3 and omega-6 fats in hepatocellular carcinoma cells .

Carcinoma represent 90 percent of all cases of liver cancer and is usually rapidly fatal. Omega-6 had no effect on cancer cells, but omega-3 – as  (DHA) and (EPA) – induced apoptosis (programmed cell death).

In the second study, omega-3 fats have been shown as effective in the treatment of cholangiocarcinoma tumor cells, an aggressive and fatal type of cancer that forms in the liver bile ducts.

Researchers said omega-3 fatty acids may not only be an effective treatment for liver cancer, but could also protect the liver from hepatic steatosis, chronic liver disease thought to be a precursor to cancer.

These above are just a few specific examples of cancer and Omega3 .Omega are great in any cancer.

Omega-3 Builds Muscle Mass in Cancer Patients, thus PREVENT and HELP IN CACHEXIA: http://articles.mercola.com/sites/articles/archive/2011/03/24/omega3-builds-muscle-mass-in-cancer-patients.aspx

Omega 3 fats are highly recommended and among the few fats accepted in cancer (either from seeds, nuts, avocado or fish, although fish oils might be contaminated with heavy metals from the fish )

These and many other roles that help in cancer, including stimulation of the immune system, reducing inflammation , direct relationship to sun tolerance by the body and regulation of vitamin D levels (often those with cancer can not tolerate the sun, therefore can not benefit either its energy or the important natural Vitamin D, the sun being main source of vitamin D, but omega 3 fixes this, also regulating levels of vitamin D)

http://articles.mercola.com/sites/articles/archive/2010/07/27/omega3s-reduce-your-risk-of-breast-cancer.aspx

See also:

For prostate cancer risk and omega3 controversy see this articles:

http://articles.mercola.com/sites/articles/archive/2013/07/31/omega-3-fats.aspx

http://articles.mercola.com/sites/articles/archive/2001/07/21/flaxseed-part-two.aspx

science.naturalnews.com/…/520717_The_relation_of_alpha_linolenic_acid_ to_the_risk_of.html
The relation of alpha linolenic acid to the risk of prostate cancer: a systematic review and meta analysis.
science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html
Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids prostate cancer (PCa) risk, whereas a diet rich in omega3 decreases risk.

Much more about  OMEGA 3 as well a POWERFUL (stage IV) cancer treatment based on AWARDED 2 NOBEL PRIZES  in my book.

============================================================================================

Vitamin K

 (i.e: in algae, greens, grapes, etc) Also see antagonists such as cinnamon, etc,
science.naturalnews.com/V/Vitamin_K_and_cancer.html

Vitamin K antagonists

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Antioxidanţi exogeni importanţi

RESVERATROLUL (alimente culoare neagra)

•        Este cel mai puternic anti-oxidant natural cunoscut

•          Există într-o cantitate de 50-100 mg / g pieliţă de strugure, mai mult decât dublul oricărei alte surse identificate

•          Se găseşte în general în fructele de culoare violet -neagră (dar nu numai) : sucul de struguri negri, sâmburii de struguri, dude, mure, coacăze, prune, dar şi din varza de Bruxelles (care poate fi consumată şi fiartă), fragi

•          Are un efect direct de distrugere tumorală, acţionând împotriva oricărui tip de localizare tumorală, dar şi asupra celor hormono-dependente (sân, ovar, testicul, ficat, stomac)

•          Blochează formarea de vase de sânge în jurul tumorii, dar şi metastazarea celulelor maligne

•          Conferă o protecţie deosebită materialului genetic nuclear (ADN), ferindu-l de mutaţii

•          Are un potenţial anti-oxidant de 50 de ori superior vitaminelor C şi E la un loc

•          Este de 10-20 de ori mai puternic decât vitamina E în protecţia anti-oxidativă a LDL-colesterolului

•          Scade nivelul sangvin al colesterolului total şi al trigliceridelor

•          Este un vasodilatator arterial

•          Se opune expansiunii fibroase a cicatricei post-infarct miocardic

•          Încă din 1985 se ştie că are capacitatea de a inhiba agregarea plachetară indusă de trombină şi ADP (Kimura) => efect anti-trombotic

•          Are efecte anti-SIDA, antivirale (virusurile hepatitice, dar nu numai !), antialergice (inhibând eliberarea Histaminei), antibacteriene şi antiinflamatoare

•          Măreşte elasticitatea articulaţiilor, stimulând sinteza fibrelor de colagen (astfel având şi un rol cosmetic)

•          Preîntâmpină ridarea şi îmbătrânirea pielii, menţinându-i elasticitatea şi tinereţea

•          Efect protectiv faţă de boala Alzheimer prin blocarea proteinei NF-Kb, împiedicând astfel microglia să distrugă neuronii

•          Există studii încurajatoare referitoare la potenţialul terapeutic al său în ceea ce priveşte şi alte boli neuro-degenerative

•          Resveratrolul poate reduce dezvoltarea melanomului malign, poate distruge cancerul de sân şi poate micşora nivelul colesterolului total sangvin

•          Creşte durata de viaţă şi potenţialul reproductiv la animalele de experienţă pe care a fost testat (15-30%)

•          Se comportă şi ca un fito-estrogen (vezi soia)

•          Previne şi combate eficient osteoporoza

Acum sper ca intelegeti de ce strugurii negrii se consuma intregi, cu tot cu pielite si samburi!

Mai mult , strugurii negrii au si multi lti antioxidanti si agenti anticancerigeni.

LICOPENUL(alimente culoare roşie)

•          (Alte denumiri : licopena, licopina)

•          Este cel mai important reprezentant al familiei carotenoizilor

•          Este singurul anti-oxidant a cărui cantitate sporeşte de circa 4-6 ori prin fierbere !!! (bulionul / pasta de roşii de ex.)(dar se distrug alte vitamine precum C si enzime prin fierbere, deci nu e un lucru recomandat!)

•          Licopen întâlnim în aproape orice aliment de culoare roşie : rosii, căpşuni, grape-fruit roşu, ardei iute şi capia, gogoşari, guava, papaya, ulei de cătină, dar şi-n struguri

•          Cea mai mare cantitate de licopen se găseşte în rosii (300-400 mg/kg) ; pentru calităţile cromatice, gustative şi medicinale ale tomatelor, italienii le-au denumit  „pomo d’oro“ (măr de aur), iar germanii „paradiesapfel“ (măr al paradisului)( de fapt rosiile sunt din aceeasi familie cu strugurii si fructele de padure: „berries”)

•          Datorită înapoierii culturale, cât şi a conservatorismului prost înţeles şi aplicat, tomatele au fost introduse şi cultivate la noi în ţară pe scară largă abia în urmă cu un secol !!!

•        Uleiul de măsline amplifică substanţial absorbţia licopenului ; prezintă biodisponibilitate şi efect antioxidant scăzut în medii apoase, în timp ce mediile lipidice îl fac puternic reactiv şi biodisponibil

•        Licopenul modifică profilul acizilor graşi din plasma umană

•        La concentraţii relativ scăzute de licopen, acesta are efecte antioxidante în protecţia celulelor, a membranelor celulare şi ADN

•        Are o capacitate anti-oxidantă de 10 ori mai puternică comparativ cu vitamina E

•        Este o substanţă cu efecte anti-tumorale marcate în general, şi specific în cancerul de prostată, sân, vezică urinară, pancreas, cervical, ovarian, uterin, intestinal, plămâni, piele

•        Unii cercetători merg până acolo încât afirmă că forţa licopenului este aşa de mare încât ajunge o singură roşie de dimensiuni medii consumată zilnic, pentru a ne da o protecţie sigură şi totală anti-tumorală pentru tot restul vieţii

•        Reduce semnificativ riscul aterosclerozei (incidenta infarctului miocardic scade cu 20-50% la persoanele ce consumă frecvent fructe şi legume bogate în licopen) şi procesele de îmbătrânire celulară

•        Alături de vitaminele C şi E, previne degenerescenţa maculară a retinei şi diminuează riscul de cataractă cu 50%

•        În consumul de licopen se pun speranţe mari pentru prevenirea bolilor neoplazice, degenerative şi a maladiei Alzheimer

B-CAROTENUL( alimente culoare galbenă)

–     beta-carotenul aparţine familiei carotenoidelor

•    (apocarotenalul şi nu b-carotenul este responsabil de colorarea în galben-portocaliu a alimentelor care-i conţin !)

•          A fost asociat decenii întregi cu vitamina A deoarece în organism este transformat rapid în aceasta. Deşi este o coincidenţă de denumire, nu cred că este greşit ca această vitamină să se bucure de o consideraţie deosebită din partea noastră, ca fiind vitamina…A !

•          Astfel s-a purces la utilizarea b-carotenului ca supliment, iar astăzi este de departe cea mai cunoscută şi cea mai cercetată carotenoidă, dar toate suplimentele studiate nu au avut acţiunea b-carotenului, dpdv teoretic

•          Este posibil ca, indiferent de conţinutul suplimentelor, acestea să interacţioneze cu absorbţia b-carotenului natural şi cu carotenoidele conţinute în dietă. Este de asemenea posibil ca, în loc de a avea loc un efect antioxidant, să existe unul pro-oxidant

•          Vitamina A luată constant perioade îndelungate la doze mari prezintă riscuri notabile, însă precursorul său, b-carotenul se pare că nu prezintă nici un risc, chiar la doze mamut (x 100 doza zilnică) ; majoritatea fructelor şi legumelor conţin cantităţi mult mai mari de b-caroten decât de vitamină A

•          Carotenoizii au efecte identice cu ale vitaminei A, dar riscul dezvoltării unei intoxicaţii este aproape nul la doze enorme

•          Necesar : 5000-8000 UI = 2-3 mg/zi (un morcov mediu conţine ceva mai mult de 10.000 UI)

Funcţii :

•          Antiinfecţioasă

•          Substanţă anti-oxidantă redutabilă

•          Atenuează mult simptomele şi efectele secundare ale curelor citostatice şi / sau radioterapice( de aceea sucuri de morcov sunt bune daca faceti chimio/radio)

•          Reglementează somnul şi tensiunea arterială

•          Creşte numărul şi stimulează activitatea limfocitelor T „ajutătoare” (LTh)

•          Frenator folicular în sindromul premenstrual

•          Stimulează şi creşte sinteza progesteronului

•          Ajută la formarea smalţului dentar

•          Utilă în tratamentul retinitei pigmentare

•          Asigură vederea normală (participă la sinteza rodopsinei, pigment fotosensibil din ochi) şi protecţia epiteliilor. Determină inducţia, diferenţierea şi creşterea ţesuturilor epiteliale, le menţine integritatea şi previne keratinizarea

•          Rol important în reproducere

•          Stabilizează membranele celulare

•          Stimulează ARN-ul mesager, cât şi sinteza unor proteine şi glicoproteine

•          Determină secreţia lizozimului salivar

•          Stimulează creşterea osoasă şi dentară

•          Este un antagonist tiroidian

•          Protector împotriva radiaţiilor de orice fel

•          Împreună cu vitamina C, reduce mult secreţia de mucus bronşic la astmatici şi la bronşiticii cronici (BPCO)

•          I s-a demonstrat eficienţa şi-n SIDA, studiile fiind încă în curs

•          Utilă în tratamentul calviţiei androgenice, a ridurilor şi a acneei juvenile

•          Îmbunătăţeşte toleranţa corpului la grefe

•          Scade riscul dezvoltării unor boli cardio-vasculare la persoanele care asociază şi un stil de viaţă sănătos ; reduce semnificativ frecvenţa infarctului miocardic, iar recuperarea post-infarct este de mai scurtă durată, iar riscul recidivelor este mai mic. Acelaşi efect îl prezintă şi Coenzima Q10

•          Este vitamina longevităţii întârziind mult apariţia senilităţii

•          Protejează ADN-ul celular de acţiunea nocivă a toxinelor

•          Surse (mg / 100 g produs) : lucernă (4,818), morcov (3-9), migdale (5-6), salată (4-8), măceşe (5), alune de padure (4,4), spanac (2,5), varză (2), sfeclă roşie (1,78), mango (1,454), papaya (1,25), pătrunjel, nucă (1,2), pepene galben (1,03) caisă (0,606), măsline (0,2), tomate (0,133), ulei de cătină, germeni de grâu, leguminoase, rădăcinoase, citrice, prune, usturoi, ceapă, fasole verde, tot ce-i verde, zmeură, pere, mere, smochine, creson, mazăre, bostan, dude, gutui, avocado, castane, cireşe, banane, nap, castravete, ananas, coacăze, mei, ovăz, orez, păpădie, struguri, ţelină, aloe

•          Diareea cronică, medicaţia de scădere a colesterolului, uleiul mineral, contraceptivele orale, alcoolul, fumatul, stresul, sarcina şi alăptarea, hepatitele şi ciroza hepatică generează deficite semnificative de vitamină A

LUTEINA

•          Este un alt membru al familiei carotinoizilor (“lutheus” = galben)

•        Surse de luteină : loboda, salata, spanacul, dovleacul galben, morcov, rebarbarum, praz, mazăre, vegetale verzi şi orice fruct sau legumă care are culori intense

•          Se fixează în mod special la nivelul retinei, în special a maculei (pata galbenă) şi la nivelul cristalinului

•          Cercetările privind degenerarea maculei la vârste înaintate au dovedit rolul protector important al luteinei (6 mg/zi luteină timp de 6 luni, scade incidenţa degenerării maculei cu 43%)

•          Există posibilitatea prevenirii cataractei prin utilizarea de AO, dar mai ales prin luteină (scăzând la o 1/3 riscul apariţiei acesteia)

•          La diabetici şi hipertensivi protecţia prin luteină este excepţională la nivelul arterelor retiniene

•          Efectul antioxidant al luteinei se manifestă nu numai la nivelul aparatului vizual, dar şi la nivelul altor organe, precum şi în cadrul unor lanţuri metabolice putând fi utilizată cu rezultate bune de către toţi cei cărora li s-a recomandat consumul de AO (pentru prevenirea bolilor cardiovasculare, cancerului prostatic etc.)

•          Oxidarea grăsimilor este inhibată în cea mai mare măsură de luteină, cu consecinţe deosebite în serul sangvin şi la nivelul ochiului ; scade colesterolul sangvin

•        Efectul luteinei este amplificat de licopen

Alţi carotenoizi mai puţin cunoscuţi

Zeaxantina

•          Are culoarea galbenă

•        Surse : avocado, varza,morcov,boabele de porumb, ardeii roşii, mango, portocale, spanac, gălbenuşul de ou (şi-n general acolo unde există şi b-carotenul)

•          Previne degenerescenţa maculară a retinei alături de luteină (în special datorită capacităţii de a filtra lumina ultravioletă şi cea albastră) şi cataracta

 

Cantaxantina

•          Are o culoare roşie intensă

•        Surse : ciupercile comestibile

Este un colorant alimentar, cu termostabilitate ridicată şi nu este fotosensibilă

 

Apocarotenal

•          Imprimă culoarea portocalie spre roşu a alimentelor

•        Surse : citrice (în special in mandarine), legume verzi (în special în spanac)

•          Este o provitamină A, ca şi b-carotenul. Are jumatate din activitatea vitaminei A şi a b-carotenului

 

(Acidul-)bapo-8-carotenal

•          Nuanţele de culoare obţinute de esterul acidului apocarotenal din alimente variază de la galben-lămâie până la galben-orange

•        Surse : este larg răspândit în natură, în special în plante, cum ar fi citricele, legumele verzi, iarba, lucerna, dar şi-n gălbenuşul de ou

•          În organismul uman este parţial convertit în vitamina A prin oxidare, contribuind la furnizarea de vitamina A

E demonstrat faptul ca avocado creste efectul carotenoizilor intre 200-400%(de 2-4 ori)(deci puteti creste  eficienta sucurilor de legume adaugand PUTIN avocado in sucuri)

Picnogenolii

•          “Picnogenolii” este o denumire generică mai corectă decât “picnogenolul”, deoarece defineşte un grup de substanţe înrudite (şi nu una singură), cu efecte sinergice, înmănunchiate

•          Aparţin familiei chimice a polifenolilor, clasei flavonoidelor. Este un complex de substanţe (proantocianine), printre care se menţionează : acidul cafeic, ferulic, fenolic, catechina, epicatechina etc.

•        Surse : seminţele de struguri, extractul de pin maritim, scoarţa de salcie, aloe vera, ceai verde, viţa nobilă, unele fructe şi verdeţuri

•          Picnogenolii au demonstrat o putere de peste 30 de ori superioară vitaminelor A, C şi E la un loc în combaterea radicalilor liberi, în menţinerea integrităţii membranelor celulare, a ADN-ului, împiedicând peroxidarea lipidică

•        Au efect antioxidant excepţional faţă de ionul superoxid, inhibând şi enzima generatoare a acestui RL (xantin-oxidaza)

•          Picnogenolii împreună cu vitaminele A,C şi E conduc la o puternică acţiune antioxidantă

•          Contribuie la reglarea tonusului muscular al arterelor, combătând vaso-spasmul

•          Participă la sinteza lichidului biliar şi la eliminarea bilei din vezica biliară (efect coleretic-colecistokinetic)

•          Reduc formarea nitrozaminelor

•          Efect hepatoprotector în caz de intoxicaţie prin expunere la substanţe toxice (solvenţi organici)

•          Inhibă formarea leucotrienelor, care sunt printre mediatorii cei mai importanţi ai reacţiilor alergice, astmului bronşic şi ai inflamaţiilor acute

•          Întăresc peretele capilar, menţinându-i supleţea (elasticitatea) şi integritatea

•          Posedă o activitate anti-mutagenă, de protejare a materialului genetic, dar şi anti-tumorală directă

•          Imunostimulator de excepţie

•          Efectele sale benefice au fost studiate în bolile degenerative cardio-vasculare, fragilitatea capilară, insuficienţa venoasă periferică

•          Nu prezintă efecte adverse pe termen lung, fiind lipsit de toxicitate

•          Protejează împotriva căderii părului

•          Previne şi combate retinopatia (microangiopatia retiniană) diabetică şi non-diabetică

•          Acţiune antienzimatică (în vitro) împotriva : elastazei, hialuronidazei, colagenazei, a1-antitripsinei, xantinoxidazei, b-glucuronidazei

 

Acidul ascorbic (vitamina C)

•          Întăreşte peretele vascular

•          Fixator de Calciu mai puternic decât vitaminele D

•          Scade colesterolul total şi creşte HDL-C

•          Previne şi distruge placa de aterom (la peste 300 mg/zi în prezenţa unui CT<160 mg%)

•          Previne cataracta, îi opreşte evoluţia şi chiar determină regresia ei

•          Creşte longevitatea şi scade numărul deceselor de orice cauză

•          Indispensabilă pentru absorbţia Fierului

•          Funcţie depurativ-detoxifiantă

•          Măreşte rezistenţa organismului la efort

•          Cresc performanţele intelectuale şi indicele IQ (alături de vitamina B1)

•          Asigură integritatea tegumentelor şi mucoaselor

•          Stimulează atât imunitatea anti-cancer, cât şi pe cea antiinfecţioasă

•          Contribuie la buna funcţionare a glandelor suprarenale

•          Fortifică musculatura şi ţesutul conjunctiv

•          Stimulează eliminarea Pb din oase

•          Participă la sinteza colagenului

•          Combate sterilitatea masculină

•          Rol desensibilizant (anti-alergic)

•          Grăbeşte vindecarea hepatitelor acute şi opreşte evoluţia celor cronice spre ciroză sau cancer hepatic

In dozele adecvate(peste 10 grame zilnic) poate prelungi viata bolnavilor decancer de pina la 16 ori

Complexul vitaminic E

•        Nu este vorba de o singură substanţă, ci de… (cel puţin) 8 : (a,b,g,d-) tocoferol şi (a,b,g,d-) tocotrienol + bioflavonoide

•        Surse (mg la 100 g / ml produs) :  migdale (25,8), ulei de măsline, nuci (12,3), germenii de grâu, măsline (8), zmeura (4,5), salata (4), ardei (3,1), avocado (3), spanac (1,7), piersici (0,6), uleiul de cătină, creson, polen, pătrunjel, aloe vera, mazăre, ridiche neagră, seminţele de in, bostan şi de floarea soarelui, roşii, alune de padure

•        Necesar : 20-30 mg / zi (eficienţa sa însă va creşte exponenţial la doze de >100 mg/zi)

•        Observaţie : orice aliment care conţine vitamina E, va conţine automat şi vitamina D şi F !

•          Clorul, pastilele cu fier anorganic şi anticoncepţionalele orale necesită cantităţi mari de vitamină E, deoarece o inactivează ; medicaţia hipocolesterolemiantă, uleiul mineral (folosit ca laxativ), fumatul, sarcina şi alăptarea, pancreatitele, hepatitele, boala Crohn etc. se însoţesc de deficite ale vitaminei E !

•          Este un anti-oxidant excepţional, “ocrotind” de oxidare acizii graşi nesaturaţi, vitaminele A şi C din lumenul intestinal, dar şi membranele celulare. Ne protejează şi de efectele oxidante remarcabile ale ozonului, smogului, de radiaţii şi de efectele nocive ale chimioterapiei (alături de vitamina A)

•          Protejează vitamina A de oxidare şi globulele roşii de factorii hemolitici

•          Potenţează funcţiile vitaminei C (evident în cataractă)

•          Grăbeşte vindecarea arsurilor şi a rănilor

•          Are calităţi diuretice şi hipotensoare

•          Diminuă frecvenţa crizelor epileptice la copii ; se pare că ar fi utilă şi în tratamentul bolii Parkinson

•          Vitamină anti-tumorală redutabilă în general, dar şi specifică pentru cancerele prostatic, col uterin, sân  şi pulmonar

•          Intervine în metabolismul şi-n sinteza acizilor nucleici şi a proteinelor

•          Măreşte capacitatea funcţională a muşchilor, ameliorând circulaţia capilară şi utilizând optim oxigenul la acest nivel. Este implicată şi în producerea de energie la nivel celular

•          Asigură buna funcţionare a aparatului reproducător şi este esenţială pentru sistemul nervos central

•          Reduce problemele legate de ciclul menstrual (tratând eficient sindromul premenstrual) şi de menopauză

•          Intervine şi-n eritropoeza (formarea globulelor roşii ale sângelui) fiziologică şi protejează eritrocitele împotriva distrugerii (hemoliză)

•          Are şi proprietăţi anti-coagulante, inhibând agregarea plachetară

•          Funcţiile acestei vitamine sunt potenţate în mare măsură de prezenţa Seleniului şi asta preponderent în cancer

•          Alături de Seleniu grăbeşte liza pigmentului de uzură neuronal – lipofuscina

•          Creşte producţia de interleukină 2, la rându-i stimulatoare a producţiei de limfocite T

•          Scade VLDL şi LDL-C (pe acesta din urmă în special în prezenţa vitaminei A), crescând în schimb HDL-C. Împiedică oxidarea LDL-C

•          Aportul corespunzător de vitamină E reduce semnificativ frecvenţa şi amplitudinea crizelor de angină pectorală

Acizii graşi omega 3

•        Surse : ulei de in,  nucă, spirulină,avocado,ulei ficat cod sau somon

•          Sursele de origine vegetală conţin doar reprezentantul inferior al clasei (ALA), în vreme ce uleiurile de peşte conţin toţi reprezentanţii (ALA, EPA şi DHA), alături de cantităţi importante de colesterol

•          Scad Colesterolul Total sangvin şi LDL-C cu densitate mică (care este extrem de aterogen !)

•          Au  un efect anti-trombotic

•          Produc o creştere uşoară a HDL-C

•          Scad fibrinogenul şi agregarea plachetară şi cresc timpul de sângerare

•          Reduc întinderea zonei de necroză cardiacă post-infarct recent

•          DHA este esenţial pentru dezvoltarea sistemului nervos, a retinei şi a sistemului imunitar al copilului

•          DHA şi ALA sunt  constituenţi importanţi ai membranei neuronale şi a vaselor de sânge din creier. În stările asociate cu deficitul lor, se întâlneşte o încetinire a creşterii staturo-ponderale, leziuni ale pielii, degenerescenţă  a organelor interne, o funcţie vizuală anormală şi chiar neuropatie periferică

•          Întârzie şi reduce apariţia simptomelor senilităţii prin blocarea generării de radicali liberi de natură lipidică

•          Acţiune anti-mutagenă şi de protejare a materialului genetic

•          Efect antiinflamator mediu

•          Reduc simptomele şi blochează acţiunea citokinelor (TNFa, PAF, IL1-8 etc.)

•          Modulatori deosebit de puternici ai răspunsului imun

•          Efecte importante în bolile auto-imune, infecţii virale şi / sau bacteriene, cancer, SIDA

•          Au capacitatea de a distruge vasele care alimentează cu sânge tumora

•          Previn apariţia preeclamsiei şi a eclamsiei gravidice. Previn apariţia depresiei de post-partum. Efect antidepresiv general

•          Prezintă efecte benefice şi-n sindromul Raynaud, după o utilizare îndelungată

•          Un raport w3 / w6 > 1/3 provoacă vasodilataţie arteriolară şi prevenind o eventuală HTA

•          În bolile glomerulare renale, aportul de AG w 3 a redus pierderile de proteinuria

•          Vindecă ulcerul gastric şi duodenal ; efecte excelente au fost raportate şi-n terapia colonului iritabil

•          Regularizează ciclul menstrual, diminuă frecvenţa şi intensitatea durerilor menstruale

•          Sunt substanţe anti-alergice, reducând frecvenţa şi intensitatea crizelor de astm bronşic

•          Au efect hipoglicemiant (util şi la diabetici), transformând glucoza sangvină în glicogen de depozit ; reduc rezistenţa la insulină

•          Previn şi combat eficient migrenele

•          Rol calmant şi de mărire a rezistenţei sistemului nervos la stres. Somnul se instalează mai repede la cei care au în alimentaţie surse bogate de AG w 3, iar frecvenţa insomniilor scade semnificativ

•          Stimulează sinteza colagenului din piele

Acizii graşi omega 6

 

•        Surse :  germeni de grâu, dovleac, rapiţă, susan

•          Extrem de sensibili la lumină, aer şi termic (prăjire), în prezenţa cărora putând suferi procese de izomerizare cis-trans (devenind din anti-aterogene, pro-aterogene), cât şi oxidări multiple la dublele legături, devenind din anti-oxidante, pro-oxidante

•          Reprezentanţi : acidul arahidonic, acidul g-linolenic şi acidul linoleic

•          Indispensabili pentru producerea de energie, pentru transferul oxigenului din aer până la nivelul hemoglobinei

•          Precursori ai prostaglandinelor

•          Anti-aterosclerotice “per se”

•          Asigură buna funcţionare a sistemului nervos central, intrând în structura membranelor neuronale

•          Ameliorează simptomele ulcerului gastric şi duodenal

•          Menţin tonicitatea şi troficitatea pielii

•          Carenţa acestor acizi determină astenie, piele uscată, deficit imunitar, retard al creşterii şi sterilitate

•          Un raport w6 / w3 > 1/3 are efecte pro-inflamatorii (raportul optim este < 1/3)!!!!!!!!!!

 

Coenzima Q10

•          Este un anti-oxidant exo-, dar şi endogen

•        Surse : frunzele de tutun, uleiul de cătină, alune de padure, varză, conopidă, broccoli, spanac, germenii de grâu, orez integral, uleiurile vegetale

•          Exerciţiul fizic, vitamina E şi Seleniul îi stimulează sinteza

•        Necesar : (se presupune că ar fi în jurul valorii de) 30-90 mg/zi (1 mg pentru fiecare an vârstă / zi)

•          Se concentrează în organele cu metabolism intens (creier, miocard, ficat…)

•          Cei care au o dietă vegan, aduc mai multă ubiquinonă prin alimentaţie decât omnivorii

•        Normo- şi subponderalii prezintă concentraţii mai ridicate de CoQ10 comparativ cu cei care au supragreutate

•          Sinteza ubiquinonei se reduce odată cu vârsta

•          Catalizator în procesul de formare a energiei celulare, răspunzătoare de 95% din energia ce se formează în corp ; menţine integritatea membranelor mitocondriale

•          Previne oxidarea LDL-C şi scade riscul aterosclerotic

•          Adjuvant extrem de util în tratamentul insuficienţei cardiace şi al cardiopatiei ischemice. Determină rărirea frecvenţei atacurilor anginoase cu 45-50%, mărind capacitatea de efort a pacienţilor cardiaci. Reduce hipertrofia miocardică şi-i satisface necesarul energetic. Creşte fluxul sangvin coronarian, îmbunătăţeşte metabolismul şi asigură furnizarea de energie pentru miocard, coboară TA

•          Determină producerea de anticorpi şi creşte concentraţia plasmatică a imunoglobulinelor G

•          Boala Parkinson prezintă concentraţii mici de ubiquinonă în creier, iar în SIDA există concentraţii plasmatice extrem de reduse ; pacienţii HIV pozitivi, vor rămâne asimptomatici dacă au o concentraţie optimă a CoQ10 în sânge

•          Utilă în distrofia musculară progresivă, ameliorând în bună măsură simptomatologia

•          Anti-oxidant puternic, imuno-stimulator (sporind numărul de limfocite TK) ; substanţă cu proprietăţi anti-tumorale (absorbind radicalii liberi), anti-SIDA şi antileucemică

•          Îmbunătăţeşte funcţia cerebrală şi creşte capacitatea de memorizare

•          Diminuă depozitele de grăsime

•          Măreşte rezistenţa musculară la efort

•          Grăbeşte vindecarea plăgilor, menţine supleţea, elasticitatea şi tinereţea tegumentelor

•          Capabilă să vindece ulcerul gastroduodenal

•          Adjuvant important, util şi extrem de eficace în refacerea ficatului (hepatite A, B, C etc., acute sau cronice)

•          Utilă în tratamentul schizofreniei, a bolii Alzheimer şi a sclerozei multiple

•          Întârzie procesele de îmbătrânire şi se opune celor degenerative

•          Medicaţia statinică, prin inhibarea enzimei HMG-CoA reductazei, determină o diminuare a producţiei de CoQ10 , iar  medicaţia b-blocantă inhibă enzimele activate de către CoQ10 şi produce o scădere a concentraţiei sale

Bioflavonoidele

•          Sunt cunoscute mai ales ca vitamina C2, P, sau flavone

•          Au fost descoperite de către omul de ştiinţă maghiar Albert Szent-Gyorgyi, descoperitorul vitaminei C, care a observat că bioflavonoidele au un efect sinergic cu vitamina C, având o mare importanţă în întărirea peretelui capilar

•          Se găsesc în multe fructe (coaja de măr şi pară, pieliţa internă a citricelor, fructele care au culori intense) şi verdeţuri

•          Bioflavonoidele diferă de la fruct la fruct, de la frunză la frunză, atât ca tip, cât şi ca şi cantitate şi putere antioxidantă

•          Cele mai cunoscute bioflavonoide sunt quercetina, rutina, hesperidina, naringina, baicalina şi picnogenolul

•          Substanţe potenţatoare de efect pentru minerale şi vitamine (mărind efectul acestora de 20-200 de ori)

•          Cresc rezistenţa capilarelor, reglându-le permeabilitatea ; se opun formării de varice şi hemoroizi

•          Efect antianginos şi tonicardiac ; anti-aterosclerotice de primă mână şi anti-trombotice

•          Sunt esenţiale pentru protecţia în calea oxidării vitaminei C

•          Au activitate majoră antivirală şi anticancerigenă

•          Efect antiinflamator moderat şi desensibilizant (maladii alergice şi astm bronşic) ; reduc cantitatea de Histamină din sânge

•          Acţiune marcată împotriva virusurilor poliomielitei, hepatitelor A şi B, influenţei şi HIV

•          Tonice-trofice şi regeneratoare hepatice

•          In vitro quercetina şi picnogenolul inhibă complet replicarea virusului HIV

•          Reduc problemele legate de ciclul menstrual (dureri, durata sângerării, neregularităţi etc.) ; au şi efect anti-abortiv

•          Tulburările de natură aterosclerotică ale vaselor urechii interne de care suferă diabeticii, par să răspundă tratamentului combinat vitamină C + bioflavonoide

•          Administrate împreună prezintă efecte benefice în tratamentul distrofiei musculare şi al depresiei cronice

•          Studii legate de administrarea lor concomitentă, cu rezultate benefice sunt în curs, în tratamentul schizofreniei

•          Bioflavonoidele din afine sunt eficace în tulburările de vedere, putând ameliora vederea crepusculară, reducând fenomenele de neacomodare la întuneric ; se opun degenerărilor retiniene de origine hipertensivă, diabetică şi miopiei evolutive

•          Rănile se vindecă mai repede şi fără cicatrici

•          Preîntâmpină ridarea şi îmbătrânirea pielii, menţinându-i elasticitatea şi tinereţea

•          Carenţa de bioflavonoide este asemănătoare cu carenţa de vitamină C : pierderi de sânge, fragilitate capilară, hematoame ; constituie un factor predispozant pentru reumatism

•          Antagonişti : fumatul, stresul, antiinflamatoarele steroidiene şi nesteroidiene, antibioticele, cafeaua

•          Cresc absorbţia bioflavonoidelor : vitamina C, Calciul şi Magneziul

Niacina (vitamina B3, PP, NAD)

•          Este o vitamină relativ stabilă termic, la lumină şi oxigen

•        Surse (mg%) :  caise  (6,7), cartof (1), piersica (0,9), germenii şi tărâţele de grâu, nuci, orez, polen, grape-fruit, anason, ovăz, varza, gutui, lămâi, mere, pere, vinete, roşii, sfeclă roşie, smochine, seminţele de in, aloe

•        Necesar : 15-20 mg/zi. Dacă dieta conţine suficient Triptofan şi vitamină B6, corpul şi-o poate sintetiza şi singur

•          Cel mai important factor de creştere al HDL-C. Reduce şi nivelul trigliceridelor

•          Creşte capacitatea de memorizare

•          Intensifică metabolizarea glucozei, rol important în diabetul zaharat

•          Participă la integritatea mucoaselor, prevenind instalarea a multiple dermatoze

•          Ameliorează simptomele artritei

•          Carenţa sa dă pelagra (cei 3 “D” : dermatită, demenţă, diaree)

•          Participă la producerea energiei în corp

•          Ajută la sinteza unor hormoni : insulină, testosteron, estrogeni

•          Combate un eventual miros neplăcut al gurii (halena)

•          Combate şi / sau ameliorează durerile ulceroase

•          În doze mari stimulează eliberarea de Histamină

•          În sarcină, alăptare, sau în cazurile în care se prestează un exerciţiu fizic intens, se suplimentează dieta cu cantităţi corespunzătoare de vitamină B3 Alcoolul este şi el răspunzător de scăderea nivelului plasmatic al acestei vitamine !

Germaniul (organic !)

Este un mineral alcalin(ceeaza alcalinitate), la fel ca potasiu, cesiu, etc.

–          necesar : 0,4 – 1,5 mg/zi(mult mai mult in cancere- detalii in carte)

–          funcţii : util în prevenţia şi tratamentul multor boli :

– cancere cu diverse localizări : colon, prostată, sân, plămâni, ovar, col uterin. Este un element cu proprietăţi anti-tumorale importante. Stimulează concomitent superoxid-dismutaza, glutation-peroxidaza şi catalaza !!!

– leucemii, SIDA

– astm bronşic

– gastrite şi ulcere

– diabet

– angină pectorală, HTA, ATS, infarct miocardic, sd. Raynaud

– boli psihice : psihoze depresive, schizofrenie, boala Parkinson, epilepsie, autism

– boli oculare : cataractă, glaucom, inflamaţii şi dezlipire de retină

– are o acţiune anti-virală şi anti-fungică

                   – stimulează imunitatea : creşte producţia de gama-interferon şi de limfokine, cât şi numărul de macrofage, limfocite Tk şi de supresoare

– combate osteoporoza şi artrita reumatoidă

– suprimă durerile menstruale

– rol fundamental în cura de dezintoxicare după acumularea de Mercur în corp

– element anti-oxidant puternic şi anti-mutagen

– scade concentraţia colesterolului sangvin

– măreşte biodisponibilitatea oxigenului la nivel celular

–          surse : usturoi, tărâţele de cereale şi cerealele integrale, aloe, nuci, uleiuri vegetale, fasole, morcovi, broccolli, conopidă, roşii, rădăcina de Ginseng.

Seleniul

–          necesar : 100 micrograme/zi. Carnea, alcoolul şi fumatul îi blochează absorbţia! Prin ejaculare se pierd cantităţi sporite de Seleniu !

–          funcţii : – element anti-oxidant cu proprietăţi anti-tumorale excepţionale

– previne acumularea unor metale grele în corp şi le grăbeşte eliminarea : Mercur, Arsen, Cupru (dacă acesta riscă să ajungă la niveluri plasmatice toxice !), Cadmiu

                    – stimulează sinteza şi efectele glutation-peroxidazei, enzimă extrem de potentă anti-tumoral, care nu poate funcţiona în absenţa Seleniului

– stimulează sinteza de imunoglobuline

– împiedică degenerarea musculaturii striate

– ameliorează simptomele legate de menopauză, în special bufeurile de căldură

– anti-ATS de forţă

– protejează Fe2+ din hemoglobină împotriva oxidării la Fe3+

– anti-reumatic puternic

– măreşte apărarea imună

– protejează ficatul de infiltraţia grasă

– controlează funcţia secretorie normală a pancreasului

– previne şi tratează eficient mătreaţa

–          surse : usturoi, ceapă, praz, afine, cereale integrale, grâu încolţit, aloe vera, seminţe de susan şi fistic, sparanghel, leguminoase, zarzavaturi, roşii.

Zincul

–          necesar : 15-20 mg/zi. Atenţie : stresul poate produce eliminarea a peste 8 mg de Zinc pe zi pe cale urinară, în timp ce cura citostatică îi reduce absorbţia şi-i măreşte eliminarea ! Medicaţia diuretică, alcoolismul cronic, dieta bogată în Fosfor (carne şi lactate) şi fibre vegetale (la un aport de peste 50 g pe zi !),  sarcina şi alăptarea sunt stări însoţite de pierderi mari de Zinc.

–          funcţii : – grăbeşte vindecarea şi cicatrizarea rănilor

– potenţează alături de Cupru şi Mangan efectele enzimei SOD

– intră în compoziţia a peste 70 de metalo-proteine (printre care şi a insulinei, heparinei) şi a unor enzime

– este un anti-oxidant redutabil pentru radicalii liberi

– previne şi tratează eficient depresiile, alături de Cupru

– metal cu efecte puternice anti-tumorale

– este un antagonist al Calciului

– intervine în formarea normală a globulelor sangvine (rol anti-anemic şi anti-leucemic). Stimulează creşterea numărului de leucocite (globule albe) în periferie

– este esenţial pentru maturarea limfocitelor T din timus

– stimulează hipofiza şi glandele sexuale, reglându-le în acelaşi timp şi funcţia

– previne şi combate eficient degenerarea maculară (petei galbene de pe retină)

– creşte potenţa şi măreşte dorinţa sexuală (prin stimularea secreţiei testosteronului – pe care prolactina l-ar inhiba – la ambele sexe)

– reduce semnificativ frecvenţa răcelilor

– este extrem de util în boala Wilson, ajutând la eliminarea excesului de Cupru

– protejează organismul de efectele toxice ale Cadmiului şi Plumbului, inhibându-le absorbţia lor din intestin

– creşte apetitul

– participă la metabolismul energetic, la sinteza şi degradarea proteinelor, cât şi la sinteza acizilor nucleici. Ajută la metabolismul vitaminei A

– intervine şi-n menţinerea echilibrului acido-bazic

– favorizează eliminarea CO2-ului din celule

– participă la sinteza colagenului

– asigură sensibilitatea gustativă, olfactivă şi tactilă normală

– ajută la dispariţia petelor galbene şi a striurilor unghiale

– favorizează contracţiile musculare

– creşte concentraţia testosteronului şi a hormonului FSH

– sunt studii în curs referitoare la utilitatea sa în tratamentul schizofreniei, acneei juvenile şi al ulcerului gastric şi duodenal. Rezultatele preliminare însă sunt promiţătoare

– intensifică activitatea enzimei alcool-ehidrogenaza şi o inhibă pe cea a anhidrazei carbonice

– anti-artritic bun

– inhibă secreţia de prolactină, care dacă ar stimula producţia de testosteron, ar determina o hipertrofie prostatică. Menţine astfel glanda la dimensiuni normale şi se opune adenomului de prostată

– este un inductor enzimatic pentru enzimele care contribuie la transcrierea corectă a informaţiei genetice de la nivelul ADN şi ARN. S-a descoperit că în sângele bolnavilor de SIDA cantitatea de Zinc este foarte redusă, şi se presupune că suplimentarea cu Zinc a dietei acestor pacienţilor ar avea rezultate notabile împotriva bolii

– participă la metabolismul vitaminei A

– asocierea Zinc + Cupru realizează beneficii notabile în dereglările hipofizo-ovariene şi testiculare

– asocierea Zinc + Cobalt + Nichel este utilă în tulburări pancreatice şi hipofizare

– asocierea Zinc + Magneziu + Fier contribuie la creşterea capacităţii de memorizare

– asocierea Zinc + Magneziu + vitamina B6 determină creşterea concentraţiei plasmatice a testosteronului

–          surse : nucile, castanele, stridiile, afinele, germenii de grâu, cereale integrale, piersici, portocale, banane, cireşe, ciuperci, pere, mere, smochine, susan, migdale, aloe vera, cerealele integrale, sfecla roşie, roşia, spanac, varză, salată, usturoi, caisă, vinete, seminţe de dovleac, migdale.

   Organele corpului uman care concentrează Zincul sunt : prostata, rinichii, ficatul, oasele, creierul, retina şi muşchii. S-a constatat că în SIDA şi diabetul zaharat există concentraţii sangvine foarte scăzute ale Zincului, iar suplimentarea alimentaţiei cu acest metal a adus beneficii semnificative.

Cuprul

–          necesar : 2,5 – 3 mg/zi

–          funcţii : – anti-oxidant puternic şi anti-cancerigen

– previne alterarea şi oxidarea AG nesaturaţi şi-n special a celor 3 AG w 3

– contribuie la integritatea membranelor celulare

– previne şi tratează eficient depresiile alături de Zinc

– determină creşterea uşoară a HDL-C

– asigură pigmentarea normală a părului

– limitează formarea radicalilor liberi

– asigură creşterea şi dezvoltarea armonioasă a organelor şi celulelor

– creşte apărarea imună

– menţine vasele arteriale curate (de ateroscleroză) şi elastice

– are şi efect anti-coagulant

– implicat în formarea globulelor roşii, ajutând la transferul Fierului pe Hemoglobină şi prin aceea că intervine în absorbţia şi-n utilizarea Fierului pentru hematopoeză

– ajută la formarea calusului în fracturi

– menţine integritatea mielinei (beneficii în boala numită “scleroză în plăci” ?)

– participă la sinteza colagenului alături de vitamina C

– reduce simptomele artritei

Deficitul de cupru antrenează şi următoarele tulburări : defecte de absorbţie şi metabolizare ale Fierului, anemia refractară la Fier, apariţia enteropatiei specifice cu pierderi consecutive de Cupru, creşte rata apariţiei de sugari distrofici şi prematuri, a sindromului mielodisplazic, a osteoporozei, se constată anomalii neurologice, reducerea diferenţierilor celulare, aritmii cardiace, hipotiroidie, transformare megaloblastică a seriei albe.

–          surse : nuci, castane, varză, migdale, alune, cireşe, mere, portocale, struguri, grape-fruit, polen, gutui, lămâi, pere, piersici, smochine, caise, măsline, cereale integrale, sfeclă roşie, nap, ceapă, spanac, cicoare, praz, cartof, roşii.

Manganul

–          necesar : 3 mg/zi. Un aport crescut de Fier este responsabil uneori de un deficit al Manganului

–          funcţii : – anti-oxidant puternic, având proprietăţi anti-tumorale marcate

            – împreună cu Zincul şi Cuprul, este metalul care stimulează funcţia enzimei SOD

– necesar structurii osoase normale şi a cartilajului articular

– este implicat în sinteza hormonilor sexuali

– asigură funcţionarea optimă a creierului, stimulând sinteza DOPA-minei. Această substanţă, în afara faptului că este un puternic anti-reumatic, este în stare să amelioreze simptomele bolii Parkinson (boală caracterizată tocmai prin scăderea numărului neuronilor producători de DOPA-mină)

– util în tratamentul schizofreniei şi al unor boli neurologice

– stimulează absorbţia vitaminelor C, H şi B1

– anti-alergic şi desensibilizant redutabil

– asocierea Mangan + Cobalt este utilă în tratamentul tulburărilor de memorie, sindromul premenstrual, anxietate, colite spastice, tulburărilor de menopauză

–          surse : nuci, castanealoe vera, tărâţe, sparanghel, spanac, vinete, pere, mere, cereale integrale, usturoi, ceapă, praz, varză, sfeclă roşie, ţelină, morcov, păpădie, polen, caisă, cartof, ciuperci, cicoare, pătrunjel, portocale, smochine, căpşuni, struguri, uleiul de cătină.

Molibdenul

•          Este un mineral care se găseşte în cantităţi extrem de reduse în aproape toate ţesuturile vegetale şi animale

•          Prezintă efecte AO, fiind deci important în tratamentul maladiilor degenerative şi a îmbătrânirii

•        Surse : legumele, cerealele integrale şi verdeţurile cu frunze verde închis, salată, fasole

•        Necesar : 75-500 mg/zi

•          > 500 mg/zi există riscul pierderilor de Cu, diaree, anemie şi întârzierea creşterii ; la 15 mg/zi poate apărea guta

•          Carenţa este consecinţa acumulării de SO32-, radical toxic pentru sistemul nervos ; alimentele rafinate şi conservate consumate perioade mai lungi conduc inexorabil la carenţe de Mo

•          Intră în compoziţia a 2 enzime : xantinoxidaza (care ajută la mobilizarea Fe din rezervele hepatice, favorizând transformarea Fe2+ în Fe3+) şi aldehidoxidaza (necesară pentru oxidarea grăsimilor). Ambele enzime sunt implicate în transferul de electroni

•          Mo are un rol important în metabolismul Cu şi al N

•          Este implicat în faza finală a formării urinei

•          Previne anemia feriprivă

•          Smalţul dentar conţine Mo şi s-a descoperit că mineralul previne cariile dentare

•          Anumite studii au arătat o legătură între o cantitate optimă de Mo din alimentaţie şi incidenţa redusă a cancerului esofagian (asociat şi unei doze optime de vitamină C)

•          Combate impotenţa

MSM (Metil-sulfonil-metanul)

•        Este un AO puternic, capabil să împiedice acţiunea RL, dezintegrându-i ; acţionează atât direct, cât şi prin faptul că este o sursă de sulf ; MSM furnizează Sulful necesar AA sulfuraţi (MET, CYS, TAU), consideraţi la rându-le drept AO puternici

•          Se găseşte în cantităţi importante în spirulină şi aloe vera

•          Reacţionează la nivelul mucoaselor cu toxinele, contribuind la dezactivarea şi eliminarea acestora

•          Ameliorează permeabilitatea membranelor celulare, favorizând intrarea în celule a substanţelor nutritive şi a vitaminelor, dar şi eliminarea reziduurilor (este printre cele mai puternice substanţe cu efect dezintoxicant cunoscute)

•          Este un analgezic natural, blocând conducerea impulsurilor dureroase de-a lungul fibelor nervoase (fibrele C)

•          Blochează inflamaţia şi procesele inflamatorii, potenţând efectele cortizolului

•          Joacă un rol fundamental în menţinerea integrităţii articulaţiilor şi elasticităţii ţesutului conjunctiv

•          Dilată vasele de sânge, ameliorând circulaţia

•          Accelerează procesele de vindecare şi de reparaţie celulară

•          Reprezintă un bun miorelaxant în diverse forme de durere cronică

•          Este un adjuvant al mecanismelor naturale de apărare ale corpului, reglând metabolismul prostaglandinelor, participând la formarea anticorpilor

•          Este considerat un element sinergic pentru vitaminele A, B, C, D şi E, CoQ10, amino-acizi, Se, Ca, Mg

•          Diminuează simptomele unui mare număr de reacţii alergice, în special cele alimentare, dar şi cele prin mecanism de contact, sau prin inhalare. A fost stabilită o relaţie directă între cantitatea de MSM şi rezistenţa la alergeni ; este un inhibitor de Histamină

•          Controlează reacţiile inflamatorii în cadrul reacţiilor auto-imune

•          Reduce necesarul de medicamente anti-H2 în tratamentul ulcerului gastro-duodenal ; combate constipaţia asociată ulcerului

•          Măreşte rezistenţa organismului la stres (obiectiv şi subiectiv)

•          Toate tipurile de afecţiuni dermatologice, mai ales cele de natură alergică, răspund pozitiv la un regim dietetic integrat cu MSM ; s-a demonstrat că administrarea de MSM pe cale orală este eficace contra acneei, a pielii uscate, iritate şi a descuamărilor cutanate (sub formă de ungvent)

Alţi antioxidanţi exogeni importanţi

 

Betanina

•          Este colorantul roşu din sfecla roşie

•          Imuno-stimulator important

•          Are predilecţie pentru cancerele hormono-dependente

 

Sulforafanul

•          Izotiocianat, prezent din abundenţă în vegetalele aparţinând familiei crucifere, ceai verde, ceapă

•          Antioxidant puternic, mai puţin studiat

•          Capabil de distrugere tumorală directă, în special asupra tumorilor hormono-dependente : sân, col uterin, prostată

•          Imunostimulator

•          Prezintă şi efecte antiinflamatoare

•          Hepatoprotector şi detoxifiant

 

Indol-3-carbinolul

•          Prezent mai ales în vegetalele crucifere(varza, conopida, etc.)

•          Împiedică dezvoltarea tumorală malignă

•          Împiedică angiogeneza tumorală

 

Capsicaina (capsaicina)

•          Se găseşte şi se extrage şi ardeiul iute şi capia

•          Substanţă puternic iritantă pentru mucoase (uneori şi pentru tegumente)

•          I s-au descoperit proprietăţi anti-tumorale gastrice

•          Anti-reumatic de excepţie (există ungvente cu capsicaină)

 

Limonena

•          O întâlnim în citrice, dar mai ales în grape-fruit

•          Inhibă dezvoltarea tumorilor mamare

 

Antocianinele

•          Sunt coloranţi naturali roşii-albăstrui

•          Se găsesc în fructele de pădure, struguri, sfecla roşie, grape-fruit

•          Reduc frecvenţa tuturor cancerelor, indiferent de localizare, atât prin acţiune directă de distrugere tumorală, cât şi prin reciclarea glutationului

 

Acidul elagic

•          Există în din struguri, căpşuni şi cireşe

•          Este capabil să neutralizeze acţiunea substanţelor pro-carcinogene

 

Sulfidele

•          Le întâlnim la reprezentanţii familiei liliacee (usturoi, ceapă, praz)

•          Inhibă creşterea tumorală, fiind şi captatori excepţionali ai RL de oxigen

 

(Tri-)terpenele

•          Surse : crucifere(varza conopida,etc.), cereale integrale şi citrice

•          Diminuă multiplicările rapide, particulare celulelor tumorale

 

Substanţele indolice

•          Există aproape exclusiv în vegetalele crucifere

•          Utili în prevenţia şi terapia cancerului de sân, blocând acţiunea excesului de estrogen natural, factor implicat în creşterea tumorală

 

N-acetil-cisteina

•          Este un medicament fluidifiant al secreţiilor bronşice

•          Reprezintă un precursor, dar şi un stimulator al sintezei Glutationului

 

EDTA (Acidul etilen-diamino-tetraacetic)

•          Este un medicament utilizat drept kelator în ordine descrescătoare pentru : Cr,  Fe3+, Hg, Cu, Pb, Zn, Al, Fe2+, Mn, Ca, Mg

 

Ionul de H

•          Există în apa unor izvoare de munte, din câteva zone ale lumii ; persoanele care beau acea apă fac parte dintre marii longevivi

•          A fost puţin studiat, dar se pare că este pe cale să detroneze resveratrolul din poziţia sa de lider al substanţelor AO

 

Nota:

Dozele de mai sus sunt recomandarile pentru persoane sanatoase.

Un pacient cu cancer are nevoie de doze de 10 ori mai mari, si nu glumesc.

Vedeti exemplul cu coenzima(click aici).

Niciodata nu se incepe cu dozele maxime ci cu doze mici si se cresc dozele pe masura ce organismul se adapteaza

 

Nu recomand suplimente antioxidante deoarece pot contine si multe substante chmice adaugate.Pulsa ca pot fi mai scumpe decat ce puteti obtine pe cale alimentara. Luati-va antioxidantii din fructe legume ierburi si verdeturi bine spalate cu apa filtrata.

 

Multa sanatate,

Cristian

 

CONSIDERATI UTILE CELE DE AICI?
SPUNETI SI ALTORA!(butoane sub aceasta pagina si in DREAPTA SUS!)

Combate radiatiile si efectele RADIOTERAPIEI!(Glutation, Vitamine, alge marine, ceai verde)

Un lucru pe care, personal consider ca multi il trec cu vederea si poate fi o cauza mult mai semnificativa de cancer, la noi in tara, poate mai importanta decat stilul de viata modern, „vestic”, il constituie radiatiile abosrbite in 1986 de la Cernobil.

Am fost expusi la radiatii de la Cernobil( a se vedea aici) la care se adauga orice radiografii, scanari CT, PET  și Radioterapia, raze X si multe altele, inclusiv mamografii.

Trebuie sa ne protejam  și sa inversam orice prejudiciu care ar putea fi realizat de către radiații.

Ce putem  face pentru a ne proteja , mai ales in cazul in care facem RADIOTERAPIE?

Există mai multe lucruri pe care le puteți face. Aici am prezentat cateva.

Alte tratamente cu rezultate foarte bune in zona Cerobil si Japonia am dat si in carte.

Continuă lectura