Intervenția nutrițională cu acizii grași omega-3 într-un caz de histiocitom fibros malign al plămânilor

Prezentăm un caz de bărbat în vârstă de 78 de ani cu un histiocitom fibros malign cu leziuni multiple în ambii plămâni. 

După diagnosticare, el a refuzat chimioterapia convențională și a ales o intervenție nutrițională prin creșterea consumului de acizi grași omega-3 și scăderea aportului de acizi grași omega-6.

Am estimat că a consumat 15 g de acid eicosapentaenoic cu acizi grași omega-3 (EPA) și acid docosahexaenoic (DHA) pe zi, iar raportul dintre acidul linoleic și acizii grași omega-3 în lanțul său lung a fost 0,81 .

Scanările tomografice computerizate computerizate și razele X pulmonare au evidențiat remarcabil o scădere lentă și constantă a dimensiunii și a numărului de noduli bilaterali.

Nu are efecte secundare evidente din consumul unor cantități mari de uleiuri de pește și alge bogate în DHA și EPA și rămâne asimptomatic.

PMID: 16201843 

DOI: 10.1207 / s15327914nc5202_2

https://www.ncbi.nlm.nih.gov/pubmed/16201843

Pardini RS ¹ , Wilson D , Schiff S , Bajo SA , Pierce R.

Informații despre autor

1

Departamentul de Biochimie, Colegiul de Agricultură, Biotehnologie și Resurse Naturale, Universitatea Navada, Reno, NV 89557, SUA.ronp@cabnr.unr.edu

interviu Prof. dr. John Ionescu fondator și director științific al Clinicii Speciale Neukirchen din Bavaria

Prof. dr. John Ionescu, fondator și director științific al Clinicii Speciale Neukirchen din Bavaria, este o personalitate în domeniul terapiilor integrative.

Deşi plecat încă din anii 70 din România doctorul Ionescu nu-şi uită ţara, ci doreşte ca ultimele descoperiri ale medicinii să fie integrate şi în terapiile clinicilor din România.

 

– Una dintre cele mai mari probleme de sănătate ale ultimelor decenii este cancerul. Cât de importantă este nutriţia în cancer?
– Pe lângă starea de inflamaţie cronică care poate fi cauzată de diverse antigene alimentare[1] şi care trebuie eliminate încă de la începutul terapiei, în faza iniţială, pacientul cu boală malignă are nevoie de un bun terapeut şi respectiv dietetician, care a înţeles particularităţile metabolice comune tuturor celulelor canceroase.
În acest sens, este esenţială înţelegerea rolului zaharurilor, în special al glucozei şi al fructozei, în metabolismul energetic al celulelor canceroase. Aceste celule se hrănesc predominant cu zahărul pe care-l iau din sângele pacientului şi, ca atare, orice creştere a glicemiei sangvine conduce la o accelerare a multiplicării tumorale. Acest zahăr, absolut necesar celulei canceroase, este metabolizat pe cale glicolizei aerobe (efectul Warburg), cu producerea unei cantităţi crescute de acid lactic pe care celula canceroasă îl elimină în spaţiul extracelular cu toate mijloacele pe care le are la dispoziţie. Eliminarea continuă a protonilor de hidrogen şi a acidului lactic asociată cu acumularea intracelulară de elemente reductive precum glutationul, cisteina, NADH-ul şi, în special, glucoza, conferă celulei canceroase o stare de redoză asociată cu un pH alcalin, care îi permite o multiplicare permanentă, similară cu cea a celulelor embrionale sănătoase.

 

Pe de altă parte, starea de acidoză extracelulară[2] realizată prin mecanismul menţionat mai sus conduce atât la avansarea rapidă a tumorii în ţesuturile înconjurătoare prin activarea proteazelor corespunzătoare, cât şi la anihilarea activităţii celulelor sistemului imun, care rămân paralizate la locul tumorii, contribuind la creşterea în dimensiuni a acesteia.

Ca atare, scopul unei terapii nutriţionale adecvate nu poate fi decât reducerea consumului de glucoză a celulelor tumorale, eliminarea redozei intracelulare necesare multiplicării şi compensarea corespunzătoare a acidităţii extracelulare.

a se citi :

CAUZELE CANCERULUI

Antioxidantii si multirezistenta cancer ; Beneficiile dietei paleo-ketogenica si suplementelor naturale in lupta impotriva cancerului

LUPTA IMPOTRIVA CANCERULUI: IMPORTANȚA CORECTARII PHului CELULEI

 

 

Atât intervenţiile din clinica noastră, cât şi studiile universitare la nivel mondial pe care le-am prezentat în cadrul conferinţei demonstrează rezultate clinice deosebite în acest sens. Spre exemplu, corectarea acidozei extracelulare poate fi efectuată extrem de simplu prin administrarea lactatului sau a bicarbonatului de sodiu, fapt care reface capacitatea celulelor sistemului imun de a ataca periferia tumorii şi reduce dramatic capacitatea de metastazare a tumorii.
Studiile americane şi europene în acest domeniu aduc dovezi zdrobitoare în acest sens – desigur, o veste proastă pentru firmele de medicamente, întrucât bicarbonatul care costă 2 lei poate fi folosit extrem de eficient în combaterea acidozei extracelulare şi a metastazărilor, reducând frecvenţa şi intensitatea acestora.

 

A se citi si:

https://tratamenteanticancer.wordpress.com/2015/12/14/cum-se-face-amestecul-lamaie-bicarbonat-sodiu/

https://tratamenteanticancer.wordpress.com/2013/05/03/tratament-cancer-bicarbonat-sodiu/

NOTE: dieta alcaliniZANTA, apa (ionizata ) alcalina, minerale precum CESIU, POTASIU CALCIU RUBIDIU sunt de asemenea optiuni.

Retineti ca UN SINGUR tratament cancer pentru alcalinizare PUTERNIC este suficient, nu trebuie toate

 

În mod similar, pentru inhibarea redozei celulare avem nevoie de o dietetică pro-oxidativă, deci cu elemente nutriţionale cu un potenţial redox ridicat, cum ar fi anumite sucuri de legume.

 

exemple includ vitamina C intravenos  sau vitamina C lipozomala sau mixul oxidativ proteina sulfurica-omega 3 / lactate bio-ulei in bio al dr Budwig 

Acestea consumă rezerva reductivă a celulelor tumorale şi, asociate cu inhibitori de carboanhidrază (o altă enzimă extrem de puternic exprimată în celulele tumorale) reduc eliminarea protonilor din celulă, crescând acidifierea intracelulară şi inducând apoptoza (moartea) celulei canceroase.

Celulele canceroase se hrănesc cu glucoză
– Care sunt cele mai importante aspecte care trebuie avute în vedere la nutriţia pacientului afectat de cancer?
– Cel mai important factor îl reprezintă reducerea consumului de glucoză, ori aici sunt diferite tehnici care vizează atât reducerea intrării glucozei în celula canceroasă, prin inhibarea transportorilor de glucoză, cât şi, în primul rând, prin reducerea nivelurilor sangvine ale glucozei circulante. Este esenţial pentru un pacient cu boală canceroasă ca medicul să îi prescrie o dietă hipocalorică, o dietă ketogenă, cel puţin pentru perioada imediat dinainte şi după terapia anticanceroasă clasică, cu 3 zile înainte de chimioterapie şi 3 zile după chimioterapie. Această dietă ketogenă, la rândul ei, are un potenţial redox pro-oxidativ, consumând rezervele celulei tumorale şi făcând-o mult mai sensibilă atât la chimioterapie, cât şi la terapia cu radiaţii.

Acelaşi coleg care îi demonstrează pacientului unde se află tumorile în corpul său prin testul aşa-numit PET-CT, prin care este perfuzată în sânge o moleculă de glucoză marcată, care se duce ca un trasor, ca un magnet, la locul tumorii, ar trebui să îi explice pacientului că este imperios necesar să renunţe la carbohidraţii simpli de genul pâinii, făinoaselor, pastelor, prăjiturilor, dulciurilor de tot felul,fructelor dulci bogate în fructoză sau legumelor bogate în zahăr, precum sfecla(aici este GRESIT intr-ucat desi este ADEVARAT ca fructele si sfecla au glucoza (si glucoza este interceptata si hraneste in primul rand o celula de cancer) acestea (spre deosebire de fainoase cereale si zaharuri simple) au si ANTICANCERIGENI;cure exclusive de fructe(DOAR PENTRU CEI SUPRAPONDERALI!!!)  au vindecat bolnavi de cancer etapa 4 )

 

, care nu fac decât să hrănească celula tumorală. Din păcate, nu se întâmplă acest lucru. Acelaşi coleg care-i arată pacientului cu ajutorul glucozei marcate unde sunt tumorile sale, după administrarea chimioterapiei îi face perfuzii cu glucoză „pentru a-i fortifica organismul”. Asta se întâmplă în Institutele Oncologice din România, din Olanda, din America, din Germania etc.

– Ce înseamnă pentru pacienţi ignorarea acestor recomandări alimentare?
– Această ignoranţă privind aspectele metabolice centrale ale celulei canceroase poate fi fatală pacientului şi poate anula toate efectele terapiei clasice. Deci, faptul că pacientul pleacă acasă fără nici o recomandare nutriţională şi este lăsat să mănânce ce vrea sau faptul că însuşi medicul face greşeli şi îi administrează după chimioterapie injecţii cu fier, care se duce ca un magnet la locul tumorii, favorizează în mod evident creşterea tumorală. Orice perfuzie cu fier sau alimentele bogate în fier, cum ar fi carnea roşie, nu fac decât să accelereze dramatic creşterea masei tumorale.

– Ce ar trebui să ştie oamenii în ceea ce priveşte prevenţia cancerului?
– Nutriţia pacientului neoplazic sau profilaxia bolii trebuie să aibă în vedere administrarea unor alimente cu indice glicemic scăzut, eliminarea factorilor nutritivi precum zaharurile simple şi glucidele simple, precum făinoasele sau dulciurile, administrarea legumelor, fibrelor, anumitor forme de proteine cu Omega 3, cum ar fi carnea proaspătă de peşte, uleiurile cu Omega 3 şi altele(mixul dr Budwig mai ales), care să întărească apărarea imunitară, oferind în acelaşi timp cât mai puţine elemente pentru energetica celulei tumorale. În acest sens, dietele ketogene bogate în proteine şi lipide sunt deosebit de importante, întrucât celulele tumorale nu pot folosi aceste elemente în metabolismul energetic.

Cancerul şi chimizarea mâncării
– Cum arată dieta dumneavoastră personalizată?
– Întrucât nu sufăr de alergii, nu am nevoie de o dietă personalizată pe care să o urmăresc în fiecare zi pentru a mă feri de contactul cu alimentele nocive. Ştim însă cu toţii că, de la o anumită vârstă, după ce ai trecut de 40-50 de ani, metabolismul energetic se schimbă, iar tendinţa de acumulare a grăsimii abdominale şi viscerale creşte rapid, pe măsura scăderii producţiei de HGH şi testosteron. Paralel, scade treptat şi masa musculară. Contra acestor evoluţii mă apăr printr-o dietă cu indice glicemic scăzut, din care elimin pe cât posibil pâinea, făinoasele şi dulciurile, ceea ce-mi permite două lucruri esenţiale:
Să menţin o insulinemie şi o glicemie constante;
Să evit accesele de foame şi, ca atare, să pot lucra 14-18 ore fără să mănânc.

Pe măsură ce înaintăm în vârstă, nu este o idee bună să forţăm ultimele rezerve ale corpului cu creşteri inutile de insulină, care pot induce în timp rezistenţă la insulină, sindromul metabolic şi depunerea grăsimii periferice, cu toate efectele secundare – obezitate, diabet, boli cardiovasculare etc.
Din păcate, nici în cazul diabetului soluţiile de tratament nu sunt cauzale, preferându-se administrarea de insulină ani de zile în cazul diabetului de tip I, sau a substanţelor anti-diabetice la diabetul de tip II, în loc să se identifice şi să se elimine tocmai alimentele cu indice glicemic crescut, ce produc aceste descărcări masive de insulină. Prin urmare, cel mai simplu lucru pentru mine este să rămân la această dietă cu indice glicemic scăzut şi să prestez o activitate fizică, în măsura timpului disponibil.

https://tratamenteanticancer.wordpress.com/2016/01/07/progress-nutritional-si-abordari-impotriva-diabetului/

– Cum priviţi consumul de carne de către pacienţii cu cancer?
– Carnea roşie este dăunătoare pacienţilor cu cancer din cauza concentraţiei crescute în fier şi glutamină. În plus, nitrozaminele şi alte organotoxine care apar în urma procesului de prăjire al cărnii au un potenţial carcinogenic foarte ridicat. Se poate folosi, în schimb, carnea de peşte proaspăt, care e bogată în Omega 3, o dată la două sau la trei zile.
Dar, în primul rând, aceşti pacienţi trebuie să adopte dietele hipoglicemice bazate pe sucuri de legume, cu un potenţial redox corespunzător celulei canceroase. Proteine se pot obţine şi din carnea de găină sau curcan, dacă păsările nu au primit în hrană xenobioticele, estrogenii şi pesticidele care accelerează în special creşterea tumorilor endocrine, ovariene sau de prostată.

Explozia pe care o înregistrăm în cancer în ultimii patruzeci de ani are de-a face tocmai cu chimizarea mâncării şi cu acumularea în corp a diverşilor poluanţi, de la pesticidele, solvenţii, detergenţii şi coloranţii cu care venim în contact şi până la aliajele metalice dentare sau oalele de gătit din oţel, inox, cupru sau aluminiu, care eliberează zi de zi metalele respective (nichel, crom, cositor, cadmiu, fier, plumb, mercur, zinc ş.a.) în mâncarea gătită pe care o ingerăm. Studiile noastre arată că aceste metale ating nivele maxime în tumorile maligne investigate! Prin urmare, recomandăm la curs folosirea oalelor de ceramică, de sticlă Jena sau de lut.

 

si elemente chelante precum alga marina Chlorella sau zeoliti sunt de luat in calcul

– Ce rol are stresul în bolile canceroase?
– Atât stresul oxidativ cât şi stresul psiho-social joacă un rol important în slăbirea capacităţilor sistemului imun. Pacienţii aflaţi sub un stres continuu acuză boli infecţioase recidivante şi prezintă un risc crescut pentru apariţia primelor focare tumorale. Dar şi polimorfismele genetice care avansează de la o generaţie la alta, în special cele privind enzimele de dezintoxicare, pot fi o cauză majoră pentru acumularea de substanţe toxice în corp, care la rândul lor pot induce stresul oxidativ mai sus amintit.

sa nu uitam ca stressul PRELUNGIT este SUPRESORUL IMUNITAR NR 1:

https://tratamenteanticancer.wordpress.com/2013/11/10/how-stress-causes-cancer-2/

 

Detalii şi soluţii terapeutice pentru aspectele discutate aici vor fi prezentate pe larg la cursurile de Oncologie Integrativă şi Detoxificare din 18.03.2017, respectiv 17.06.2017 din Bucureşti, sub egida UMF „Carol Davila”, tel: 0757.205.791.
Interviu realizat de Remus Florescu
De pe Adevarul.ro
Articol publicat in Revista Familia Ortodoxa nr. 98/Martie 2017
[1] Substanţe care pot provoca formarea anticorpilor şi sensibilizarea limfocitelor circulante. (n.red.)
[2] Mărirea acidităţii sângelui, datorită unor tulburări funcţionale în organism. (n.red.)

• Publicat de: Sanziana Busuioc
• 10 Apr 2017

Comentat /completat de fratele vostru – sa ne rugam sa fie bine!
Doamne ajuta!

p.s. :desigur, recomandarile de mai sus sunt referitor alimentatie(doar UNUL din pilonii unui protocol de cancer)

REPET ALTE LUCRURI IMPORTANTE :

FOARTE MULT CONTEAZA STAREA EMOTIONALA, FLORA INTESTINALA SANATOASA(reglementeaza si echilibrul hormonal si sistemul imunitar), ODIHNA ADECVATA NOAPTEA(care regleaza Melatonina si hormonii) si EXPUNEREA LA SOARE PENTRU IMUNITATE SI DACA INTARITI IMUNITATEA IN TIMP UTIL SCAPATI SI DE CANCER SI MULTE ALTE PROBLEME.

FACETI CE PUTETI, ALOCATI-VA RESURSELE LA CE E PRIORITAR  – in primul rand luati:

 -vitamina D3 15000 ui pe zi(INCLUSIV SI MAI ALES DIN MINIM 30 minute de stat la soare),

– Vitamina C  lipozomala  MINIM 6 grame pe zi(exemplu AURORA SAU LIV ON sau LIPOLIFE GOLD – cautati pe google) ,

– Melatonina 20-30 mg seara(INCLUSIV SOMN INAINTE DE ORA 22)

– miere cu ALOE ARBORESCENS si o ciuperca imunitara(https://www.hawlik-vitalpilze.de/),

– probiotice(inclusiv( zeama de ) muraturi de fermentatie naturala, NU in otet) si

– RECOMANDARILE DR BUDWIG  (inclusiv cele pentru cancere avansate  si casexie si NU uitati ficatul)  LA BAZA.

si clorura de cesiu puteti adauga functie de ce buget aveti

incercati de asemenea sa mergeti la Sfantul Maslu si sa va Spovediti si Impartasiti in mod regulat. 

primite cu cateva zile in urma de la unii dintre voi:

„Hristos a inviat!

Vineri am fost la Sfantul Maslu, iar apoi toata ziua mama s-a simtit mai in putere, a putut trebalui toata ziua prin gospodarie si nu a simtit nevoia sa se mai aseze in pat. Se simte mai intarita si mai intremata, parca a luat un pic in greutate. Si ieri a fost la fel, si astazi, multumim Domnului, am fost la biserica si a fost bine, chiar a putut sa-si reia locul la cor. Continuam asa cum ati recomandat si cu ajutorul lui Dumnezeu nadajduim sa fie bine.

Ma gandeam, credeti ca e necesar sa comand preventiv sulfat de hidrazina, pentru orice eventualitate?

Doamne ajuta!” Dorin

„O cheama Zinaida i-am facut Sfintu Maslu a doua zi de Paste si miercuri s-a trezit a zimbit era lucida credeam ca a trecut cu bine si a doua zi joi iarasi in starea de somn atunci n-a mincat vreo 2 zile apoi parca si-a revenit putin o hraneam cu lingura de vreo 4 ori pe zi minca pina vineri si de simbata nu mai maninca „

si daca vreti va dau exemple de oameni peste care a dat sau a TRECUT masina de la mine din oras care ajunsesera fie cu capul sfarmat(o colega de liceu) fie in scaun cu rotile( un politist) care s-au recuperat complet dupa Sfantul Maslu(sa ne intelegem bine:medicii nu au dat nici o sansa)

Mare este puterea Domnului!!

Suplimente, medicamente si ‘alicamente’ bazice impotriva cancerului: sinergia si cocktailuri pentru o strategia integrala

 

Traducere facuta de Nicoleta

 

Poate fi frustranta sarcina de a decide intre cantitatea mare de suplimente disponibile pe piata. La aceasta trebuie sa adaugam cantitatea mare de marci, prezentari si doze ale fiecareia, ceea ce face foarte complicat sa alegem suplimentele cele mai eficiente, doza cea mai potrivita cat si brand-urile cu cel mai bun raport calitate / pret. Inainte de a propune o lista de suplimente anticancer despre care cred ca indeplinesc parametrii de calitate si eficacitate, voi explica pe ce ma bazez pentru a le propune. Orice decizie trebuie sa provina dintr-o reflexie anterioara, o colectie de date aprofundate si o strategie elaborata plecand de la acestea. Acea alegere poate sa nu fie cea mai exacta si nici nu intentionez sa fie exhaustiva (i se pot adauga zeci de ierburi si multe alte suplimente), dar se bazeaza pe un rationament bine fundamentat. Desigur, as fi incantat de orice corecturi sau adaugari. 

1. Dieta alimentatia ANTIcancer este cea mai importanta

Suplimentele ANTIcancer nu ar trebui sa inlocuiasca dieta ANTIcancer, ci doar sa o sustina, furnizand alte molecule care au fost demonstrate a fi eficace in doze mari(care nu pot fi obtinuti in doze adecvate din dieta precum vitamina C IntraVenos(30-50 grame zilnic administrate in interval de 90 minute) sau vitamina C lipozomala, vitamina D3 (5000-20000, UI mai ales iarna),  melatonina (6-50 mg, seara) , suplimente imunitare (ciuperci medicinale (ABM, AHCC,reishi , maitake ,shiitake ,chaga, cordyceps, etc) , Transfer factor,  imunomodulatori precum 714 X, etc) , suplimente deparazitare, minerale puternic alcalinizante precum Clorura de cesiu (chiar si calciu si potasiu, seleniu,etc) , omega 3 in doze mari(asa cum include dieta dr Budwig , alte grasimi permise in cancer) si compusi care nu sunt de obicei gasiti intr-o dieta tipic occidentala sau nu pot fi procurati in dozele necesare pentru efect anticancerigen din alimente integrale. Pe cat posibil, o mare parte dintre suplimente ar trebui sa consiste din alimente, fie sub forma de extract, fie sub forma de uleiuri sau infuzii care sa asigure toate substantele nutritive esentiale ale alimentului sau ale plantei din care provin insa in doza mai mare si in cantitate mai mica. Chiar si in astfel de cazuri, nu ar trebui sa inlocuiasca produsele alimentare, ci sa o consolideze pe aceasta. De fapt, lista suplimentelor include, de asemenea, unele alimente care ar trebui sa fie consumate, cred ca, aproape in fiecare zi deoarece pot fi considerate medicamente autentice, prin urmare,  de aici numele sau de „alicamente”. In realitate, intr-o  dieta ANTIcancer, ar trebui sa nu fie introdus nici un aliment care sa nu poate fi numit asa in masura mai mare sau mai mica.

Desi s-au identificat molecule naturale de actiune antitumorala, trebuie sa luam in considerare faptul ca un produs alimentar contine, de asemenea, tot ceea ce „noi nu stim ca nu stim”, adica, mii de posibili compusi cu o eficacitate terapeutica datorita factorilor inca necunoscuti. Exceptiile sunt vitamine, minerale, antioxidanti si molecule specifice, care s-au dovedit a fi eficiente, fie in probe solide, fie datorita elementelor care demonstreza un potential beneficiu si absenta efectelor sale secundare.

2. Sinergia

Cel mai important cuvant atunci cand se planifica o strategie/protocol ANTIcancer este sinergie. Definitia grafica  a sinergiei este 1 + 1 > 2 , adica doua substante care, luate impreuna, furnizeaza o valoare terapeutica mai mare decat ar furniza-o individual. In timp ce masurile conventionale se aplica, in general, din una in una, datorita efectelor sale secundare, dieta si suplimente au avantajul de a fi instrumente terapeutice inofensive sau cu putine efecte secundare. Este adevarat ca deja se aplica linii terapeutice din 2 medicamente sau un medicament impreuna cu o doza de radiatie, dar asta e tot ce isi poate permite medicina alopata. Exista studii, desi inca off-label, care cerceteaza strategii similare cu cele propuse aici, dar cu medicamentele conventionale. Este vorba de un multidrog utilizat pentru alte boli si cu putine efecte secundare, aplicate concomitent cu dozele de chimioterapie metronomice (doze mici, dar zilnice) care „par” promitatoare. Cu toate acestea, foarte putini medici indraznesc sa le prescrie pacientilor lor (studiile sunt preliminare) si prefera sa se aplice tratamentul standard , previzibil si neproblematic ( pentru ei ). Principala critica impotriva strategiei de suplimente alimentare este ca „nu este dovedita”. Un argument inutil, din moment ce nimeni nu poate pretinde ca un bolnav sa stea cu mainile in san in timp ce i comunica ca nimic nu se mai poate face pentru el.

 Si pentru ca  trebuie sa asteptam mult  pana sa se faca aceasta probe, finantate de companii farmaceutice (cand ?!) , (de fapt, nu se va face niciodata daca paradigma actuala nu se schimba, pentru ca nimeni nu va investi banii necesari pentru a obtine date semnificative statistic pentru lucruri din care nu se paote obtine profit substatial), nu avem nici o alta alternativa decat sa ne bazam pe studii in vitro, pe animale sau in cel mai bun caz, pe un numar limitat de pacienti sau feedback pacienti (MARTURII). Desigur, in unele cazuri, dovezile sunt suficient de solide pentru a justifica studii pe scara larga. 

O alta critica folosita impotriva suplimentelor este ca studiile stiintifice care sunt bazate pe anumite doze terapeutice, sunt foarte greu de realizat cu prezentari comerciale obisnuite. In timp ce acest lucru este adevarat, in unele cazuri, este de asemenea adevarat ca:

• Apar prezentari comerciale cu doze din ce in ce mai mari
•  Sinergia, despre care am vorbit, permite ca doua suplimente, luate in doze relativ mici, sa se sustin reciproc si sa produca un efect considerabil sporit- exemple : piperina pontenteaza biodisponibilitatea cucurminei pina la 2000%; catechinele din ceaiul verde si curcumina din curcuma se sustin reciproc. Acelasi lucru este valabil si cu unele suplimente de biodisponibilitate scazuta, care cresc cu mai multe ordine de marime daca sunt consumate impreuna cu anumite alimente, care pot fi considerate de la sine ca medicamente. De exemplu, berberina si scortisoara. 

Ca de obicei, diferenta dintre o buna si o proasta decizie sunt rezultatul cunostiintelor si al informatiilor: cateva suplimente izolate, luate fara a urma nici o regula, in doze aleatorii sunt putin probabil sa aiba efect, si da,  sunt multi care il vor folosi ca argument de catre cei care neaga ca aceasta strategie ar pentru avea vreo eficacitate

3. Cocktail

Al doilea cuvant pe care trebuie sa il retinem este cocktail: trebuie sa profitam de efectele secundare minime sau inexistente pentru a suplimenta cu cat mai multe substante, de forma agresiva si sub forma de multe abordari biochimice, dar intotdeauna cu aceeasi focalizare: de a folosi ceea ce s-a dovedit a nu avea nici un efect secundar sau foarte putine si care ofera un avantaj, desi aparent „indirect”. 

In acest moment pot aparea intrebari rezonabile: Si cum stii ca nu exista interactiuni nedorite intre unele dintre aceste suplimente? De unde stii ca rezultatul final nu va fi mai rau? Raspunsul meu este: Nu stiu. Nu pretind a fi sigur de eficienta lor, dar nu voi sta fara sa fac nimic asteptand  sa fie testate toate combinatiile de molecule, pentru ca acea zi nu va veni niciodata.  Acest rezultat negativ prin combinarea multe suplimente este posibil, dar putin probabil pentru ca vorbim de lucruri naturale : ma concentrez pe studii care au demonstrat ca sinergia este benefica in anumite combinatii de molecule. Si, de asemenea, ma bazez pe experienta anterioara a persoanelor care au aplicat protocoale similare si au obtinut rezultate excelente. Este evident ca nu toate combinatiile posibile de substante pot fi fost testate, dar epidemiologia si istoria ne invata ca fiecare molecula prezenta in natura, a fost supusa unui test „dublu-orb”, care a durat mii de ani, in timpul careia observarea si bunul simt au declarat eficacitatea si siguranta lor relativa. Eu cred ca vorbesc cu oameni inteligenti. Oameni capabili sa ia o decizie cu responsabilitate, inclusiv cu deplina cunostinta,  care s-a informat in mod liber si care sunt capabili de a cantari echilibrul intre posibile riscuri si beneficii. Persoanele care isi asuma responsabilitatea cu privire la ceea ce se intampla in viata lor si decid sa ia fraiele vietii lor. Sarcina mea este de a informa si de a oferi uneltele pentru ca astfel de decizii sa poata fi facute cu mai multa incredere si responsabilitate. Este posibil, desigur, ca dozele sa fie prea mari sau sa apara interactiuni nedorite si inca necunoscute, dar aceste probleme sunt aproape intotdeauna usoare si trecatoare. 

4. Orice substanta eficienta trebuie luata in considerare

Este important sa nu avem prejudicii atunci cand se studiaza orice actiune sau molecula. Bunul simt ar trebui sa prevaleze intotdeauna si nu trebuie sa ne pierdem motivarea de ce facem acest lucru: sa construim o strategie de actiuni practice care sa conduca la remisie, fara sau cu minime efecte secundare. Din acest motiv, lista pe care o voi prezenta, va include unele medicamente conventionale, dar care au demostrat un amestec de eficacitate si tolerabilitate. Pentru ca nu sunt impotriva drogurilor/medicamentelor sintetice in sine, ci numai impotriva celor otravitoare si ineficiente. 

5. Bani

Stiu din experienta cat de scump poate fi un plan de suplimente. Daca adaugam achizitionarea de alimente ecologice, costul lunar poate fi destul de mare. Nu am nici o solutie pentru acest lucru, din pacate. Nici chiar acolo unde sistemul public de sanatate nu a fost jefuit, statul nu isi asuma aceste cheltuieli.  Incercati sa gasiti marci si formulari cu cel mai bun raport calitate / pret, dar chiar si asa, costul final va fi mare pentru un buzunar de mijloc. 

Lista de suplimente impotriva cancerului

Am pregatit o liste cu explicatii alegerii fiecarui supliment, deoarece consider esential sa clarific motivele deciziilor luate, precum si datele pe care ma bazez. Problema este ca documentul deja are 30 de pagini, ceea ce il face imposibil de gestionat iar eu as intarzia foarte mult in a-l termina, asa ca voi publicat doua liste de informatii sumare, insa voi dezvolta in viitor, in alte articole, toate motivele pentru care sfatuiesc fiecare supliment precum si studiile in care ma bazez.

In ceea ce priveste cele doua liste:

• Una este o lista de suplimente in care explic dozele, reguli de administrare si posibile marci propuse
• A doua este o lista de suplimente si medicamente pe care este bine sa le avem la indemana si pe care inca nu le luam, dar  care ar putea fi interesante pentru altii. Sugerez ca fiecare cititor sa investigheze pe cont propriu, daca considera util vreun medicament si sa il introduca in planul lui de tratament. 
• In aceste liste, voi prezenta terapiile pe care le consider cele mai promitatoare si sper ca pot dezvolta mai tarziu,  insa repet avertismentul: nu am, desigur, certitudinea ca aceasta combinatie este cea mai eficienta. Toata lumea ar trebui sa cantareasca ce-l poate ajuta cel mai bine. Nu am nimic de-a face cu niciunul dintre brandurile de suplimente pe care le sugerez. Am pus doar ca un exemplu ceea ce noi am ales si, cu toate ca, in unele cazuri, sunt destul de sigur ca am ales bine, in altele, cum ar fi extractul de ceai verde, nu am incredere ca calitatea acestuia coincide cu ceea ce producatorul spune si orice sugestie este bine venita. 

Clasificarea suplimentele

A- molecule naturale izolate sau extracte de alimente care fac parte din grupul pe care noi il intelegem ca suplimente.

B- Alicamente: produse alimentare care urmeaza sa fie luate intr-un mod similar medicamentelor insa in anumite doze minime pentru proprietatile sale benefice intrinseci si a capacitatii sale de a potenta alte masuri.

C- Medicamente disponibile, care au demonstrat un beneficiu in studii preliminare fara a produce efecte adverse sau cu efecte secundare usoare. In aceasta lista vor exista medicamente care pot fi obtinute printr-o achizitie on-line.

 D- Protocoale standard: propuse de companii private si / sau medici privati, indepartati de curentul oficial si care par a fi tratamente anticancer eficiente.

E- Medicamente care se pot obtine cu retete, cu relative putine efecte secundare si care pot fi prescrise numai de catre un medic.

 F- Molecule sintetice care inca nu sunt disponibile sau cu o disponibilitatea foarte dificila, dar cu o foarte mare eficacitate potentiala. 

Lista 1: suplimentele principale

Naltrexona in doze mici sau LDN (low dose naltrexone)

Tip: C

Brand: BuyLDN (a se vedea lista tarilor in care se trimite)

Doza: 4,5 mg / zi: 1 capsula pe zi, inainte de culcare.

Posibil efect: stimulare imunitatii. 

Comentarii: Primul supliment in aceasta lista nu este o molecula naturala, ci sintetica: un medicament in doze mari, adesea prescrise impotriva alcoolismului. In doze mici, nu are efecte secundare si s-a dovedit cu pacientii reali, ca are o eficacitatea antitumorala extraordinara (numai in doze mici. Cresterea dozei se va anula efectele pozitive). Este un obligatoriu in orice strategie de suplimentarea impotriva cancerului un supliment imunitar.

Nota mea: Alte suplimente imunitare NATURALE:ciuperci medicinale (ABM, AHCC,reishi , maitake ,shiitake ,chaga, cordyceps, etc), MGN3, BIOBRAN, BetaGlucani, Transfer factor,  imunomodulatori precum 714 X, etc.

 

Coriolus versicolor (PSP50)

Tip: A

Doza: 3 capsule pe zi, 1 mic dejun – 1 pranz – 1 cina

Brand: Oriveda

Posibile mecanisme de actiune: Folosit in tratamentul conventional in tari precum Japonia, este un excelent immunopotentiator.

Nota mea: alt brand bun de ciuperci(de la bolnavi stiut): https://www.hawlik-vitalpilze.de/

Nu uitati ca imunitatea tine si de starea emotionala a bolnavului, sanatatea florei intestinale, echilibrul hormonal, odihna adecvata noaptea, expunerea la soare ziua, LIPSA DE PARAZITI A ORGANELOR(suplimente deparazitare)  si de alti parametrii.

 

Ulei de peste sau de krill 

Tip: A

Brand: Enerzona

Doza: minim de 6 grame pe zi. Acesta doza poate fi mult mai mare in cazul in care o analiza AA de sange / EPA determina aceasta nevoie.

Doza: 2 linguri la micul dejun.

Posibile mecanisme de actiune: posibila actiune antitumorala, antiinflamatorie.

Comentarii: Unul dintre cele mai studiate suplemente si a carei eficienta a fost demonstrata.

Nota mea: NEAPARAT de inclus 6-8 linguri ulei de in ecologic zilnic (asa cum include dieta dr Budwig ) si  alte grasimi permise in cancer(ulei de canepa, de canabis si alte uleirui permise; nu uitati minim 2-3 punmi de nucifere samburi si seminte GERMINATE zilnic(exceptie caju si arahide)

 

Ulei de nuca de cocos

Tip: B

Brand: Coconoil

Doza: 3-6 lingurite pe zi, 1-2 lingurite la fiecare masa

Posibile mecanisme de actiune: multiple (antitumoral, facilitator al cetoza, etc.)

Nota mea: poate fi de asemenea folosite pentru gatit, in plus fata de doza prescrisa aici si suplimentat cu ulei de masline virgin.

 

Vitamina D3

Tipul: A

Brand: Puritan’s Pride

Doza: 10.000UI / zi la inceput. Se reduce la 2.000-5.000UI / zi cand se ajunge la un nivel de sange intre 80 si 100 ng / ml

Doze: 2 capsule la micul dejun

Posibile mecanisme de actiune: multiple

Comentarii: unul dintre cele mai studiate suplimente si a carei eficienta a fost demonstrata

vitamina D3 (5000-20000, UI mai ales iarna;stat la saore zilnic minim 30 minute NECESAR)

 

Melatonina

Tip: A

Brand: Pure Encapsulations

Doza: 20 mg / zi, 1 cu 1 ora inainte de culcare

Posibile mecanisme de actiune: modularea sistemului imunitar si al inflamatie.

Comentarii: hormon foarte studiat, de a carei eficacitate exista dovezi foarte puternice.

Nota mea: melatonina (6-50 mg, seara)

 

Vitamina K2

Tip : A

Brand: Now Foods

Doza: este legata de consumul de vitamina D3. Aproximativ 100 de mcg / zi pentru fiecare 1000 UI de D3 consumat. In cazul in care se iau alte suplimente si alimente din aceasta lista care sunt surse abundente de K2, se poate lua doar 300mcg / zi.

Se iau 3 capsule pe zi. 1 mic dejun, 1 pranz, 1 cina, sau  toate 3, la un moment dat sau doar 1 pe zi in cazul in care capsula este de  300mcg.

Posibile mecanisme de actiune: multiple.

Comentarii: se prezinta ca o vitamina cu o actiune la fel de importanta ca si D3 si care permite sa se moduleze efectele potentiale asociate cu consumul excesiv de D3.

Nota mea: Atentie ca multe alimente verzi si lactate din dieta Budwig contin deja vitamina K si excesul de vitamina K NU este recomandat

 

Bisglicinatul de magneziu (inlocuieste calciu, magneziu si zinc)

Tip: A

Doza: 1500 mg pe zi, 3 comprimate: 1 mic dejun – 1 pranz – 1 cina

Brand: Bulk Powders

Posibile mecanisme de actiune: Magneziul este implicat intr-un numar foarte mare de reactii in organism si lucreaza sinergic cu vitaminele D3 si K2.

 

Glicina

Tip: A

Doza: 15-20g pe z,  3 comprimate: 1 mic dejun – 1 pranz – 1 cina

Brand: Bulk Powders

Posibile mecanisme de actiune: S-a dovedit a fi un aminoacid antiangiogenetic care permite sa se obtina glutation din metionina.

 

Lizina

Tip: A

Doza: 30g pe zi, 1 mic dejun – 1 pranz – 1 cina

Posibile mecanisme de actiune: Este unul dintre cei doi aminoacizi cetogeni care se poate transforma in cetone, nu in glucoza, cu ajutorul caruia se intra mai bine in cetoza. In plus, si-a dovedit capacitatea de a creste imunitatea. 

Nota mea: Prolina si ceai verde extract (EGCG) sunt parte din formula Dr Rath din carte si e bine sa luati aceasta formula a Dr Matias Rath

 

 Extract de ceai verde

Tip: A

Brand: Puritan’s Pride

Doza: trebuie asigurat un minim intre 1500 si 3000 mg pe zi (in aceasta prezentare, 6 capsule pe zi, 2 mic dejun – 2 masa – 2 cina ).

Posibile mecanisme de actiune: face dificila glutaminoliza tumorala si creste eficienta insulinei. 

Comentarii: Nu sunt sigur ca am ales bine brand-ul. Este marca cu cea ai mare cantitate de catechine pe capsula pe care am gasit-o, dar culoarea nu arata asa cum ar trebui sa aiba un extract de calitate si pretul sau este, de asemenea, suspect de scazut. As aprecia orice sugestii.

 

Curcuma

Tip: A

Brand: Theracurmin

Doza: In aceasta prezentare, 1200mg, 2 capsule pe zi, 1 mic dejun – 1 cina

Mecanisme de actiune posibile: multiple (anti-inflamatoare, directe anti-tumorale)

Comentarii: Multiple studii sustin eficacitatea sa, desi biodisponibilitatea curcuminei este foarte scazuta. Dintre toate observatiile de piata (lipozomi, nanoparticule, etc.), cu diferite solutii pentru a creste o astfel de utilizare a metabolitilor, Theracurmin pare a fi cel mai puternic, dar din aprilie 2015, a fost introdus pe piata Novasolcurcumin care sustine ca a crescut biodisponibilitatea. 

Are o puternica actiune sinergica cu ulei de peste si resveratrol, printre altele. Creste efectul sau daca se consuma in paralel cu piperul negru pina la 2000%(cucurmin 95)

 

Scortisoara 

Tip: B

Doza: aproximativ 3 grame pe zi (unele persoane pot experimenta o iritatie gastrica atunci cand este luata in exces si trebuie redusa doza atunci cand apare acest disconfort. In unele studii, scortisoara este considerata un protector al stomacului). Se ia ¼ lingurita la fiecare masa si inainte de alte suplemente.

Posibile mecanisme de actiune: impiedica sa fie utilizat zaharul, limiteza varfurile glucozei, limiteaza autofagia tumorala.

Comentarii: imbunatateste absortia berberinei. Este un antimalaric si de asemenea, un antitumoral.

 

Piperul negru

Tip: B

Doza: aproximativ 3 grame pe zi (unele persoane pot experimenta iritatie gastrica atunci cand sunt luate in exces si trebuie redusa doza in cazul in care se produce acest disconfort). Se ia ¼ lingurita cu fiecare masa si inainte de celelalte suplemente.

Posibile mecanisme de actiune: antitumorala directa.

Comentarii: Piperul negru este un cunoscut intensificator de mai multe medicamente si molecule. De exemplu, creste in mai mult de 1000 de ori absorbtia curcumei.

Nota mea: Ghimbir si alte condimente anticancerigene de asemenea , in special integrate in  tratamente cu miere 

Nu uitati de miere si Aloe Arborescens ,excelent pentru imunitate

Resveratrol

Tip: A

Brand: Terraternal

Doza: In aceasta prezentare, 150-300mg / zi, echivalentul a 200 de ori o doza orala de resveratrol, 1 sau 2 capsule pe zi

Posibile mecanisme de actiune: face dificila glicoliza tumorala si autofagia tumorala

Comentarii : probabil cel mai scump supliment, insa unul dintre cele mai studiate, care blocheaza mecanisme de baza ale unei tumori si a carei eficienta este dincolo de orice indoiala, cu conditia ca formulele folosite sa fie cele pe care organismul le poate utiliza. 

Are, probabil, o buna absorbtie insa nu este folosit 100% (desi exista un studiu care sugereaza ca acest lucru nu este asa si ca utilizarea este mult mai mare decat se credea anterior).

Acest supliment se administreaza sublingual pentru a fie metabolizat prin membranele mucoase ale cavitatii bucale. In acest mod creste foarte mult biodisponibilitatea.  Din acesta este motiv este atat de scump. 

Asa cum se intampla si cu curcumina, creste si mai mult disponibilitatea resveratrolului prin consumul de piper negru. Trebuie sa folosim formule care utilizeaza Trans-resveratrol si nu Cis-resveratrol. 

resveratrol + pterostilben este mai efecient decat resveratrol trans

Nota mea: recomand consumul de alimente bogate in resveratrol, precum struguri negrii NUMAI pe stomacul gol

 

Vitamina B6, B12 si B9 (acid folic)

Tip:A

Brand: Vitamina B6, Vitamina B12 si Folati

Dozare: 100-200 mg de B6, 800-1600 micrograme de folati, de la 1000 la 2000 de micrograme de vitamina B12 pe zi, pana cand o analiza  a homocisteinei sangelui arata niveluri mai mici la 6 micromoli / l. 

Odata atinse aceste nivele, continuati cu 100 mg de B6, 800 mcg de folati si 1000 micrograme de vitamina B12 pe zi: 1 sau 2 tablete pe zi.

Posibile mecanisme de actiune: favorizeaza metilarea si ulterioara sinteza a antioxidanti endogeni glutation si SAM. Studiile sugereaza ca reduc expansiunea si agresivitatea celulelor tumorale, singure sau ca adjuvant la chimioterapiei.

Comentarii: cele 3 vitamine sunt implicate in procese similare, prin urmare, ele sunt grupate intr-o singura achizitie. A fost foarte studiat relatia lor cu homocisteina si cu toate bolile cronice, inclusiv cancerul.

Este important sa fugim de formele sintetice  oxidate ale vitaminelor si sa folosim pe cele biologic active (piridoxal-5-fosfat pentru vitamina B6, methylcobalamin pentru vitamina B12 desi exista si alte modalitati de valide -L-5-methylenetetrahydrofolate pentru B9). In cazul folatilor, daca trebuie utilizati pentru perioade lungi de timp, este indicata forma oxidat a vitaminei B9 deoarece acidul folic poate conduce la aparitia de mai multe probleme decat beneficii.

 

Probiotice (mai ales in si dupa chimio terapie)

Tip: A

Brand: Nutrition Now

Doza: pentru acea prezentare, 3 comprimate pe zi: dimineata si la pranz .

Posibile mecanisme de actiune: intarirea sistemului imunitar.

Comentariile: este indicat sa se combine suplimentarea cu alimentele integrale care fermenteaza. Sunt tot mai multe studii care sustin ca exista o stransa legatura intre sistemul imunitar si microbiome intestinale sanatoase precum si intre rezistenta sistemului imunitar si cancerul.

In plus, probiotice ar putea rezolva problemele proastei absortii nutritive ale unor pacienti, astfel incat pot fi considerate ca stimulatori puternici ale altor suplimente. Trebuie alese branduri cu mai mult de 10 de tulpini diferite de bacterii, mai mult de 10.000 de milioane de organisme vii, atunci cand sunt ingerate si, de asemenea, care contin prebiotice, cum ar fi inulina. 

 

Probiotice

Tip: B

Doza: mancati, daca este posibil, in fiecare zi, cantitati rezonabile de chefir, varza acra (sau alte legume fermentate NATURAL si NU in otet) si muraturi naturale(fermentatie , nu in otet).dieta Budwig  este excelenta si aici.

Posibile mecanisme de actiune: intarirea sistemului imunitar.

Observatii: Este un aspect atat de important  incat consider ca este necesar ca actiunea probiotica trebuie sa se „prescrie” pe doua fronturi: cu suplimente si alimente.

 

Enzime proteolitice si pancreatice

Tip: A

Brand: Zymactive (sau Wobenzym)

Doza: In aceasta prezentare, un minim de 3 capsule pe zi, inainte de micul dejun si intre mese, pentru a evita pe cat posibil, sa fi utilizate de stomac in timpul digestiei si pentru a ajunge in fluxul sanguin.

Posibile mecanisme de actiune: neclare.  Posibil sa dizolve membranele tumorale, stimuleaza sistemul imunitar si impiedica angiogeneza.

Comentariile: s-au efectuat teste pe grupuri reduse de pacientii cu adenocarcinoame pancreatice, a caror timp de supravietuire s-a triplat prin utilizarea enzimelor la doze mari (adica minim 60 pilule pe zi, 10 cu o ora inainte de fiecare masa si 10 cu mese) 

 

Berberina

Tip: A

Brand: LeanerLiving

Doza: 1500-2000mg pe zi,  3 comprimate pe zi, 1 la scurt timp inainte de fiecare masa. Indicat cu scortisoara (explicatia mai jos).

Mecanisme posibile de actiune: creste eficienta insulinei, reduce inflamatia, reduce activitatea enzimelor tumorale, are o directa eficacitate antiproliferativa.

Comentarii: un alt indispensabil. Berberina are o biodisponibilitate scazuta, dar creste odata cu utilizarea concomitenta a scortisoarei. 

 

Chlorella (Nota mea: Spirulina de asemenea, AFA pentru ficat; Kombu si Kelp si cele brune sunt si mai bune)

Tip: A

Brand:  Source Naturals

Doza: intre 2 si 5 grame pe zi (poate provoca diaree la inceputul tratamentului pentru ca este un puternic detoxifiant  de impuritati si metale grele. In cazul in care se intampla acest lucru, se reduce doza initiala pana dispare problema). Se iau: 4 la 9 comprimate pe zi, 2-3 inainte de micul dejun si 0-3 intre mese. Se poate cumpara si sub forma de pulbere.

Posibile mecanisme de actiune: potentiator imunitar, antialergic (histamina este un promotor puternic al cancerului), efect probiotic. 

Comentarii: suplimentul ar trebui sa provina din agricultura ecologica si „“Broken Cell  Wall” sau organismul o nu poate folosi. Sinergie cu curcuma si spirulina.

 

Spirulina

Tip: A

Brand: Solgar

Doza: intre 2 si 5 grame pe zi, de la 4 la 9 sau mai multe comprimate pe zi, 2-3 inainte de micul dejun si 0-3 intre mese. Se poate cumpara si sub forma de pulbere.

Posibile mecanisme de actiune: excelent potentator al celulelor NK (natural killer) ale sistemului imunitar, efect probiotic.

Comentarii: Este o sursa foarte mare de beta-caroten, precursor ale vitaminei A. In plus, contine doze mari de vitamina B12.

 

 R-alfa-acid lipoic

Tip: A

Brand: AOR

Doza: 600-1200mg / zi, (pentru aceasta marca, 2-4 pe zi), ultima doza mica se ia impreuna cu naltrexona (LDN) si melatonina.

Posibile mecanisme de actiune: blocarea glicolizei, similar cu dicloracetat de sodiu.

Comentarii: are o puternica sinergie cu LDN si Hydroxycitrate (extract din Garcinia Cambogia), s-a fost folosit in unele teste, cu pacientii bolnavi de cancer avansat si s-a obtinut o crestere in mod spectacular a mediei de supravietuire si, in unele cazuri, remisii complete si durabile. Este convenabil sa se utilizeze forma naturala a acidului R-lipoic care este mai bine utilizata de catre organism.

 

Hydroxycitrate (extract din Garcinia Cambogia)

Tip: A

Brand: Pure Body Naturals

Doza:. 3 grame / zi, (aceasta marca 1-2 capsule inainte de fiecare masa).

Posibile mecanisme de actiune: nu sunt sigur inca. 

Comentarii: asa cum am spus, are o puternica sinergie cu acid alfa-lipoic. A a fost testat pe unii pacienti cu cancer avansat, cu rezultate foarte bune. 

 

Extract de usturoi invechit

Tip: A

Brand: Kyolic

Doza: 2400-3600mg / zi (pentru aceasta marca, 4-6 capsule pe zi in timpul meselor)

Posibile mecanisme de actiune: anti-inflamator, amplificator al sistemului imunitar, efect antiproliferativ direct.

Comentarii : are o puternica sinergie cu LDN. Usturoiul invechit are un procentaj mai mari de diferite molecule decat usturoi normal, dar nu are alicina(si alicina e foarte importanta). De aceea, este recomandabil sa se consume cantitati mari de usturoi, ulei de usturoi si / sau usturoi incoltit in fiecare zi. 

 

Vitamina C in doze mari (Nota mea: vitamina C IntraVenos(30-50 grame zilnic administrate in interval de 90 minute) sau liposomala)

Tip: A

Doza: Depinde de starea pacientului si de tolerabilitatea (excesul cauzeaza diaree si trebuie redusa doza). Daca se administreaza intravenos, se incepe cu o doza intre 30 si 50 de grame pe zi, dar se poate ajunge la 2g / kg greutate corporala. Daca se ia lipozomi de inalta calitate, se spune ca aproximativ 6 grame de vitamina C (continuti in aproximativ 30 de mililitri de lipozomi), este echivalentul a 50 de grame injectat, dar ar fi mult de discutat pentru ca ambele tipuri de administrare a vitaminei C au mecanisme diferite, astfel incat este dificil sa se stabileasca echivalente. Se pot lua 3-6 doze pe zi de lipozomi de inalta calitate, pentru a ajunge la 30-50 ml pe zi (6-10 grame de vitamina C). Este important sa fiti constanti si sa distribuiti pe tot parcursul zilei.

Posibile mecanisme de actiune: citostatic direct prin generarea peroxidului de hidrogen.

Comentarii: aplicarea sa a dat rezultate aparent extraordinare in clinici medicale din intreaga lume care ofera tratamente complementare, in special cu DHA (acid docosahexaenoic, un omega 3). Problema este ca, datorita absorbtiei intestinale inferioare ale formelor orale conventionale, pana in prezent a fost aplicata doar intravenos, care cere ajutor medical si impiedica aplicarea autonoma de catre pacient. In prezent au fost testate formule orale lipozomale (care pot fi fabricate la domiciliu, folosind un agent de curatare cu ultrasunete, si in conformitate cu doua posibile „retete”), cu o capacitate de absorbtie mult mai mare. 

 

Lamaie

Tip: B

Doza: cel putin doua lamai organice pe zi, suc sau apa si stevie/miere si bicarbonat sodiu. Se poate bea oricand insa intotdeauna una in dimineata cu micul dejun

Posibile mecanisme de actiune: antitumoral direct, limiteaza varfurile glucozei

Comentarii: este recomandabil sa se foloseasca din agricultura ecologica. Se poate utiliza, de asemenea si coaja rasa pentru gatit.

 

Lista 2: suplimentele complementare

Sunt suplimentele care ar putea face parte din orice strategii de suplimentarea impotriva cancerului. Pentru aceste nu am inca referinte de marci.  De asemenea, sunt incluse aici si medicamentele care au nevoie de retete prescrise si protocoale farmacologice non-standard. 

 

Maitake MD

Alt extract de ciuperci, in acest caz Maitake, cu efecte similare. 

 

Extract de struguri

Tip: A

Multe studii in vitro si pe soareci confirma eficacitatea sa ca potential supresor tumoral. 

repet : includeti struguri negri pe stomacul gol sau in posturi ca cel Brandt dar nu mai mult de 3 zile

 

Quercetina

Tip: A

Si mai multe studii au confirmat eficacitatea sa antitumoral si prezinta sinergie cu ceai verde si, mai presus de toate, poate fi folosit ca un mijloc de crestere a nivelurilor exploatabile ale resveratrolului. 

 

Coenzima Q10 (Nota mea: 400-1200 mg pe zi)

Tip: A

Un cofactor prezent in membranele interioare ale mitocondriilor, ale caror niveluri scazute sunt asociate cu prezenta a numeroase boli cronice.

 

Boswellia

Tip: A

Un efect citotoxic direct, anti-inflamatorii si un bun agent anti-edem in tumorile cerebrale.

 

MCP (modified citrus pectin)

Tip: A

Este vorba de pectina din citrice modificata pentru ca organismului sa o poata absorbi. Unele studii recunoaste eficacitatea acesteia pentru a preveni sau pentru a impiedica metastaza, reducand agresivitatea unor forme de cancer. 

 

Extract de crucifere

Tip: A

Plantele din familia varzei contin molecule studiate pe larg ca potent antitumoral cu o citotoxicitate directa iar studii recente sugereaza ca poate reduce nivelul proteinei CXCR4 implicate in fenomenele de metastaze.

Este indicat sa se consume de cat mai multe ori pe saptamana gulii, varza de Bruxelles, varza, conopida, broccoli insa nu ar fie rau sa se ia si un bun extract din aceste plante.

 

Silimarina(din ciulinul laptelui)

Tip: A

Protector si detoxifiant hepatic puternic, cu multiple actiuni anti-tumorale directe, anti-inflamatorii si anti-angiogenice. Un bun adjuvant in cazul in care se ia in timpul chimioterapiei pentru a spori eficacitatea acesteia, protejand in acelasi timp, intr-o anumita masura, organismul sanatos (in special ficatul) de reactii adverse.

 

Canabis(inclusiv ulei)

Tip:A

Unul dintre cei mai promitatori compusi, supus cercetarilor in intreaga lume. Exista un zgomot de fond foarte important, cu oameni care pretind ca s-au vindecat utilizand canabinoidele. Este o veste plina de speranta insa trebuie sa fie luata cu precautie. 

 

Asthaxantin

Tip:A

Un carotenoid natural intalnit ca pigment in animale carora le confera culoarea rosie caracteristica: microalge, creveti, somon, unele pasari. 

Este considerat cel mai puternic antioxidant exogen si sunt suspicii ca ar modula inflamatia si sistemul imunitar.

 

Infuziile, sucuri si extracte de diferite plante

Tipuri: B

S-ar putea dedica mai multe articole care sa prezinte nenumaratele plante care au aratat un grad mai mare sau mai mic de eficacitate antitumorala : Artesimin, Astragalus, cimbru, Graviola(NU se ia cu dieta Budwig), aloe vera si mai ale ARBORESCENS, papadie, vasc etc.

 

DCA (dicloracetat de sodiu)

Tip: C

Va fi tratat pe larg intr-un viitor articol.

 

GC-MAA

Tip: D

Este un activator de macrofage, celule ale sistemului imunitar innascut, care actioneaza, in ceea ce priveste cancerul, ca un fel de Dr.Jeckyl si Mr. Hyde.

 In cazul in care datele privind eficacitatea acestuia sunt adevarate, am putea vorbi despre un avans foarte important pentru ca macrofagele sunt excelente dr Jekyll, aliati ai corpului sanatos. Aceasta informatie ne avertizeaza cu privire la avantajele de a activa macrofagele cu alimentele naturale, cum ar fi laptele fermentat (de ex. chefir).

 

Clorochina

Tip: E

Un medicament folosit in mod traditional impotriva malariei (cauzata de un parazit), care blocheaza autofagia tumorala si dificulteaza metastazele. In colaborare cu chimioterapie, dar, de asemenea, si cu alte masuri naturale, la soareci a demonstrat o mare capacitate sinergica. 

 

Plerixaforul

Tip: E

Un medicament imunostimulator, cu efecte secundare reduse, utilizate pentru a consolida imunitatea in transplanturi de maduva osoasa in pacientii cu leucemie, care dovedeste (din nou), rolul decisiv al sistemului imunitar in controlul cancerului. 

Este unul dintre mecanismele de actiune care pare sa blocheze proteina CXCR4, vitala pentru recurenta si metastaze. In acelasi timp, este unul dintre cel mai util medicament atunci cand se combina cu chimioterapia sau cu alte strategii mai putin agresive propuse aici.

 

3-brompiruvat

Tip: F

Probabil astazi, molecula cea mai promitatoare cu o gama de actiune mai larga si puternica, care ar putea revolutiona tratamentul cancerului, mai ales daca sunt aplicate cu amplificatori ai sistemului imunitar precum GC -Maf

Nota mea: https://tratamenteanticancer.wordpress.com/2015/08/10/nagalaza-testare-sange-detectie-si-monitorizare-tratament-cancer-si-alte-boli-asociate-cu-deficiente-ale-sistemului-imunitarautism-boli-virale-etc/ si terapiile adjuvante, cum ar fi dieta si LDN.

Este un puternic blocant al glicolizei si a demonstrat rezultate spectaculoase in soareci si in unii pacient stadiu IV care au reusit o remisie completa. 

 

2-deoxyglucose

Tip: F

Este un analog al glucozei, folosit pentru a marca tumorilor in timpul testelor de tomografie cu emisie de pozitroni (PET), care a demonstrat o eficacitate antitumorala prin blocarea drumului glucozei catre tumoare.

 

Fenilbutirat

Tip: F

Blocheaza glutaminoliza.

 

Concluzii

In definitiv, o strategie integrala care combina dieta cetonica cu suplemente ale carei obiective sunt:

• Asigurarea unui minim de glucoza, cu un optim de vitamine, minerale si fitochimicalele
• Reducerea absorbtiei de zahar si mentinerea nivelurilor scazute ale glucozei din sange
• Cresterea metabolismului grasimilor sanatoase si permise in cancer si a dozelor acestorA(PENTRU ENERGIE SI alte beneficii antitumorale ) si producerea corpilor cetonici
• cresterea PHului celulelor de cancer peste 7,5 (exemplu clorura de cesiu)
• cresterea alcalinitatii in general pentru a impiedica raspandirea rapida a cancerului
• detoxifiere (inclusiv cu zeoliti si clisme CAFEA zilnice)
• Impiedicarea glicolizei
• Reducerea la minimum a contributiei metioninei
• Impiedicarea glutaminolizei
• Reducerea nivelului de insulina
• Reducerea nivelului de homocisteina
• Reducerea autofagiei tumorale
• Reduce inflamatia
• Consolidarea sistemului imunitar (celule NK, macrofage si limfocite)
• Reducerea angiogenezei
• Folosirea de numeroase citotoxice combinate

 

Articol original pe cancerintegral.com. modificat putin de mine insa e interesant cum oameni din tari diferite, ajung la concluzii similare.

 

Doamne ajuta!

ULEIUERI si GRASIMI PERMISE IN CANCER,

Acest articol este un extras din cartea Dr italian Giuseppe Nacci , M.D. , „1000 plante impotriva cancerului FARA chimio-terapie”,una din cele mai solid argumentate carti de profil , 650 pagini in care se citeaza  2,050 publicatii stintifice officiale si 2,100 referinte bibliografice. Extrasul se refera la ULEIUERI si GRASIMI PERMISE IN CANCER, care ar trebui sa fie SURSA PRINCIPALA DE ENERGIE ( de preferat in locul carbohidratilor)

CLICK AICI pentru  limba ROmana(traducerea “aproximativa” utilizand google translate).

Chapter 1.c: FATS and OILS (“Fatty acids”)
Looking at Table 1 (Chapter 1.a) you will notice that oils are extremely rich as a source of energy
(900 Kilocalories per 100 grams olive oil, sunflower oil, flax oil, grape seed oil, corn oil, etc).

Fats are very rich in energy as well.
Chemically speaking, both can be considered to be “fatty acids”.

There are “essential” oils and fats that contain vitamins (vitamin E, vitamin F, etc…). They are called “essential” because our body is not able to synthesize them.
However, in the modern world we talk a lot about severe health problems linked to a diet rich in fats and oils. Actually, the problem needs to be further explained.
Chemically speaking, fats and oils contain three types of “fatty acids”:
Saturated fatty acids (dangerous for health);
monounsaturated fatty acids (not dangerous for health, can contain vitamins);
polyunsaturated fatty acids (not dangerous for health, can contain vitamins).
Saturated fats, or “bad” fats, are present in most animal fats, in margarine and in fats used in pastry shops.

GM food has been recently suspected to contain them as well (1207).

Saturated fatty acids, or “bad” fats, cause very severe alterations to cell membranes. They replace vitamin F (“good” fatty acids), and cause severe forms of cell wall impermeability for many
substances like glucose (that might cause Type 2 Diabetes), apoptotic vitamins (that might cause cancer and tumours in general), and other substances that are vital for cells, such as vitamin C (that
might cause heart attack, strokes etc).
On the contrary, unsaturated fats (“good fats”) are made of cis-cis fatty acids, that are typically contained in cold-worked vegetable oils.
Unsaturated fats (vegetable oils) are present in many plant seeds (SEE Table 2), and in some animals, such as some fat fish that live in cold waters (salmons, herrings – caution to heavy metals contamination).
Fatty acids are vital for muscle cells to produce energy for them during physical activity and to relax them (1208).
Furthermore, “good” fatty acids control blood coagulation (1209).
They also influence the release of CCK, a hormone that tells our brain that we ate enough and that we should stop eating (1210).
They contribute to maintaining conduction speed in motor and sensory nerves as well (1211).
They can keep our skin healthy (1212).
They reduce high blood pressure (1213).
They reduce lung cancer (Pardini R.S.: Nutritional Intervention with Omega-3 Fatty Acids in a case of Malignant
Fibrous Histiocytoma of the Lungs, Nutrition and Cancer 2005, 52 (2) , pp.: 121-129
It is useful to give a bit more detail about the vitamins contained in these oils.

Alpha-Lipoic Acid
Alpha-lipoic acid is an essential fatty acid that contains organic sulphur (as it is organic, that is, linked to biological molecules, it is not toxic). It directly helps making brain, muscle and skeleton
energy available during physical activity (1208), controlling diabetes as well (1214).

Alpha-Linolenic Acid
Alpha-linolenic acid is a cis polyunsaturated fatty acid present in cold-pressed flax seed oil. It is transformed into EPA and DHA (Omega 3 fats), that are difficult to find in food.

Table 2: percentage of vitamins contained in oils

Conclusions are:

Flax Seeds Excellent omega 3
Pumpkin Seeds Excellent for vitamin E

SoyBean (NO GM) Good but Avoid soy

Sunflower(NO GM) Excellent but AVOID Polyunsaturated oils (e.g. corn oils ) as they promote the growth of small blood vessels (by promoting the production of “bad” prostaglandins). This is bad for cancer because it allows tumors to grow new blood vessels. However, these small blood vessels are good for preventing strokes. Thus, ONLY WHILE  on a cancer diet avoid polyunsaturated oils.
Walnuts(NO GM) Good 
Rice (NO GM) Good if organic, coldworked
Safflower Good
Grape Seed Good
Sweetcorn (NO GM) Inadvisable for Multiple Sclerosis, also AVOID in cancer
Extravirgin Olive Excellent for vitamin E! moderation
Sesame Good
Rapeseed (NO GM) Good if does not contain  erucic acid
Almonds Advisable! (whole RAW almonds are also great source of protein an vitamin B17)

Peanuts (NO GM) Inadvisable
Palm  Negative 

Palmist Negative

Coconut  Negative(whole raw young coconut allowed in cancer)

Other oils that are great in cancer :

Seabuckthorn oil.  cannabis oil hemp oil, avocado oil(whole avocado) and hazelnuts (whole raw hazelnuts – alune de PADURE).

Other sources of good, healthy fats are various fruit seeds( melons, grapes, berries, prunes, nectarines, apricots(vitamin b17), apples, pears, and others).

Note. If you are not a very weak person that should immediately go on a diet rich in omega3, water-soluble fats(see my book for details) , it is advisable you eat whole RAW foods rather than oils extracted from them.

Best of health,

Cristian

P.S.: Dozele, ca in orice lucru destinat sa combata  cancerul(o conditie severa) sunt RIDICATE: MINIM 6-8 linguri ulei omega 3 hidrosolubil zilnic(NON GMO, atentie inclusiv la in), minim un Pumn de nuci sau migdale zilnic, 40-50 samburi caise zilnic(care NU sunt toxici in aceste doze a se vedea si aici click si aici click si in cartea versiunea in engleza),etc.

Nu mai spun ca unii dintre dvs inca se infometeaza cu Breuss. MARE RESEALA!

 

Scientific Studies Database PAGE 2

CLICK AICI pt. limba ROmana

Vitamins in alphabetical order

Vitamin A

There is an extensive bibliography on the anti-tumoral action of Vitamin A.

In particular the combined use of Retinoids in the proper proportions (beta-Carotene: retinol = 4 : 1), establishes a synergism which is higher than the sum of the single components.

Studies on Vitamin A and cancer (MANY STUDIES -see NOTES below)

science.naturalnews.com/V/Vitamin_A_and_cancer.html

NOTE 1:

The link above contains multiple studies and the same do many links below( I will try to indicate by ”MANY STUDIES”)

Vitamin A, retinoids and carotenoids as chemopreventive agents for prostate cancer.
2004 The Journal of urology

An international evaluation of the cancer preventive potential of vitamin A.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of dietary vitamin A, carotenoids, and other antioxidants with the risk of ovarian cancer.

2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of vitamin A, vitamin C and zinc with laryngeal cancer.
2003 Indian journal of cancer

Vitamin A, retinol, and carotenoids and the risk of gastric cancer: a prospective cohort study.
2007 The American journal of clinical nutrition

The effect of Vitamin A status in children treated for cancer.
2008 Pediatric hematology and oncology

Oxidant, Vitamin A and angiogenic markers in laryngeal cancer patients.
2003 The Journal of laryngology and otology

The role of antioxidants and Vitamin A in ovarian cancer: results from the Women’s Health Initiative.
2008 Nutrition and cancer

Can Vitamin A modify the activity of docetaxel in MCF-7 breast cancer cells?
2007 Folia histochemica et cytobiologica / Polish Academy of Sciences, Polish Histochemical and Cytochemical Society

Does Vitamin A prevent high-dose-methotrexate-induced D-xylose malabsorption in children with cancer?
2004 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Intake of vitamin A-rich foods and lung cancer risk in Taiwan: with special reference to garland chrysanthemum and sweet potato leaf consumption.
2007 Asia Pacific journal of clinical nutrition

Vitamin A and apoptosis in prostate cancer.
2002 Endocrine-related cancer

Skin, sun, and vitamin A: from aging to cancer.
2001 The Journal of dermatology

Vitamin A metabolism is impaired in human ovarian cancer.
2009 Gynecologic oncology

Effectiveness of Vitamin A acetate for enhancing the production of lung cancerspecific monoclonal antibodies.
1999 Cytotechnology

A population-based case-control study of carotenoid and Vitamin A intake and ovarian cancer (United States).
2001 Cancer causes & control : CCC

Recent advances in the use of Vitamin A (retinoids) in the prevention and treatment of cancer.
2000 Nutrition (Burbank, Los Angeles County, Calif.)

Fruit, vegetable, Vitamin A intakes, and prostate cancer risk.
2008 Prostate cancer and prostatic diseases

The Vitamin A family can significantly decrease the expression of ERbeta of ERs positive breast cancer cells in the presence or absence of ER ligands and paclitaxel.
2009 Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology

Induction of apoptosis in maxillary sinus cancer cells by 5-fluorouracil, Vitamin A and radiation (FAR) therapy.
1999 European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology – Head and Neck Surgery

EUROSCAN, a randomized trial of Vitamin A and N-acetylcysteine in patients with head and neck cancer or lung cancer. For the EUropean Organization for Research and Treatment of cancer Head and Neck and Lung cancer Cooperative Groups.
2000 Journal of the National cancer Institute

Rapid HPLC method for the determination of Vitamin A and E and cotinine concentration in human serum in women with CIN and cervical cancer.
2009 Ginekologia polska

Estradiol and tamoxifen differently affects the inhibitory effects of Vitamin A and their metabolites on the proliferation and expression of alpha2beta1 integrins in MCF-7 breast cancer cells.
2009 Advances in medical sciences

Renaissance of the biologically active Vitamin A derivatives: established and novel directed therapies for cancer and chemoprevention.
2003 Current pharmaceutical design

Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
2009 Asian Pacific journal of cancer prevention : APJCP

Whole-food sources of Vitamin A more effectively inhibit female rat sexual maturation, mammary gland development, and mammary carcinogenesis than retinyl palmitate.
2007 The Journal of nutrition

[Chemoprevention and FAR therapy regimen comprised of 5-fluorouracil, Vitamin Aand radiation].
2001 Gan to kagaku ryoho. cancer & chemotherapy

Prostate cancer:

• Vitamin A and apoptosis in prostate cancer. 2002 Endocrine-related cancer
• Fruit, vegetable, Vitamin A intakes, and prostate cancer risk. 2008 Prostate cancer and prostatic diseases
• Vitamin A, retinoids and carotenoids as chemopreventive agents for prostate cancer. 2004 The Journal of urology

Breast cancer:

• The Vitamin A family can significantly decrease the expression of ERbeta of ERs positive breast cancer cells in the presence or absence of ER ligands and paclitaxel. 2009 Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology
• Estradiol and tamoxifen differently affects the inhibitory effects of Vitamin A and their metabolites on the proliferation and expression of alpha2beta1 integrins in MCF-7 breast cancer cells. 2009 Advances in medical sciences
• Can Vitamin A modify the activity of docetaxel in MCF-7 breast cancer cells?                                                                                             2007 Folia histochemica et cytobiologica / Polish Academy of Sciences, Polish Histochemical and Cytochemical Society
• Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology                                                                                                   2009 Asian Pacific journal of cancer prevention : APJCP

Ovarian cancer:

• A population-based case-control study of carotenoid and Vitamin A intake and ovarian cancer (United States).                            2001 Cancer causes & control : CCC
• The role of antioxidants and Vitamin A in ovarian cancer: results from the Women’s Health Initiative.                                              2008 Nutrition and cancer
• Association of dietary vitamin A, carotenoids, and other antioxidants with the risk of ovarian cancer.                                           2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Vitamin A metabolism is impaired in human ovarian cancer. 2009 Gynecologic oncology

Lung cancer:

• Effectiveness of Vitamin A acetate for enhancing the production of lung cancer specific monoclonal antibodies. 1999 Cytotechnology
• Intake of vitamin A-rich foods and lung cancer risk in Taiwan: with special reference to garland chrysanthemum and sweet potato leaf consumption. 2007 Asia Pacific journal of clinical nutrition

Lung, head & neck cancers:

• EUROSCAN, a randomized trial of Vitamin A and N-acetylcysteine in patients with head and neck cancer or lung cancer. For the EUropean Organization for Research and Treatment of cancer Head and Neck and Lung cancer Cooperative Groups.

2000 Journal of the National cancer Institute

Cervical cancer:

• Rapid HPLC method for the determination of Vitamin A and E and cotinine concentration in human serum in women with CIN and cervical cancer. 2009 Ginekologia polska

Gastric cancer:

• Vitamin A, retinol, and carotenoids and the risk of gastric cancer: a prospective cohort study.                                                           2007 The American journal of clinical nutrition

Laryngeal cancer:

• Association of vitamin A, vitamin C and zinc with laryngeal cancer. 2003 Indian journal of cancer
• Oxidant, Vitamin A and angiogenic markers in laryngeal cancer patients. 2003 The Journal of laryngology and otology

Children cancer:

• The effect of Vitamin A status in children treated for cancer. 2008 Pediatric hematology and oncology
• Does Vitamin A prevent high-dose-methotrexate-induced D-xylose malabsorption in children with cancer?                                 2004 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Maxillary sinus cancer :

• Induction of apoptosis in maxillary sinus cancer cells by 5-fluorouracil, Vitamin A and radiation (FAR) therapy. 1999 European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology – Head and Neck Surgery
• [Chemoprevention and FAR therapy regimen comprised of 5-fluorouracil, Vitamin A and radiation].                                                                       2001 Gan to kagaku ryoho. cancer & chemotherapy

Studies on Vitamin A and Antiproliferative

http://science.naturalnews.com/V/Vitamin_A_and_Antiproliferative.html

Studies on Vitamin A and radiation

http://science.naturalnews.com/V/Vitamin_A_and_radiation.html

There is an extensive bibliography on the anti-tumoral action of Vitamin  A cited from LAST PAGE OF THIS DATABASE: 1,3,10,13,14,17,18,23,24,28,34,35,36,45,56,69,73,76,92,93,94,100,106,111,120,121,129,131,137,138,165,187,200,202,203,208,209,21,213,214,216,218,222,235,255,256,257,263,264,265,266,280,282,286,287,288,298,303,304,305,307,313,315,322,324,325,326,334,338,340,341,347,38,352,354,362,363,365,382,383,385,390,398,402,404,405,409,410,420,425,426,427,445,446,447,448,454,457,461,463,468,469,470,471,473,477,48,493,508,512):

This studies conclude  the following about vitamin A:

Anti-tumoral action in general

Vitamin A and retinoids have an anti neoplastic action, shown both in vivo and in vitro, in various tumors: basilomas, scaly carcinomas, melanomes, skin cancers, fungoid mycosis, acute promyelocyte leucemia, ovarian cancer, breast cancer, lung cancer, cancer of the bladder and follicular carcinomas of the thyroid.

Preventative action

Anti-tumoral action has been amply shown indirectly, that is with a preventative purpose. Various  studies have, in fact, shown that low plasmatic levels of beta-Carotene, vitamin C and vitamin E are  connected to an increase in the incidence of lung cancer.

Direct action at a receptor level

Only beta RAR (retinoic acid receptor) seems to be involved. More specifically, vitamin A (and its derivatives) acts by binding with specific receptors. In this way it can both inhibit the proteic synthesis of DNA and RNA, and also perform an anti-promotion  action to determine the return of a cellular differentiation. Furthermore its ability to inhibit oncogenes should be noted.

Apoptosis induction 

Apoptosis means the activation of specific endonucleases which break up the DNA, acting at a level of nucleosomic sites that make up the primary structural unit of the nuclear cromatine of the cell.  Vitamin A and retinoids in general can induce apoptosis in neoplastic cells, by activating intracellular proteolytic enzymes, called caspase 2 and caspase 3, which provoke deterioration  through proteolysis by a transcription factor, called Spl.  If this basal cellular transcription is altered, death is caused by Apoptosis.

In prostate cancer the retinoids intervene by reducing the level of the bcl 2 gene, whose function is to protect the cells from death by Apoptosis.  Carotenoids induced apoptosis in prostate cancer

Dă clic pentru a accesa carotenoidi%20sono%20fattori%20attivi%20contro%20il%20cancro%20della%20prostata.pdf

Inhibition of the cellular cycle

The retinoids block the passage of the cell from phase G1 to phase S (reducing the activity of a protein, called cycline D1): this passage, if not blocked, would lead the cell to mytosis.

Reduction of phosphorilation

Retinoids intervene in cancer causing a reduction in phosphorilation of the pRb, thus increasing survival; this protein is active in suppressing cellular growth.

Synergetic interaction with interferons

A second mechanism, apart from the proteic-enzymatic one, can be traced back to the intervention of interferons which, like retinoids, act as anti-proliferative factors. In practice, acting synergetically, they induce the expression of proteins capable of inhibiting neoplastic cellular proliferation.

The intake of natural carotenoids from food 

There is very little evidence to show that carotenoids taken in from food can increase the levels ofvitamin A: an extra portion a day of green leaf vegetables is not able to increase the hematic level of vitamin A; on the contrary, a mixed diet of foods particularly rich in beta-Carotene will give a  significant increase in vitamin A present in the blood. (627).

If you oblige healthy individuals to eat carrots (270g), broccoli (600g) or tomato juice (180g) it does not establish any significant changes in the hematic levels of the carotenoids: you only find an extremely wide variation (even up to 3-4 times) in the efficiency of gastro-intestinal absorption of  the carotenoids and therefore in their subsequent bio-availability at a hematic level (628) .

Optimal values of anti-oxidants in normal individuals

Vitamin C: >50 microMols/Liter

Vitamin E: >30 microMols/Liter

Vitamin A: >2.2 microMols/Liter

Beta-Carotene: >0.4 microMols/Liter

Association of vitamin A with vitamin E

In an experimental model of a cellular membrane the possibility of a positive interaction between anti-oxidant liposollubles such as beta-Carotene and alpha-Tocopherol has been investigated; the  result showed that there exists a synergetic action between beta-Carotene and alpha-Tocopherol  together which inhibits the processes of lipidic peroxidization compared with when they are used alone (629). THUS:

NOTE 2:

Vitamins , antioxidants and nutrients should be taken from WHOLE, ORGANIC(note below) FOODS (as much as possible) NOT from supplements as they need other vitamins and nutrients for proper usage.

Whole-food sources of Vitamin A more effectively inhibit female rat sexual maturation, mammary gland development, and mammary carcinogenesis than retinyl palmitate. 2007 The Journal of nutrition

The difference between synthetic vitamins and natural vitamins

The difference between synthetic vitamins and natural ones can be easily exemplified by the experimental case of synthetic beta-Carotene (made up entirely of isomeric trans-beta-Carotene), and of natural beta-Carotene (made up of both isomeric trans-beta-Carotene and isomeric cis-beta- Carotene): the study showed a strong discrimination between the two isomers, with a serious decrease (impoverishment) induced at the level of Lycopene present in the LDLs (…). THUS:

3.It would be better to use the NATURAL (juice) ORGANIC WHOLE foods rich in vitamin A such as  Daucus carota (carrots ), avocados, cantalopues, etc. rather than the synthetic pharmaceutical vitamin products based on Vitamin A.

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B Vitamins 

Studies on Vitamins B and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B_and_cancer.html

Plasma folate, vitamin B-6, vitamin B-12, and risk of breast cancer in women.
2008 The American journal of clinical nutrition

Low intake of vitamin B-6 is associated with increased risk of colorectal cancer in Japanese men.
2007 The Journal of nutrition

Dietary folate, methionine, riboflavin, and vitamin B-6 and risk of sporadic colorectal cancer.
2008 The Journal of nutrition

A prospective study of dietary folate and Vitamin B and colon cancer according to microsatellite instability and KRAS mutational status.
2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Effect of combined folic acid, vitamin B6, and vitamin B12 on cancer risk in women: a randomized trial.
2008 JAMA : the journal of the American Medical Association

Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer.
2003 Journal of the National cancer InstituteCancer incidence and mortality after treatment with folic acid and vitamin B12.
2009 JAMA : the journal of the American Medical AssociationOne-carbon metabolism-related nutrients and prostate cancer survival.
2009 The American journal of clinical nutritionCirculating concentrations of folate and vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study.
2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Reduced thiamine (vitamin B1) levels following gastrectomy for gastric cancer.
2002 Gastric cancer : official journal of the International Gastric cancer Association and the Japanese Gastric cancer Association

Dietary folate intake, MTHFR genetic polymorphisms, and the risk of endometrialcancer among Chinese women.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Localized folate and vitamin B-12 deficiency in squamous cell lung cancer is associated with global DNA hypomethylation.
2000 Nutrition and cancer

Dietary factors of one-carbon metabolism and prostate cancer risk.
2006 The American journal of clinical nutrition

Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.
2009 BMC cancer

Folate, vitamin B(6), and vitamin B(12) intake and the risk of breast cancer among Mexican women.
2006 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Dietary folate intake and breast cancer risk: results from the Shanghai Breast cancerStudy.
2001 Cancer research

Vitamin B6 and risk of colorectal cancer: a meta-analysis of prospective studies.
2010 JAMA : the journal of the American Medical Association

One-carbon metabolism and prostate cancer risk: prospective investigation of seven circulating B vitamins and metabolites.
2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Folate, vitamin B6, multivitamin supplements, and colorectal cancer risk in women.
2006 American journal of epidemiology

Relationship of folate, vitamin B-6, vitamin B-12, and methionine intake to incidence of colorectal cancers.
2002 Nutrition and cancer

Association of the B-vitamins pyridoxal 5′-phosphate (B(6)), B(12), and folate with lungcancer risk in older men.
2001 American journal of epidemiology

Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.
2009 Nutrition and cancer

The role of vitamin B6 in the prevention of haematological toxic effects of linezolid in patients with cancer.
2008 The Journal of antimicrobial chemotherapy

Methylenetetrahydrofolate reductase, diet, and risk of colon cancer.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer

Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
2005 International journal of cancer. Journal international du cancer

Vitamin B(12) and Folate Status in Head and Neck Cancer.
2002 Asian Pacific journal of cancer prevention : APJCP

Vitamins B2 and B6 and genetic polymorphisms related to one-carbon metabolism as risk factors for gastric adenocarcinoma in the European prospective investigation intocancer and nutrition.
2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Associations of dietary methyl donor intake with MLH1 promoter hypermethylation and related molecular phenotypes in sporadic colorectal cancer.
2008 Carcinogenesis

Vitamin B6 points PC6 injection during acupuncture can relieve nausea and vomiting in patients with ovarian cancer.
2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society

Studies on Vitamins B and Antineoplastic

http://science.naturalnews.com/V/Vitamin_B_and_Antineoplastic.html

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Vitamin B2 – Riboflavin

Studies on Riboflavin and cancer

science.naturalnews.com/R/Riboflavin_and_cancer.html(MANY STUDIES in this link )

Dietary folate, methionine, riboflavin, and vitamin B-6 and risk of sporadic colorectal cancer.
2008 The Journal of nutrition

Augmented antioxidant status in Tamoxifen treated postmenopausal women with breast cancer on co-administration with Coenzyme Q10, Niacin and Riboflavin.
2008 Cancer chemotherapy and pharmacology

Anti-angiogenic potential of CoenzymeQ10, Riboflavin and niacin in breast cancerpatients undergoing tamoxifen therapy.
2008 Vascular pharmacology

Biochemical characterization of Riboflavin carrier protein (RCP) in prostate cancer.
2009 Frontiers in bioscience : a journal and virtual library

Effect of coenzyme Q10, Riboflavin and niacin on serum CEA and CA 15-3 levels in breast cancer patients undergoing tamoxifen therapy.
2007 Biological & pharmaceutical bulletin

Serum cytokine levels of interleukin-1beta, -6, -8, tumour necrosis factor-alpha and vascular endothelial growth factor in breast cancer patients treated with tamoxifen and supplemented with co-enzyme Q(10), Riboflavin and niacin.
2007 Basic & clinical pharmacology & toxicology

Multidrug transporter ABCG2/breast cancer resistance protein secretes Riboflavin(vitamin B2) into milk.
2007 Molecular and cellular biologyIntracellular processing of Riboflavin in human breast cancer cells.
2008 Molecular pharmaceuticsAmeliorating effect of coenzyme Q10, Riboflavin and niacin in tamoxifen-treated postmenopausal breast cancer patients with special reference to lipids and lipoproteins.
2007 Clinical biochemistry

Effect of Coenzyme Q(10), Riboflavin and Niacin on Tamoxifen treated postmenopausal breast cancer women with special reference to blood chemistry profiles.
2009 Breast cancer research and treatment

Design of riboflavin-presenting PAMAM dendrimers as a new nanoplatform for cancer-targeted delivery.
2010 Bioorganic & medicinal chemistry letters

Co-enzyme Q10, Riboflavin and niacin supplementation on alteration of DNA repair enzyme and DNA methylation in breast cancer patients undergoing tamoxifen therapy.
2008 The British journal of nutrition

Riboflavin deficiency and esophageal cancer: a case control-household study in the Caspian Littoral of Iran.
2005 Cancer detection and prevention

[Apoptosis of human gastric cancer cell induced by photochemical riboflavin].
2003 Ai zheng = Aizheng = Chinese journal of cancer

Riboflavin carrier protein: a serum and tissue marker for breast carcinoma.
2001 International journal of cancer. Journal international du cancer

Studies on Riboflavin and Anti-Angiogenic

http://science.naturalnews.com/R/riboflavin_and_Anti-Angiogenic.html

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Vitamin B3(Niacin)

Studies on Niacin and cancer

Augmented antioxidant status in Tamoxifen treated postmenopausal women with breast cancer on co-administration with Coenzyme Q10, Niacin and Riboflavin.
2008 Cancer chemotherapy and pharmacology

Serum cytokine levels of interleukin-1beta, -6, -8, tumour necrosis factor-alpha and vascular endothelial growth factor in breast cancer patients treated with tamoxifen and supplemented with co-enzyme Q(10), riboflavin and niacin.
2007 Basic & clinical pharmacology & toxicology

Anti-angiogenic potential of CoenzymeQ10, riboflavin and Niacin in breast cancerpatients undergoing tamoxifen therapy.
2008 Vascular pharmacology

Effect of coenzyme Q10, riboflavin and Niacin on serum CEA and CA 15-3 levels in breast cancer patients undergoing tamoxifen therapy.

2007 Biological & pharmaceutical bulletin

Anti-invasive activity of Niacin and trigonelline against cancer cells.

2005 Bioscience, biotechnology, and biochemistry

Biochemical assessment of Niacin deficiency among carcinoid cancer patients.

2005 The American journal of gastroenterology

Ameliorating effect of coenzyme Q10, riboflavin and Niacin in tamoxifen-treated postmenopausal breast cancer patients with special reference to lipids and lipoproteins.

2007 Clinical biochemistry

Effect of Coenzyme Q(10), Riboflavin and Niacin on Tamoxifen treated postmenopausal breast cancer women with special reference to blood chemistry profiles.

2009 Breast cancer research and treatment

Co-enzyme Q10, riboflavin and Niacin supplementation on alteration of DNA repair enzyme and DNA methylation in breast cancer patients undergoing tamoxifen therapy.

2008 The British journal of nutrition

Niacin supplementation decreases the incidence of alkylation-induced nonlymphocytic leukemia in Long-Evans rats.

2008 Nutrition and cancer

Niacin and carcinogenesis.

2003 Nutrition and cancer

Other studies:

ANTI-invasive activities of Niacin against cancer  cells; http://science.naturalnews.com/…/3260742_Anti_invasive_activity_of_niacin_and_ trigonelline_against_cancer_cells.html

Studies on Niacin and breast cancer

http://science.naturalnews.com/N/Niacin_and_breast_cancer.html (MANY STUDIES in this link)  

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Vitamin B5 ( pantotenic acid )

Pantothenic Acid for Stage IV Breast Cancer Treatment Information

https://www.medify.com/…/pantothenic–acid-treatment-stage-iv-breast-ca…‎

Vitamins B2, B3,B5 are respiratory vitamins(increasing cellular oxygenation –  see above Otto Wartburg proposals).

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Vitamin B6

Studies on Vitamin B6 and cancer – Natural News Science (MANY STUDIES)

science.naturalnews.com/V/Vitamin_B6_and_cancer.html

Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer.

2003 Journal of the National cancer Institute

Effect of combined folic acid, vitamin B6, and vitamin B12 on cancer risk in women: a randomized trial.

2008 JAMA : the journal of the American Medical Association

The role of Vitamin B6 in the prevention of haematological toxic effects of linezolid in patients with cancer.

2008 The Journal of antimicrobial chemotherapy

Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.

2009 BMC cancer

Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.

2009 Nutrition and cancer

Methionine and Vitamin B6 intake and risk of pancreatic cancer: a prospective study of Swedish women and men.

2007 Gastroenterology

Plasma Vitamin B6 and the risk of colorectal cancer and adenoma in women.

2005 Journal of the National cancer Institute

Plasma folate, vitamin B6, vitamin B12, and homocysteine and pancreatic cancer risk in four large cohorts.

2007 Cancer research

Folate, vitamin B6, multivitamin supplements, and colorectal cancer risk in women.

2006 American journal of epidemiology

Folate, vitamin B6, vitamin B12, and vitamin B2 intake, genetic polymorphisms of related enzymes, and risk of colorectal cancer in a hospital-based case-control study in Japan.

2005 Nutrition and cancer

Folate and Vitamin B6 intake and risk of colon cancer in relation to p53 expression.

2008 Gastroenterology

Vitamin B6 points PC6 injection during acupuncture can relieve nausea and vomiting in patients with ovarian cancer.

2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society

Vitamin B6 and risk of colorectal cancer: a meta-analysis of prospective studies.

2010 JAMA : the journal of the American Medical Association

Dietary Vitamin B6 intake and the risk of colorectal cancer.

2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Prospective study of plasma Vitamin B6 and risk of colorectal cancer in men.

2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Vitamin B6 intake, alcohol consumption, and colorectal cancer: a longitudinal population-based cohort of women.

2005 Gastroenterology

Relationship between dietary and supplemental intake of folate, methionine, Vitamin B6 and folate receptor alpha expression in ovarian tumors.
2010 International journal of cancer. Journal international du cancer

Vitamin B12 

Studies on Vitamin B12 and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B12_and_cancer.html

Common polymorphisms in methylenetetrahydrofolate reductase gene are associated with risks of cervical intraepithelial neoplasia and cervical cancer in women with low serum folate and vitamin B12. 2011 Cancer causes & control : CCC Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer. 2003 Journal of the National cancer Institute Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study. 2005 International journal of cancer. Journal international du cancer Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study. 2005 International journal of cancer. Journal international du cancer The risk of cervical cancer in relation to serum concentrations of folate, vitamin B12, and homocysteine. 2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Effect of combined folic acid, vitamin B6, and Vitamin B12 on cancer risk in women: a randomized trial. 2008 JAMA : the journal of the American Medical AssociationPlasma Vitamin B12 concentrations and the risk of colorectal cancer: a nested case-referent study. 2008 International journal of cancer. Journal international du cancerDietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women. 2009 BMC cancer Circulating concentrations of folate and Vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study. 2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan. 2009 Nutrition and cancer Cancer incidence and mortality after treatment with folic acid and vitamin B12. 2009 JAMA : the journal of the American Medical Association Plasma folate, vitamin B6, vitamin B12, and homocysteine and pancreatic cancer risk in four large cohorts. 2007 Cancer research Plasma homocysteine, folate, and Vitamin B12 levels in patients with laryngeal cancer. 2008 Archives of otolaryngology–head & neck surgery A polymorphism of the methionine synthase gene: association with plasma folate, vitamin B12, homocyst(e)ine, and colorectal cancer risk. 1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Folate, vitamin B6, vitamin B12, and vitamin B2 intake, genetic polymorphisms of related enzymes, and risk of colorectal cancer in a hospital-based case-control study in Japan. 2005 Nutrition and cancer Elevated serum Vitamin B12 levels associated with CRP as a predictive factor of mortality in palliative care cancer patients: a prospective study over five years. 2000 Journal of pain and symptom management Folate, Vitamin B12 and postmenopausal breast cancer in a prospective study of French women. 2006 Cancer causes & control : CCC Vitamin B12 deficiency: a new risk factor for breast cancer? 1999 Nutrition reviews Toxic epidermal necrolysis related to pemetrexed and carboplatin with Vitamin B12and folic acid supplementation for advanced non-small cell lung cancer. 2009 Onkologie Methylenetetrahydrofolate reductase C677T genotype affects promoter methylation of tumor-specific genes in sporadic colorectal cancer through an interaction with folate/vitamin B12 status. 2008 World journal of gastroenterology : WJG Efficacy and safety of two doses of pemetrexed supplemented with folic acid andVitamin B12 in previously treated patients with non-small cell lung cancer. 2008 Clinical cancer research : an official journal of the American Association forcancer Research Serum Vitamin B12 and folate status among patients with chemotherapy treatment for advanced colorectal cancer. 2009 Upsala journal of medical sciences Circulating folate, vitamin B12, homocysteine, Vitamin B12 transport proteins, and risk of prostate cancer: a case-control study, systematic review, and meta-analysis. 2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Dietary folate and Vitamin B12 intake before diagnosis decreases gastric cancermortality risk among susceptible MTHFR 677TT carriers. 2010 Nutrition (Burbank, Los Angeles County, Calif.) Increased serum CA-15.3 levels in patients with megaloblastic anemia due to Vitamin B12 deficiency. 2004 Oncology

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Vitamin B17(Laetrile/ Amygdalin) (very important vitamin)

Note: “Laetrile” acronym for “LAEvomandeloniTRILE-glucoside”) as Amygdalin:

Laetrile has two molecules of glucose, Amygdalin has more.

Indeed, the chemical structure of Laetrile is D-1 mandelonitrile–beta-glucuronide, while

for Amygdalin it is D-mandelonitrile-bi-glucoside.

B17/ Amygdalin studies science.naturalnews.com/amygdalin.html

How laetrile or B17 from apricot seeds kills only cancer cells ·

apricot studies science.naturalnews.com/apricot.html

Apricot Seeds Kill Cancer Cells without Side Effects

See also:  http://worldwithoutcancer.org.uk/success.html,   http://www.apricotsfromgod.info/journal.htm

Much more details(including treatments & CLINICAL CASES with B17) in my BOOK + DR. Binzel’s FREE EBOOK on this topic + Dr. Nacci’s FREE EBOOK on this topic.

NOTE:

Vitamins(including B group) should be taken from WHOLE FOODS(organic) (as much as possible),NOT from supplements as vitamins and nutrients need other vitamins and nutrients for proper usage within the body.

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Vitamin C (very important vitamin)

Vitamin C   is a respiratory vitamin- it stimulates the body to produce its own hydrogen peroxide (increasing cellular oxygenation –  see above Otto Wartburg proposals).

Therefore, megadoses of vitamin C are administered orally and intravenously by orthomolecular physicians to treat cancer.

60 references to studies published that show the importance of vitamin C:

www.doctoryourself.com / biblio_cameron.html http://www.doctoryourself.com/cancer_hoffer.html.

Even National Institutes of Health is actively sponsoring studies of high-dose intravenous vitamin C therapy.

Other MULTIPLE studies on  vitamin’s C cancer fighting effects: science.naturalnews.com/…/4854330_Vitamin_C_in_alternative_cancer_ treatment_historical_background.html

High doses of vitamin C and cancer :

science.naturalnews.com/…/2387600_High_dose_vitamin_C_supplement_ use_is_associated_with_self.html

Vitamin C and breast cancer studies: science.naturalnews.com / V / Vitamin_C_and_breast_cancer.html

Vitamin C , vitamin E & selenium in breast cancer : science.naturalnews.com/…/1198191_Dietary_beta_carotene_vitamin_C_and _E_intake_and_breast.html science.naturalnews.com/…/3959947_Association_between_breast_cancer_ and_vitamin_C_vitamin_E_and.html

Vitamin C ,vitamin A & zinc  in laryngeal cancer  :

science.naturalnews.com/…/4508069_Association_of_vitamin_A_vitamin_C _and_zinc_with_laryngeal.html

Vitamin C &  gastric cancer:

science.naturalnews.com/…/3603872_Plasma_and_dietary_vitamin_C_levels _and_risk_of_gastric.html science.naturalnews.com/…/3628783_Prediagnostic_plasma_vitamin_C_ levels_and_the_subsequent_risk_of.html science.naturalnews.com/…/4790082_Effect_of_physiological_ concentrations_of_vitamin_C_on_gastric_cancer.html

Also see: http://orthomolecular.org/resources/omns/v04n19.shtml

There is also an extensive bibliography on the anti-tumoral action of vitamin C (25,33,47,54,83,91,122,129,181,197,202,218,244,246,270,299,311,335,339,367,404,405,414,415,416,489,496,510,511)

See the bibliography and studies quoted on last page of databse.

The pioneers of this oncological therapy were Pauling, who received the Nobel Prize for Chemistry, and the Italian Pantellini.

Ascorbic acid is mainly known for its ability to reduce metallic ions in various enzymatic processes and above all for its ability to act as an anti-oxidant agent, thus able to remove free radicals, reducing the damage caused at a genome level.

Furthermore it may be able to block the formation of nitrose at a gastro-intestinal level, as well as carrying out a preventative action on the formation of adenomatose polyps.

Even though ascorbic acid is well known for its collagene forming action, and the well known effect of scurvy in cases of reduction or absence of this acid in the diet, this vitamin is also important, alongside vitamins A and E as a first class anti-tumoral agent.

It is a respiratory vitamin(like vitamins E, B3, B5)  that helps increase cellular oxygen levels.

It reinforces the intercellular bond and forestalls the destructive action of the hyalurons produced by many neoplastic cells.

Vitamin C stimulates the Natural killer lymphocytes, it supports the macrophagic activity, the chemio-tactical mobility of the white corpuscles, the production of antibodies and the response of the T cytotoxic lymphocytes to the antigenes.

Cameron found very low plasmatic levels of vitamin C in cancer patients (0.26 mg/100 mL) compared to normal plasmatic values (54).

In other studies, 154 cancer patients, undergoing analysis, were found to have low levels of vitamin C not only in their plasma (0.31 mg/100 mL), but also in their leucocytes (15.9 mg/10 E+8), with a positive correlation (r = 0.42) between these two values; in particular the authors attributed the alteration in the immune response to the tumor, especially of the phagocytosis, to the low  concentration of vitamin C in the white blood corpuscles.

Already in 1974 Goetz had shown that vitamin C, in vitro, was capable of stimulating the motility and the chemiotaxis of neutrophiles.

The doses advised in literature for anti-neoplastic therapy are about 3-10 grams a day, reaching even 40 grams daily, because the vitamin is not toxic, at high doses it only has a laxative effect.

It would be better to take it by eating fresh fruit, but it is difficult to reach such high doses of the vitamin in this way.

Integrating fresh fruit (kiwi, oranges, lemons and grapefruit) with the juice of sea buckthorn  , Rosa canina (dog rose, wild rose) to reach a daily dosage of at least 5- 8 grams of natural vitamin C, avoiding however, the use of pharmaceutically prepared vitamin C tablets.

Natural vitamin C, rich in its metabolites and other components called bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercitin, Pychnogenol, etc) is more powerful and efficient, and furthermore is devoid of unpleasant gastric effects which are the result of high doses of synthetic vitamin C.

Natural vitamin C is moreover, characterized by a significant reduction in the formation of Calcium oxalate in the kidneys, as opposed to synthetic vitamin C. It is also easier for the intestine to absorb and has greater bio-availability, above all through its most important metabolites such as tronic acid, lixonic acid, xilonic acid etc.

This bio-availability has a critical importance in the immune defense system because the white blood cells tend to absorb Natural vitamin C 4 times more than they absorb synthetic vitamin C.

Recently, Myrciaria paraensis (camu-camu) has appeared on the European market. It is a small exotic fruit, similar to a small orange, but it contains 50 times more natural vitamin C than Citrus aurantium (orange), and it could therefore provide the daily dosage, of at least 3 grams, of vitamin  .

Also Malpighia punicifolia (acerola), a cherry from the Antilles, is very rich in vitamin C, containing 50 to 100 times more than citrus fruits.

Natural vitamin C is therefore efficient because it is naturally associated to the bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercetine,Quercitine, Pychnogenol, etc..) and other molecules, in plants often characterized by an immune  stimulating activity (Echinacea purpurea, Plantago major, Capsicum frutescens)

Note: With very high therapeutic doses (>8-10 grams a day), Magnesium (e.g. Dolomite) must also be taken to avoid the risk of kidney stones.

Here enclosed scientific papers extracted from Catherine Kousmine (“Save your body”, page 129,

“Effects of C vitamin on our body according to Linus Pauling, edition Tecniche Nuove):

“…an intake of 1500 milligrams of ascorbic acid by mouth determines a concentration of 1.5 milligrams of C vitamin for each 100 millilitres of blood. By increasing the intake, the concentration suddenly increases up to 2.5 milligrams and then goes back to 1.5 millilitres for each 100 millilitres blood. There are enzymes which help the conversion of most

ascorbates into useful oxidation products. If the intake remains high, the body increases the amount of enzymes useful to the conversion; otherwise, if the ascorbic acid dose is suddenly reduced for some days, an excess of conversion enzymes and then a too law level of vitamin C in the blood occur. This means a number of disorders, as for example a higher sensibility to infections. The adaptation to a lower proportion takes place by reducing the number of conversion enzymes: it is necessary to gradually decrease the dose of vitamin C. By taking 100 milligrams a day and in presence of a plasmatic level of 1 milligram for every 100 millilitres blood, urines do not contain ascorbic acid because it is reabsorbed by renal tubules. If the intake is higher than 100 milligrams, i.e. 1-2 grams a day, 25% go in urines and the rest is kept by the body. Healthy people, who lack in vitamin C for some months, have to take 2-4 grams in order to eliminate them through urines. In case of cancer patients, who are used to take high doses of ascorbic acid, an interruption of some days requires an intake of 50 grams (fifty grams) of vitamin C so that this one can be found in urines”.

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Vitamin D (very important vitamin)

Cancer prevention by a MINIMUM of 77% cases just by sun exposure(best source of vitamin D): http://www.naturalnews.com/035063_vitamin_D_cancer_facts.html

Cancer treatment with sun exposure(vitamin D): http://www.polymvasurvivors.com/what_you_know.html#Vitamin%20D%20

NOTE:The link above contains multiple studies on sun exposure(vitamin D) and it’s crucial importance in fighting cancer:

Circulating Vitamin D levels in the Blood Dramatically Reduces Death Rate

* For each 10ng/ml increase in the blood level of vitamin D, the death rate from cancer would be reduced by 29%. The relationship between vitamin D and colorectal cancer is particularly strong.

The death rate for colorectal cancer would be reduced by 49%. (30 – Read Supporting Scientific Article on PUBMED)

Vitamin D Levels est during Summer = Improves survival by 40%

* A Norwegian study showed that cancer patients diagnosed in the summer when vitamin D levels are highest had up to 40% better survival rates than patients diagnosed in the winter when vitamin D levels are at their lowest.

(56 – Read Supporting Scientific Article on PUBMED )

Low Vitamin D Levels Greater chance of Death & Metastasis

* Breast cancer patients with low levels of vitamin D followed over eleven years had a 70% greater chance of dying and twice the rate of developing metastasis than patients with high levels of vitamin D.

(90 – Read Supporting Scientific Article )

Lung Cancer Patients Higher levels = Dramatic increase in Survival

* Early stage lung cancer patients who were diagnosed in the summer and had the highest levels of vitamin D had a five-year survival of 73% compared to 30% for those diagnosed in the winter with low vitamin D levels.

(60 – Read Supporting Scientific Article on PUBMED)

Lung – Colon – Prostate – Renal – Endometrial Cancers Higher Vit D levels dramatically decreases Risk

*High vitamin D levels (based on latitude and UV exposure) also correlate with a decreased risk of developing lung cancer, a 45% and 65% reduction in men and women respectively. Similar effects have been demonstrated in colon, prostate, renal and endometrial cancer. (33 – Read Supporting Scientific Article on BMJ(British Medical Journal))

Advanced Colorectal Patients Risk of death greatly Improved

* The risk of death for colorectal cancer patients with advanced disease but with high levels of vitamin D was reduced by over 60% compared to patients with low vitamin D levels. (64 – Read Supporting Scientific Article on PUBMED)

Prostate Cancer Patients

*Prostate Cancer Patients with Vitamin D levels in the mid and high range had a 60% and 85% respectively reduced risk of death from the condition compared to patients with low levels of Vitamin D. This is almost a seven fold increase in the risk of death in those with low vs high vitamin D levels. (152 – Read Supporting Scientific Article on PUBMED)

There is also an extensive bibliography on the anti-tumoral action of vitamin D- see last page of this database:  (28,157,160,188,208,209,231,240,246, 254,302,323,479,489)

Vitamin D is BEST taken from skin exposure to the sun, MINIMUM 30 mins daily,without any sunscreen or protection. Avoid burning and avoid the interval 11 am – 16 pm.

Natural vitamin D, contained in some plants, is however preferable to the synthetic type, because the latter is about 10 times more capable of binding with Magnesium, taking it away from the organism, thus causing all the damage that the loss of this incurs (osteoporosis, kidney stones).

Vitamin D induces the inhibition of neoplastic cellular growth: this has been shown in vitro in neoplastic cellular lines; especially of the hematopoietic system, of the CNS, of the prostate; the  colon, the ovaries and the breasts.

This action is thought to be expressed at various levels, in particular:

a) by means of apoptosis induction, through the activation of p21, that is, the inhibitor of the kinase proteins;

b) the inhibition of neoplastic cellular growth, which would be blocked in the G1 phase because of the action of the IGF1 inhibitor;

c) by means of cellular differentiation.

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Vitamin E(respiratory vitamin)

This liposoluble substance consists of a group of various components, called Tocopherols.

Seven of these exist in nature; alpha-Tocopherol, beta-Tocopherol, gamma- Tocopherol, delta-Tocopherol, epsilon-Tocopherol, zetaTocopheros and eta-Tocopherol.

Studies on Vitamin E and cancer

Selenium and Vitamin E supplements for prostate cancer: evidence or embellishment?
2002 Urology

Supplemental Vitamin E intake and prostate cancer risk in a large cohort of men in the United States.
1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

The role of Vitamin E in the prevention of cancer: a meta-analysis of randomized controlled trials.

2007 Annals of Saudi medicine

Long-term use of supplemental multivitamins, vitamin C, vitamin E, and folate does not reduce the risk of lung cancer.
2008 American journal of respiratory and critical care medicine

Vitamin E and selenium supplementation and risk of prostate cancer in the Vitamins and lifestyle (VITAL) study cohort.
2008 Cancer causes & control : CCCVitamin E suppresses telomerase activity in ovarian cancer cells.
2007 Cancer detection and preventionEffects of long-term Vitamin E supplementation on cardiovascular events and cancer: a randomized controlled trial.
2005 JAMA : the journal of the American Medical AssociationSupplemental and dietary Vitamin E intakes and risk of prostate cancer in a large prospective study.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Antioxidants vitamin C and Vitamin E for the prevention and treatment of cancer.
2006 Journal of general internal medicine

Vitamin E: the evidence for multiple roles in cancer.
2003 Nutrition and cancer

Vitamin E supplements and risk of prostate cancer in U.S. men.
2004 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Serum and dietary Vitamin E in relation to prostate cancer risk.
2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Association of variants in two Vitamin E transport genes with circulating Vitamin Econcentrations and prostate cancer risk.
2009 Cancer research

Association between breast cancer and vitamin C, Vitamin E and selenium levels: results of a case-control study in India.
2005 Asian Pacific journal of cancer prevention : APJCP

A short-term dietary supplementation with high doses of Vitamin E increases NK cell cytolytic activity in advanced colorectal cancer patients.
2007 Cancer immunology, immunotherapy : CII

SELECT: the next prostate cancer prevention trial. Selenum and Vitamin E cancerPrevention Trial.
2001 The Journal of urology

Vitamin C and Vitamin E supplement use and colorectal cancer mortality in a large American cancer Society cohort.
2001 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Vitamin E and breast cancer.
2004 The Journal of nutrition

SELECT: the selenium and Vitamin E cancer prevention trial.
2003 Urologic oncology

The selenium and Vitamin E cancer prevention trial.
2003 World journal of urology

Selenium and Vitamin E cancer prevention trial.
2004 Annals of the New York Academy of Sciences

Selenium and Vitamin E cancer Prevention Trial: a nutrient approach to prostatecancer prevention.
2009 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

A nutrient approach to prostate cancer prevention: The Selenium and Vitamin Ecancer Prevention Trial (SELECT).
2010 Nutrition and cancer

Null activity of selenium and Vitamin E as cancer chemopreventive agents in the rat prostate.
2010 Cancer prevention research (Philadelphia, Pa.)

Vitamin E and cancer.
2007 Vitamins and hormones

Clinical models for testing chemopreventative agents in prostate cancer and overview of SELECT: the Selenium and Vitamin E cancer Prevention Trial.
2003 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

Cancer Preventive Effects of Vitamin E.
2011 Current pharmaceutical biotechnology

Vitamin E but not St. John’s wort mitigates leukopenia caused by cancerchemotherapy in rats.
2006 Translational research : the journal of laboratory and clinical medicine

Vitamin C and Vitamin E supplement use and bladder cancer mortality in a large cohort of US men and women.
2002 American journal of epidemiology

Review of Vitamin E and selenium in the prevention of prostate cancer: implications of the selenium and Vitamin E chemoprevention trial.
2002 Integrative cancer therapies

Vitamin E and breast cancer prevention: current status and future potential.
2003 Journal of mammary gland biology and neoplasia

Vitamin E and breast cancer prevention: current status and future potential.
2003 Journal of mammary gland biology and neoplasia

Molecular epidemiologic studies within the Selenium and Vitamin E cancerPrevention Trial (SELECT).
2001 Cancer causes & control : CCC

Selenium and vitamin E: cell type- and intervention-specific tissue effects in prostate cancer.

2009 Journal of the National cancer Institute

Tocopherol transfer protein sensitizes prostate cancer cells to vitamin E.
2010 The Journal of biological chemistry

Selenium and Vitamin E for prostate cancer: post-SELECT (Selenium and Vitamin Ecancer Prevention Trial) status.
2011 Molecular medicine (Cambridge, Mass.)

Vitamin E and cancer: An insight into the anticancer activities of Vitamin E isomers and analogs.
2008 International journal of cancer. Journal international du cancer

Vitamin E intake, alpha-tocopherol status, and pancreatic cancer in a cohort of male smokers.
2009 The American journal of clinical nutrition

The roles of alpha-vitamin E and its analogues in prostate cancer.
2007 Vitamins and hormones

Combined lycopene and Vitamin E treatment suppresses the growth of PC-346C human prostate cancer cells in nude mice.
2006 The Journal of nutrition

Vitamin E, alpha- and gamma-tocopherol, and prostate cancer.
1999 Seminars in urologic oncology

Synergistic effect of Vitamin E and selenium in human prostate cancer cell lines.
2004 Prostate cancer and prostatic diseases

Gamma (gamma) tocopherol upregulates peroxisome proliferator activated receptor (PPAR) gamma (gamma) expression in SW 480 human colon cancer cell lines.
2003 BMC cancer

Vitamin E succinate inhibits colon cancer liver metastases.
2002 The Journal of surgical research

Lycopene and Vitamin E interfere with autocrine/paracrine loops in the Dunning prostate cancer model.
2004 The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Higher baseline serum concentrations of Vitamin E are associated with lower total and cause-specific mortality in the Alpha-Tocopherol, Beta-Carotene cancerPrevention Study.
2006 The American journal of clinical nutrition

Alpha-tocopheryl succinate, the most effective form of Vitamin E for adjuvant cancertreatment: a review.
2003 Journal of the American College of Nutrition

gamma-Tocopherol or combinations of Vitamin E forms induce cell death in human prostate cancer cells by interrupting sphingolipid synthesis.
2004 Proceedings of the National Academy of Sciences of the United States of America

Experimental basis for cancer prevention by vitamin E.
2000 Cancer investigation

Vitamin E analogues as a novel group of mitocans: anti-cancer agents that act by targeting mitochondria.
2007 Molecular aspects of medicine

Vitamin E succinate decreases lung cancer tumor growth in mice.
2005 The Journal of surgical research

RRR-alpha-vitamin E succinate potentiates the antitumor effect of calcitriol in prostatecancer without overt side effects.
2009 Clinical cancer research : an official journal of the American Association forcancer Research

Serum levels of folate, lycopene, ß-carotene, retinol and Vitamin E and prostatecancer risk.
2010 European journal of clinical nutrition

Trace elements and Vitamin E status in Nigerian patients with prostate cancer.
2010 African health sciences

Designing the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2005 Journal of the National cancer Institute

Vitamin E analogues and immune response in cancer treatment.
2007 Vitamins and hormones

Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Vitamin E serum levels and gastric cancer: results from a cohort of patients in Tuscany, Italy.
2000 Cancer letters

Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Effects of topical L-selenomethionine with topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
2003 Journal of the American Academy of Dermatology

Vitamin E and the Y4 agonist BA-129 decrease prostate cancer growth and production of vascular endothelial growth factor.
2002 The Journal of surgical research

Pro-apoptotic mechanisms of action of a novel Vitamin E analog (alpha-TEA) and a naturally occurring form of Vitamin E (delta-tocotrienol) in MDA-MB-435 human breast cancer cells.
2004 Nutrition and cancer

Minority recruitment to the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2005 Clinical trials (London, England)

Salubrious effect of vitamin C and Vitamin E on tamoxifen-treated women in breastcancer with reference to plasma lipid and lipoprotein levels.
2000 Cancer letters

Design of Physicians’ Health Study II–a randomized trial of beta-carotene, vitamins E and C, and multivitamins, in prevention of cancer, cardiovascular disease, and eye disease, and review of results of completed trials.
2000 Annals of epidemiology

Vitamin E succinate promotes breast cancer tumor dormancy.
2000 The Journal of surgical research

Mitocans as anti-cancer agents targeting mitochondria: lessons from studies withVitamin E analogues, inhibitors of complex II.
2007 Journal of bioenergetics and biomembranes

Centralized blood processing for the selenium and Vitamin E cancer prevention trial: effects of delayed processing on carotenoids, tocopherols, insulin-like growth factor-I, insulin-like growth factor binding protein 3, steroid hormones, and lymphocyte viability.
2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

The therapeutic and preventive effect of RRR-alpha-vitamin E succinate on prostatecancer via induction of insulin-like growth factor binding protein-3.
2007 Clinical cancer research : an official journal of the American Association forcancer Research

Vitamin E and prostate cancer: is Vitamin E succinate a superior chemopreventive agent?
2005 Nutrition reviews

Aspirin prevents stroke but not MI in women; Vitamin E has no effect on CV disease or cancer.
2006 Cleveland Clinic journal of medicine

Effect of Vitamin E on tamoxifen-treated breast cancer cells.
2006 Surgery

Dietary administration of the proapoptotic Vitamin E analogue alpha-tocopheryloxyacetic acid inhibits metastatic murine breast cancer.
2006 Cancer research

Combination of Vitamin E and selenium causes an induction of apoptosis of human prostate cancer cells by enhancing Bax/Bcl-2 ratio.
2008 The Prostate

Non-hormonal treatment of hot flushes in breast cancer survivors: gabapentin vs. vitamin E.
2009 Climacteric : the journal of the International Menopause Society

Vitamin E analog alpha-TEA, methylseleninic acid, and trans-resveratrol in combination synergistically inhibit human breast cancer cell growth.
2008 Nutrition and cancer

Poly(L-lactide)-vitamin E TPGS nanoparticles enhanced the cytotoxicity of doxorubicin in drug-resistant MCF-7 breast cancer cells.
2010 Biomacromolecules

Vitamin E succinate induced apoptosis and enhanced chemosensitivity to paclitaxel in human bladder cancer cells in vitro and in vivo.
2010 Cancer science

Tocopherol-associated protein suppresses prostate cancer cell growth by inhibition of the phosphoinositide 3-kinase pathway.
2005 Cancer research

Differential retention of alpha-vitamin E is correlated with its transporter gene expression and growth inhibition efficacy in prostate cancer cells.
2007 The Prostate

Vitamin E succinate (VES) induces Fas sensitivity in human breast cancer cells: role for Mr 43,000 Fas in VES-triggered apoptosis.
1999 Cancer research

Effects of topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
2000 Nutrition and cancer

Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
2003 Cancer research

Vitamin E analogs trigger apoptosis in HER2/erbB2-overexpressing breast cancercells by signaling via the mitochondrial pathway.
2005 Biochemical and biophysical research communications

Vitamin E and prostate cancer.
2002 The Urologic clinics of North America

Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
2003 Cancer research

Peptide YY augments gross inhibition by Vitamin E succinate of human pancreaticcancer cell growth.
2000 The Journal of surgical research

Supplemental and dietary vitamin E, beta-carotene, and vitamin C intakes and prostatecancer risk.
2006 Journal of the National cancer Institute

Vitamin E in the primary prevention of cardiovascular disease and cancer: the Women’s Health Study: a randomized controlled trial.
2005 JAMA : the journal of the American Medical Association

Dietary intakes of retinol, beta-carotene, vitamin D and Vitamin E in the European Prospective Investigation into cancer and Nutrition cohort.
2009 European journal of clinical nutrition

Mitochondrial targeting of Vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II.
2011 The Journal of biological chemistry

Impact of supplemental site grants to increase African American accrual for the Selenium and Vitamin E cancer Prevention Trial.
2010 Clinical trials (London, England)

The Outcome of Selenium and Vitamin E cancer Prevention Trial (SELECT) reveals the need for better understanding of selenium biology.
2009 Molecular interventions

Critical roles for JNK, c-Jun, and Fas/FasL-Signaling in Vitamin E analog-induced apoptosis in human prostate cancer cells.
2008 The Prostate

Effect of selenium and Vitamin E on risk of prostate cancer and other cancers: the Selenium and Vitamin E cancer Prevention Trial (SELECT).
2009 JAMA : the journal of the American Medical Association

Tocotrienols, the Vitamin E of the 21st century: its potential against cancer and other chronic diseases.
2010 Biochemical pharmacology

Mixed PEG-PE/vitamin E tumor-targeted immunomicelles as carriers for poorly soluble anti-cancer drugs: improved drug solubilization and enhanced in vitro cytotoxicity.
2008 European journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft für Pharmazeutische Verfahrenstechnik e.V

Nanoparticles of poly(lactide)/vitamin E TPGS copolymer for cancer chemotherapy: synthesis, formulation, characterization and in vitro drug release.
2006 Biomaterials

Involvement of JNK/p73/NOXA in Vitamin E analog-induced apoptosis of human breast cancer cells.
2008 Molecular carcinogenesis

Vitamin E supplementation in cardiovascular disease and cancer prevention: Part 1.
2005 The Annals of pharmacotherapy

A peptide conjugate of Vitamin E succinate targets breast cancer cells with high ErbB2 expression.
2007 Cancer research

Vitamin E succinate inhibits NF-kappaB and prevents the development of a metastatic phenotype in prostate cancer cells: implications for chemoprevention.
2007 The Prostate

The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

Vitamin E succinate and cancer treatment: a Vitamin E prototype for selective antitumour activity.
2003 British journal of cancer

Vitamin E down-modulates iNOS and NADPH oxidase in c-Myc/TGF-alpha transgenic mouse model of liver cancer.
2004 Journal of hepatology

Vitamin E inhibits the high-fat diet promoted growth of established human prostate LNCaP tumors in nude mice.
1999 The Journal of urology

Vitamin E analog alpha-TEA and celecoxib alone and together reduce human MDA-MB-435-FL-GFP breast cancer burden and metastasis in nude mice.
2004 Breast cancer research and treatment

Development of gamma (gamma)-tocopherol as a colorectal cancer chemopreventive agent.
2003 Critical reviews in oncology/hematology

Vitamin E analogues: a new class of inducers of apoptosis with selective anti-cancer effects.

2004 Current cancer drug targets

The role of PC-SPES, selenium, and Vitamin E in prostate cancer.
2002 Oncology (Williston Park, N.Y.)

Differential response of human ovarian cancer cells to induction of apoptosis byVitamin E Succinate and Vitamin E analogue, alpha-TEA.
2004 Cancer research

A short-term dietary supplementation of high doses of Vitamin E increases T helper 1 cytokine production in patients with advanced colorectal cancer.
2002 Clinical cancer research : an official journal of the American Association forcancer Research

Role of caspase-8 activation in mediating vitamin E-induced apoptosis in murine mammary cancer cells.
2003 Nutrition and cancer

High dietary level of synthetic Vitamin E on lipid peroxidation, membrane fatty acid composition and cytotoxicity in breast cancer xenograft and in mouse host tissue.
2003 Cancer cell international

[Roles of ERK1/2 MAPK in Vitamin E succinate-induced apoptosis in human gastriccancer SGC-7901 cells].
2003 Wei sheng yan jiu = Journal of hygiene research

Alpha-vitamin E derivative, RRR-alpha-tocopheryloxybutyric acid inhibits the proliferation of prostate cancer cells.
2007 Asian journal of andrology

Roles of Fas signaling pathway in Vitamin E succinate-induced apoptosis in human gastric cancer SGC-7901 cells.
2002 World journal of gastroenterology : WJG

Mechanisms mediating the antiproliferative and apoptotic effects of Vitamin E in mammary cancer cells.
2005 Frontiers in bioscience : a journal and virtual library

Cellular and molecular effects of alpha-tocopheryloxybutyrate: lessons for the design of Vitamin E analog for cancer prevention.
2004 Anticancer research

Vitamin E and cancer.
2009 Nutrition and cancer

Pentoxifylline and Vitamin E treatment for prevention of radiation-induced side-effects in women with breast cancer: a phase two, double-blind, placebo-controlled randomised clinical trial (Ptx-5).
2009 European journal of cancer (Oxford, England : 1990)

Vitamin E prevents lipid raft modifications induced by an anti-cancer lysophospholipid and abolishes a Yap1-mediated stress response in yeast.
2010 The Journal of biological chemistry

Analogs of Vitamin E epitomized by alpha-tocopheryl succinate for pancreatic cancertreatment: in vitro results induce caution for in vivo applications.
2010 Pancreas

Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
2009 Asian Pacific journal of cancer prevention : APJCP

[The study of apoptosis induction effect of Vitamin E succinate on Tca8113 human tongue cancer cells].
2008 Hua xi kou qiang yi xue za zhi = Huaxi kouqiang yixue zazhi = West China journal of stomatology

Antioxidant status and risk of cancer in the SU.VI.MAX study: is the effect of supplementation dependent on baseline levels?
2005 The British journal of nutrition

Double-blind placebo-controlled randomised trial of Vitamin E and pentoxifylline in patients with chronic arm lymphoedema and fibrosis after surgery and radiotherapy for breast cancer.
2004 Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology

Vitamin E succinate induces apoptosis in human prostate cancer cells: role for Fas inVitamin E succinate-triggered apoptosis.
2000 Nutrition and cancer

Vitamin E and other antioxidants inhibit human prostate cancer cells through apoptosis.
2000 The Prostate

Vitamin E succinate suppresses prostate tumor growth by inducing apoptosis.
2006 International journal of cancer. Journal international du cancer

In vitro and in vivo evaluation of anticancer actions of natural and synthetic Vitamin Eforms.
2008 Molecular nutrition & food research

Vitamin E analogues as anticancer agents: lessons from studies with alpha-tocopheryl succinate.
2006 Molecular nutrition & food research

Prospective study of serum Vitamin E levels and esophageal and gastric cancers.
2003 Journal of the National cancer Institute

Systematic review on „vitamin E and prevention of colorectal cancer”.
2010 Pakistan journal of pharmaceutical sciences

Vitamin E analogues as mitochondria-targeting compounds: from the bench to the bedside?
2009 Molecular nutrition & food research

Vitamin E and cancer: An insight into the anticancer activities of …

science.naturalnews.com/…/1364373_Vitamin_E_and_cancer_An_insight_ into_the_anticancer_activities.html

Studies on Vitamin E and radiation

http://science.naturalnews.com/V/Vitamin_E_and_radiation.html

There is also an extensive bibliography on the anti-tumoral action of vitamin E -see last page of DATABASE (6,19,20,30,45,91,95,112, 125,129,142,165,167,190,202,228,229,246,261,280,332,404,405,452,494)

This liposoluble substance consists of a group of various components, called Tocopherols. Seven of these exist in nature; alpha, beta, gamma, delta, epsilon, zeta and eta.

Alpha-Tocopherol has an anti-oxidant effect on the lipidic membranes in synergy with Melatonin(see below), carrying out a preventative action on the peroxidization of the cellular membrane induced by  ionizing radiation and by chemical carcinogenes.

Vitamin E also carries out an anti-oxidative action in a wider sense, acting as a ‘scavenger’ of the free radicals, similar to vitamin C.

It performs a stimulating activity on the immune system; it induces cellular differentiation; it inhibits, in a selective way, cellular growth intervening at DNA and RNA synthesis level.

Various studies have shown its ability to induce apoptosis in cellular lines of breast carcinomas and lymphoma B.

It is inactivated by Iron, therefore it is essential that any medicines based on Iron are not taken at the same time as vitamin E, but at a distance of at least 10-12 hours.

Aluminum, which is often present in pharmaceutical products, also deactivates vitamin E.

The uncooked oil of Triticum sativum (wheat germ; note: has vitamin B12) contains about twice as much vitamin E as the uncooked oil from the seeds of Helianthus annuus (sunflower) and the latter  contains about five times as much as uncooked olive oil. Furthermore all these seeds are rich in  essential unsaturated fats, an important part of the diet for cancer patients.

In anti-neoplastic therapy much is being discussed about: the raw seeds of Helianthus annuus

(which the author personally considers useful in therapy), wheat shoots (the author is not in favor),

Saccharomyces cerevisiae (yeast, of which the author is not in favor), and the shoots of soya  lecithin (of dubious use and the author is against their use because of the transgenic risk).

Natural vitamins are by far preferable to industrially produced ones: Dracontium loretense, for example, which is considered one of the best  plants for its specific anti-oxidant potential, is of superior quality in its anti-oxidant ability compared to synthetic vitamin E (566). WHOLE, ORGANIC FOODS are BEST due to COMPLEXES of NUTRIENTS !

As an already extracted natural product, together with or without other vitamins, vitamin E must be  given in addition to high quantities of raw seeds of Helianthus annuus (also containing vitamin A,all the vitamin B compounds, vitamin D, Manganese, Zinc and Magnesium) and of high quantities of raw Triticum sativum (which is rich in the precious alpha-lipoic acid): both are also very rich in vitamin B6 (pyridoxin), the latter is important for the immune system, but it is difficult to find in other compatible foods for a suitable diet for cancer patients. Pyridoxin, in fact, is contained especially in Saccharomyces cerevisiae, the latter is a food which the author does not regard favorably for an anti-neoplastic diet, because it contains high quantities of folic acid.

Natural Octacosanol, extracted from the oil of Triticum sativum, has a synergetic action with vitamin E, but it is, in any case, better to consume it with all the uncooked oil of Triticum sativum  and/or Triticum sativum itself rather than taking it already extracted, as a pharmaceutical product  (because it loses its active principles).

Vitamin E is particularly efficient in combination with Selenium, which is contained in Aloe species, Solanum lycopersicum (tomatoes), Equisetum species, Allium cepa (onions).

There is also an extensive bibliography on Selenium (see last page of DATABASE) :

79,108,112,129,133,136,143,156,228,229,276,338,339,364,367,404,405,407,443,452,458,501,510,511)

Both vitamin E and Selenium are in their turn synergetic with Zinc in inhibiting the production of inflammatoryprostoglandins and leukotrienes.

Vitamin E conversion factors

Vitamin E is expressed in milligrams of tocopheral equivalents (T.E.)

1 milligram of Tocopherol is equal to:

= 1 milligram of D-alpha Tocopherol

= 2 milligrams of D-beta Tocopherol

= 5 milligrams of D-gamma Tocopherol

==========================================================================================================

Vitamin F  (very important vitamin-see omega 3)

Polyunsaturated fatty acids :

arachidonic acid, Linoleic cis-cis natural acid (vitamin F1) as: alpha-lipoic acid,  alpha-linolenic acid,etc.

Alpha-lipoic acid

Food Sources: 

spinach, broccoli, tomato, peas, Brussels sprouts, rice bran

Mechanism of action:

Alpha-lipoic acid acts as a lipophilic free radical scavenger. Dihydrolipoic acid (DHLA), a reduced form of lipoic acid, has more potent antioxidant effects. It can assist in repairing oxidative damage and regenerate endogenous antioxidants such as vitamin C, vitamin E, and glutathione.

Both DHLA and lipoic acid also have metal chelating capacities.

As a lipoamide, alpha-lipoic acid functions as a cofactor in various multienzyme systems involved in the decarboxylation of alpha-keto acids such as pyruvate. ^{(13) (14) (15)}

Alpha-lipoic acid caused cell cycle arrest in G0/G1 in FaDu and Jurkat human tumor cell lines ⁽¹⁾.

Alpha lipoic acid was also found to scavenge reactive oxygen species in MCF-7 breast cancer cells ⁽¹⁶⁾.

Reduction of reactive oxygen species was then followed by cancer cell growth arrest and apoptosis ⁽¹⁶⁾.

For studies quoted see http://www.mskcc.org/cancer-care/herb/alpha-lipoic-acid  

Other studies:

Studies on Alpha–Lipoic Acid and cancer –  MANY STUDIES

science.naturalnews.com/A/Alpha–Lipoic_Acid_and_cancer.html

The natural antioxidant Alpha-Lipoic Acid induces p27(Kip1)-dependent cell cycle arrest and apoptosis in MCF-7 human breast cancer cells.
2010 European journal of pharmacology

Restoration of functional defects in peripheral blood mononuclear cells isolated fromcancer patients by thiol antioxidants Alpha-Lipoic Acid and N-acetyl cysteine.
2000 International journal of cancer. Journal international du cancer

alpha-Lipoic acid: the potential for use in cancer therapy.
2008 Neoplasma

Alpha-Lipoic Acid reduces matrix metalloproteinase activity in MDA-MB-231 human breast cancer cells.
2010 Nutrition research (New York, N.Y.)

Alpha-Lipoic Acid induces apoptosis in human colon cancer cells by increasing mitochondrial respiration with a concomitant O2-*-generation.
2005 Apoptosis : an international journal on programmed cell death

Mechanism of alpha-lipoic acid-induced apoptosis of lung cancer cells.
2009 Annals of the New York Academy of SciencesThe long-term survival of a patient with pancreatic cancer with metastases to the liver after treatment with the intravenous alpha-lipoic acid/low-dose naltrexone protocol.
2006 Integrative cancer therapiesCytoprotection against Cr(6+)-induced DNA damage by alpha-lipoic acid: implications in reducing occupational cancer risk.
2009 Mutagenesis

Studies on Alpha-Lipoic Acid and Apoptotic

http://science.naturalnews.com/A/Alpha-Lipoic_Acid_and_Apoptotic.html

Alpha-Linolenic Acid(omega 3)

Food sources:flaxseed, avocado, walnuts, almonds, fishoils

Mechanism of action:

The alpha-linolenic acid (vitamin F), for instance, is a cis-polyunsaturated fatty acid that is contained in linseed cold-pressed oil: it is transformed into EPA and DHA (Omega-3 fatty acids) and is quite effective against malignant tumours, as shown by Pardini ( 1647 );

Moreover, Noguchi has proved that Omega-3 fatty acids, unlike Omega-6 fatty acids, help reduce tumour masses, although

Omega-6 fatty acids are unsaturated fatty acids, too ( 1654 ).

see  last page of DATABASE for studies quoted.

Studies on Alpha–Linolenic Acid and cancer –  MANY STUDIES

science.naturalnews.com/A/Alpha–Linolenic_Acid_and_cancer.html

Alpha-Linolenic Acid and the risk of prostate cancer. What is the evidence?
2004 The Journal of urology

Low Alpha-Linolenic Acid content of adipose breast tissue is associated with an increased risk of breast cancer.
2000 European journal of cancer (Oxford, England : 1990)

A prospective study of dietary Alpha-Linolenic Acid and the risk of prostate cancer(United States).
2006 Cancer causes & control : CCC

The relation of Alpha-Linolenic Acid to the risk of prostate cancer: a systematic review and meta-analysis.
2009 The American journal of clinical nutrition

Alpha-Linolenic Acid and risk of prostate cancer: a case-control study in Uruguay.
2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

HER2 (erbB-2)-targeted effects of the omega-3 polyunsaturated fatty acid, Alpha-Linolenic Acid (ALA; 18:3n-3), in breast cancer cells: the „fat features” of the „Mediterranean diet” as an „anti-HER2 cocktail”.
2006 Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National cancer Institute of MexicoInhibitory effect of conjugated Alpha-Linolenic Acid from bifidobacteria of intestinal origin on SW480 cancer cells.
2009 LipidsProspective studies of dietary Alpha-Linolenic Acid intake and prostate cancer risk: a meta-analysis.
2010 Cancer causes & control : CCC

Dietary Alpha-Linolenic Acid is associated with reduced risk of fatal coronary heart disease, but increased prostate cancer risk: a meta-analysis.
2004 The Journal of nutrition

Growth-inhibitory and proapoptotic effects of Alpha-Linolenic Acid on estrogen-positive breast cancer cells.
2009 Annals of the New York Academy of Sciences

Comparison of stearidonic acid and Alpha-Linolenic Acid on PGE2 production and COX-2 protein levels in MDA-MB-231 breast cancer cell cultures.
2005 The Journal of nutritional biochemistry

Conjugation with Alpha-Linolenic Acid improves cancer cell uptake and cytotoxicity of doxorubicin.
2009 Bioorganic & medicinal chemistry letters

Does Alpha-Linolenic Acid in combination with linoleic acid influence liver metastasis and hepatic lipid peroxidation in BOP-induced pancreatic cancer in Syrian hamsters?
2000 Prostaglandins, leukotrienes, and essential fatty acids

Conjugation with alpha linolenic acid improves cancer cell uptake …

science.naturalnews.com/…/542108_Conjugation_with_alpha_linolenic_acid _improves_cancer_cell_uptake_and.html

Conjugation with alpha linolenic acid improves cancer cell uptake and cytotoxicity of doxorubicin.

Inhibitory effect of conjugated alpha linolenic acid from …

science.naturalnews.com/…/2462104_Inhibitory_effect_of_conjugated_alpha _linolenic_acid_from_bifidobacteria_of.html

Inhibitory effect of conjugated alpha linolenic acid from bifidobacteria of intestinal origin on SW480 cancer cells. Publication: Lipids Publication Date: 2009

Growth inhibitory and proapoptotic effects of alpha linolenic acid on …

science.naturalnews.com/…/1637124_Growth_inhibitory_and_proapoptotic_ effects_of_alpha_linolenic_acid_on.html

We tested the anticarcinogenic effect of Alpha–Linolenic Acid (ALA) as a single compound. To test the role of ALA in breast cancer cells (MCF-7), we analyzed …

omega 3 fatty acids on cancer risk: a systematic review.

science.naturalnews.com/…/2859351_Effects_of_omega_3_fatty_acids_on_ cancer_risk_a.html

Effects of omega 3 fatty acids on cancer risk: a systematic review.

Modulation of prostate cancer genetic risk by omega 3 and omega 6 …

science.naturalnews.com/…/3442786_Modulation_of_prostate_cancer_ genetic_risk_by_omega_3_and.html

Modulation of prostate cancer genetic risk by omega 3 and omega 6 fatty acids.

Effect of altering dietary omega 6 omega 3 fatty acid ratios on prostate cancer…

science.naturalnews.com/…/3897405_Effect_of_altering_dietary_omega_6_ omega_3_fatty_acid.html

Effect of altering dietary omega 6 omega 3 fatty acid ratios on prostate cancer membrane composition, cyclooxygenase 2, and prostaglandin E2.

Prostate tumor growth and recurrence can be modulated by omega3 intake…

science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html

Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids …prostate cancer (PCa) risk, whereas a diet rich in omega–3 decreases risk.

Beneficial effects of omega 3 long chain fatty acids in breast cancer …

science.naturalnews.com/…/296576_Beneficial_effects_of_omega_3_long_ chain_fatty_acids_in.html

Beneficial effects of omega 3 long chain fatty acids in breast cancer and cardiovascular diseases: voltage gated sodium channels as a common feature?

Fatty fish and fish omega 3 fatty acid intakes decrease the breast cancer risk…

science.naturalnews.com/…/1200161_Fatty_fish_and_fish_omega_3_fatty_ acid_intakes_decrease.html

Although it is believed that fish omega–3 fatty acids may decrease breast cancer risk, epidemiological evidence has been inconclusive. This study examined the …

Omega 3 fatty acid supplements in women at high risk of breast cancer …

science.naturalnews.com/…/759291_Omega_3_fatty_acid_supplements_in_ women_at_high_risk.html

BACKGROUND : Preclinical evidence of the preventive benefits of omega–3 (n-3) polyunsaturated fatty acids (PUFAs) in breast cancer continues to fuel interest …

Decreased severity of ovarian cancer and increased survival with omega 3.

science.naturalnews.com/…/256930_Decreased_severity_of_ovarian_cancer_ and_increased_survival_in_hens.html

Prevention and treatment of pancreatic cancer by omega 3 & curcumin in …

science.naturalnews.com/…/2359858_Prevention_and_treatment_of_ pancreatic_cancer_by_curcumin_in_combination.html

Prevention and treatment of pancreatic cancer by curcumin in combination with omega 3 fatty acids. Publication: Nutrition and cancer. Publication Date: 2008

The importance of the omega 6 omega 3 fatty acid ratio in colorectal cancer…

science.naturalnews.com/…/1101398_The_importance_of_the_omega_6_ omega_3_fatty_acid.html

A ratio of 2.5/1 reduced rectal cell proliferation in patients with colorectal cancer, whereas a ratio of 4/1 with the same amount of omega–3 PUFA had no effect.

Impact of postoperative omega 3 fatty acid supplemented parenteral …

science.naturalnews.com/…/1136096_Impact_of_postoperative_omega_3_ fatty_acid_supplemented_parenteral_nutrition.html

Impact of postoperative omega 3 fatty acid supplemented parenteral nutrition on clinical outcomes and immunomodulations in colorectal cancer patients.

Omega 3 reduce colorectal cancer risk in men by 66 percent  …

science.naturalnews.com/…/383170_Calcium_Mobilization_in_Ovarian_ Cancer_Cells_in_Response_to_Lysophospholipids.html

Herbal Remedy Essiac® and Cancer (press release) · Omega–3 fatty acids slash colorectal cancer risk in men by 66 percent · Fatty Acid Blood Tests Help …

Growth of human gastric cancer cells in nude mice is delayed by omega 3…

science.naturalnews.com/…/1201162_Growth_of_human_gastric_cancer_ cells_in_nude_mice_is.html

Growth of human gastric cancer cells in nude mice is delayed by a ketogenic diet supplemented with omega 3 fatty acids and medium chain triglycerides.

Neuroblastoma studies

science.naturalnews.com/Neuroblastoma.html

Cancer science. Omega–3 fatty acid supplementation delays the progression of neuroblastoma in vivo. International journal of cancer. Journal …

[omega 3 Fatty acid containing diet (Racol) reduces toxicity of chemotherapy…

science.naturalnews.com/…/945592_omega_3_Fatty_acid_containing_diet_ Racol_reduces_toxicity_of.html

omega 3 Fatty acid containing diet (Racol) reduces toxicity of chemoradiation therapy for patients with esophageal cancer].

 

Modulation of lipid rafts by Omega 3 fatty acids in inflammation and …

science.naturalnews.com/…/2597039_Modulation_of_lipid_rafts_by_Omega _3_fatty_acids_in.html

Modulation of lipid rafts by Omega 3 fatty acids in inflammation and cancer: implications for use of lipids during nutrition support.

 

Omega 3, on their own, are crucial for a cancer patient:

-promote optimal functioning of the immune system.

A team of researchers based in China have published results of their work on omega-3 and breast cancer risk in the British Medical Journal (BMJ), stating that a high intake of fatty acids found in fish is associated with a reduction 14 percent risk of breast cancer later in life. To conduct their study, the scientists reviewed and analyzed a group of 26 studies in the United States, Europe and Asia, which included more than 800,000 participants and 20,000 cases of breast cancer.

–promote natural cancer cell death and reduce cancer risk.

After analyzing the data to determine the impact of long chain fats on the risk of breast cancer, the researchers determined the difference between the lowest and highest intake category of marine polyunsaturated fatty acids was associated with a 14 percent reduced risk of disease .

In addition, for an increase of 0.1 grams per day of consumption of omega-3 fatty acids in fish, the risk of breast cancer is reduced by 5 percent. These results clearly demonstrate a significantly reduced risk of affecting the development of breast cancer and future progression of the disease.

The study authors concluded „Our current study provides solid evidence and robust as omega3 are inversely associated with risk of breast cancer. „

Scientists understand from previous studies that omega-3 fats, DHA and EPA, are preferentially accumulated in cell membranes and prevent cancer development through increased transfer of nutrients and oxygen to the cell nucleus.

With DHA metabolism, metabolites are produced that destroy cancer cells. Researchers believe that this mechanism is responsible for reducing the risk of cancers found in this study. (1200-2400 mg EPA / DHA combined daily to reduce the risk associated with breast cancer and other disease lines). Higher doses are required in cancer.

-omega 3 stops spread of cancer

Omega-6, found in most vegetable oils, increased spread of cancer cells in the bone marrow.

Nevertheless, the spread was blocked by omega-3

Comments Dr. Mercola:

„The evidence continues to grow. Not all unsaturated fats are good for you. Even if they come from such healthy foods such as sunflower seeds grown biodynamic. See, most vegetable oils are rich in omega-6 fat and very few people are deficient in these fats.

In addition, omega-3 provides a wonderful benefits for patients with prostate cancer by blocking the functioning of omega-6 fats that cancer cells use them as an energy source. (Therefore, only during cancer your treatment, avoid omega 6 polyunsaturated fats because the thinner blood vessels supply  tumors. thin blood vessels in the body are necessary for optimal functioning, but avoid in cancer high doses of omega-6) This happens at the molecular level when omega-3  (EPA  andDHA), reduces the action of a metabolite of the omega-6 arachidonic acid, prostaglandin E2. The sole point agreed on this study is that scientists have recommended omega 3 as a preventative for prostate cancer as and to stop its development. „

Dr. Clapp compiled a comprehensive program that has achieved national recognition USA. 1990 –was a very successful year of the program – had 16 cures of prostate cancer based on deep cleansing of toxins accumulated in the body and prostate, then flooding the cells with omega 3 fats. „Perhaps the most interesting aspect of this study is clear evidence of the mechanisms that omega-6 fats stimulate the growth of prostate cancer, clearly debunking industry hypethat  vegetable oils are good for our health. „

We saw dramatically reduced overall inflammation (measured by CRP testing) and especially in reducing enlarged prostate, reducing also any tumor mass, monitored / evaluated by Doppler sonograms.

-Omega 3 promote apatosis(programmed cell death) in cancer cells

Two new studies suggest that omega-3 fatty acids may inhibit the growth of liver cancer cells, suggesting that this may be an effective therapy to treat and prevent liver cancer.

Like vitamin B17/laetrile,and other natural substances(turmeric, ginger) omega 3 trigger apatosis.

The first study examined the effects of omega-3 and omega-6 fats in hepatocellular carcinoma cells .

Carcinoma represent 90 percent of all cases of liver cancer and is usually rapidly fatal. Omega-6 had no effect on cancer cells, but omega-3 – as  (DHA) and (EPA) – induced apoptosis (programmed cell death).

In the second study, omega-3 fats have been shown as effective in the treatment of cholangiocarcinoma tumor cells, an aggressive and fatal type of cancer that forms in the liver bile ducts.

Researchers said omega-3 fatty acids may not only be an effective treatment for liver cancer, but could also protect the liver from hepatic steatosis, chronic liver disease thought to be a precursor to cancer.

These above are just a few specific examples of cancer and Omega3 .Omega are great in any cancer.

Omega-3 Builds Muscle Mass in Cancer Patients, thus PREVENT and HELP IN CACHEXIA: http://articles.mercola.com/sites/articles/archive/2011/03/24/omega3-builds-muscle-mass-in-cancer-patients.aspx

Omega 3 fats are highly recommended and among the few fats accepted in cancer (either from seeds, nuts, avocado or fish, although fish oils might be contaminated with heavy metals from the fish )

These and many other roles that help in cancer, including stimulation of the immune system, reducing inflammation , direct relationship to sun tolerance by the body and regulation of vitamin D levels (often those with cancer can not tolerate the sun, therefore can not benefit either its energy or the important natural Vitamin D, the sun being main source of vitamin D, but omega 3 fixes this, also regulating levels of vitamin D)

http://articles.mercola.com/sites/articles/archive/2010/07/27/omega3s-reduce-your-risk-of-breast-cancer.aspx

See also:

For prostate cancer risk and omega3 controversy see this articles:

http://articles.mercola.com/sites/articles/archive/2013/07/31/omega-3-fats.aspx

http://articles.mercola.com/sites/articles/archive/2001/07/21/flaxseed-part-two.aspx

The relation of alpha linolenic acid to the risk of prostate cancer: a …

science.naturalnews.com/…/520717_The_relation_of_alpha_linolenic_acid_ to_the_risk_of.html

The relation of alpha linolenic acid to the risk of prostate cancer: a systematic review and meta analysis.

Prostate tumor growth and recurrence can be modulated by omega3 intake…

science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html

Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids …prostate cancer (PCa) risk, whereas a diet rich in omega–3 decreases risk.

Much more about  OMEGA 3 as well a POWERFUL (stage IV) cancer treatment based on AWARDED 2 NOBEL PRIZES  in my book.

============================================================================================

Vitamin K

 (i.e: in algae, greens, grapes, etc) Also see antagonists such as cinnamon, etc,

Studies on Vitamin K and cancer (MANY STUDIES)

science.naturalnews.com/V/Vitamin_K_and_cancer.html

Dietary Vitamin K intake in relation to cancer incidence and mortality: results from the Heidelberg cohort of the European Prospective Investigation into cancer and Nutrition (EPIC-Heidelberg).
2010 The American journal of clinical nutrition

Dietary intake of Vitamin K and risk of prostate cancer in the Heidelberg cohort of the European Prospective Investigation into cancer and Nutrition (EPIC-Heidelberg).
2008 The American journal of clinical nutrition

Serum undercarboxylated osteocalcin as biomarker of Vitamin K intake and risk of prostate cancer: a nested case-control study in the Heidelberg cohort of the European prospective investigation into cancer and nutrition.
2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

Vitamin K and childhood cancer: analysis of individual patient data from six case-control studies.
2002 British journal of cancer

Vitamin K and childhood cancer: a report from the United Kingdom Childhoodcancer Study.
2003 British journal of cancer

Long-term use of Vitamin K antagonists and incidence of cancer: a population-based study.
2011 BloodCancer prevention and Vitamin K antagonists: an overview.
2010 Thrombosis researchSorafenib combined Vitamin K induces apoptosis in human pancreatic cancer cell lines through RAF/MEK/ERK and c-Jun NH2-terminal kinase pathways.

2010 Journal of cellular physiology

Efficacy of low- molecular- weight- heparin versus Vitamin K antagonists for long term treatment of cancer-associated venous thromboembolism in adults: a systematic review of randomized controlled trials.
2009 Thrombosis research

A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).
2004 Hepato-gastroenterology

Low-molecular-weight heparins are superior to Vitamin K antagonists for the long term treatment of venous thromboembolism in patients with cancer: a cochrane systematic review.
2008 Journal of experimental & clinical cancer research : CR

Oxygen free radical generating mechanisms in the colon: do the semiquinones ofVitamin K play a role in the aetiology of colon cancer?
2001 Biochimica et biophysica acta

A study of the prevalence of Vitamin K deficiency in patients with cancer referred to a hospital palliative care team and its association with abnormal haemostasis.
2008 Journal of clinical pathology

Production of superoxide and dissipation of mitochondrial transmembrane potential by vitamin K2 trigger apoptosis in human ovarian cancer TYK-nu cells.
2006 Apoptosis : an international journal on programmed cell death

Vitamin K2-mediated apoptosis in cancer cells: role of mitochondrial transmembrane potential.
2008 Vitamins and hormones

Vitamins K2, K3 and K5 exert antitumor effects on established colorectal cancer in mice by inducing apoptotic death of tumor cells.
2007 International journal of oncology

Inhibitory effects of vitamin K3 on DNA polymerase and angiogenesis.
2008 International journal of molecular medicine

A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).2004 Hepato-gastroenterology

Vitamins K2, K3 and K5 exert antitumor effects on established colorectal cancer in mice by inducing apoptotic death of tumor cells.2007 International journal of oncology

Production of superoxide and dissipation of mitochondrial transmembrane potential by vitamin K2 trigger apoptosis in human ovarian cancer TYK-nu cells. 2006 Apoptosis : an international journal on programmed cell death

Vitamin K2-mediated apoptosis in cancer cells: role of mitochondrial transmembrane potential. 2008 Vitamins and hormones Inhibitory effects of vitamin K3 on DNA polymerase and angiogenesis. 2008 International journal of molecular medicine

Vitamin K antagonists

Cancer prevention and Vitamin K antagonists: an overview.

2010 Thrombosis research

Long-term use of Vitamin K antagonists and incidence of cancer: a population-based study.

2011 Blood

Efficacy of low- molecular- weight- heparin versus Vitamin K antagonists for long term treatment of cancer-associated venous thromboembolism in adults: a systematic review of randomized controlled trials.

2009 Thrombosis research

Low-molecular-weight heparins are superior to Vitamin K antagonists for the long term treatment of venous thromboembolism in patients with cancer: a cochrane systematic review.

2008 Journal of experimental & clinical cancer research : CR

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Scientific Studies Database PAGE 2

CLICK AICI pentru  limba ROmana (DACA nu e deja tradusa)

PAGE 2 CONTENTS:

• Vitamins in alphabetical order:

1. Vitamin A
2. B Vitamins
   Vitamin B2 – Riboflavin, Vitamin B3(Niacin), Vitamin B5 ( pantotenic acid )(respiratory vitamins), Vitamin B6,B12, B17
3. Vitamin C (very important vitamin, also respiratory vitamin)
4. Vitamin D (very important vitamin)
5. Vitamin E (respiratory vitamin)
6. Vitamin F(including Omega 3)
7. Vitamin K & Vitamin K antagonists

Vitamins in alphabetical order

Vitamin A

There is an extensive bibliography on the anti-tumoral action of Vitamin A.

In particular the combined use of Retinoids in the proper proportions (beta-Carotene: retinol = 4 : 1), establishes a synergism which is higher than the sum of the single components.

Studies on Vitamin A and cancer (MANY STUDIES -see NOTES below)

science.naturalnews.com/V/Vitamin_A_and_cancer.html

NOTE 1:

The link above contains multiple studies and the same do many links below( I will try to indicate by ”MANY STUDIES”)

• Vitamin A, retinoids and carotenoids as chemopreventive agents for prostate cancer.
  2004 The Journal of urology
• An international evaluation of the cancer preventive potential of vitamin A.
  1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Association of dietary vitamin A, carotenoids, and other antioxidants with the risk of ovarian cancer.
  2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Association of vitamin A, vitamin C and zinc with laryngeal cancer.
  2003 Indian journal of cancer
• Vitamin A, retinol, and carotenoids and the risk of gastric cancer: a prospective cohort study.
  2007 The American journal of clinical nutrition
• The effect of Vitamin A status in children treated for cancer.
  2008 Pediatric hematology and oncology
• Oxidant, Vitamin A and angiogenic markers in laryngeal cancer patients.
  2003 The Journal of laryngology and otologyThe role of antioxidants and Vitamin A in ovarian cancer: results from the Women’s Health Initiative.
  2008 Nutrition and cancer
• Can Vitamin A modify the activity of docetaxel in MCF-7 breast cancer cells?
  2007 Folia histochemica et cytobiologica / Polish Academy of Sciences, Polish Histochemical and Cytochemical Society
• Does Vitamin A prevent high-dose-methotrexate-induced D-xylose malabsorption in children with cancer?2004 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer
• Intake of vitamin A-rich foods and lung cancer risk in Taiwan: with special reference to garland chrysanthemum and sweet potato leaf consumption.
  2007 Asia Pacific journal of clinical nutrition
• Vitamin A and apoptosis in prostate cancer.
  2002 Endocrine-related cancer
• Skin, sun, and vitamin A: from aging to cancer.
  2001 The Journal of dermatology
• Vitamin A metabolism is impaired in human ovarian cancer.
  2009 Gynecologic oncology
• Effectiveness of Vitamin A acetate for enhancing the production of lung cancerspecific monoclonal antibodies.
  1999 Cytotechnology
• A population-based case-control study of carotenoid and Vitamin A intake and ovarian cancer (United States).
  2001 Cancer causes & control : CCC
• Recent advances in the use of Vitamin A (retinoids) in the prevention and treatment of cancer.
  2000 Nutrition (Burbank, Los Angeles County, Calif.)
• Fruit, vegetable, Vitamin A intakes, and prostate cancer risk.
  2008 Prostate cancer and prostatic diseases
• The Vitamin A family can significantly decrease the expression of ERbeta of ERs positive breast cancer cells in the presence or absence of ER ligands and paclitaxel.
  2009 Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology
• Induction of apoptosis in maxillary sinus cancer cells by 5-fluorouracil, Vitamin A and radiation (FAR) therapy.
  1999 European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology – Head and Neck Surgery
• EUROSCAN, a randomized trial of Vitamin A and N-acetylcysteine in patients with head and neck cancer or lung cancer. For the EUropean Organization for Research and Treatment of cancer Head and Neck and Lung cancer Cooperative Groups.
  2000 Journal of the National cancer Institute
• Rapid HPLC method for the determination of Vitamin A and E and cotinine concentration in human serum in women with CIN and cervical cancer.
  2009 Ginekologia polska
• Estradiol and tamoxifen differently affects the inhibitory effects of Vitamin A and their metabolites on the proliferation and expression of alpha2beta1 integrins in MCF-7 breast cancer cells.
  2009 Advances in medical sciences
• Renaissance of the biologically active Vitamin A derivatives: established and novel directed therapies for cancer and chemoprevention.
  2003 Current pharmaceutical design
• Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
  2009 Asian Pacific journal of cancer prevention : APJCP
• Whole-food sources of Vitamin A more effectively inhibit female rat sexual maturation, mammary gland development, and mammary carcinogenesis than retinyl palmitate.
  2007 The Journal of nutrition
• [Chemoprevention and FAR therapy regimen comprised of 5-fluorouracil, Vitamin Aand radiation].
  2001 Gan to kagaku ryoho. cancer & chemotherapy

Prostate cancer:

• Vitamin A and apoptosis in prostate cancer. 2002 Endocrine-related cancer
• Fruit, vegetable, Vitamin A intakes, and prostate cancer risk. 2008 Prostate cancer and prostatic diseases
• Vitamin A, retinoids and carotenoids as chemopreventive agents for prostate cancer. 2004 The Journal of urology

Breast cancer:

• The Vitamin A family can significantly decrease the expression of ERbeta of ERs positive breast cancer cells in the presence or absence of ER ligands and paclitaxel. 2009 Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology
• Estradiol and tamoxifen differently affects the inhibitory effects of Vitamin A and their metabolites on the proliferation and expression of alpha2beta1 integrins in MCF-7 breast cancer cells. 2009 Advances in medical sciences
• Can Vitamin A modify the activity of docetaxel in MCF-7 breast cancer cells?                                                                                             2007 Folia histochemica et cytobiologica / Polish Academy of Sciences, Polish Histochemical and Cytochemical Society
• Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology                                                                                                   2009 Asian Pacific journal of cancer prevention : APJCP

Ovarian cancer:

• A population-based case-control study of carotenoid and Vitamin A intake and ovarian cancer (United States).                            2001 Cancer causes & control : CCC
• The role of antioxidants and Vitamin A in ovarian cancer: results from the Women’s Health Initiative.                                              2008 Nutrition and cancer
• Association of dietary vitamin A, carotenoids, and other antioxidants with the risk of ovarian cancer.                                           2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Vitamin A metabolism is impaired in human ovarian cancer. 2009 Gynecologic oncology

Lung cancer:

• Effectiveness of Vitamin A acetate for enhancing the production of lung cancer specific monoclonal antibodies. 1999 Cytotechnology
• Intake of vitamin A-rich foods and lung cancer risk in Taiwan: with special reference to garland chrysanthemum and sweet potato leaf consumption. 2007 Asia Pacific journal of clinical nutrition

Lung, head & neck cancers:

• EUROSCAN, a randomized trial of Vitamin A and N-acetylcysteine in patients with head and neck cancer or lung cancer. For the EUropean Organization for Research and Treatment of cancer Head and Neck and Lung cancer Cooperative Groups.

2000 Journal of the National cancer Institute

Cervical cancer:

• Rapid HPLC method for the determination of Vitamin A and E and cotinine concentration in human serum in women with CIN and cervical cancer. 2009 Ginekologia polska

Gastric cancer:

• Vitamin A, retinol, and carotenoids and the risk of gastric cancer: a prospective cohort study.                                                           2007 The American journal of clinical nutrition

Laryngeal cancer:

• Association of vitamin A, vitamin C and zinc with laryngeal cancer. 2003 Indian journal of cancer
• Oxidant, Vitamin A and angiogenic markers in laryngeal cancer patients. 2003 The Journal of laryngology and otology

Children cancer:

• The effect of Vitamin A status in children treated for cancer. 2008 Pediatric hematology and oncology
• Does Vitamin A prevent high-dose-methotrexate-induced D-xylose malabsorption in children with cancer?                                 2004 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

Maxillary sinus cancer :

• Induction of apoptosis in maxillary sinus cancer cells by 5-fluorouracil, Vitamin A and radiation (FAR) therapy. 1999 European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology – Head and Neck Surgery
• [Chemoprevention and FAR therapy regimen comprised of 5-fluorouracil, Vitamin A and radiation].                                                                       2001 Gan to kagaku ryoho. cancer & chemotherapy

Studies on Vitamin A and Antiproliferative

http://science.naturalnews.com/V/Vitamin_A_and_Antiproliferative.html

Studies on Vitamin A and radiation

http://science.naturalnews.com/V/Vitamin_A_and_radiation.html

There is an extensive bibliography on the anti-tumoral action of Vitamin  A cited from LAST PAGE OF THIS DATABASE: 1,3,10,13,14,17,18,23,24,28,34,35,36,45,56,69,73,76,92,93,94,100,106,111,120,121,129,131,137,138,165,187,200,202,203,208,209,21,213,214,216,218,222,235,255,256,257,263,264,265,266,280,282,286,287,288,298,303,304,305,307,313,315,322,324,325,326,334,338,340,341,347,38,352,354,362,363,365,382,383,385,390,398,402,404,405,409,410,420,425,426,427,445,446,447,448,454,457,461,463,468,469,470,471,473,477,48,493,508,512):

This studies conclude  the following about vitamin A:

Anti-tumoral action in general

Vitamin A and retinoids have an anti neoplastic action, shown both in vivo and in vitro, in various tumors: basilomas, scaly carcinomas, melanomes, skin cancers, fungoid mycosis, acute promyelocyte leucemia, ovarian cancer, breast cancer, lung cancer, cancer of the bladder and follicular carcinomas of the thyroid.

Preventative action

Anti-tumoral action has been amply shown indirectly, that is with a preventative purpose. Various  studies have, in fact, shown that low plasmatic levels of beta-Carotene, vitamin C and vitamin E are  connected to an increase in the incidence of lung cancer.

Direct action at a receptor level

Only beta RAR (retinoic acid receptor) seems to be involved. More specifically, vitamin A (and its derivatives) acts by binding with specific receptors. In this way it can both inhibit the proteic synthesis of DNA and RNA, and also perform an anti-promotion  action to determine the return of a cellular differentiation. Furthermore its ability to inhibit oncogenes should be noted.

Apoptosis induction 

Apoptosis means the activation of specific endonucleases which break up the DNA, acting at a level of nucleosomic sites that make up the primary structural unit of the nuclear cromatine of the cell.  Vitamin A and retinoids in general can induce apoptosis in neoplastic cells, by activating intracellular proteolytic enzymes, called caspase 2 and caspase 3, which provoke deterioration  through proteolysis by a transcription factor, called Spl.  If this basal cellular transcription is altered, death is caused by Apoptosis.

In prostate cancer the retinoids intervene by reducing the level of the bcl 2 gene, whose function is to protect the cells from death by Apoptosis.  Carotenoids induced apoptosis in prostate cancer

Dă clic pentru a accesa carotenoidi%20sono%20fattori%20attivi%20contro%20il%20cancro%20della%20prostata.pdf

Inhibition of the cellular cycle

The retinoids block the passage of the cell from phase G1 to phase S (reducing the activity of a protein, called cycline D1): this passage, if not blocked, would lead the cell to mytosis.

Reduction of phosphorilation

Retinoids intervene in cancer causing a reduction in phosphorilation of the pRb, thus increasing survival; this protein is active in suppressing cellular growth.

Synergetic interaction with interferons

A second mechanism, apart from the proteic-enzymatic one, can be traced back to the intervention of interferons which, like retinoids, act as anti-proliferative factors. In practice, acting synergetically, they induce the expression of proteins capable of inhibiting neoplastic cellular proliferation.

The intake of natural carotenoids from food 

There is very little evidence to show that carotenoids taken in from food can increase the levels ofvitamin A: an extra portion a day of green leaf vegetables is not able to increase the hematic level of vitamin A; on the contrary, a mixed diet of foods particularly rich in beta-Carotene will give a  significant increase in vitamin A present in the blood. (627).

If you oblige healthy individuals to eat carrots (270g), broccoli (600g) or tomato juice (180g) it does not establish any significant changes in the hematic levels of the carotenoids: you only find an extremely wide variation (even up to 3-4 times) in the efficiency of gastro-intestinal absorption of  the carotenoids and therefore in their subsequent bio-availability at a hematic level (628) .

Optimal values of anti-oxidants in normal individuals

Vitamin C: >50 microMols/Liter

Vitamin E: >30 microMols/Liter

Vitamin A: >2.2 microMols/Liter

Beta-Carotene: >0.4 microMols/Liter

Association of vitamin A with vitamin E

In an experimental model of a cellular membrane the possibility of a positive interaction between anti-oxidant liposollubles such as beta-Carotene and alpha-Tocopherol has been investigated; the  result showed that there exists a synergetic action between beta-Carotene and alpha-Tocopherol  together which inhibits the processes of lipidic peroxidization compared with when they are used alone (629). THUS:

NOTE 2:

Vitamins , antioxidants and nutrients should be taken from WHOLE, ORGANIC(note below) FOODS (as much as possible) NOT from supplements as they need other vitamins and nutrients for proper usage.

Whole-food sources of Vitamin A more effectively inhibit female rat sexual maturation, mammary gland development, and mammary carcinogenesis than retinyl palmitate. 2007 The Journal of nutrition

The difference between synthetic vitamins and natural vitamins

The difference between synthetic vitamins and natural ones can be easily exemplified by the experimental case of synthetic beta-Carotene (made up entirely of isomeric trans-beta-Carotene), and of natural beta-Carotene (made up of both isomeric trans-beta-Carotene and isomeric cis-beta- Carotene): the study showed a strong discrimination between the two isomers, with a serious decrease (impoverishment) induced at the level of Lycopene present in the LDLs (…). THUS:

3.It would be better to use the NATURAL (juice) ORGANIC WHOLE foods rich in vitamin A such as  Daucus carota (carrots ), avocados, cantalopues, etc. rather than the synthetic pharmaceutical vitamin products based on Vitamin A.

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B Vitamins 

Studies on Vitamins B and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B_and_cancer.html

• Plasma folate, vitamin B-6, vitamin B-12, and risk of breast cancer in women.
  2008 The American journal of clinical nutrition
• Low intake of vitamin B-6 is associated with increased risk of colorectal cancer in Japanese men.
  2007 The Journal of nutrition
• Dietary folate, methionine, riboflavin, and vitamin B-6 and risk of sporadic colorectal cancer.
  2008 The Journal of nutrition
• A prospective study of dietary folate and Vitamin B and colon cancer according to microsatellite instability and KRAS mutational status.
  2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Effect of combined folic acid, vitamin B6, and vitamin B12 on cancer risk in women: a randomized trial.
  2008 JAMA : the journal of the American Medical Association
• Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer.
  2003 Journal of the National cancer Institute
• Cancer incidence and mortality after treatment with folic acid and vitamin B12.
  2009 JAMA : the journal of the American Medical Association
• One-carbon metabolism-related nutrients and prostate cancer survival.
  2009 The American journal of clinical nutrition
• Circulating concentrations of folate and vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study.
  2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Reduced thiamine (vitamin B1) levels following gastrectomy for gastric cancer.
  2002 Gastric cancer : official journal of the International Gastric cancer Association and the Japanese Gastric cancer Association
• Dietary folate intake, MTHFR genetic polymorphisms, and the risk of endometrialcancer among Chinese women.
  2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Localized folate and vitamin B-12 deficiency in squamous cell lung cancer is associated with global DNA hypomethylation.
  2000 Nutrition and cancer
• Dietary factors of one-carbon metabolism and prostate cancer risk.
  2006 The American journal of clinical nutrition
• Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.
  2009 BMC cancer
• Folate, vitamin B(6), and vitamin B(12) intake and the risk of breast cancer among Mexican women.
  2006 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Dietary folate intake and breast cancer risk: results from the Shanghai Breast cancerStudy.
  2001 Cancer research
• Vitamin B6 and risk of colorectal cancer: a meta-analysis of prospective studies.
  2010 JAMA : the journal of the American Medical Association
• One-carbon metabolism and prostate cancer risk: prospective investigation of seven circulating B vitamins and metabolites.
  2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Folate, vitamin B6, multivitamin supplements, and colorectal cancer risk in women.
  2006 American journal of epidemiology
• Relationship of folate, vitamin B-6, vitamin B-12, and methionine intake to incidence of colorectal cancers.
  2002 Nutrition and cancer
• Association of the B-vitamins pyridoxal 5′-phosphate (B(6)), B(12), and folate with lungcancer risk in older men.
  2001 American journal of epidemiology
• Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.
  2009 Nutrition and cancer
• The role of vitamin B6 in the prevention of haematological toxic effects of linezolid in patients with cancer.
  2008 The Journal of antimicrobial chemotherapy
• Methylenetetrahydrofolate reductase, diet, and risk of colon cancer.
  1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
  2005 International journal of cancer. Journal international du cancer
• Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study.
  2005 International journal of cancer. Journal international du cancer
• Vitamin B(12) and Folate Status in Head and Neck Cancer.
  2002 Asian Pacific journal of cancer prevention : APJCP
• Vitamins B2 and B6 and genetic polymorphisms related to one-carbon metabolism as risk factors for gastric adenocarcinoma in the European prospective investigation intocancer and nutrition.
  2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Associations of dietary methyl donor intake with MLH1 promoter hypermethylation and related molecular phenotypes in sporadic colorectal cancer.
  2008 Carcinogenesis
• Vitamin B6 points PC6 injection during acupuncture can relieve nausea and vomiting in patients with ovarian cancer.
  2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society

Studies on Vitamin B and Antineoplastic

http://science.naturalnews.com/V/Vitamin_B_and_Antineoplastic.html

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Vitamin B2 – Riboflavin

Studies on Riboflavin and cancer

science.naturalnews.com/R/Riboflavin_and_cancer.html(MANY STUDIES in this link )

• Dietary folate, methionine, riboflavin, and vitamin B-6 and risk of sporadic colorectal cancer.
  2008 The Journal of nutrition
• Augmented antioxidant status in Tamoxifen treated postmenopausal women with breast cancer on co-administration with Coenzyme Q10, Niacin and Riboflavin.
  2008 Cancer chemotherapy and pharmacology
• Anti-angiogenic potential of CoenzymeQ10, Riboflavin and niacin in breast cancerpatients undergoing tamoxifen therapy.
  2008 Vascular pharmacology
• Biochemical characterization of Riboflavin carrier protein (RCP) in prostate cancer.
  2009 Frontiers in bioscience : a journal and virtual library
• Effect of coenzyme Q10, Riboflavin and niacin on serum CEA and CA 15-3 levels in breast cancer patients undergoing tamoxifen therapy.
  2007 Biological & pharmaceutical bulletin
• Serum cytokine levels of interleukin-1beta, -6, -8, tumour necrosis factor-alpha and vascular endothelial growth factor in breast cancer patients treated with tamoxifen and supplemented with co-enzyme Q(10), Riboflavin and niacin.
  2007 Basic & clinical pharmacology & toxicology
• Multidrug transporter ABCG2/breast cancer resistance protein secretes Riboflavin(vitamin B2) into milk.
  2007 Molecular and cellular biology
• Intracellular processing of Riboflavin in human breast cancer cells.
  2008 Molecular pharmaceutics
• Ameliorating effect of coenzyme Q10, Riboflavin and niacin in tamoxifen-treated postmenopausal breast cancer patients with special reference to lipids and lipoproteins.
  2007 Clinical biochemistry
• Effect of Coenzyme Q(10), Riboflavin and Niacin on Tamoxifen treated postmenopausal breast cancer women with special reference to blood chemistry profiles.
  2009 Breast cancer research and treatment
• Design of riboflavin-presenting PAMAM dendrimers as a new nanoplatform for cancer-targeted delivery.
  2010 Bioorganic & medicinal chemistry letters
• Co-enzyme Q10, Riboflavin and niacin supplementation on alteration of DNA repair enzyme and DNA methylation in breast cancer patients undergoing tamoxifen therapy.
  2008 The British journal of nutrition
• Riboflavin deficiency and esophageal cancer: a case control-household study in the Caspian Littoral of Iran.
  2005 Cancer detection and prevention
• [Apoptosis of human gastric cancer cell induced by photochemical riboflavin].
  2003 Ai zheng = Aizheng = Chinese journal of cancer
• Riboflavin carrier protein: a serum and tissue marker for breast carcinoma.
  2001 International journal of cancer. Journal international du cancer

Studies on Riboflavin and Anti-Angiogenic

http://science.naturalnews.com/R/riboflavin_and_Anti-Angiogenic.html

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Vitamin B3(Niacin)

Studies on Niacin and cancer

• Augmented antioxidant status in Tamoxifen treated postmenopausal women with breast cancer on co-administration with Coenzyme Q10, Niacin and Riboflavin.
  2008 Cancer chemotherapy and pharmacology
• Serum cytokine levels of interleukin-1beta, -6, -8, tumour necrosis factor-alpha and vascular endothelial growth factor in breast cancer patients treated with tamoxifen and supplemented with co-enzyme Q(10), riboflavin and niacin.
  2007 Basic & clinical pharmacology & toxicology
• Anti-angiogenic potential of CoenzymeQ10, riboflavin and Niacin in breast cancerpatients undergoing tamoxifen therapy.
  2008 Vascular pharmacology
• Effect of coenzyme Q10, riboflavin and Niacin on serum CEA and CA 15-3 levels in breast cancer patients undergoing tamoxifen therapy.
  2007 Biological & pharmaceutical bulletin
• Anti-invasive activity of Niacin and trigonelline against cancer cells.
  2005 Bioscience, biotechnology, and biochemistry
• Biochemical assessment of Niacin deficiency among carcinoid cancer patients.
  2005 The American journal of gastroenterology
• Ameliorating effect of coenzyme Q10, riboflavin and Niacin in tamoxifen-treated postmenopausal breast cancer patients with special reference to lipids and lipoproteins.
  2007 Clinical biochemistry
• Effect of Coenzyme Q(10), Riboflavin and Niacin on Tamoxifen treated postmenopausal breast cancer women with special reference to blood chemistry profiles.
  2009 Breast cancer research and treatment
• Co-enzyme Q10, riboflavin and Niacin supplementation on alteration of DNA repair enzyme and DNA methylation in breast cancer patients undergoing tamoxifen therapy.
  2008 The British journal of nutrition
• Niacin supplementation decreases the incidence of alkylation-induced nonlymphocytic leukemia in Long-Evans rats.
  2008 Nutrition and cancer
• Niacin and carcinogenesis.
  2003 Nutrition and cancer

• Other studies:

ANTI-invasive activities of Niacin against cancer  cells; http://science.naturalnews.com/…/3260742_Anti_invasive_activity_of_niacin_and_ trigonelline_against_cancer_cells.html

Studies on Niacin and breast cancer

http://science.naturalnews.com/N/Niacin_and_breast_cancer.html (MANY STUDIES in this link)  

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Vitamin B5 ( pantotenic acid )

Pantothenic Acid for Stage IV Breast Cancer Treatment Information

https://www.medify.com/…/pantothenic–acid-treatment-stage-iv-breast-ca…‎

Vitamins B2, B3,B5 are respiratory vitamins(increasing cellular oxygenation –  see above Otto Wartburg proposals).

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Vitamin B6

Studies on Vitamin B6 and cancer – Natural News Science (MANY STUDIES)

science.naturalnews.com/V/Vitamin_B6_and_cancer.html

• Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer.
  2003 Journal of the National cancer Institute
• Effect of combined folic acid, vitamin B6, and vitamin B12 on cancer risk in women: a randomized trial.
  2008 JAMA : the journal of the American Medical Association
• The role of Vitamin B6 in the prevention of haematological toxic effects of linezolid in patients with cancer.
  2008 The Journal of antimicrobial chemotherapy
• Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women.
  2009 BMC cancer
• Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan.
  2009 Nutrition and cancer
• Methionine and Vitamin B6 intake and risk of pancreatic cancer: a prospective study of Swedish women and men.
  2007 Gastroenterology
• Plasma Vitamin B6 and the risk of colorectal cancer and adenoma in women.
  2005 Journal of the National cancer Institute
• Plasma folate, vitamin B6, vitamin B12, and homocysteine and pancreatic cancer risk in four large cohorts.
  2007 Cancer research
• Folate, vitamin B6, multivitamin supplements, and colorectal cancer risk in women.
  2006 American journal of epidemiology
• Folate, vitamin B6, vitamin B12, and vitamin B2 intake, genetic polymorphisms of related enzymes, and risk of colorectal cancer in a hospital-based case-control study in Japan.
  2005 Nutrition and cancer
• Folate and Vitamin B6 intake and risk of colon cancer in relation to p53 expression.
  2008 Gastroenterology
• Vitamin B6 points PC6 injection during acupuncture can relieve nausea and vomiting in patients with ovarian cancer.
  2009 International journal of gynecological cancer : official journal of the International Gynecological cancer Society
• Vitamin B6 and risk of colorectal cancer: a meta-analysis of prospective studies.
  2010 JAMA : the journal of the American Medical Association
• Dietary Vitamin B6 intake and the risk of colorectal cancer.
  2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Prospective study of plasma Vitamin B6 and risk of colorectal cancer in men.
  2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Vitamin B6 intake, alcohol consumption, and colorectal cancer: a longitudinal population-based cohort of women.
  2005 Gastroenterology
• Relationship between dietary and supplemental intake of folate, methionine, Vitamin B6 and folate receptor alpha expression in ovarian tumors.
  2010 International journal of cancer. Journal international du cancer

Vitamin B12 

Studies on Vitamin B12 and cancer – Natural News Science(MANY STUDIES)

science.naturalnews.com/V/Vitamin_B12_and_cancer.html

Common polymorphisms in methylenetetrahydrofolate reductase gene are associated with risks of cervical intraepithelial neoplasia and cervical cancer in women with low serum folate and vitamin B12. 2011 Cancer causes & control : CCC Plasma folate, vitamin B6, vitamin B12, homocysteine, and risk of breast cancer. 2003 Journal of the National cancer Institute Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study. 2005 International journal of cancer. Journal international du cancer Plasma folate, vitamin B12, and homocysteine and prostate cancer risk: a prospective study. 2005 International journal of cancer. Journal international du cancer The risk of cervical cancer in relation to serum concentrations of folate, vitamin B12, and homocysteine. 2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Effect of combined folic acid, vitamin B6, and Vitamin B12 on cancer risk in women: a randomized trial. 2008 JAMA : the journal of the American Medical Association Plasma Vitamin B12 concentrations and the risk of colorectal cancer: a nested case-referent study. 2008 International journal of cancer. Journal international du cancer Dietary intake of folate, vitamin B6, and vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Brazilian women. 2009 BMC cancer Circulating concentrations of folate and Vitamin B12 in relation to prostate cancerrisk: results from the European Prospective Investigation into cancer and Nutrition study.2008 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Dietary intake of folate, vitamin B2, vitamin B6, vitamin B12, genetic polymorphism of related enzymes, and risk of breast cancer: a case-control study in Japan. 2009 Nutrition and cancer Cancer incidence and mortality after treatment with folic acid and vitamin B12. 2009 JAMA : the journal of the American Medical Association Plasma folate, vitamin B6, vitamin B12, and homocysteine and pancreatic cancer risk in four large cohorts. 2007 Cancer research Plasma homocysteine, folate, and Vitamin B12 levels in patients with laryngeal cancer. 2008 Archives of otolaryngology–head & neck surgery A polymorphism of the methionine synthase gene: association with plasma folate, vitamin B12, homocyst(e)ine, and colorectal cancer risk. 1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Folate, vitamin B6, vitamin B12, and vitamin B2 intake, genetic polymorphisms of related enzymes, and risk of colorectal cancer in a hospital-based case-control study in Japan. 2005 Nutrition and cancer Elevated serum Vitamin B12 levels associated with CRP as a predictive factor of mortality in palliative care cancer patients: a prospective study over five years. 2000 Journal of pain and symptom management Folate, Vitamin B12 and postmenopausal breast cancer in a prospective study of French women. 2006 Cancer causes & control : CCC Vitamin B12 deficiency: a new risk factor for breast cancer? 1999 Nutrition reviews Toxic epidermal necrolysis related to pemetrexed and carboplatin with Vitamin B12and folic acid supplementation for advanced non-small cell lung cancer. 2009 Onkologie Methylenetetrahydrofolate reductase C677T genotype affects promoter methylation of tumor-specific genes in sporadic colorectal cancer through an interaction with folate/vitamin B12 status. 2008 World journal of gastroenterology : WJG Efficacy and safety of two doses of pemetrexed supplemented with folic acid andVitamin B12 in previously treated patients with non-small cell lung cancer. 2008 Clinical cancer research : an official journal of the American Association forcancer Research Serum Vitamin B12 and folate status among patients with chemotherapy treatment for advanced colorectal cancer. 2009 Upsala journal of medical sciences Circulating folate, vitamin B12, homocysteine, Vitamin B12 transport proteins, and risk of prostate cancer: a case-control study, systematic review, and meta-analysis. 2010 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology Dietary folate and Vitamin B12 intake before diagnosis decreases gastric cancermortality risk among susceptible MTHFR 677TT carriers. 2010 Nutrition (Burbank, Los Angeles County, Calif.) Increased serum CA-15.3 levels in patients with megaloblastic anemia due to Vitamin B12 deficiency. 2004 Oncology

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Vitamin B17(Laetrile/ Amygdalin) (very important vitamin)

Note: “Laetrile” acronym for “LAEvomandeloniTRILE-glucoside”) as Amygdalin:

Laetrile has two molecules of glucose, Amygdalin has more.

Indeed, the chemical structure of Laetrile is D-1 mandelonitrile–beta-glucuronide, while for Amygdalin it is D-mandelonitrile-bi-glucoside.

In 1845, fifteen years after the first French scientific experiences, the French scientific journal “Gazette Medicale de Paris”, (1188) and afterwards the German journal “Journal für die Chirurgie und Augenheil-kunde”(1189), described the first case of metabolic therapy with vitamin B17 to “cure cancer”, created by Russian doctor Inosmetzeff, professor at the Imperial University of Russia in Moscow. Therapy was performed on a twenty-one-year-old boy affected by cancer, and consisted of 46 grams of Amygdalin administered for 3 months. Inosmetzeff had also cured a 48-year-old woman with extended metastases due to womb cancer. In 1845 this woman was still alive, 11 years after metabolic therapy with Amygdalin. In both cases, Inosmetzeff said that he never noticed any side effects with vitamin B17.

For instance, doctor Francisco Contreras, the current managing director of the Oasis of Hope hospital in Tijuana, Mexico, treated more that 60,000 patients with a vegetarian therapy and vitamin B17 in 35 years of activity (1187). http://www.mednat.cancro/Contreras.pdf

Doctor Ernesto Contreras has been using Laetrile since 1963, and thinks that

“…The majority of most frequent cancers, such as lung, breast, colon, ovarian, stomach,esophagus, prostate cancer, and lymphoma, can improve dramatically with Laetrile” (1187).

Clinical Case history

 

Amygdalin taken orally has been known to be a poison since ancient times, though amygdalin-laden black and brown bitter seeds were described as antitumor agents in the pharmacopeia of ancient China (1497)

Egyptian, Greek, Roman and Arabic physicians also used amygdalin to treat tumors (1498).

In a study conducted in 1958, Prof Marco Tasca, head of the radiology department of the Civil Hospital in Sanremo, treated 21 Italian terminally ill patients – 3 suffering from seminomas, 4 from breast cancers, 1 from womb cancer, 2 from laryngitis cancers, 7 from lung cancers, 1 from cancer of the oesophagus, 2 from stomach cancers, 1 from Hodgkin’s disease – with intramuscular injections of Laetrile. He noticed that patients showed good drug tolerance, their clinical conditions improved during the entire treatment period and only one month – on average – after the interruption of the therapy the neoplastic pathology resumed its progression. He pointed out only two complications:

hemorrhage and icterus. The former probably caused by necrotic eschars coming off the tissues, the latter induced by a direct toxic action on hepatic cells, which rarely happens though (5% of his case histories). The article is available in PDF format (1373) at http://www.fiocco59.altervista.org/images/tasca.pdf  or http://www.mednat.org/cancro/tasca.pdf.

In the 1966 report, Proceedings of the Ninth International Cancer Congress, Rossi cites a ten-year trial in Europe involving 150 patients that found „50 percent of all cases in treatment showed objective improvement” and concluded that laetrile was „an extremely useful chemotherapeutic drug.”(1382) http://fiocco59.altervista.org/vitamina_b_17.htm

In 1994, professor Dr .Binzel published the results he obtained treating patients with Laetrile between 1974 and 1991. His case history included 180 patients with primary cancer (with no metastasis and limited to only one organ or tissue). 131 patients were still alive in 1991, when the report was published. At that time, 58 patients had been followed for 2 to 4 years, while 80 of them had had a medical follow-up for 5 to 18 years. Out of the 42 patients that had died by 1991, 23 had died from cancer, 12 from “unrelated causes” and 7 of “unknown causes” (Binzel E.P.: “Alive and Well”). http://www.mednat.org/cancro/ALIVE_AND_WELL.pdf

Among patients with metastasis, 32 out of 108 had died from their disease, 6 from “unrelated causes” and 9 from “unknown causes”. Out of the 61 patients that were still alive in 1991, 30 had had a medical follow-up of 2-4 years, 31 had been followed for 5-18 years.

Doctor John A. Richardson’s case history of 1976 reports over 6,000 cases that show a positive effect of vitamin B17 against cancer. (1187)

There are 4,800 cases reported and carefully studied by doctor Ernesto Contreras. Those were selected among 10,000 case sheets collected in 14 years of experiences with Laetrile.

Doctor Paul Wedel from Oregon reported about 4,000 cases of metabolic treatment. He survived cancer himself with vitamin B17 and a diet similar to the gersonian one (1187).

1,000 cases were reported by doctor Manuel Navarro of Santo Tomas University in Manila, the Philippines. The Mexican government is even monitoring about 100 patients that are being treated with metabolic therapy and vitamin B17, under the guidance of doctor Mario Soto de Leon, medical director of the Cydel Clinic in Tijuana (1187).

In Germany, doctor Hans Nieper reported about 1,000 cases. (http://www.mwt.net/~drbrewer  )

It is interesting to notice that cases such as that of Mr. Glen Rutherford from Kansas, who healed completely in Tijuana, are recorded in tribunal archives as “cures” (1187).

Clinical Trial of Chemotherapeutic treatment of advanced cancers with Leatrile

Guidetti Ettore

Rossi Benedetto

Deckers Christian

Presented at the 9th International Cancer Congress in Tokyo, October 1966

From 1954 to 1966 we gave 150 patients the above-mentioned therapy, chiefy at San Cottolengo Hospital, Turin; DosioHospital, Milan; and Louvain University Cancer Institute. All patients were in the terminal stage of the disease, the majority of them prey to cachezia, and all other therapies had failed.

The following table summarizes the cases treated, classified according to the site of the tumor, and showing the number of patients for each degree of reaction to therapy. We use the sign ++ to denote patients who reacted in an objectively favourable manner, by which we mean diminution of volume of the tumor or at least all interruption of its evolution, improvement in the roentgenographic picture, and improvement in laboratory findings. The mark + and + indicates patients who showed a more or less distinct subjective improvement, and the mark – those who reacted negatively to the treatment.

Cases corresponding to ++ represent about 20% of those treated.

We again underline the fact that the majority of these cases were simultaneously subjected to an immunotype therapy, which might have some bearing on the number of positive results observed, grouped under the signs ++ and + totalling about half the number of cases treated.

Cancer Site        No. cases            ++    +    +-           –

–

Toruli tactiles                    26                           5             6             6             9

Breast                                  25                           3             8             7             7

Uterus                                 24                           7             7             4             6

Rectum                                               20                           2             9            2             7

Ovary (with infusion)    10                           2             2             2             4

Other types                       30                           9             7             2             12

Totals                                   135                        28           39           23           45

We have separately considered neoplasms of the pleura with effusion (15 cases), where the product was used direct by injection in the pleural cavity. In these cases we observed our best results, as generally we obtained reduction and then on occasion complete disappearance of the effusion, associated with a distinct improvement in the patients’ condition.

Conclusion:

On the basis of our clinical trial, we are able to state that L-mandelonitrile-beta-diglucoside may be considered an extremely useful chemotherapeutic drug for palliative medical treatment of malign neoplasms, from the standpoint both of its therapeutic effect and its very low toxicity.

B17/ Amygdalin studies science.naturalnews.com/amygdalin.html

How laetrile or B17 from apricot seeds kills only cancer cells ·

apricot studies science.naturalnews.com/apricot.html

Apricot Seeds Kill Cancer Cells without Side Effects

• Monographic Summary of AMYGDALIN
• Monographic Analysis of Amygdalin (over 70 pages)
• Nutritional Implications by Ernest T. Krebs, Jr. John Beard Memorial Foundation (Privately published: 1964).
• The Nitrilosides(Vitamin B-17) Their Nature, Occurence and Metabolic Significance (Antineoplastic Vitamin B-17) Ernst T. Krebs, Jr. Reprinted from the Journal of Applied Nutrition, Volume 22, Numbers 3 and 4, 1970.
• The Ultimate Cancer Conspiracy Vitamin B17 Laetrile by Joe Vialls.
• Ernest T. Krebs Jr. Biography (Discoverer of B-17).
• Success Stories – Laetrile Testimonials, Case Histories, Success Stories.
• THE UNITARIAN OR TROPHOBLASTIC THESIS OF CANCER by Ernest T. Krebs, Jr.,* Ernst T. Krebs, Sr.,** and Howard H. Beard*** (Reprinted From the Medical Record, 163:149-174, July 1950) MEDICAL RECORD AN INTERNATIONAL JOURNAL OF MEDICINE AND SURGERY Founded 1843 Vol. 163, No.7 JULY, 1950 Whole No.2809
• The Four Optional forms of Cancer Therapy Dr. Robert D. Sullivan, Department of Cancer Research at the Lahey Clinic Foundation.
• The Laetrile Quacks The names, professional standings, medical achievements, and clinical findings of some of the more prominent doctors who endorse Laetrile; the beneficial side-effects produced by its use; a suggested anti-cancer diet; and a brief description of vitamin B15.
• The Hoax Of The “Proven” Cancer Cures The effects of surgery and radiation in the treatment of cancer; a comparison showing that those who receive no treatment at all live just as long, if not longer, than those who are treated.
• Laetrile and Cyanide Read about the life saving substance called cyanide.
• Ralph Moss on Cancer Politics – Ralph Moss on Chemotherapy, Laetrile, Coley’s Toxins, Burzynski, & Cancer Politics – Laura Lee radio show, 1994.
• Metabolic Therapy in Cancer – METABOLIC THERAPY IN CANCER
  AND ITS OBJECTIVE The use of Amygdalin (Vitamin B-17) LAETRILE (Vitamin B-17) TREATMENT by Harold W. Manner, Ph.D. Metabolic Research Foundation.
• The Truth about Surgeries and Biopsies – According to Clinical Oncology for Medical Students and Physicians (published jointly by the University of Rochester School of Medicine and the American Cancer Society)
• Laetrile: Cancer Cure Or Quackery? – Excerpts from: Los Angeles Herald-Examiner, Sunday, July 21, 1974.

 

See also:  http://worldwithoutcancer.org.uk/success.html,   http://www.apricotsfromgod.info/journal.htm

Much more details(including treatments & CLINICAL CASES with B17) in my BOOK + DR. Binzel’s FREE EBOOK on this topic + Dr. Nacci’s FREE EBOOK on this topic.

NOTE:

Vitamins(including B group) should be taken from WHOLE FOODS(organic) (as much as possible),NOT from supplements as vitamins and nutrients need other vitamins and nutrients for proper usage within the body.

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Vitamin C (very important vitamin)

Vitamin C   is a respiratory vitamin- it stimulates the body to produce its own hydrogen peroxide (increasing cellular oxygenation –  see above Otto Wartburg proposals).

Therefore, megadoses of vitamin C are administered orally and intravenously by orthomolecular physicians to treat cancer.

60 references to studies published that show the importance of vitamin C:

www.doctoryourself.com / biblio_cameron.html http://www.doctoryourself.com/cancer_hoffer.html.

Even National Institutes of Health is actively sponsoring studies of high-dose intravenous vitamin C therapy.

Other MULTIPLE studies on  vitamin’s C cancer fighting effects: science.naturalnews.com/…/4854330_Vitamin_C_in_alternative_cancer_ treatment_historical_background.html

High doses of vitamin C andi cancer :

science.naturalnews.com/…/2387600_High_dose_vitamin_C_supplement_ use_is_associated_with_self.html

Vitamin C and breast cancer studies: science.naturalnews.com / V / Vitamin_C_and_breast_cancer.html

Vitamin C , vitamin E & selenium in breast cancer : science.naturalnews.com/…/1198191_Dietary_beta_carotene_vitamin_C_and _E_intake_and_breast.html science.naturalnews.com/…/3959947_Association_between_breast_cancer_ and_vitamin_C_vitamin_E_and.html

Vitamin C ,vitamin A & zinc  in laryngeal cancer  :

science.naturalnews.com/…/4508069_Association_of_vitamin_A_vitamin_C _and_zinc_with_laryngeal.html

Vitamin C &  gastric cancer:

science.naturalnews.com/…/3603872_Plasma_and_dietary_vitamin_C_levels _and_risk_of_gastric.html science.naturalnews.com/…/3628783_Prediagnostic_plasma_vitamin_C_ levels_and_the_subsequent_risk_of.html science.naturalnews.com/…/4790082_Effect_of_physiological_ concentrations_of_vitamin_C_on_gastric_cancer.html

Also see: http://orthomolecular.org/resources/omns/v04n19.shtml

There is also an extensive bibliography on the anti-tumoral action of vitamin C (25,33,47,54,83,91,122,129,181,197,202,218,244,246,270,299,311,335,339,367,404,405,414,415,416,489,496,510,511)

See the bibliography and studies quoted on last page of databse.

The pioneers of this oncological therapy were Pauling, who received the Nobel Prize for Chemistry, and the Italian Pantellini.

Ascorbic acid is mainly known for its ability to reduce metallic ions in various enzymatic processes and above all for its ability to act as an anti-oxidant agent, thus able to remove free radicals, reducing the damage caused at a genome level.

Furthermore it may be able to block the formation of nitrose at a gastro-intestinal level, as well as carrying out a preventative action on the formation of adenomatose polyps.

Even though ascorbic acid is well known for its collagene forming action, and the well known effect of scurvy in cases of reduction or absence of this acid in the diet, this vitamin is also important, alongside vitamins A and E as a first class anti-tumoral agent.

It is a respiratory vitamin(like vitamins E, B3, B5)  that helps increase cellular oxygen levels.

It reinforces the intercellular bond and forestalls the destructive action of the hyalurons produced by many neoplastic cells.

Vitamin C stimulates the Natural killer lymphocytes, it supports the macrophagic activity, the chemio-tactical mobility of the white corpuscles, the production of antibodies and the response of the T cytotoxic lymphocytes to the antigenes.

Cameron found very low plasmatic levels of vitamin C in cancer patients (0.26 mg/100 mL) compared to normal plasmatic values (54).

In other studies, 154 cancer patients, undergoing analysis, were found to have low levels of vitamin C not only in their plasma (0.31 mg/100 mL), but also in their leucocytes (15.9 mg/10 E+8), with a positive correlation (r = 0.42) between these two values; in particular the authors attributed the alteration in the immune response to the tumor, especially of the phagocytosis, to the low  concentration of vitamin C in the white blood corpuscles.

Already in 1974 Goetz had shown that vitamin C, in vitro, was capable of stimulating the motility and the chemiotaxis of neutrophiles.

The doses advised in literature for anti-neoplastic therapy are about 3-10 grams a day, reaching even 40 grams daily, because the vitamin is not toxic, at high doses it only has a laxative effect.

It would be better to take it by eating fresh fruit, but it is difficult to reach such high doses of the vitamin in this way.

Integrating fresh fruit (kiwi, oranges, lemons and grapefruit) with the juice of sea buckthorn  , Rosa canina (dog rose, wild rose) to reach a daily dosage of at least 5- 8 grams of natural vitamin C, avoiding however, the use of pharmaceutically prepared vitamin C tablets.

Natural vitamin C, rich in its metabolites and other components called bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercitin, Pychnogenol, etc) is more powerful and efficient, and furthermore is devoid of unpleasant gastric effects which are the result of high doses of synthetic vitamin C.

Natural vitamin C is moreover, characterized by a significant reduction in the formation of Calcium oxalate in the kidneys, as opposed to synthetic vitamin C. It is also easier for the intestine to absorb and has greater bio-availability, above all through its most important metabolites such as tronic acid, lixonic acid, xilonic acid etc.

This bio-availability has a critical importance in the immune defense system because the white blood cells tend to absorb Natural vitamin C 4 times more than they absorb synthetic vitamin C.

Recently, Myrciaria paraensis (camu-camu) has appeared on the European market. It is a small exotic fruit, similar to a small orange, but it contains 50 times more natural vitamin C than Citrus aurantium (orange), and it could therefore provide the daily dosage, of at least 3 grams, of vitamin  .

Also Malpighia punicifolia (acerola), a cherry from the Antilles, is very rich in vitamin C, containing 50 to 100 times more than citrus fruits.

Natural vitamin C is therefore efficient because it is naturally associated to the bioflavenoids (Citrin, Hesperidin, Campherol, Galangine, Isoamnetin, Rutine, Hyperoxide, Quercetine,Quercitine, Pychnogenol, etc..) and other molecules, in plants often characterized by an immune  stimulating activity (Echinacea purpurea, Plantago major, Capsicum frutescens)

Note: With very high therapeutic doses (>8-10 grams a day), Magnesium (e.g. Dolomite) must also be taken to avoid the risk of kidney stones.

Here enclosed scientific papers extracted from Catherine Kousmine (“Save your body”, page 129,

“Effects of C vitamin on our body according to Linus Pauling, edition Tecniche Nuove):

“…an intake of 1500 milligrams of ascorbic acid by mouth determines a concentration of 1.5 milligrams of C vitamin for each 100 millilitres of blood. By increasing the intake, the concentration suddenly increases up to 2.5 milligrams and then goes back to 1.5 millilitres for each 100 millilitres blood. There are enzymes which help the conversion of most

ascorbates into useful oxidation products. If the intake remains high, the body increases the amount of enzymes useful to the conversion; otherwise, if the ascorbic acid dose is suddenly reduced for some days, an excess of conversion enzymes and then a too law level of vitamin C in the blood occur. This means a number of disorders, as for example a higher sensibility to infections. The adaptation to a lower proportion takes place by reducing the number of conversion enzymes: it is necessary to gradually decrease the dose of vitamin C. By taking 100 milligrams a day and in presence of a plasmatic level of 1 milligram for every 100 millilitres blood, urines do not contain ascorbic acid because it is reabsorbed by renal tubules. If the intake is higher than 100 milligrams, i.e. 1-2 grams a day, 25% go in urines and the rest is kept by the body. Healthy people, who lack in vitamin C for some months, have to take 2-4 grams in order to eliminate them through urines. In case of cancer patients, who are used to take high doses of ascorbic acid, an interruption of some days requires an intake of 50 grams (fifty grams) of vitamin C so that this one can be found in urines”.

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Vitamin D (very important vitamin)

• Cancer prevention by a MINIMUM of 77% cases just by sun exposure(best source of vitamin D): http://www.naturalnews.com/035063_vitamin_D_cancer_facts.html

• Cancer treatment with sun exposure(vitamin D): http://www.polymvasurvivors.com/what_you_know.html#Vitamin%20D%20

NOTE:The link above contains multiple studies on sun exposure(vitamin D) and it’s crucial importance in fighting cancer:

Circulating Vitamin D levels in the Blood Dramatically Reduces Death Rate

* For each 10ng/ml increase in the blood level of vitamin D, the death rate from cancer would be reduced by 29%. The relationship between vitamin D and colorectal cancer is particularly strong.

The death rate for colorectal cancer would be reduced by 49%. (30 – Read Supporting Scientific Article on PUBMED)

Vitamin D Levels est during Summer = Improves survival by 40%

* A Norwegian study showed that cancer patients diagnosed in the summer when vitamin D levels are highest had up to 40% better survival rates than patients diagnosed in the winter when vitamin D levels are at their lowest.

(56 – Read Supporting Scientific Article on PUBMED )

Low Vitamin D Levels Greater chance of Death & Metastasis

* Breast cancer patients with low levels of vitamin D followed over eleven years had a 70% greater chance of dying and twice the rate of developing metastasis than patients with high levels of vitamin D.

(90 – Read Supporting Scientific Article )

Lung Cancer Patients Higher levels = Dramatic increase in Survival

* Early stage lung cancer patients who were diagnosed in the summer and had the highest levels of vitamin D had a five-year survival of 73% compared to 30% for those diagnosed in the winter with low vitamin D levels.

(60 – Read Supporting Scientific Article on PUBMED)

Lung – Colon – Prostate – Renal – Endometrial Cancers Higher Vit D levels dramatically decreases Risk

*High vitamin D levels (based on latitude and UV exposure) also correlate with a decreased risk of developing lung cancer, a 45% and 65% reduction in men and women respectively. Similar effects have been demonstrated in colon, prostate, renal and endometrial cancer. (33 – Read Supporting Scientific Article on BMJ(British Medical Journal))

Advanced Colorectal Patients Risk of death greatly Improved

* The risk of death for colorectal cancer patients with advanced disease but with high levels of vitamin D was reduced by over 60% compared to patients with low vitamin D levels. (64 – Read Supporting Scientific Article on PUBMED)

Prostate Cancer Patients

*Prostate Cancer Patients with Vitamin D levels in the mid and high range had a 60% and 85% respectively reduced risk of death from the condition compared to patients with low levels of Vitamin D. This is almost a seven fold increase in the risk of death in those with low vs high vitamin D levels. (152 – Read Supporting Scientific Article on PUBMED)

There is also an extensive bibliography on the anti-tumoral action of vitamin D- see last page of this database:  (28,157,160,188,208,209,231,240,246, 254,302,323,479,489)

Vitamin D is BEST taken from skin exposure to the sun, MINIMUM 30 mins daily,without any sunscreen or protection. Avoid burning and avoid the interval 11 am – 16 pm.

Natural vitamin D, contained in some plants, is however preferable to the synthetic type, because the latter is about 10 times more capable of binding with Magnesium, taking it away from the organism, thus causing all the damage that the loss of this incurs (osteoporosis, kidney stones).

Vitamin D induces the inhibition of neoplastic cellular growth: this has been shown in vitro in neoplastic cellular lines; especially of the hematopoietic system, of the CNS, of the prostate; the  colon, the ovaries and the breasts.

This action is thought to be expressed at various levels, in particular:

a) by means of apoptosis induction, through the activation of p21, that is, the inhibitor of the kinase proteins;

b) the inhibition of neoplastic cellular growth, which would be blocked in the G1 phase because of the action of the IGF1 inhibitor;

c) by means of cellular differentiation.

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Vitamin E (respiratory vitamin)

This liposoluble substance consists of a group of various components, called Tocopherols.

Seven of these exist in nature; alpha-Tocopherol, beta-Tocopherol, gamma- Tocopherol, delta-Tocopherol, epsilon-Tocopherol, zetaTocopheros and eta-Tocopherol.

Studies on Vitamin E and cancer

• Selenium and Vitamin E supplements for prostate cancer: evidence or embellishment?
  2002 Urology
• Supplemental Vitamin E intake and prostate cancer risk in a large cohort of men in the United States.
  1999 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• The role of Vitamin E in the prevention of cancer: a meta-analysis of randomized controlled trials.
  2007 Annals of Saudi medicine
• Long-term use of supplemental multivitamins, vitamin C, vitamin E, and folate does not reduce the risk of lung cancer.
  2008 American journal of respiratory and critical care medicine
• Vitamin E and selenium supplementation and risk of prostate cancer in the Vitamins and lifestyle (VITAL) study cohort.
  2008 Cancer causes & control : CCC
• Vitamin E suppresses telomerase activity in ovarian cancer cells.
  2007 Cancer detection and prevention
• Effects of long-term Vitamin E supplementation on cardiovascular events and cancer: a randomized controlled trial.
  2005 JAMA : the journal of the American Medical Association
• Supplemental and dietary Vitamin E intakes and risk of prostate cancer in a large prospective study.
  2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Antioxidants vitamin C and Vitamin E for the prevention and treatment of cancer.
• 2006 Journal of general internal medicine
• Vitamin E: the evidence for multiple roles in cancer.
• 2003 Nutrition and cancer
• Vitamin E supplements and risk of prostate cancer in U.S. men.
• 2004 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Serum and dietary Vitamin E in relation to prostate cancer risk.
  2007 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Association of variants in two Vitamin E transport genes with circulating Vitamin Econcentrations and prostate cancer risk.2009 Cancer research
• Association between breast cancer and vitamin C, Vitamin E and selenium levels: results of a case-control study in India.
  2005 Asian Pacific journal of cancer prevention : APJCPA short-term dietary supplementation with high doses of Vitamin E increases NK cell cytolytic activity in advanced colorectal cancer patients.2007 Cancer immunology, immunotherapy : CIISELECT: the next prostate cancer prevention trial. Selenum and Vitamin E cancerPrevention Trial.
  2001 The Journal of urologyVitamin C and Vitamin E supplement use and colorectal cancer mortality in a large American cancer Society cohort.
  2001 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

  Vitamin E and breast cancer.
  2004 The Journal of nutrition

  SELECT: the selenium and Vitamin E cancer prevention trial.
  2003 Urologic oncology

  The selenium and Vitamin E cancer prevention trial.
  2003 World journal of urology

  Selenium and Vitamin E cancer prevention trial.
  2004 Annals of the New York Academy of Sciences

  Selenium and Vitamin E cancer Prevention Trial: a nutrient approach to prostatecancer prevention.
  2009 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

  A nutrient approach to prostate cancer prevention: The Selenium and Vitamin Ecancer Prevention Trial (SELECT).
  2010 Nutrition and cancer

  Null activity of selenium and Vitamin E as cancer chemopreventive agents in the rat prostate.
  2010 Cancer prevention research (Philadelphia, Pa.)

  Vitamin E and cancer.
  2007 Vitamins and hormones

  Clinical models for testing chemopreventative agents in prostate cancer and overview of SELECT: the Selenium and Vitamin E cancer Prevention Trial.
  2003 Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

  Cancer Preventive Effects of Vitamin E.
  2011 Current pharmaceutical biotechnology

  Vitamin E but not St. John’s wort mitigates leukopenia caused by cancerchemotherapy in rats.
  2006 Translational research : the journal of laboratory and clinical medicine

  Vitamin C and Vitamin E supplement use and bladder cancer mortality in a large cohort of US men and women.
  2002 American journal of epidemiology

  Review of Vitamin E and selenium in the prevention of prostate cancer: implications of the selenium and Vitamin E chemoprevention trial.
  2002 Integrative cancer therapies

  Vitamin E and breast cancer prevention: current status and future potential.
  2003 Journal of mammary gland biology and neoplasia

  Vitamin E and breast cancer prevention: current status and future potential.
  2003 Journal of mammary gland biology and neoplasia

  Molecular epidemiologic studies within the Selenium and Vitamin E cancerPrevention Trial (SELECT).
  2001 Cancer causes & control : CCC

  Selenium and vitamin E: cell type- and intervention-specific tissue effects in prostate cancer.
  2009 Journal of the National cancer Institute

  Tocopherol transfer protein sensitizes prostate cancer cells to vitamin E.
  2010 The Journal of biological chemistry

  Selenium and Vitamin E for prostate cancer: post-SELECT (Selenium and Vitamin Ecancer Prevention Trial) status.
  2011 Molecular medicine (Cambridge, Mass.)

  Vitamin E and cancer: An insight into the anticancer activities of Vitamin E isomers and analogs.
  2008 International journal of cancer. Journal international du cancer

  Vitamin E intake, alpha-tocopherol status, and pancreatic cancer in a cohort of male smokers.
  2009 The American journal of clinical nutrition

  The roles of alpha-vitamin E and its analogues in prostate cancer.
  2007 Vitamins and hormones

  Combined lycopene and Vitamin E treatment suppresses the growth of PC-346C human prostate cancer cells in nude mice.
  2006 The Journal of nutrition

  Vitamin E, alpha- and gamma-tocopherol, and prostate cancer.
  1999 Seminars in urologic oncology

  Synergistic effect of Vitamin E and selenium in human prostate cancer cell lines.
  2004 Prostate cancer and prostatic diseases

  Gamma (gamma) tocopherol upregulates peroxisome proliferator activated receptor (PPAR) gamma (gamma) expression in SW 480 human colon cancer cell lines.
  2003 BMC cancer

  Vitamin E succinate inhibits colon cancer liver metastases.
  2002 The Journal of surgical research

  Lycopene and Vitamin E interfere with autocrine/paracrine loops in the Dunning prostate cancer model.
  2004 The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

  Higher baseline serum concentrations of Vitamin E are associated with lower total and cause-specific mortality in the Alpha-Tocopherol, Beta-Carotene cancerPrevention Study.
  2006 The American journal of clinical nutrition

  Alpha-tocopheryl succinate, the most effective form of Vitamin E for adjuvant cancertreatment: a review.
  2003 Journal of the American College of Nutrition

  gamma-Tocopherol or combinations of Vitamin E forms induce cell death in human prostate cancer cells by interrupting sphingolipid synthesis.
  2004 Proceedings of the National Academy of Sciences of the United States of America

  Experimental basis for cancer prevention by vitamin E.
  2000 Cancer investigation

  Vitamin E analogues as a novel group of mitocans: anti-cancer agents that act by targeting mitochondria.
  2007 Molecular aspects of medicine

  Vitamin E succinate decreases lung cancer tumor growth in mice.
  2005 The Journal of surgical research

  RRR-alpha-vitamin E succinate potentiates the antitumor effect of calcitriol in prostatecancer without overt side effects.
  2009 Clinical cancer research : an official journal of the American Association forcancer Research

  Serum levels of folate, lycopene, ß-carotene, retinol and Vitamin E and prostatecancer risk.
  2010 European journal of clinical nutrition

  Trace elements and Vitamin E status in Nigerian patients with prostate cancer.
  2010 African health sciences

  Designing the Selenium and Vitamin E cancer Prevention Trial (SELECT).
  2005 Journal of the National cancer Institute

  Vitamin E analogues and immune response in cancer treatment.
  2007 Vitamins and hormones

  Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
  2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

  Vitamin E serum levels and gastric cancer: results from a cohort of patients in Tuscany, Italy.
  2000 Cancer letters

  Donepezil and Vitamin E for preventing cognitive dysfunction in small cell lungcancer patients: preliminary results and suggestions for future study designs.
  2005 Supportive care in cancer : official journal of the Multinational Association of Supportive Care in Cancer

  Effects of topical L-selenomethionine with topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
  2003 Journal of the American Academy of Dermatology

  Vitamin E and the Y4 agonist BA-129 decrease prostate cancer growth and production of vascular endothelial growth factor.
  2002 The Journal of surgical research

  Pro-apoptotic mechanisms of action of a novel Vitamin E analog (alpha-TEA) and a naturally occurring form of Vitamin E (delta-tocotrienol) in MDA-MB-435 human breast cancer cells.
  2004 Nutrition and cancer

  Minority recruitment to the Selenium and Vitamin E cancer Prevention Trial (SELECT).
  2005 Clinical trials (London, England)

  Salubrious effect of vitamin C and Vitamin E on tamoxifen-treated women in breastcancer with reference to plasma lipid and lipoprotein levels.
  2000 Cancer letters

  Design of Physicians’ Health Study II–a randomized trial of beta-carotene, vitamins E and C, and multivitamins, in prevention of cancer, cardiovascular disease, and eye disease, and review of results of completed trials.
  2000 Annals of epidemiology

  Vitamin E succinate promotes breast cancer tumor dormancy.
  2000 The Journal of surgical research

  Mitocans as anti-cancer agents targeting mitochondria: lessons from studies withVitamin E analogues, inhibitors of complex II.
  2007 Journal of bioenergetics and biomembranes

  Centralized blood processing for the selenium and Vitamin E cancer prevention trial: effects of delayed processing on carotenoids, tocopherols, insulin-like growth factor-I, insulin-like growth factor binding protein 3, steroid hormones, and lymphocyte viability.
  2005 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology

  The therapeutic and preventive effect of RRR-alpha-vitamin E succinate on prostatecancer via induction of insulin-like growth factor binding protein-3.
  2007 Clinical cancer research : an official journal of the American Association forcancer Research

  Vitamin E and prostate cancer: is Vitamin E succinate a superior chemopreventive agent?
  2005 Nutrition reviews

  Aspirin prevents stroke but not MI in women; Vitamin E has no effect on CV disease or cancer.
  2006 Cleveland Clinic journal of medicine

  Effect of Vitamin E on tamoxifen-treated breast cancer cells.
  2006 Surgery

  Dietary administration of the proapoptotic Vitamin E analogue alpha-tocopheryloxyacetic acid inhibits metastatic murine breast cancer.
  2006 Cancer research

  Combination of Vitamin E and selenium causes an induction of apoptosis of human prostate cancer cells by enhancing Bax/Bcl-2 ratio.
  2008 The Prostate

  Non-hormonal treatment of hot flushes in breast cancer survivors: gabapentin vs. vitamin E.
  2009 Climacteric : the journal of the International Menopause Society

  Vitamin E analog alpha-TEA, methylseleninic acid, and trans-resveratrol in combination synergistically inhibit human breast cancer cell growth.
  2008 Nutrition and cancer

  Poly(L-lactide)-vitamin E TPGS nanoparticles enhanced the cytotoxicity of doxorubicin in drug-resistant MCF-7 breast cancer cells.
  2010 Biomacromolecules

  Vitamin E succinate induced apoptosis and enhanced chemosensitivity to paclitaxel in human bladder cancer cells in vitro and in vivo.
  2010 Cancer science

  Tocopherol-associated protein suppresses prostate cancer cell growth by inhibition of the phosphoinositide 3-kinase pathway.
  2005 Cancer research

  Differential retention of alpha-vitamin E is correlated with its transporter gene expression and growth inhibition efficacy in prostate cancer cells.
  2007 The Prostate

  Vitamin E succinate (VES) induces Fas sensitivity in human breast cancer cells: role for Mr 43,000 Fas in VES-triggered apoptosis.
  1999 Cancer research

  Effects of topical and oral Vitamin E on pigmentation and skin cancer induced by ultraviolet irradiation in Skh:2 hairless mice.
  2000 Nutrition and cancer

  Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
  2003 Cancer research

  Vitamin E analogs trigger apoptosis in HER2/erbB2-overexpressing breast cancercells by signaling via the mitochondrial pathway.
  2005 Biochemical and biophysical research communications

  Vitamin E and prostate cancer.
  2002 The Urologic clinics of North America

  Synergy between selenium and Vitamin E in apoptosis induction is associated with activation of distinctive initiator caspases in human prostate cancer cells.
  2003 Cancer research

  Peptide YY augments gross inhibition by Vitamin E succinate of human pancreaticcancer cell growth.
  2000 The Journal of surgical research

  Supplemental and dietary vitamin E, beta-carotene, and vitamin C intakes and prostatecancer risk.
  2006 Journal of the National cancer Institute

  Vitamin E in the primary prevention of cardiovascular disease and cancer: the Women’s Health Study: a randomized controlled trial.
  2005 JAMA : the journal of the American Medical Association

  Dietary intakes of retinol, beta-carotene, vitamin D and Vitamin E in the European Prospective Investigation into cancer and Nutrition cohort.
  2009 European journal of clinical nutrition

  Mitochondrial targeting of Vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II.
  2011 The Journal of biological chemistry

  Impact of supplemental site grants to increase African American accrual for the Selenium and Vitamin E cancer Prevention Trial.
  2010 Clinical trials (London, England)

  The Outcome of Selenium and Vitamin E cancer Prevention Trial (SELECT) reveals the need for better understanding of selenium biology.
  2009 Molecular interventions

  Critical roles for JNK, c-Jun, and Fas/FasL-Signaling in Vitamin E analog-induced apoptosis in human prostate cancer cells.
  2008 The Prostate

  Effect of selenium and Vitamin E on risk of prostate cancer and other cancers: the Selenium and Vitamin E cancer Prevention Trial (SELECT).
  2009 JAMA : the journal of the American Medical Association

  Tocotrienols, the Vitamin E of the 21st century: its potential against cancer and other chronic diseases.
  2010 Biochemical pharmacology

  Mixed PEG-PE/vitamin E tumor-targeted immunomicelles as carriers for poorly soluble anti-cancer drugs: improved drug solubilization and enhanced in vitro cytotoxicity.
  2008 European journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft für Pharmazeutische Verfahrenstechnik e.V

  Nanoparticles of poly(lactide)/vitamin E TPGS copolymer for cancer chemotherapy: synthesis, formulation, characterization and in vitro drug release.
  2006 Biomaterials

  Involvement of JNK/p73/NOXA in Vitamin E analog-induced apoptosis of human breast cancer cells.
  2008 Molecular carcinogenesis

  Vitamin E supplementation in cardiovascular disease and cancer prevention: Part 1.
  2005 The Annals of pharmacotherapy

  A peptide conjugate of Vitamin E succinate targets breast cancer cells with high ErbB2 expression.
  2007 Cancer research

  Vitamin E succinate inhibits NF-kappaB and prevents the development of a metastatic phenotype in prostate cancer cells: implications for chemoprevention.
  2007 The Prostate

  The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
  2002 World journal of gastroenterology : WJG

  The effects of Vitamin E succinate on the expression of c-jun gene and protein in human gastric cancer SGC-7901 cells.
  2002 World journal of gastroenterology : WJG

  Vitamin E succinate and cancer treatment: a Vitamin E prototype for selective antitumour activity.
  2003 British journal of cancer

  Vitamin E down-modulates iNOS and NADPH oxidase in c-Myc/TGF-alpha transgenic mouse model of liver cancer.
  2004 Journal of hepatology

  Vitamin E inhibits the high-fat diet promoted growth of established human prostate LNCaP tumors in nude mice.
  1999 The Journal of urology

  Vitamin E analog alpha-TEA and celecoxib alone and together reduce human MDA-MB-435-FL-GFP breast cancer burden and metastasis in nude mice.
  2004 Breast cancer research and treatment

  Development of gamma (gamma)-tocopherol as a colorectal cancer chemopreventive agent.
  2003 Critical reviews in oncology/hematology

  Vitamin E analogues: a new class of inducers of apoptosis with selective anti-cancer effects.
  2004 Current cancer drug targets

  The role of PC-SPES, selenium, and Vitamin E in prostate cancer.
  2002 Oncology (Williston Park, N.Y.)

  Differential response of human ovarian cancer cells to induction of apoptosis byVitamin E Succinate and Vitamin E analogue, alpha-TEA.
  2004 Cancer research

  A short-term dietary supplementation of high doses of Vitamin E increases T helper 1 cytokine production in patients with advanced colorectal cancer.
  2002 Clinical cancer research : an official journal of the American Association forcancer Research

  Role of caspase-8 activation in mediating vitamin E-induced apoptosis in murine mammary cancer cells.
  2003 Nutrition and cancer

  High dietary level of synthetic Vitamin E on lipid peroxidation, membrane fatty acid composition and cytotoxicity in breast cancer xenograft and in mouse host tissue.
  2003 Cancer cell international

  [Roles of ERK1/2 MAPK in Vitamin E succinate-induced apoptosis in human gastriccancer SGC-7901 cells].
  2003 Wei sheng yan jiu = Journal of hygiene research

  Alpha-vitamin E derivative, RRR-alpha-tocopheryloxybutyric acid inhibits the proliferation of prostate cancer cells.
  2007 Asian journal of andrology

  Roles of Fas signaling pathway in Vitamin E succinate-induced apoptosis in human gastric cancer SGC-7901 cells.
  2002 World journal of gastroenterology : WJG

  Mechanisms mediating the antiproliferative and apoptotic effects of Vitamin E in mammary cancer cells.
  2005 Frontiers in bioscience : a journal and virtual library

  Cellular and molecular effects of alpha-tocopheryloxybutyrate: lessons for the design of Vitamin E analog for cancer prevention.
  2004 Anticancer research

  Vitamin E and cancer.
  2009 Nutrition and cancer

  Pentoxifylline and Vitamin E treatment for prevention of radiation-induced side-effects in women with breast cancer: a phase two, double-blind, placebo-controlled randomised clinical trial (Ptx-5).
  2009 European journal of cancer (Oxford, England : 1990)

  Vitamin E prevents lipid raft modifications induced by an anti-cancer lysophospholipid and abolishes a Yap1-mediated stress response in yeast.
  2010 The Journal of biological chemistry

  Analogs of Vitamin E epitomized by alpha-tocopheryl succinate for pancreatic cancertreatment: in vitro results induce caution for in vivo applications.
  2010 Pancreas

  Evaluation of plasma non-enzymatic antioxidants in breast cancer etiology.
  2009 Asian Pacific journal of cancer prevention : APJCP

  [The study of apoptosis induction effect of Vitamin E succinate on Tca8113 human tongue cancer cells].
  2008 Hua xi kou qiang yi xue za zhi = Huaxi kouqiang yixue zazhi = West China journal of stomatology

  Antioxidant status and risk of cancer in the SU.VI.MAX study: is the effect of supplementation dependent on baseline levels?
  2005 The British journal of nutrition

  Double-blind placebo-controlled randomised trial of Vitamin E and pentoxifylline in patients with chronic arm lymphoedema and fibrosis after surgery and radiotherapy for breast cancer.
  2004 Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology

  Vitamin E succinate induces apoptosis in human prostate cancer cells: role for Fas inVitamin E succinate-triggered apoptosis.
  2000 Nutrition and cancer

  Vitamin E and other antioxidants inhibit human prostate cancer cells through apoptosis.
  2000 The Prostate

  Vitamin E succinate suppresses prostate tumor growth by inducing apoptosis.
  2006 International journal of cancer. Journal international du cancer

  In vitro and in vivo evaluation of anticancer actions of natural and synthetic Vitamin Eforms.
  2008 Molecular nutrition & food research

  Vitamin E analogues as anticancer agents: lessons from studies with alpha-tocopheryl succinate.
  2006 Molecular nutrition & food research

  Prospective study of serum Vitamin E levels and esophageal and gastric cancers.
  2003 Journal of the National cancer Institute

  Systematic review on „vitamin E and prevention of colorectal cancer”.
  2010 Pakistan journal of pharmaceutical sciences

  Vitamin E analogues as mitochondria-targeting compounds: from the bench to the bedside?
  2009 Molecular nutrition & food research

Vitamin E and cancer: An insight into the anticancer activities of …

science.naturalnews.com/…/1364373_Vitamin_E_and_cancer_An_insight_ into_the_anticancer_activities.html

Studies on Vitamin E and radiation

http://science.naturalnews.com/V/Vitamin_E_and_radiation.html

There is also an extensive bibliography on the anti-tumoral action of vitamin E -see last page of DATABASE (6,19,20,30,45,91,95,112, 125,129,142,165,167,190,202,228,229,246,261,280,332,404,405,452,494)

This liposoluble substance consists of a group of various components, called Tocopherols. Seven of these exist in nature; alpha, beta, gamma, delta, epsilon, zeta and eta.

Alpha-Tocopherol has an anti-oxidant effect on the lipidic membranes in synergy with Melatonin(see below), carrying out a preventative action on the peroxidization of the cellular membrane induced by  ionizing radiation and by chemical carcinogenes.

Vitamin E also carries out an anti-oxidative action in a wider sense, acting as a ‘scavenger’ of the free radicals, similar to vitamin C.

It performs a stimulating activity on the immune system; it induces cellular differentiation; it inhibits, in a selective way, cellular growth intervening at DNA and RNA synthesis level.

Various studies have shown its ability to induce apoptosis in cellular lines of breast carcinomas and lymphoma B.

It is inactivated by Iron, therefore it is essential that any medicines based on Iron are not taken at the same time as vitamin E, but at a distance of at least 10-12 hours.

Aluminum, which is often present in pharmaceutical products, also deactivates vitamin E.

The uncooked oil of Triticum sativum (wheat germ; note: has vitamin B12) contains about twice as much vitamin E as the uncooked oil from the seeds of Helianthus annuus (sunflower) and the latter  contains about five times as much as uncooked olive oil. Furthermore all these seeds are rich in  essential unsaturated fats, an important part of the diet for cancer patients.

In anti-neoplastic therapy much is being discussed about: the raw seeds of Helianthus annuus

(which the author personally considers useful in therapy), wheat shoots (the author is not in favor),

Saccharomyces cerevisiae (yeast, of which the author is not in favor), and the shoots of soya  lecithin (of dubious use and the author is against their use because of the transgenic risk).

Natural vitamins are by far preferable to industrially produced ones: Dracontium loretense, for example, which is considered one of the best  plants for its specific anti-oxidant potential, is of superior quality in its anti-oxidant ability compared to synthetic vitamin E (566). WHOLE, ORGANIC FOODS are BEST due to COMPLEXES of NUTRIENTS !

As an already extracted natural product, together with or without other vitamins, vitamin E must be  given in addition to high quantities of raw seeds of Helianthus annuus (also containing vitamin A,all the vitamin B compounds, vitamin D, Manganese, Zinc and Magnesium) and of high quantities of raw Triticum sativum (which is rich in the precious alpha-lipoic acid): both are also very rich in vitamin B6 (pyridoxin), the latter is important for the immune system, but it is difficult to find in other compatible foods for a suitable diet for cancer patients. Pyridoxin, in fact, is contained especially in Saccharomyces cerevisiae, the latter is a food which the author does not regard favorably for an anti-neoplastic diet, because it contains high quantities of folic acid.

Natural Octacosanol, extracted from the oil of Triticum sativum, has a synergetic action with vitamin E, but it is, in any case, better to consume it with all the uncooked oil of Triticum sativum  and/or Triticum sativum itself rather than taking it already extracted, as a pharmaceutical product  (because it loses its active principles).

Vitamin E is particularly efficient in combination with Selenium, which is contained in Aloe species, Solanum lycopersicum (tomatoes), Equisetum species, Allium cepa (onions).

There is also an extensive bibliography on Selenium (see last page of DATABASE) :

79,108,112,129,133,136,143,156,228,229,276,338,339,364,367,404,405,407,443,452,458,501,510,511)

Both vitamin E and Selenium are in their turn synergetic with Zinc in inhibiting the production of inflammatoryprostoglandins and leukotrienes.

Vitamin E conversion factors

Vitamin E is expressed in milligrams of tocopheral equivalents (T.E.)

1 milligram of Tocopherol is equal to:

= 1 milligram of D-alpha Tocopherol

= 2 milligrams of D-beta Tocopherol

= 5 milligrams of D-gamma Tocopherol

==========================================================================================================

Vitamin F  (very important vitamin-see omega 3)

Polyunsaturated fatty acids :

arachidonic acid, Linoleic cis-cis natural acid (vitamin F1) as: alpha-lipoic acid,  alpha-linolenic acid,etc.

Alpha-lipoic acid

Food Sources: 

spinach, broccoli, tomato, peas, Brussels sprouts, rice bran

Mechanism of action:

Alpha-lipoic acid acts as a lipophilic free radical scavenger. Dihydrolipoic acid (DHLA), a reduced form of lipoic acid, has more potent antioxidant effects. It can assist in repairing oxidative damage and regenerate endogenous antioxidants such as vitamin C, vitamin E, and glutathione.

Both DHLA and lipoic acid also have metal chelating capacities.

As a lipoamide, alpha-lipoic acid functions as a cofactor in various multienzyme systems involved in the decarboxylation of alpha-keto acids such as pyruvate. ^{(13) (14) (15)}

Alpha-lipoic acid caused cell cycle arrest in G0/G1 in FaDu and Jurkat human tumor cell lines ⁽¹⁾.

Alpha lipoic acid was also found to scavenge reactive oxygen species in MCF-7 breast cancer cells ⁽¹⁶⁾.

Reduction of reactive oxygen species was then followed by cancer cell growth arrest and apoptosis ⁽¹⁶⁾.

For studies quoted see http://www.mskcc.org/cancer-care/herb/alpha-lipoic-acid  

Other studies:

Studies on Alpha–Lipoic Acid and cancer –  MANY STUDIES

science.naturalnews.com/A/Alpha–Lipoic_Acid_and_cancer.html

• The natural antioxidant Alpha-Lipoic Acid induces p27(Kip1)-dependent cell cycle arrest and apoptosis in MCF-7 human breast cancer cells.
  2010 European journal of pharmacology
• Restoration of functional defects in peripheral blood mononuclear cells isolated fromcancer patients by thiol antioxidants Alpha-Lipoic Acid and N-acetyl cysteine.
  2000 International journal of cancer. Journal international du cancer
• alpha-Lipoic acid: the potential for use in cancer therapy.
  2008 Neoplasma
• Alpha-Lipoic Acid reduces matrix metalloproteinase activity in MDA-MB-231 human breast cancer cells.
  2010 Nutrition research (New York, N.Y.)
• Alpha-Lipoic Acid induces apoptosis in human colon cancer cells by increasing mitochondrial respiration with a concomitant O2-*-generation.
  2005 Apoptosis : an international journal on programmed cell death
• Mechanism of alpha-lipoic acid-induced apoptosis of lung cancer cells.
  2009 Annals of the New York Academy of Sciences\
• The long-term survival of a patient with pancreatic cancer with metastases to the liver after treatment with the intravenous alpha-lipoic acid/low-dose naltrexone protocol.
  2006 Integrative cancer therapies
• Cytoprotection against Cr(6+)-induced DNA damage by alpha-lipoic acid: implications in reducing occupational cancer risk.
  2009 Mutagenesis

Studies on Alpha-Lipoic Acid and Apoptotic

http://science.naturalnews.com/A/Alpha-Lipoic_Acid_and_Apoptotic.html

Alpha-Linolenic Acid(omega 3)

Food sources:flaxseed, avocado, walnuts, almonds, fishoils

Mechanism of action:

The alpha-linolenic acid (vitamin F), for instance, is a cis-polyunsaturated fatty acid that is contained in linseed cold-pressed oil: it is transformed into EPA and DHA (Omega-3 fatty acids) and is quite effective against malignant tumours, as shown by Pardini ( 1647 );

Moreover, Noguchi has proved that Omega-3 fatty acids, unlike Omega-6 fatty acids, help reduce tumour masses, although

Omega-6 fatty acids are unsaturated fatty acids, too ( 1654 ).

see  last page of DATABASE for studies quoted.

Studies on Alpha–Linolenic Acid and cancer –  MANY STUDIES

science.naturalnews.com/A/Alpha–Linolenic_Acid_and_cancer.html

• Alpha-Linolenic Acid and the risk of prostate cancer. What is the evidence?
  2004 The Journal of urology
• Low Alpha-Linolenic Acid content of adipose breast tissue is associated with an increased risk of breast cancer.
  2000 European journal of cancer (Oxford, England : 1990)
• A prospective study of dietary Alpha-Linolenic Acid and the risk of prostate cancer(United States).
  2006 Cancer causes & control : CCC
• The relation of Alpha-Linolenic Acid to the risk of prostate cancer: a systematic review and meta-analysis.
  2009 The American journal of clinical nutrition
• Alpha-Linolenic Acid and risk of prostate cancer: a case-control study in Uruguay.
  2000 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• HER2 (erbB-2)-targeted effects of the omega-3 polyunsaturated fatty acid, Alpha-Linolenic Acid (ALA; 18:3n-3), in breast cancer cells: the „fat features” of the „Mediterranean diet” as an „anti-HER2 cocktail”.
  2006 Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National cancer Institute of Mexico
• Inhibitory effect of conjugated Alpha-Linolenic Acid from bifidobacteria of intestinal origin on SW480 cancer cells.
  2009 Lipids\
• Prospective studies of dietary Alpha-Linolenic Acid intake and prostate cancer risk: a meta-analysis.
  2010 Cancer causes & control : CCC
• Dietary Alpha-Linolenic Acid is associated with reduced risk of fatal coronary heart disease, but increased prostate cancer risk: a meta-analysis.
  2004 The Journal of nutrition
• Growth-inhibitory and proapoptotic effects of Alpha-Linolenic Acid on estrogen-positive breast cancer cells.
  2009 Annals of the New York Academy of Sciences
• Comparison of stearidonic acid and Alpha-Linolenic Acid on PGE2 production and COX-2 protein levels in MDA-MB-231 breast cancer cell cultures.
  2005 The Journal of nutritional biochemistry
• Conjugation with Alpha-Linolenic Acid improves cancer cell uptake and cytotoxicity of doxorubicin.
  2009 Bioorganic & medicinal chemistry letters
• Does Alpha-Linolenic Acid in combination with linoleic acid influence liver metastasis and hepatic lipid peroxidation in BOP-induced pancreatic cancer in Syrian hamsters?
  2000 Prostaglandins, leukotrienes, and essential fatty acids
• Conjugation with alpha linolenic acid improves cancer cell uptake …

  science.naturalnews.com/…/542108_Conjugation_with_alpha_linolenic_acid _improves_cancer_cell_uptake_and.html

  Conjugation with alpha linolenic acid improves cancer cell uptake and cytotoxicity of doxorubicin.

  Inhibitory effect of conjugated alpha linolenic acid from …

  science.naturalnews.com/…/2462104_Inhibitory_effect_of_conjugated_alpha _linolenic_acid_from_bifidobacteria_of.html

  Inhibitory effect of conjugated alpha linolenic acid from bifidobacteria of intestinal origin on SW480 cancer cells. Publication: Lipids Publication Date: 2009

  Growth inhibitory and proapoptotic effects of alpha linolenic acid on …

  science.naturalnews.com/…/1637124_Growth_inhibitory_and_proapoptotic_ effects_of_alpha_linolenic_acid_on.html

  We tested the anticarcinogenic effect of Alpha–Linolenic Acid (ALA) as a single compound. To test the role of ALA in breast cancer cells (MCF-7), we analyzed …

  omega 3 fatty acids on cancer risk: a systematic review.

  science.naturalnews.com/…/2859351_Effects_of_omega_3_fatty_acids_on_ cancer_risk_a.html

  Effects of omega 3 fatty acids on cancer risk: a systematic review.

  Modulation of prostate cancer genetic risk by omega 3 and omega 6 …

  science.naturalnews.com/…/3442786_Modulation_of_prostate_cancer_ genetic_risk_by_omega_3_and.html

  Modulation of prostate cancer genetic risk by omega 3 and omega 6 fatty acids.

  Effect of altering dietary omega 6 omega 3 fatty acid ratios on prostate cancer…

  science.naturalnews.com/…/3897405_Effect_of_altering_dietary_omega_6_ omega_3_fatty_acid.html

  Effect of altering dietary omega 6 omega 3 fatty acid ratios on prostate cancer membrane composition, cyclooxygenase 2, and prostaglandin E2.

  Prostate tumor growth and recurrence can be modulated by omega3 intake…

  science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html

  Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids …prostate cancer (PCa) risk, whereas a diet rich in omega–3 decreases risk.

  Beneficial effects of omega 3 long chain fatty acids in breast cancer …

  science.naturalnews.com/…/296576_Beneficial_effects_of_omega_3_long_ chain_fatty_acids_in.html

  Beneficial effects of omega 3 long chain fatty acids in breast cancer and cardiovascular diseases: voltage gated sodium channels as a common feature?

  Fatty fish and fish omega 3 fatty acid intakes decrease the breast cancer risk…

  science.naturalnews.com/…/1200161_Fatty_fish_and_fish_omega_3_fatty_ acid_intakes_decrease.html

  Although it is believed that fish omega–3 fatty acids may decrease breast cancer risk, epidemiological evidence has been inconclusive. This study examined the …

  Omega 3 fatty acid supplements in women at high risk of breast cancer …

  science.naturalnews.com/…/759291_Omega_3_fatty_acid_supplements_in_ women_at_high_risk.html

  BACKGROUND : Preclinical evidence of the preventive benefits of omega–3 (n-3) polyunsaturated fatty acids (PUFAs) in breast cancer continues to fuel interest …

  Decreased severity of ovarian cancer and increased survival with omega 3.

  science.naturalnews.com/…/256930_Decreased_severity_of_ovarian_cancer_ and_increased_survival_in_hens.html

  Prevention and treatment of pancreatic cancer by omega 3 & curcumin in …

  science.naturalnews.com/…/2359858_Prevention_and_treatment_of_ pancreatic_cancer_by_curcumin_in_combination.html

  Prevention and treatment of pancreatic cancer by curcumin in combination with omega 3 fatty acids. Publication: Nutrition and cancer. Publication Date: 2008

  The importance of the omega 6 omega 3 fatty acid ratio in colorectal cancer…

  science.naturalnews.com/…/1101398_The_importance_of_the_omega_6_ omega_3_fatty_acid.html

  A ratio of 2.5/1 reduced rectal cell proliferation in patients with colorectal cancer, whereas a ratio of 4/1 with the same amount of omega–3 PUFA had no effect.

  Impact of postoperative omega 3 fatty acid supplemented parenteral …

  science.naturalnews.com/…/1136096_Impact_of_postoperative_omega_3_ fatty_acid_supplemented_parenteral_nutrition.html

  Impact of postoperative omega 3 fatty acid supplemented parenteral nutrition on clinical outcomes and immunomodulations in colorectal cancer patients.

   Omega 3 reduce colorectal cancer risk in men by 66 percent  …

  science.naturalnews.com/…/383170_Calcium_Mobilization_in_Ovarian_ Cancer_Cells_in_Response_to_Lysophospholipids.html

  Herbal Remedy Essiac® and Cancer (press release) · Omega–3 fatty acids slash colorectal cancer risk in men by 66 percent · Fatty Acid Blood Tests Help …

  Growth of human gastric cancer cells in nude mice is delayed by omega 3…

  science.naturalnews.com/…/1201162_Growth_of_human_gastric_cancer_ cells_in_nude_mice_is.html

  Growth of human gastric cancer cells in nude mice is delayed by a ketogenic diet supplemented with omega 3 fatty acids and medium chain triglycerides.

  Neuroblastoma studies

  science.naturalnews.com/Neuroblastoma.html

  Cancer science. Omega–3 fatty acid supplementation delays the progression of neuroblastoma in vivo. International journal of cancer. Journal …

  [omega 3 Fatty acid containing diet (Racol) reduces toxicity of chemotherapy…

  science.naturalnews.com/…/945592_omega_3_Fatty_acid_containing_diet_ Racol_reduces_toxicity_of.html

  omega 3 Fatty acid containing diet (Racol) reduces toxicity of chemoradiation therapy for patients with esophageal cancer].

   

  Modulation of lipid rafts by Omega 3 fatty acids in inflammation and …

  science.naturalnews.com/…/2597039_Modulation_of_lipid_rafts_by_Omega _3_fatty_acids_in.html

  Modulation of lipid rafts by Omega 3 fatty acids in inflammation and cancer: implications for use of lipids during nutrition support.

   

Omega 3, on their own, are crucial for a cancer patient:

-promote optimal functioning of the immune system.

A team of researchers based in China have published results of their work on omega-3 and breast cancer risk in the British Medical Journal (BMJ), stating that a high intake of fatty acids found in fish is associated with a reduction 14 percent risk of breast cancer later in life. To conduct their study, the scientists reviewed and analyzed a group of 26 studies in the United States, Europe and Asia, which included more than 800,000 participants and 20,000 cases of breast cancer.

–promote natural cancer cell death and reduce cancer risk.

After analyzing the data to determine the impact of long chain fats on the risk of breast cancer, the researchers determined the difference between the lowest and highest intake category of marine polyunsaturated fatty acids was associated with a 14 percent reduced risk of disease .

In addition, for an increase of 0.1 grams per day of consumption of omega-3 fatty acids in fish, the risk of breast cancer is reduced by 5 percent. These results clearly demonstrate a significantly reduced risk of affecting the development of breast cancer and future progression of the disease.

The study authors concluded „Our current study provides solid evidence and robust as omega3 are inversely associated with risk of breast cancer. „

Scientists understand from previous studies that omega-3 fats, DHA and EPA, are preferentially accumulated in cell membranes and prevent cancer development through increased transfer of nutrients and oxygen to the cell nucleus.

With DHA metabolism, metabolites are produced that destroy cancer cells. Researchers believe that this mechanism is responsible for reducing the risk of cancers found in this study. (1200-2400 mg EPA / DHA combined daily to reduce the risk associated with breast cancer and other disease lines). Higher doses are required in cancer.

-omega 3 stops spread of cancer

Omega-6, found in most vegetable oils, increased spread of cancer cells in the bone marrow.

Nevertheless, the spread was blocked by omega-3

Comments Dr. Mercola:

„The evidence continues to grow. Not all unsaturated fats are good for you. Even if they come from such healthy foods such as sunflower seeds grown biodynamic. See, most vegetable oils are rich in omega-6 fat and very few people are deficient in these fats.

In addition, omega-3 provides a wonderful benefits for patients with prostate cancer by blocking the functioning of omega-6 fats that cancer cells use them as an energy source. (Therefore, only during cancer your treatment, avoid omega 6 polyunsaturated fats because the thinner blood vessels supply  tumors. thin blood vessels in the body are necessary for optimal functioning, but avoid in cancer high doses of omega-6) This happens at the molecular level when omega-3  (EPA  andDHA), reduces the action of a metabolite of the omega-6 arachidonic acid, prostaglandin E2. The sole point agreed on this study is that scientists have recommended omega 3 as a preventative for prostate cancer as and to stop its development. „

Dr. Clapp compiled a comprehensive program that has achieved national recognition USA. 1990 –was a very successful year of the program – had 16 cures of prostate cancer based on deep cleansing of toxins accumulated in the body and prostate, then flooding the cells with omega 3 fats. „Perhaps the most interesting aspect of this study is clear evidence of the mechanisms that omega-6 fats stimulate the growth of prostate cancer, clearly debunking industry hypethat  vegetable oils are good for our health. „

We saw dramatically reduced overall inflammation (measured by CRP testing) and especially in reducing enlarged prostate, reducing also any tumor mass, monitored / evaluated by Doppler sonograms.

-Omega 3 promote apatosis(programmed cell death) in cancer cells

Two new studies suggest that omega-3 fatty acids may inhibit the growth of liver cancer cells, suggesting that this may be an effective therapy to treat and prevent liver cancer.

Like vitamin B17/laetrile,and other natural substances(turmeric, ginger) omega 3 trigger apatosis.

The first study examined the effects of omega-3 and omega-6 fats in hepatocellular carcinoma cells .

Carcinoma represent 90 percent of all cases of liver cancer and is usually rapidly fatal. Omega-6 had no effect on cancer cells, but omega-3 – as  (DHA) and (EPA) – induced apoptosis (programmed cell death).

In the second study, omega-3 fats have been shown as effective in the treatment of cholangiocarcinoma tumor cells, an aggressive and fatal type of cancer that forms in the liver bile ducts.

Researchers said omega-3 fatty acids may not only be an effective treatment for liver cancer, but could also protect the liver from hepatic steatosis, chronic liver disease thought to be a precursor to cancer.

These above are just a few specific examples of cancer and Omega3 .Omega are great in any cancer.

Omega-3 Builds Muscle Mass in Cancer Patients, thus PREVENT and HELP IN CACHEXIA: http://articles.mercola.com/sites/articles/archive/2011/03/24/omega3-builds-muscle-mass-in-cancer-patients.aspx

Omega 3 fats are highly recommended and among the few fats accepted in cancer (either from seeds, nuts, avocado or fish, although fish oils might be contaminated with heavy metals from the fish )

These and many other roles that help in cancer, including stimulation of the immune system, reducing inflammation , direct relationship to sun tolerance by the body and regulation of vitamin D levels (often those with cancer can not tolerate the sun, therefore can not benefit either its energy or the important natural Vitamin D, the sun being main source of vitamin D, but omega 3 fixes this, also regulating levels of vitamin D)

http://articles.mercola.com/sites/articles/archive/2010/07/27/omega3s-reduce-your-risk-of-breast-cancer.aspx

See also:

For prostate cancer risk and omega3 controversy see this articles:

http://articles.mercola.com/sites/articles/archive/2013/07/31/omega-3-fats.aspx

http://articles.mercola.com/sites/articles/archive/2001/07/21/flaxseed-part-two.aspx

The relation of alpha linolenic acid to the risk of prostate cancer: a …

science.naturalnews.com/…/520717_The_relation_of_alpha_linolenic_acid_ to_the_risk_of.html

The relation of alpha linolenic acid to the risk of prostate cancer: a systematic review and meta analysis.

Prostate tumor growth and recurrence can be modulated by omega3 intake…

science.naturalnews.com/…/3203424_Prostate_tumor_growth_and_ recurrence_can_be_modulated_by_the.html

Evidence indicates that a diet rich in omega (omega)-6 polyunsaturated fatty acids …prostate cancer (PCa) risk, whereas a diet rich in omega–3 decreases risk.

Much more about  OMEGA 3 as well a POWERFUL (stage IV) cancer treatment based on AWARDED 2 NOBEL PRIZES  in my book.

============================================================================================

Vitamin K

 (i.e: in algae, greens, grapes, etc) Also see antagonists such as cinnamon, etc,

Studies on Vitamin K and cancer (MANY STUDIES)

science.naturalnews.com/V/Vitamin_K_and_cancer.html

• Dietary Vitamin K intake in relation to cancer incidence and mortality: results from the Heidelberg cohort of the European Prospective Investigation into cancer and Nutrition (EPIC-Heidelberg).
  2010 The American journal of clinical nutrition
• Dietary intake of Vitamin K and risk of prostate cancer in the Heidelberg cohort of the European Prospective Investigation into cancer and Nutrition (EPIC-Heidelberg).
  2008 The American journal of clinical nutrition
• Serum undercarboxylated osteocalcin as biomarker of Vitamin K intake and risk of prostate cancer: a nested case-control study in the Heidelberg cohort of the European prospective investigation into cancer and nutrition.
  2009 Cancer epidemiology, biomarkers & prevention : a publication of the American Association for cancer Research, cosponsored by the American Society of Preventive Oncology
• Vitamin K and childhood cancer: analysis of individual patient data from six case-control studies.
  2002 British journal of cancer
• Vitamin K and childhood cancer: a report from the United Kingdom Childhoodcancer Study.
  2003 British journal of cancer
• Long-term use of Vitamin K antagonists and incidence of cancer: a population-based study.
  2011 Blood
• Cancer prevention and Vitamin K antagonists: an overview.
  2010 Thrombosis research
• Sorafenib combined Vitamin K induces apoptosis in human pancreatic cancer cell lines through RAF/MEK/ERK and c-Jun NH2-terminal kinase pathways.
  2010 Journal of cellular physiology
• Efficacy of low- molecular- weight- heparin versus Vitamin K antagonists for long term treatment of cancer-associated venous thromboembolism in adults: a systematic review of randomized controlled trials.2009 Thrombosis research
• A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).
  2004 Hepato-gastroenterology
• Low-molecular-weight heparins are superior to Vitamin K antagonists for the long term treatment of venous thromboembolism in patients with cancer: a cochrane systematic review.
  2008 Journal of experimental & clinical cancer research : CR
• Oxygen free radical generating mechanisms in the colon: do the semiquinones ofVitamin K play a role in the aetiology of colon cancer?
  2001 Biochimica et biophysica acta
• A study of the prevalence of Vitamin K deficiency in patients with cancer referred to a hospital palliative care team and its association with abnormal haemostasis.
  2008 Journal of clinical pathology
• Production of superoxide and dissipation of mitochondrial transmembrane potential by vitamin K2 trigger apoptosis in human ovarian cancer TYK-nu cells.
  2006 Apoptosis : an international journal on programmed cell death
• Vitamin K2-mediated apoptosis in cancer cells: role of mitochondrial transmembrane potential.
  2008 Vitamins and hormones
• Vitamins K2, K3 and K5 exert antitumor effects on established colorectal cancer in mice by inducing apoptotic death of tumor cells.
  2007 International journal of oncology
• Inhibitory effects of vitamin K3 on DNA polymerase and angiogenesis.
  2008 International journal of molecular medicine
• A resected case of metachronous liver metastasis from lung cancer producing alpha-fetoprotein (AFP) and protein induced by Vitamin K absence or antagonist II (PIVKA-II).2004 Hepato-gastroenterology

• Vitamins K2, K3 and K5 exert antitumor effects on established colorectal cancer in mice by inducing apoptotic death of tumor cells.2007 International journal of oncology

• Production of superoxide and dissipation of mitochondrial transmembrane potential by vitamin K2 trigger apoptosis in human ovarian cancer TYK-nu cells. 2006 Apoptosis : an international journal on programmed cell death

• Vitamin K2-mediated apoptosis in cancer cells: role of mitochondrial transmembrane potential. 2008 Vitamins and hormones Inhibitory effects of vitamin K3 on DNA polymerase and angiogenesis. 2008 International journal of molecular medicine

Vitamin K antagonists

• Cancer prevention and Vitamin K antagonists: an overview.
  2010 Thrombosis research
• Long-term use of Vitamin K antagonists and incidence of cancer: a population-based study.
  2011 Blood
• Efficacy of low- molecular- weight- heparin versus Vitamin K antagonists for long term treatment of cancer-associated venous thromboembolism in adults: a systematic review of randomized controlled trials.
  2009 Thrombosis research
• Low-molecular-weight heparins are superior to Vitamin K antagonists for the long term treatment of venous thromboembolism in patients with cancer: a cochrane systematic review.
  2008 Journal of experimental & clinical cancer research : CR

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Alimente utile in cancerul functie de tip de cancer

 

Cancer san

Care a împiedicat mai bine tumorile, struguri negrii sau ceaiul verde?

Cred eu ca am raspuns deja la acest lucru dar haideti sa vedem ce spun studiile .
Un mai 2006 studiul a constatat ca DOAR polifenolul resveratrol, găsit în pielita de struguri negri/mov/rosii/roze,
este mai eficientă decât polifenolul, epigallocatechin-3-galat (EGCG), gasit in ceaiul verde in prevenirea tumorilor mamare la sobolani. Inca de la naștere, sobolanilor li s-au administrat fie resveratrol fie EGCG regulat.. La vechi 50 zile ” sobolanilor li s-au dat cancerigen, DMBA. Șobolanii cu resveratrol au avut mai putine tumori și a existat o întârziere în timp a  dezvoltarilor tumorale.

Analiza țesutului mamar a arătat că tratamentul cu resveratrol a rezultat structuri lobulare mai diferențiate. În plus, a existat o reducere semnificativă în celule proliferative în structurile ductal mamare, facand tesutul mamar mai puțin sensibil la daune de la agenți cancerigeni. (A se vedea Whitsett TG et al., Resveratrol, dar nu EGCG, în dieta suprima cancer mamare DMBA induse  la sobolani, J Carcinog 2006.)

Alte studii spun ca ceai verde este mai potent decat DOAR resveratrol.

Dar ambele sunt foarte bune, deci includeti atat struguri negrii(contin multi alti anticancerigeni puternici, nutrienti si antioxidanti) pe stomacol gol cat si ceai verde in dieta dvs.

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Capsuni si fructe de padure ajuta la lupta impotriva cancerului de san

Un studiu de la Harvard a urmărit dieta și sănătatea de 1271 de persoane iubitoare de căpșuni găsit o scădere generală de 70% din toate cazurile de cancer. Un studiu celulă 2004 a investigat efectele specifice ale zece extracte diferite de fructe și fructe de padure,  inclusiv macese, afine, coacaze negre, aronia negru, mere, cătină, ligonberries, cireșe, zmeură  pe celule de cancer de san si celulelor cancerului de colon. Extractele au scăzut proliferarea atât celulele canceroase sân  cat si a celulelor cancerului de colon. Efectul de inhibare a variat cu concentrația de fructe si boabe variate – o medie de 52% pentru celulele cancer de san. Deoarece această rată de anti-proliferare nu a putut fi găsită prin vitamina C standard de ascorbat singura, a existat o sugestie a unei sinergii între  vitamina C și alte substanțe. Pentru celulele de cancer mamar, antocianii, care sunt pigmentii roșu  albastru  din fructe și aparțin clasei de compuși cunoscuți ca flavonoide, poate contribui puternic  la inhibarea celulelor canceroase. (A se vedea Olsson M. și colab., Inhibarea Cancer Cell proliferarii in vitro de legume si fructe Extracte și corelații cu nivelele de antioxidanti, J Agric Food Chem 2004.)

Murele contin rutina, flavonoide ce poate bloca efectele unei enzime ce este responsabila de cheagurilor de sange. Acesta reduce considerabil riscul atacului de cord sau de  accident vascular cerebral.
Căpșuni cultivate ecologic, cu condiția niveluri de antioxidanti mai mari și mai bune au inhibat mai bine proliferarea celulelor canceroase decat căpșuni cultivate in mod conventional in celulele canceroase san . Într-un studiu din 2006, nivel mai ridicat de ascorbat (vitamina C) si antioxidanti gasiti in căpșuni organice au fost corelate cu o inhibare mai mare de proliferarea celulelor cancerului de san. Semnificația efectului de ascorbat asupra proliferării celulelor canceroase ar putea fi într-o acțiune sinergică cu alte compuși. (A se vedea Olsson ME și colab., Nivelele de antioxidant și inhibarea cancerului Proliferarea celulelor in vitro prin extrase din organic si  căpșuni cultivate convențional, J Agric Food Chem 2006.)
Acum e sezon de cirese, visine, afine, mure, zmeura, fructe de padure,caise, piersici, nectarine pepeni si tomate si toate acestea sunt deosebit de recomandate si anticancerigene, daca sunt consumate pe stomacul gol(ex: dimineata), MINIM 1 kg zilnic.

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Portocale.

Anchetatorii au descoperit acum că portocale conțin mai mult de 170 chimicate foto, inclusiv mai mult de 20 din familia puternica a  carotenoidelor . In plus, compusi numite limonoids – care dau citricelor gust  ușor amar  – par a fi agenti foarte activi anti- cancer , de asemenea.

oranges (Photo credit: WGyuri)

Tomatele

Hot News: carotenoide Deși au fost bine cunoscut ca fiind radical liber necrofagi, un studiu din 2006 a cautat sa determine mecanismul de acțiune al  licopenului – carotenoid din rosii (si pepene verde si struguri si grapefruit, etc) și acid retinoic asupra inhibarii IGFs  – factori de crestere  ca insulina- in proliferarea celulelor canceroase, inclusiv celule de cancer de san . În studiul celulelor, nivelurile D1 ciclina, care acționează ca un senzor de factor de creștere
părea să fie ținta de acțiune licopen lui. Slăbirea  nivelurilor D1 ciclina de catre licopen și acidul retinoic este un mecanism important reducerea rolului IGFs „în proliferarea celulelor maligne. (A se vedea Nahum A et al.,
Inhibarea Licopenul de IGF-induse de Cancer Cell crestere economica depinde de nivelul de Cyclin D1, Eur J Nutr 2006.)

Vinetele.

Coaja violet a vinetelor(pe care de obicei lumea o arunca) este dovada puterii antioxidantilor ce ii contine. Vinetele sunt una din cele mai bune surse de nasunin, antioxidant ce blocheaza raspandirea celulelor canceroase. Nasuninul poate incetini evolutia bolii Alzheimer pe principiul inlaturarii efectelor radicalilor liberi la nivelul neuronilor.

Mecanisme anti-cancer de san ale cruciferelor si   I3C 

Cruciferele inclusiv varza, conopida, broccoli  conțin fitochimicale care acționează împotriva cancerului în mai multe moduri diferite. Varza, singura,are peste 200 de produse chimice.De exemplu, indol-3-carbinol (I3C), care este derivat din cruciferee,  influențează gene de cancer I3C  si un site de receptor. Plus, fitochimicale din familia verzei sunt capabile de a induce activarea enzime faza 2 pentru a proteja împotriva carcinogenii chimici.

Într-un studiu de la Universitatea Georgetown 2006, fitochimicale indol-3-carbinol (I3C) dîn broccoli, conopidă, varză și, s-au găsit pentru a stimula producția de proteine cu ADN-ul  ​​BRCA 1 și BRCA 2 – ce repara ADN-ul deteriorate, care sunt scăzute în celulele canceroase. (A se vedea Rosen E et al., Legume pot proteja impotriva cancerului,
British Journal of Cancer 2006.)

Dr. Eliot Rosen, cercetator de plumb in studiu Georgetown, a comentat:

„Este acum clar că funcția de gene cruciale de cancer  poate fi influențată de compușii din lucrurile pe care le mâncam. Descoperirile noastre sugereaza un proces molecular clar  care va explica legatura dintre alimentatie si  prevenirea cancerului. „

Un alt studiu I3C, un studiu noiembrie 2005, a explorarat proprietățile anti-tumorale a componentei alimentare indol-3-carbinol din crucifere si a constatat ca efectele anti-tumorale ale I3C in celulele canceroase umane pot reduce expresia receptorilor de estrogen-alfa. (Vezi Wang TT și colab., Receptorului de estrogen alfa ca o tinta de -3 indol – Carbinol, J Nutr Biochem 2005.)

Într-un studiu broccoli , varza „la sobolani, varza si broccoli s-au dovedit a fi o excepțional de bogată sursă de inductori de enzime care protejează împotriva chimicale cancerigene. Extracte de  trei zile vechi de varza de broccoli au fost extrem de eficiente în reducerea incidenței si multiplicarii si ratei de dezvoltare a tumori mamare  la șobolani. (A se vedea Fahey JW, broccoli Varza:. O sursă extrem de bogata de inductori ai enzimelor care protejeaza impotriva chimice cancerigene, Proc Natl Acad. Sci USA 1997.)

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Ardeiul rosu

Ardeiul rosu poate fi o vedeta a scris benficile pe de ingrijire le aduce sistemului imunitare, fiind o excelenta sursa de Vitamina C. Vitamina C Stimuleaza producerea de celule albe.

Un Studiu Publicat in Jurnalul american de Nutritie Clinica a constatat ca persoanele ce au o dieta bogata in Vitamina C au fost mai putin predispuse la aparitia ridurilor Vitamina C Este esentiala a scris Productia de colagen.

Usturoi.

Studiile arata compusi sulf  care dau o aroma puternica acum s-au dovedit a fi protejat împotriva cancer si neutralizat cancerigeni și incetinit cresterii tumorilor. Într-o recentă Iowa  Studiul de sanatate femei, anchetatorii
a constatat ca femeile care consuma usturoi cel puțin o dată pe săptămână au, de asemenea, un 32% mai mică incidenta cancerului de san. Sulful e responsabil si cu o buna detoxifiere.

In plus, usturoiul si ceapa sunt o sursa importanta de seleniu, un puternc tratament de cancer individual – voi reveni cu un articol despre tratarea cancerului cu seleniu.

Daca doriti sa primiti pe mail  articolul GRATUIT , cand il public, si orice tratament cancer nou apasati pe butonul de mai jos(introduceti si adresa mail):

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Abonare

(nu uitati sa apasati pe buton)Veti primi direct pe mail orice e nou.

Hrean.

Această rădăcină stimuleaza o dezintoxicare excelenta. Soiuri puternice au fost studiate pentru proprietăți anti-cancer .

Ardei gras rosu si verde,vinete, tomate, usturoi,ceapa, varza, dovlecel, etc.

Sparanghelul conține un număr de fitochimicale capabile antifungice, antimutagenice, citotoxice și   antivirale. Într-un studiu de laborator, brut saponine (din clasa de fitochimicale terpenoide – precum licopenul ), obținute din  sparanghel, s-au dovedit a avea activitate. anti-tumorala  (A se vedea Shao Y și colab., Antitumorale  Activitatea de saponine brute obținute din sparanghel, cancer Lett 1999.)

ROSII+SPARANGHEL (ASPARAGUS)!

Busuiocul

Busuiocul contine cantitati mari de eugenol, compus cu capacitatea de  a bloca celule canceroase din a se DIVIZA si stimuleaza autodistrugerea celulelor canceroase în cancerul de col uterin.

BUSUIOC

Spanac.

Universitatea din Minnesota Servicii de sanatate a mediului și altele au descoperit că persoanele care includ două
sau mai multe portii de spanac pe săptămână în hrana lor au considerabil mai mici  ratele de cancer de san si pulmonar.

SPANAC

Fasolea.

Cercetarea in stiintele alimentare a stabilit că toate tipurile de fasole sunt încărcate cu inhibitori de protează,
compusi care fac greu pentru cancer a invada tesuturile adiacente(deci previn si impiedica metastazarea).

Daca puteti  leguminoase verzi sau GERMINATE este excelent( alaturi de cereale integrale GERMINATE asigura proteine complete, precum si energie din carbohidrati complecsi si multi nutrienti  – faceti o salata cu legume, leguminoase si cereale incoltite)!

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Curcuma, cancer de san

Inflamația este asociata cu o mare varietate de tipuri de cancer,inclusiv cancerul de san.

Femeile cu hiperplazie atipica in tumori benigne ale sânilor, care au mers pe a dezvolta cancer de san, s-au dovedit a avea un nivel semnificativ mai ridicat de COX-2, care este produsă în organism atunci când există inflamație.Într-un studiu-cheie pentru 2005, curcumina, care este derivat din curcuma, a fost gasit a reduce dramatic COX-2. În India, unde femeile consuma Curcumina, 79 dintr-un milion de femei dezvolta cancer de san, in timp ce in SUA, unde
Femeile nu consuma curcumina, 660 de femei per milion dezvolta de san.
Seminte de si ulei de in bogate in OMEGA 3

Omega-3 grasimi, gasite in seminte si ulei de in, nuci, avocado, catina si uleiul de peste, inhiba cresterea celulelor cancerului de san in cultură și în grefe la soareci.  Moartea celulară crescută este atribuită inhibarii COX-2, enzimă care promovează procesul de cancer. În plus, omega 3 activează PPAR, un regulator al metabolismului lipidic capabil a  modula proliferarea. Semintele de in, pe langa omega 3 contin si lignani anticancerigeni.

Grasimile omega 3 sunt singurele grasimi antitumorale.In plus, combinate corespunzator cu proteine sulfurice, cresc oxigenarea celulara si combat astfel cauza primara a cancerului(exista alt premiu Nobel acordat acestei descoperiri   precum si un tratament cancer foarte eficient bazat pe grasimi omega 3).

Am descris in carte exact cum pot fi folosite grasimile omega 3 ca tratament cancer foarte eficient 

Pina si Dr Gerson si-a reconsiderat opinia despre grasimile omega3 , dupa ce initial le-a disconsiderat.
Activitatea în celulele de san. (A se vedea Stoll BA, acizi grasi omega3 si peroxidarea lipidelor în  Inhibarea cancer de san, Br J Nutr 2002.)

Alaturi de grasimi din nuca de cocos, masline si migdale sunt singurele grasimi permise in cancer.

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Acestea sunt cateva alimente bine de inclus in alimentatia zilnica, pentru ca stiu ca va intereseaza.

Iodul (inclusiv solutia Logol)

Femeile din Japonia, care consumă  fructe de mare bogate in iod au o incidenta mai mica de cancer de san (revin cu un articol despre iod.)

Daca doriti sa primiti pe mail  articolul GRATUIT , cand il public, si orice tratament cancer nou apasati pe butonul de mai jos(introduceti si adresa mail):

Abonează-te la blog prin email

Introdu adresa de email pentru a te abona la acest blog și vei primi notificări prin email când vor fi publicate articole noi.

Adresă email:

Abonare

(nu uitati sa apasati pe buton)Veti primi direct pe mail orice e nou.

 

Cancer ovarian

 
Dincolo consuma o varietate bogată de produse proaspete și limitarea produse lactate, carne și zahăr, anumite alimente vizeaza in special celulele cancer ovarian – au demonstrat succes extraordinar în înfrângerea acestei boli ce va pune in pericol viata.
Ghimbir

Radacina de ghimbir este un aliment excelent pentru anihilarea celulelor cancerului ovarian.

Când cercetătorii au dizolvat pudra de ghimbir într-o soluție care conține culturi de cancer ovarian, celulele mutante au murit. Ghimbirul distruge celulele de cancer ovarian în două moduri. 

Mai întâi printr-un proces de auto-distrugere celulară numit apoptoza apoi prin autofagie, cazul în care celulele se auto-digera. Oamenii de stiinta de la Universitatea din Michigan au descoperit ca ghimbirul a provocat aceeasi rata de apoptoza ca medicamente comune de chimioterapie, dar fără efecte secundare. Rădăcină ghimbir controlează, de asemenea, inflamația, care este un precursor al cancerului ovarian .

Ginkgo biloba

Cercetările au arătat că planta Ginkgo Biloba reduce semnificativ riscul de a dezvolta cancer ovarian. Mai mult de 600 de femei cu cancer ovarian și 640 subiecti sanatosi de control au fost studiate. Participanții au avut o istorie de a folosi fie ginkgo, echinacea, sunătoare, ginseng sau condroitina. Numai cei care au luat suplimente de Ginkgo au avut rate mai mici de ovarian cancer – o impresionantă scădere cu 60 la suta a riscului. 

Ceai verde 

Băut o doar doua cani pe zi de ceai verde poate reduce riscul de cancer ovarian la jumătate.

Cercetatorii au urmat 61057 de femei, variind de la varsta 40 – 76, pe parcursul a 15 ani. Pentru cei care au baut o ceasca de ceai verde pe zi, riscul de cancer ovarian a fost redus cu 24 la sută, în timp ce două sau mai multe cesti redus riscul cu 46 la suta. Și pentru femeile care au consumat băuturi în mod constant de peste 30 de ani, ratele de cancer ovarian a scazut cu 75 la suta. Oamenii de stiinta cred ca nivelul ridicat de antioxidanti gasit in ceaiul verde sunt responsabili pentru rezultate. Nu numai ca acesti fitonutrienti puternici ajuta la prevenirea și repararea daunelor ADN-ului, dar ele împiedică, de asemenea, creșterea și răspândirea celulelor canceroase prin cresterea apoptoza.

Semintele de in

Un studiu de caz-control a descoperit ca femeile din SUA care au avut cel mai mare aport de lignani(continuti si in seminte de in, caise  si in alte plante) au avut, de asemenea, cele mai mici rate de cancer ovarian. Lignani, gasiti in semintele de in sunt considerati fitoestrogeni. Acești compuși sunt „conectați” în receptori de estrogen, blocand forme mai puternice de hormon care agravează cancer ovarian. Lignani din semințe de in, de asemenea, acționează ca un antioxidant.

Într-o anchetă care implică surprinzător  puii de găină hraniti cu in, cercetatorii de la Universitatea din Illinois au descoperit ca de cancer ovarian a fost redus în mod semnificativ la găinile care au consumat semințe. Potrivit profesorului Janice Bahr, „la găini hrănite de seminte de in, am constatat ca mai multe tumori au fost limitate la ovar și au răspândit mai puțin metastatic. Aceasta este o constatare importanta deoarece metastaze care insotesc cancer ovarian stadiu tardiv sunt principala cauză de deces de boală. ” 

Sursele pentru acest articol includ:

„Ceaiul verde pentru prevenirea cancerului ovarian si de tratament: o analiza sistematica a in vitro, in vivo și studii epidemiologice” Trudel D, Labbe DP, Bairati eu, Fradet V, Bazinet L, Tetu B, Laval University Cancer Research Center, Pub Med , septembrie 2012. Adus de la 5 noiembrie 2012 de la: http://www.ncbi.nlm.nih.gov/pubmed/22564714

„Consumul de ceai verde imbunatateste supravietuirea de cancer ovarian epitelial” Zhang M, Lee AH, Binns CW, Xie X, Scoala de Sanatate Publica, Universitatea de Tehnologie Curtin, Australia, Pub Med, 10 noiembrie 2010. Adus de la 5 noiembrie 2012 de la: http://www.ncbi.nlm.nih.gov/pubmed/15382073

„Ceaiul verde lupta impotriva cancerului ovarian” David Liu, dr., de consum ale alimentelor, 09 mai 2012. Adus de la 5 noiembrie 2012 de la: http://www.foodconsumer.org

„Vitamine cancer ovarian, suplimente, plante și tratament naturist” Ray Sahelian, MD. Adus de la 5 noiembrie 2012 de la: http://www.raysahelian.com/ovariancancer.html

„Cure naturale pentru cancer ovarian” Thomas rege. Adus de la 5 noiembrie 2012 de la: http://www.ehow.com/about_5480292_natural-cures-ovarian-cancer.html

„Consumul de ceai verde poate reduce riscul de cancer ginecologica” Jonathan Benson, Natural News, 08 mai 2012. Adus de la 5 noiembrie 2012 de la: http://www.naturalnews.com

„Litri de beneficii in doar doua cesti de ceai” Karen Collins, RD, NBC News. Adus de la 5 noiembrie 2012 de la: http://www.msnbc.msn.com

„Lignani” Linus Pauling Institute. Adus de la 5 noiembrie 2012 de la: http://lpi.oregonstate.edu/infocenter/phytochemicals/lignans/

„Pui de seminte de in-Fed pune in lumina cancer ovarian” Știința Daily, 11 mai 2010. Adus de la 10 noiembrie 2012 de la: http://www.sciencedaily.com/releases/2010/05/100504124740.htm

„Lignani” NYU Langone Medical Center. Adus de la 5 noiembrie 2012 de la: http://www.med.nyu.edu/content?ChunkIID=21801

 

 

Multa sanatate!

Cristian

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CATINA tratament cancer

Catina , nativa din China, Rusia și Mongolia, raspandita din abundenta si la noi in Romania. Catina a fost utilizata în China de peste 12 de secole în care a fost folosit în primul rând pentru medicina tradițională chineză consolidarea stomacului, circulația sângelui și respirație.

Catina este cunoscuta ca unul din superfructele cele mai echilibrate ale naturii. Acest „Fruct Sfânt din Himalaya” a fost prețuită de către tibetani nativ de secole pentru calitatile sale nutritive incredibile . Face parte din aceeasi familie de fructe cu strugurii, mure, coacaze, afine, rosiile,capsuni, fragi, capsuni,zmeura denuunite in engleza colectiv Berry.Catina este supranumita  „Wonder Berry”- wonder – minuni.

Catina-superfruct

Catina este nu numai un antioxidant puternic, dar, probabil, unul din cele mai complete superfructe de pe  Pământ, cu o gamă largă de beneficii de sănătate documentate în peste 200 de studii clinice și științifice și în creștere în fiecare zi.

Incluse în aceste 190 de substanțe nutritive, sunt următoarele:

• Vitaminele A, B-uri, C, D,E, K, iar P
• Omega 3, 6, 7 și 9 (acizi grasi esentiali sau AGE)
• 42 Lipide
• Acizi organici
• 17 aminoacizi(proteine descompuse deja)
• Acid folic
• Tocoferoli
• Flavonoidele
• Fenoli
• Terpene
• Taninuri
• 20 de elemente minerale

Mai detaliat profilul nutrițional al catinei:

 

-peste 190 de compuși biologic activi ;

 

–peste 60 de antioxidanți de mare valoare ORAC( ceea ce ii ofera rol preventiv in cancer dar, in special, o recomanda in caz de chimio/radioterapie);

 

-foarte bogata în vitamina C (300-1600 mg/100 g),  de 4 – l00 ori mai mult decat orice alte legume și fructe ( ceea ce o fac un „supliment” natural pentru prelungirea vietii, reduce durerea in cancer, detoxifiere cat si pentru un tratament antitumoral pe baza de miere, avand catina cu vitamina C ca ucigas de cancer – se amesteca miere cu catina );

 

-întreaga gamă de acizi grași omega în echilibru perfect(omega 3, 6, 7 si 9) – puternic anti -inflamator; in plus de omega-7  cătină oferă, de asemenea, o sursa bogata de antioxidanti puternici si substante anti-inflamatorii;acesti compusi aduc beneficii dovedite de a distruge mucoasa oferind în același timp o gamă largă de beneficii pentru sistemul cardiovascular, imunitar și tegumentar.
În numeroase modele animale, cătină albă a fost demonstrat pentru a preveni și repara ulcerații la nivelul tractului digestiv cauzate de stres comune cum ar fi zgomotul, radiațiile, vibrații, și medii acide. Aceste descoperiri impresionante au determinat continuarea cercetarii examinarea acestui efect la om.

Într-un studiu clinic care a inclus 30 de subiecți diagnosticați cu ulcer peptic, catina suplimentarea mare de o lună a fost considerat un tratament eficient pentru 96,7 la suta din cazuri si a avut o rata de vindecare 76,6 la sută. Efectele sale de restabilire asupra mucoasei orale si vaginale au fost, de asemenea, studiate la om si sunt la fel de impresionante.

 

– carotenoide (pro-vitamina A)(la fel ca si in cazul celor din morcovi si crucifere, carotenoizii catinei desfac invelisul protector al celulelor cancerului;

Efectele de carotenoide – alfa-caroten, beta-caroten, licopen, beta-criptoxantină, zeaxantina, luteina, cantaxantina, astaxantin – cu privire la invazia celulelelor tumorale hepatice la soareci laborator au fost investigate. Toate carotenoidele examinate au inhibatat invazia într-un mod dependent de doza adminstrata . Celulele canceroase cultivate anterior cu hipoxantin (HX) și xantin oxidaza (XO) a demonstrat o activitate extrem de invaziva. Cu toate acestea, carotenoidele, 5 um de beta-caroten și astaxantina, au suprimat această capacitate invazivă a radicalilor liberi. Rezultatele sugerează că acțiunea anti-invaziva a tumorilor este un rezultat al proprietăților antioxidante al acestor carotenoizi. „

Avocado poate creste arbsorbitia carotenoizilor care distrug invelisul enzimatic de pe celulele canceroase, deci CRESC EFICIENTA sucurilor de legume de 2-4 ori . http://whfoods.org/genpage.php?tname=foodspice&dbid=5 A NU SE EXAGERA CU AVOCADO, dar a se avea in vedere acest aspect.

 

-amestec de tocoferoli și tocotrienoli (vitamina E), fitosteroli, flavonoide, fenoli, scoarţa arbustului conţine tocoferol şi sitosterol, terpene și cu cel puțin 20 de cofactori minerali;

 

-carbohidrati complecsi, acizi organici, pectine(ca si cele din mere calmeaza nevoia de dulce), celuloza, proteine(aminoacizii),  fosfor, calciu, magneziu, potasiu, sodiu, fier, tot complexul vitaminelor B.

 

Catina este bogata în macronutrienti si micronutrienti. Catina contine si vitamina D precum și K.

 

Catina, de asemenea, contine in mod natural 5-HT (serotonina), un neurotransmitator care ajuta la reglarea emotiilor.

 

Catina numeroase beneficii pentru sănătate includ cardiovasculare, imunitate, impotriva cancerului, memorie, o creștere, anti-inflamatorii si sanatatea pielii. Catina este cunoscuta ca fructele cele mai echilibrate naturii.

Vitaminizantă de excepţie, antioxidantă redutabilă şi puternica stimulatoare a sistemului imunitar, cătina are încă multe valenţe necunoscute publicului larg.
Cercetări efectuate în Marea Britanie au descoperit capacitatea cătinei de a inhiba dezvoltarea unor tumori.

Conform cercetărilor efectuate de specialişti de prestigiu precum Ing. Ştefan Manea, prof. univ. dr. chim. Ion Brad, dr. Luminiţa Brad şi Ing. Florica Radu, cătina care creşte în ţara noastră are un conţinut chimic de excepţie, poate cel mai bogat din întreaga lume. Planta a fost studiată şi ameliorată în Staţiunea Horticolă „Mărăcineni”, unde s-au obţinut varietăţi cu ţepi reduşi. Trebuie menţionat că nicăieri în lume nici varietatea naturală, nici soiurile ameliorate nu au conţin atât de multe principii active, vitamine, minerale, microelemente, flavonoizi şi aminoacizi la nivelul cătinei care creşte în România, mai ales în zona subcarpatică.

La noi în ţară este răspândită îndeosebi în regiunea deluroasă a Munteniei şi Moldovei, fiind mai rară în stepele litorale ale Mării Negre.  Cea mai largă răspândire a cătinei a fost observată în zona bazinului râului Buzău, unde creşte din abundenţă atât de-a lungul albiilor râurilor şi izvoarelor, cât şi pe versanţii care urcă la mari înălţimi. Este o plantă căreia-i priesc terenurile sălbatice, cu vegetatie diversă şi însorite.

 

Efectele secundare negative sunt de-a dreptul neînsemnate

 

Un studiu de caz despre acizii grasi (grasimile / uleiul de catina).

 Uleiul de cătină utilizat cu un succes deosebit chiar şi în cazuri grave de arsuri termice şi chimice, sau afecţiuni ale pielii determinate de expunerea la iradieri. Merită amintit un caz relatat de ing. Ştefan Manea, referitor la un accident de muncă grav petrecut în anul 1985 la Combinatul Chimic Giurgiu.

Accidentul respectiv s-a soldat cu şase victime, cu arsuri de gradele 3 şi 4 pe suprafeţe de peste 50% din piele. Trei dintre bolnavi au fost diagnosticaţi fără şanse de supravieţuire şi au rămas internaţi la Sitalul Judeţean Giurgiu în aşteptarea fatidicului deznodământ.

Ing. Ştefan Manea apelează urgent la profesorul universitar Ioan Brad, unul dintre cei mai mari experţi în cătină la nivel mondial. Acesta este adus de urgenţă cu elicopterul de la Facultatea de Medicină din Târgu Mureş, unde pe atunci era profesor. Ioan Brad a venit cu ulei de cătină cu care le-a uns şi pansat rănile celor trei muribunzi. Rezultatul a fost de-a dreptul miraculos. Cei trei ingineri nu au intrat în blocaj renal în faza următoare, iar arsurile s-au vindecat. Cei trei au scăpat de la moarte şi trăiesc sănătoşi şi în prezent.

Uleiul de cătină este folosit şi în tratamentul ulcerului gastric şi duodenal, diareei, urticariei şi stărilor alergice, maladiilor neuroendocrine, reumatismului, afecţiunilor circulatorii şi hepatice, alcoolismului, anemiei, asteniei şi chiar stresului.

Utilizările cătinei în geriatrie au dat rezultate spectaculoase.

De asemenea, este eficientă şi în tratarea hipertensiunii arteriale, afecţiunilor coronariene, oftalmologice şi gingivale. Este un redutabil antidepresiv!, antiacneic, contra keratozei, leucoplaziei, antimutagen, antioxidant în mastite, rinite, fotofobii, boli cronice pulmonare. Este folosită cu succes şi în tratamentul psoriazisului, în prevenirea cancerului şi ca imunostimulator, prevenirea adenoamelor, tratamentul cancerului mamar, antibacteriană, inhibitoare a poftei de mâncare în cazurile de obezitate, reglatoare a lipoproteineinemiei, tratamentul leucemiilor şi limfoamelor, afecţiuni osteoarticulare, O.R.L., inflamaţii digestive, constipaţie, afecţiuni ale aparatului urinar şi al celui genital, afecţiuni hematologice, afecţiuni virale, afecţiuni ale sistemului nervos şi psihice.

În plus, în cătina albă se găsesc o seamă de substanţe cu efect hormonal prin ele însăşi sau substanţe care reprezintă precursori ai anumitor hormoni.

În uleiul de cătină este probabilă existenţa prostaglandinelor (acestea sunt considerate hormoni celulari, respectiv modulatori ai activităţii hormonale care stimulează musculatura netedă), dar mai precis se găsesc acizi graşi nesaturaţi cu două sau trei duble legături care sunt precursorii siguri ai prostaglandinelor. O altă susbstanţă cu efect hormonal care se găseşte în cantităţi mari în cătină este serotonina. Ea are ca precursor triptofanul – aminoacid esenţial care se găseşte în cantităţi mari îndeosebi în fructele uscate şi pulverizate de cătină.

 

Cu siguranta multe mai sunt de spus si multe studii se vor face despre efectele catinei in boli, inclusiv in cancer. Bolnavii de cancer abia asteapta rezultatele studiilor si cine stie cand acestea vor fi publicate, daca vor fi…

Contine o sumedenie de substante anticancerigene si sau necesare in cancer( vitamine A- caroteni, C, D, E, toti acizii grasi omega, antioxidanti, minerale, proteine, carbohidrati complexi, etc ), NU are efecte adverse, se gaseste din abundenta, INCLUDETI-O in alimentatia dvs. ( in stare naturala, fructe INTREGI, proaspete sau adaugata in sucuri de legume, verdeturi sau fructe sau intr-un tratament pe baza de miere (miere + catina + ce mai doriti- scortisoara, ghimbir, vanilie, turmeric, etc.) ).

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Iata si Dr. OZ vrobeste despre catina:

 

Daca va plac cele de aici, apasati va rog, pe (CLICK AICI), (CLICK AICI) .

Asa puteti arata dumneavoastra ca apreciati efortul depus de mine.

Pofta buna si va rog de asemenea sa completati chestionarul din josul paginii TRATAMENTE RECOMANDATE(CLICK AICI), pentru a va intelege mai bine nevoile dvs. si a putea sa va ajut mai bine!

 

Multa Sanatate,

Cristian

 

Acum as vrea sa aud si de la dumneavoastra:

Ati incercat oricare din cele de mai sus? Ce efecte au avut asupra sanatatii dumneavoastra? Lasati un comentariu mai jos!

CONSIDERATI UTILE CELE DE AICI? 

SPUNETI SI ALTORA! ( sunt butoane sub aceasta pagina precum si in DREAPTA SUS!).